ライフサイエンスコーパス: IQ

1 IQ assessed in late adolescence is a robust risk factor

2 IQ is decreased in deletion and duplication carriers, bu

3 IQ is relatively more important in predicting scores on

4 IQ motif-containing GTPase activating protein 1 (IQGAP1)

5 IQ motif-containing GTPase-activating protein 1 (IQGAP1)

6 IQ scores were available for 150 patients (60 had receiv

7 IQ slopes did not differ between groups (P = .342).

8 IQ slopes did not differ between groups (P = .509).

9 IQ was lower in the XRT group (by 12.5 points) versus th

10 IQ was lower in the XRT group (by 8.7 points) versus the

11 IQ was measured at age 18-20 as part of the Swedish mili

12 IQ, language, and motor function were measured at 7 year

13 IQ-1, a specific inhibitor of p300/beta-catenin interact

14 s costing $266 billion in the USA vs 873 000 IQ points lost and 3290 cases costing $12.6 billion in t

15 1409 individuals drawn from the top 0.0003 (IQ >170) of the population distribution of intelligence

16 ucation was associated with 1.94 (SE = 0.18) IQ units.

17 d540 to pound4495]), with a net gain of 1.22 IQ points in each analysis.

18 n day care centres decreased by 43% to 4.3% (IQ range 3.05-5.96).

19 exposure associated with a decrement of 3.70 IQ points (95% confidence interval: 0.83, 6.56).

20 ve MS (PPMS) scored a significant 4.6 to 6.9 IQ points lower than controls up to 20 years prior to fi

21 mance, as well as global conceptual ability (IQ) (P < 0.001) scores, differed significantly across th

22 al implantation of the PanOptix IOL (AcrySof IQ PanOptixTM; Alcon Research, Fort Worth, Texas, USA) p

23 A discounted lifetime value of an additional IQ point based on earnings was estimated to be pound3297

24 known to share genetic liability with ADHD: IQ, social communication, pragmatic language, and conduc

25 veral baseline covariates such as adolescent IQ, family background, and educational level.

26 ations (rpart,92 = -.35, p < .001) and adult IQ (rpart,88 = .33, p = .001) even after controlling for

27 ower score (95% CI, -2.48 to -0.74) in adult IQ, a 2.07-point lower score (95% CI, -3.14 to -1.01) in

28 Participants were matched for age, IQ, head motion, and eye status (open or closed) in the

29 Although IQ increased similarly in patients with schizophrenia an

30 Although an IQ motif promotes binding to CaM, an acidic sequence in

31 n Classification in MLD level 0 or 1) and an IQ of at least 85, when age at disease onset was older t

32 ogical families, the adopted siblings had an IQ 4.41 (SE = 0.75) points higher than their nonadopted

33 5 mug/L were four times as likely to have an IQ score < 80, the clinical cut-off for borderline intel

34 Probabilities of having an IQ in the normal (>/=70) or intellectual disability (<70

35 ; taking education costs and the value of an IQ point itself into account), and presented in terms of

36 sessed to calculate the monetary value of an IQ point.

37 RT (P = .203) and XRT groups (P = .060), and IQ slopes did not differ (P = .890).

38 y-one children with ADHD and thirty age- and IQ-matched typically developing (TD) controls underwent

39 functioning youths with ASDs and 19 age- and IQ-matched, typically developing youths (age range, 9-17

40 s of specific methyl-CpGs with attention and IQ.

41 and the different correlation between BV and IQ in cases and control subjects may induce artifactual

42 difference in specificity between PQ-CSF and IQ-CSF, the latter showed a significant improvement in s

43 ia Verbal Learning Test, second edition] and IQ [Wechsler Abbreviated Scale of Intelligence]) were pe

44 of whom 287 and 211 had data for the GCI and IQ analyses, respectively.

45 95% CI -4.12, -0.59) lower offspring GCI and IQ scores, respectively.

46 ety of datasets to show that personality and IQ predict grades and scores on achievement tests.

