1 f ghrelin in primary human T cells activates IkB, and increases Th1 cytokines and IL-17 secretion.

2 ression, and increased inhibitor of kappa B (IkB) expression in the brain.

3 oteins that are retained in the cytoplasm by IkB-like proteins; (ii) the NFATc proteins that sense ca

4          Moreover, degradation of endogenous IkB-alpha (an NF-kappaB inhibitor) was stimulated only b

5                                     Finally, IkB (NF-kappa-B inhibitor), a known substrate of beta-Tr

6 F-kB in sepsis through selective ablation of IkB kinase.

7  but not simian RRV mediated accumulation of IkB-alpha protein and decreased transcription of NF-kapp

8 tory mechanism of P3G involves the arrest of IkB-alpha activation and reduction in JNK(MAPK) phosphor

9 fier binding domain)-binding domain peptide (IkB kinase (IKK)-NEMO-binding domain (NBD) peptide), whi

10  the NF-kappaB-inducing kinase, NIK, and the IkB kinase, IKKalpha.

11 n of PKC was associated with a change in the IkB kinase (IKK)/nuclear factor (NF)-kappaB pathway, we

12 reatment before LPS activation inhibited the IkB phosphorylation.

13            For instance, targeting NF-kB via IkB kinase 2 with specific inhibitors may block an array