1 In conclusion, our study adds to the growing understanding of
2 In conclusion, our study demonstrated that NGS target sequenc
3 In conclusion, our study demonstrates a physiological role of
4 In conclusion, our study demonstrates that PRMs from the lect
5 In conclusion, our study does not support a causal effect of
6 In conclusion, our study establishes a non-genomic mechanism
7 In conclusion, our study establishes a system-wide model of a
8 In conclusion, our study expands the diagnostic tools for det
9 In conclusion, our study failed to find an association betwee
10 In conclusion, our study generated five mAbs against MGA and
11 In conclusion, our study has identified new susceptibility al
12 In conclusion, our study has uncovered the existence of a miR
13 In conclusion, our study highlights a role for CD73 as a prog
14 In conclusion, our study identified a novel PBC susceptibilit
15 In conclusion, our study identified miR-27a downregulation as
16 In conclusion, our study identified SHARPIN as a novel regula
17 In conclusion, our study identifies a previously unknown prot
18 In conclusion, our study identifies SOCS1 mimicking as a feas
19 In conclusion, our study indicates that Nod2-dependent respon
20 In conclusion, our study is the first to report the efficacy
21 In conclusion, our study presented genetic and functional evi
22 In conclusion, our study provides a comprehensive list of kid
23 In conclusion, our study provides a detailed description of t
24 In conclusion, our study provides new insight into the pathog
25 In conclusion, our study provides strong evidence that the re
26 In conclusion, our study provides supporting evidence to expl
27 In conclusion, our study puts forward mutations in RCBTB1 as
28 In conclusion, our study quantitatively demonstrated the invo
29 In conclusion, our study reports that activin A, produced dur
30 In conclusion, our study results suggest that plasma Abeta pe
31 In conclusion, our study revealed a very considerable depende
32 In conclusion, our study reveals major cell types (particular
33 In conclusion, our study reveals novel common genetic risk fa
34 In conclusion, our study reveals that EVs are an important co
35 In conclusion, our study reveals that PH most likely plays an
36 In conclusion, our study reveals the critical role of miR-118
37 In conclusion, our study sheds light on the structural and bi
38 In conclusion, our study shows an unexpected role of lipids a
39 In conclusion, our study shows that D-4F, but not apoA-I-cont
40 In conclusion, our study shows that FH and FH19-20 binding to
41 In conclusion, our study shows that RIM1/2 potently enhance t
42 In conclusion, our study suggested that chemokine receptor CC
43 In conclusion, our study suggests a common pattern of molecul
44 In conclusion, our study suggests that histologic grade may s
45 In conclusion, our study suggests that I-PTH plays a critical
46 In conclusion, our study suggests that IgA vasculitis is an a
47 In conclusion, our study suggests that in addition to quittin
48 In conclusion, our study suggests that mTOR activity in hepat
49 In conclusion, our study suggests that Wnt signaling enhances
50 In conclusion, our study unveils a novel mechanism by which h