47 ounted for documented differences in sex and IQ in affected individuals with de novo mutations by mat

48 Finally, age, sex, and IQ were important sources of cerebellar volume variabili

49 raumatized youth of comparable age, sex, and IQ.

50 school achievement (SA) at age 16 years and IQ at ages 18 to 20 years and the deviation of that perf

51 The mean school grades at age 16 years and IQ test scores at military conscription at age 18 years.

52 indexed by school grades at age 16 years and IQ test scores at military conscription.

53 Correlations in SA and IQs between the pre-SZ probands and their siblings were

54 No differences were found in the SA and IQs of siblings of BPI vs matched control probands.

55 , the siblings of the SZ probands had SA and IQs that did not differ from population means and were s

56 sess homologous CaM-binding motifs, known as IQ motifs in their C termini, which associate with calmo

57 ein kinase (MAPK) scaffold proteins, such as IQ motif containing GTPase activating protein 1 (IQGAP1)

58 cting cognitive/behavioral measures, such as IQ.

59 detailed neuropsychological battery assessed IQ and domain-specific neurocognitive functions.

60 ated using a meta-analysis of PTB-associated IQ loss, and well-established relationships of IQ loss w

61 w executive function, tremors, below-average IQ, and FXTAS.

62 Persistence, above and beyond IQ, is associated with long-term academic outcomes.

63 They bind IQ motifs within the myosin neck region and amplify conf

64 ion had a range of intellectual ability, but IQ scores were 26 points lower than noncarrier family me

65 the majority of the error-prone TLS of dG-C8-IQ, whereas pol eta is involved primarily in its error-f

66 t at nucleotide incorporation opposite dG-C8-IQ.

67 iate from the traditional mode of calmodulin-IQ recognition.

68 homology-Bin/amphiphysin/Rvs (F-BAR) Cdc15p, IQ motif containing GTPase-activating protein (IQGAP) Rn

69 d hair were negatively associated with child IQ scores.

70 ation effect; P = 6.20 x 10-3) and childhood IQ (7.9% mediation effect; P = .041) and by PEs being me

71 eight (rho = -0.155; P = .015) and childhood IQ (rho = -0.188; P = .003) were associated with the pre

72 After adjusting for maternal IQ, childhood IQ, and childhood socioeconomic status, each 5-microg/dL

73 To evaluate the impact of the Clarity IQ technology on reducing radiation risk in patients und

74 We collected IQ scores of children from the TTS cohort with both pre-

75 A composite IQ score of general cognitive ability was calculated.

76 episodic memory ( r = .26), and crystallized IQ ( r = .18).

77 been defined based on premorbid and current IQ, but little is known about the neuroanatomical differ

78 16p11.2 locus beyond the effect of decreased IQ.

79 ficantly improved postoperative IQ and delta IQ (adjusted regression coefficient [RC] = 3.4, 95% conf

80 PA was related to postoperative IQ and delta IQ, using linear regression analyses.

81 e to controls, and the severely deteriorated IQ group had widespread volumetric reductions.

82 rough Quality in Pharmaceutical Development (IQ) Drug Metabolism Leadership Group.

83 rmance of a new PET/CT system, the Discovery IQ with 5-ring detector blocks.

84 inly for estrogen receptor-positive disease (IQ-OR, 1.44; 95% CI, 1.16 to 1.79; P for heterogeneity =

85 ic interactions with the myosin-1C divergent IQ motifs, which are contacts that deviate from the trad

86 cifically bind to two sequentially divergent IQ motifs of the Dictyostelium myosin-1C.

87 dent sample, we obtained proportions in each IQ-based subgroup that were similar to our previous work

88 s (n = 89) had significantly lower estimated IQ ( P < .001) and PS scores ( P = .02) compared with pa

89 by US public health insurance had estimated IQs that were significantly lower ( P < .001) than those

90 To derive this, expected IQ was modeled based on probability of early (age <4 yea

91 de polymorphism heritability for the extreme IQ trait was 0.33 (0.02), which is the highest so far fo

92 ter adjustment for sociodemographic factors, IQ, other traumas, and childhood sexual abuse.

