ライフサイエンスコーパス: Id-2

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します

1 ner, and higher PU.1 levels induce Mac-1 and Id-2.

2 Id-2 and Id-3 are only detectable in nuclei; levels incr

3 fied cytoplasmic Id-1 expression and nuclear Id-2 and Id-3 expression in the proliferating layers of

4 rthermore, we observed a columnar pattern of Id-2 and Id-3 staining, which may relate to the epiderma

5 he 129/Sv epithelium results in a decline in Id-2 and Id-3 to below the limits of immunodetection.

6 These results suggest that Id-2 and the bHLH factors that it partners play importan

7 in cells of increasing maturity; conversely, Id-2 and, to a limited extent, Id-3 gene expression incr

8 itors of DNA binding/differentiation (Id)-1, Id-2, and Id-3 are among the most significantly up-regul

9 We demonstrated that the expression of Id-1, Id-2, and Id-3 genes was significantly induced at the ea

10 of the cancer cells exhibited abundant Id-1, Id-2, and Id-3 immunoreactivity.

11 is study we compared the expression of Id-1, Id-2, and Id-3 in the normal pancreas, in pancreatic can

12 We observed Id-1, Id-2, and Id-3 mRNA expression in proliferating human ke

13 pecific gene expression (Cadherin-6, EphA-7, Id-2, and RZR-beta) developed normally.

14 In culture, Id-2 antisense transcripts blocked differentiation.

15 Ubiquitinated Id-1/Id-2 can then bind to p62 and be transported to autolyso

16 Our results suggest that Id-1, ITF-2, and Id-2 comprise a network of interacting molecular switche

17 ted and migration was strikingly enhanced in Id-2-expressing cells.

18 laminin, is responsible for the increase in Id-2 expression during differentiation.

19 We also show that Id-2 expression is inversely correlated with the rate of

20 In conclusion, Id-2 expression not only follows a pattern opposite to t

21 Id-2 expression ran counter to that of Id-1 not only dur

22 grown under hypoxic conditions in culture), Id-2 expression was maintained.

23 and atypical papillary ducts in CP, Id-1 and Id-2 immunoreactivity was as significantly elevated as i

24 demonstrated the presence of faint Id-1 and Id-2 immunostaining in pancreatic ductal cells in the no

25 ntroduced in aggressive breast cancer cells, Id-2 is able to reduce their proliferative and invasive

26 in murine mammary epithelial cells and that Id-2 is expressed at a higher level in differentiated hu

27 inately expressed and suggests that Id-1 and Id-2 might be regulating very different events during he

28 o Id-1, we found that, in vitro and in vivo, Id-2 mRNA and protein were up-regulated as mammary epith

29 ns, we used an adenovirus vector to maintain Id-2 protein expression in cultured cytotrophoblasts.

30 Moreover, little Id-2 protein expression is detectable in human biopsies

31 oblasts transduced to constitutively express Id-2 retained characteristics of undifferentiated cells:

32 The Id-2 transcriptional inhibitor was essential for product

33 urther determined that this up-regulation of Id-2 was a necessary step toward a fully differentiated

34 The cells expressed Id-3 constitutively; Id-2 was downregulated, at the mRNA and protein levels,

35 We show further that, in contrast to Id-1, Id-2 was expressed highly in differentiated mammary epit

36 ty of immunostaining indicated that Id-1 and Id-2 were increased significantly in the cancer cells by

37 ; transcription factors/regulators Pax-5 and Id-2, which are involved in normal B-cell development; a

WebLSDに未収録の専門用語（用法）は "新規対訳" から投稿できます。