93 unds, and 3) sleep quality mediates the fish-IQ relationship.

94 Fluid IQ measured with the Kaufman Brief Intelligence Test, no

95 ition, effect sizes (Cohen's d) were 0.1 for IQ (mean difference from control 1.2, 95% CI -0.3 to 2.7

96 stimulation alone, effect sizes were 0.1 for IQ (mean difference with controls 1.7, -0.3 to 3.7), 0.3

97 ling pairs and 9,000 twin pairs assessed for IQ and for the presence of ID.

98 Similar results were obtained for IQ (HR, 0.53; 95% CI, 0.37-0.77 for the deviation from t

99 elated with the neural transition frequency, IQ scores of individuals with ASD are instead predicted

100 ers and cognitive and behavioral scores from IQ testing, and parental measures of development were te

101 [95% confidence interval = -1.70, -.17] full IQ unit per 100 mg daily caffeine intake).

102 imary outcome was global cognitive function (IQ).

103 es of attention, memory, executive function, IQ, and processing speed.

104 tive evaluation of intellectual functioning (IQ), working memory, and processing speed (PS) was condu

105 Varying levels of IQx (up to 0.29 ng/g), IQ (up to 0.93 ng/g), MeIQx (up to 0.08 ng/g), MeIQ (up

106 nalyses included the moderators age, gender, IQ, and scan site.

107 of the first (PQ-CSF) and second generation (IQ-CSF) RT-QuIC assays, and investigated the diagnostic

108 racts with a conserved isoleucine-glutamine (IQ) motif in the C terminus of the channel, while Ca(2+)

109 lin via its N-terminal isoleucine-glutamine (IQ) motif.

110 g twins failed to show significantly greater IQ decline relative to their abstinent siblings.

111 tified (P = .130), whereas in the XRT group, IQ declined by 1.1 points per year (P = .004).

112 and the unexposed siblings (44% female) had IQ testing at mean ages of 10.6 and 10.9 years, respecti

113 ficantly enriched for associations with high IQ.

114 ms (OR, 0.93 [95% CI, 0.91-0.96]) and higher IQ (OR, 1.04 [95% CI, 1.02-1.07]) in childhood compared

115 ated with less sleep disturbances and higher IQ scores in schoolchildren, 2) such relationships are n

116 ated to both fewer sleep problems and higher IQ scores.

117 dose-response relationship indicated higher IQ scores in children who always (4.80 points) or someti

118 ICC, 0.992; 95% CI: 0.986, 0.996), and IDEAL IQ and the GE 3.0-T unit (ICC, 0.966; 95% CI: 0.939, 0.9

119 rs (mDIXON Quant [Philips Healthcare], IDEAL IQ [GE Healthcare]).

120 motional problems, and cognitive impairment (IQ score <80) during childhood (ages 6, 11, and 15 years

121 Behavioral impulsivity, IQ, MID task performance, and VS BOLD were regressed aga

122 Linear mixed models examined change in IQ over time since radiation therapy (RT) by RT group, c

123 In the PBRT group, no change in IQ over time was identified (P = .130), whereas in the X

124 omic status in adulthood and with changes in IQ and socioeconomic mobility between childhood and midl

125 Associations between changes in IQ and the total brain, cerebral gray matter, cerebral w

126 7); these SD scores correspond to changes in IQ of 6 to 8 points.

127 phrenia and control participants, changes in IQ were negatively correlated with changes in lateral ve

128 n greater blood lead levels and a decline in IQ and socioeconomic status from childhood to adulthood

129 status at age 38 years and with declines in IQ and with downward social mobility.

130 ars at diagnosis are at risk for deficits in IQ and PS in the absence of cranial radiation, regardles

131 no statistically significant differences in IQ scores in later childhood.

132 about 40% of between-subject variability in IQ.

133 determinants of health to the discrepancy in IQs, which was 13%.

134 Additionally, the WMD in IQs with NBW were 14, 10 and 7 for ELBW, VLBW, and MLBW

135 The pooled weight mean difference (WMD) in IQs between NBW and LBW individuals was 10 (95% CI 9.26-

136 on between low birth weight and individuals' IQ scores (IQs).

137 tribution) were similar to those influencing IQ in the normal range.

138 Diagnosis of a psychotic disorder, initial IQ, longitudinal IQ trajectory, and timing of the last p

139 about its origin and links to intelligence (IQ) still remains.

140 d a hypomethylated region mapping to Iqgap2 (IQ motif-containing GTPase activating protein 2) and F2r

141 chsler Adult Intelligence Scale-IV (WAIS-IV; IQ range, 40-160).

142 chiatric assessment with respect to the last IQ test.

143 ith 3 different types of hydrophobic lenses: IQ SN60WF (494 eyes) and ReSTOR SN6AD1 (169 eyes) (Alcon

144 were independent of age, educational level, IQ, APOE genotype, subjective memory complaints, vascula

145 psychotic disorder, initial IQ, longitudinal IQ trajectory, and timing of the last psychiatric assess

146 grades and 0.97% (95% CI, 0.15%-1.78%) lower IQ test scores.

147 io [OR], 1.11 [95% CI, 1.04-1.19]) and lower IQ (OR, 0.98 [95% CI, 0.95-1.00]).

148 es showed that the association between lower IQ and schizophrenia is not the result of shared familia

149 were more likely to have borderline or lower IQ compared with children of mothers consuming <100 mg/d

150 r had a mean (SE) of 4.4 (0.72) points lower IQ than those without severe disorder (P < .001), and th

151 se had a mean (SE) of 5.6 (1.2) points lower IQ than those without severe disorder (P < .001).

152 After adjusting for maternal IQ, childhood IQ, and childhood socioeconomic status, ea

153 Mean IQ scores between exposed siblings (scores: full scale =

154 8 individuals from the top 0.0003 ( 170 mean IQ) of the population distribution of intelligence and 8

155 Differences in mean IQ scores between sibling pairs were: full scale = -0.2

156 of adolescents less than 1 SD below the mean IQ range than those without a disorder.

157 The mean IQs of the extremely low birth weight (ELBW, <1000 g), v

158 suggests that observed declines in measured IQ may not be a direct result of marijuana exposure but

159 ns after delivery and intelligence measures (IQ) in adulthood.

160 subclinical hypothyroidism trial, the median IQ score of the children was 97 (95% confidence interval

161 In the hypothyroxinemia trial, the median IQ score was 94 (95% CI, 91 to 95) in the levothyroxine

162 o polybrominated diphenyl ethers (11 million IQ points lost and 43 000 cases costing $266 billion in

163 redictive power was similar to the TC model (IQ-OR, 1.45; 95% CI, 1.21 to 1.73; mC, 0.55), but SNP88

164 dings on the Full-Scale IQ (FSIQ), Nonverbal IQ, and Verbal IQ; the presence of ASD or other DSM-IV d

165 arting age for IBI and in achieving a normal IQ based on starting age.

166 erse outcomes, including a lower-than-normal IQ in offspring.

167 en accepted as the extreme low of the normal IQ distribution.

168 HR, 1.07; 95% CI, 0.78-1.46 for the observed IQ).

169 at factors influencing mild ID (lowest 3% of IQ distribution) were similar to those influencing IQ in

170 actors influencing severe ID (lowest 0.5% of IQ distribution) differ from those influencing mild ID o

171 er performance on a school-age assessment of IQ, a measure whose relevance for occupational success i

172 assessment of the nationwide annual costs of IQ losses from aircraft lead emissions.

173 s responsible for the normal distribution of IQ, but that severe ID is not.

174 No effects of IQ were found.

175 lies, adoption was associated with a gain of IQ of 3.18 (SE = 0.34) points.

176 zed that some of the missing heritability of IQ might lie hidden in the human leukocyte antigen (HLA)

177 sults strongly support the implementation of IQ-CSF in clinical practice.

178 gical functioning (mean [SD]) on measures of IQ (81.22 [15.97] vs 91.28 [14.31]; Cohen d, -0.70), ver

179 nificant impairment on mean (SD) measures of IQ (95.25 [16.58] vs 100.45 [14.77]; Cohen d, -0.22; P =

180 parents and adoptees, and direct measures of IQ to measure symptoms of autism spectrum disorder, inat

181 loss, and well-established relationships of IQ loss with LEP.

182 Our results confirm the high sensitivity of IQ-CSF for detecting human prions with a sub-optimal sen

183 independent predictive power beyond that of IQ.

184 ys, and investigated the diagnostic value of IQ-CSF across the broad spectrum of human prions.

185 healthy comparison subjects group-matched on IQ, gender, and age performed a passive avoidance task w

186 healthy comparison subjects group-matched on IQ, gender, and age performed a passive avoidance task w

187 ated the causal effect of DNA methylation on IQ using the offspring genotype at sites close to the me

188 attention-deficit/hyperactivity disorder or IQ, age range was associated with gray matter difference

189 on) differ from those influencing mild ID or IQ scores in the normal range.

190 nity binding site, which leaves the original IQ-motif exposed, thereby destabilizing the lever arm.

191 nfluenced by environmental factors, parental IQs and other factors contribute to residual confounding

192 of cost (in sterling, relevant to 2013) per IQ point gained in the offspring.

193 SD; 95% CI, -0.69 to -0.40), and Performance IQ (-0.41 SD; 95% CI, -0.56 to -0.27); these SD scores c

194 primary outcomes were verbal and performance IQ scores on the Wechsler Preschool and Primary Scale of

195 l intelligence quotient (IQ) and performance IQ scores over a period of 5 years were significantly su

196 54) were analysed for verbal and performance IQ with WPPSI-III and 315 (iodine group, n=159; placebo

197 two of these sites on childhood performance IQ which was replicated for one of the sites.

198 CI -2.9 to 1.5; p=0.77), and for performance IQ were 97.5 (12.5) in the iodine group and 99.1 (13.4)

199 consumption and verbal, but not performance, IQ.

200 ion was observed with treatment arm (placebo IQ-OR, 1.46; 95% CI, 1.13 to 1.87; tamoxifen IQ-OR, 1.25

201 AED reduction also predicted >/= 10-point IQ increase (p = 0.019).

202 cational attainment and 0.86 with population IQ.

203 est NPA significantly improved postoperative IQ and delta IQ (adjusted regression coefficient [RC] =

204 the latest NPA was related to postoperative IQ and delta IQ, using linear regression analyses.

205 through Ca(2+)/CaM binding to the CaV1.2 pre-IQ domain.

206 aM) and subsequent binding of CaM to the pre-IQ domain of the channels.

207 alysis on the basis of current and premorbid IQ differences.

208 These findings hold independent of proband IQ.

209 ramide signaling on an ERK scaffold protein, IQ motif containing GTPase activating protein 1 (IQGAP1)

210 found for Full Scale intelligence quotient (IQ) (-0.52 SD; 95% CI, -0.68 to -0.37), Verbal IQ (-0.54

211 s, gait disturbances, intelligence quotient (IQ) and adaptive function, as well as paternal and mater

212 n association between Intelligence Quotient (IQ) and PBDEs.

213 full-scale or global intelligence quotient (IQ) and performance IQ scores over a period of 5 years w

214 frequency of use and intelligence quotient (IQ) change.

215 age 4 and full scale intelligence quotient (IQ) from the Wechsler Abbreviated Scale of Intelligence

216 We compared change in intelligence quotient (IQ) over time in pediatric patients with brain tumors tr

217 was driven mainly by intelligence quotient (IQ) points loss and intellectual disability due to polyb

218 sociated with reduced intelligence quotient (IQ) score in offspring.

219 <2500 g) have a lower intelligence quotient (IQ) than those with normal birth weights (NBW, >/=2500 g

220 nt (MQ), but not with intelligence quotient (IQ), age, or sex.

221 ographic confounders, intelligence quotient (IQ), and other traumas.

222 Intelligence quotient (IQ), grades, and scores on achievement tests are widely

223 corresponding to a 6 intelligence quotient (IQ)-point difference.

224 ation with respect to intelligence quotient (IQ-scores).

225 iation with full-scale intelligent quotient (IQ) (beta=0.07, P=0.03, r(2)=0.005).

226 with published GWA analyses of normal-range IQ or educational attainment.

227 isk overall (interquartile range odds ratio [IQ-OR], 1.37; 95% CI, 1.14 to 1.66; mC, 0.55), but mainl

228 rded SA and 106187 individuals with recorded IQ born in Sweden between January 1, 1972, and December

229 between increasing psychosis RPS and reduced IQ (matrix reasoning: corrected P = .003 for RPS model 2

230 ween developmental PBDE exposure and reduced IQ.

231 s may first manifest in childhood as reduced IQ and later contribute to PEs in early adulthood.

232 n inverted-J-shaped association with child's IQ at age 7 years, with a peak difference (vs. undetecta

233 evated lead concentrations impair children's IQ and can lead to lower earnings potentials.

234 nship between caffeine intake and children's IQ at 5.5 years (-.94 [95% confidence interval = -1.70,

235 caffeine intake during pregnancy, children's IQ at age 5.5, and individual and family characteristics

236 ognitive aptitude is calculated from the SA, IQ, and educational attainment in biological relatives.

237 intrafamilial control subjects with the same IQ level.

238 tistic Disorder (age 18-45 years; full scale IQ >70; ABC-Irritability subscale 13).

239 ered standardized scores for both Full Scale IQ and Verbal Comprehension, Perceptual Reasoning, Worki

240 cantly associated with changes in Full Scale IQ or Index scores.

241 cantly negatively associated with Full Scale IQ, and with all Index scores (except Processing Speed).

242 not with a greater improvement in Full Scale IQ.

243 ted with high-functioning autism (full-scale IQ >100).

244 Findings on the Full-Scale IQ (FSIQ), Nonverbal IQ, and Verbal IQ; the presence of

245 nce interval (CI): -3.9, -0.5] in Full-Scale IQ and 2.9 points (95% CI: -4.4, -1.3) in Verbal Compreh

246 The main outcome was full-scale IQ as assessed by the WPPSI-III.

247 The mean (SD) full-scale IQ at first cognitive assessment was lower in patients w

248 we observed similar decrements in Full-Scale IQ with each standard deviation increase of use for two

249 ental domains: cognitive ability (Full-Scale IQ), social behavior (Social Responsiveness Scale), and

250 low birth weight and individuals' IQ scores (IQs).

251 Lower mean (SE) IQ was observed among adolescents with past-year bipolar

252 atively, a substantial improvement was seen (IQ-OR, 1.64; 95% CI, 1.36 to 1.97; mC, 0.60).

253 ith and without these genetic events on sex, IQ, and age before comparing them on multiple behavioral

254 of high-functioning ASD males and age-/sex-/IQ-matched controls, we first identified age-associated

255 Since IQ is inherited and influenced by environmental factors,

256 s to a 5- to 10-point increase on a standard IQ test.

257 In the craniospinal subgroup, IQ remained stable in both the PBRT (P = .203) and XRT g

258 In the focal subgroup, IQ scores remained stable in the PBRT group (P = .401) b

259 IQ-OR, 1.46; 95% CI, 1.13 to 1.87; tamoxifen IQ-OR, 1.25; 95% CI, 0.96 to 1.64; P for heterogeneity =

260 mammalian brain and heart, lack a C-terminal IQ motif.

261 r predictors of important life outcomes than IQ.

262 ersonality is generally more predictive than IQ on a variety of important life outcomes.

263 The IQ (primary outcome) and indexes of Verbal Comprehension

264 The IQ strata were assigned probabilities of achieving an in

265 (MRI) brain volumes were compared among the IQ-based subgroups using analysis of covariance with int

266 We introduced the IM-AA mutation into the IQ-like motif (IM) of the Ca(2+) sensor binding site.

267 Deletion of the IQ motif (amino acids 29-58) results in loss of calmodul

268 h germanium oxide-based PET/CT scanners, the IQ with 5-ring detector blocks has the highest overall p

269 ve in vitro binding assays, we show that the IQ domain of IQGAP1 is both necessary and sufficient for

270 nalysis with pure proteins revealed that the IQ region of IQGAP1 binds directly to the intracellular

271 rmore, we determined that CaM binding to the IQ motif was required for channel function, indicating t

272 ain volumetric differences correspond to the IQ-based subgroups.

273 n showed that the interaction occurs via the IQ domain of IQGAP1 and the TEAD-binding domain of YAP.

274 The primary outcome was the IQ score at 5 years of age (or at 3 years of age if the

275 ion skills beyond what is expected for their IQ level.

276 -sensing protein, is constitutively bound to IQ domains of the C termini of human Kv7 (hKv7, KCNQ) ch

277 (top 0.0003 of the population equivalent to IQ > 147) and 3,253 unselected population controls.

278 ves: To confirm previous findings related to IQ-based subgroups of patients with schizophrenia in an

279 In each trial, IQ scores were missing for 4% of the children.

280 ) (-0.52 SD; 95% CI, -0.68 to -0.37), Verbal IQ (-0.54 SD; 95% CI, -0.69 to -0.40), and Performance I

281 ler discrepancy between nonverbal and verbal IQ and a greater likelihood of having achieved fluent la

282 ociated with decreased full-scale and verbal IQ scores compared with duplication carriers without the

283 ll-Scale IQ (FSIQ), Nonverbal IQ, and Verbal IQ; the presence of ASD or other DSM-IV diagnoses; BMI;

284 Mean WPPSI-III scores for verbal IQ were 89.5 (SD 9.8) in the iodine group and 90.2 (9.8)

285 nd was associated with impairments in verbal IQ, attention, executive function, language and visuospa

286 ld not be explained by differences in verbal IQ, intracranial volume, anxiety/depression, or attentio

287 exposures were associated with lower verbal IQ in minimally adjusted models; but after adjustment fo

288 The divergence of verbal IQ trajectories between those who subsequently developed

289 laterality positively correlated with verbal IQ.

290 ality were assessed at age 9-11 years, while IQ was assessed at age 12.

291 Moreover, in contrast to the controls whose IQ is correlated with the neural transition frequency, I

292 ods: through static estimates of cohort-wide IQ deficits and through dynamic economy-wide effects usi

293 PBRT was not associated with IQ decline or impairment, yet IQ slopes did not differ b

294 d eye movement stage 2 sleep, correlate with IQ and are thought to promote long-term potentiation and

295 ll tissue volumes in infancy correlated with IQ (r >/= 0.35, P < .05) and language (r >/= 0.29, P < .

296 Seven-year volumes correlated with IQ (r = 0.28, P = .04 for cortical GMV), language (r = 0

297 nction; however, a negative correlation with IQ at age 11 years (beta=-0.08, Z=-3.3, P=0.001) was obs

298 Here, we show that LGR5 interacts with IQ motif-containing GTPase-activating protein 1 (IQGAP1)

299 ygous for a 2 base pair (bp) deletion within IQ calmodulin-binding motif-containing protein-1 (IQCB1)

300 ssociated with IQ decline or impairment, yet IQ slopes did not differ between the PBRT and XRT groups