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3 solated from the ticks Ixodes scapularis and Ixodes affinis, the cotton rat (Sigmodon hispidus), and
4 ected on B. burgdorferi in unfed or engorged Ixodes dammini (also known as I. scapularis) ticks, furt
5 tory birds in the long-distance dispersal of Ixodes dammini ticks and in the spread of Lyme disease,
6 of nine mice infected by Ixodes scapularis (Ixodes dammini) tick inoculation of an isolate from Nant
7 Wisconsin and Minnesota, Ixodes scapularis (Ixodes dammini) ticks are the vector of three microorgan
9 Most of the strains isolated from the tick Ixodes dentatus in Missouri and Georgia belonged to the
13 se peptides were identified in the available Ixodes genome and expressed sequence tag (EST) database.
16 burgdorferi, is transmitted by ticks of the Ixodes genus and, if untreated, can cause significant mo
18 ted of a single species, the nidiculous tick Ixodes hexagonus (prevalence = 24.3%; mean intensity = 7
22 ting Borrelia sp. nov. isolated from nymphal Ixodes minor collected in South Carolina showed their cl
23 wo enzootic tick vectors, Ixodes affinis and Ixodes minor, rarely bite humans but are more important
26 pathogenic Ehrlichia bacteria isolated from Ixodes ovatus (IOE) reveal, however, that this paradigm
27 y related virulent ehrlichiae transmitted by Ixodes ovatus (IOE) ticks given intraperitoneally or int
28 rlichiosis caused by Ehrlichia bacteria from Ixodes ovatus (IOE) to determine whether OMP immunizatio
29 ng infection with a strain of Ehrlichia from Ixodes ovatus (IOE) were evaluated using a model that cl
30 lowing lethal infection with highly virulent Ixodes ovatus ehrlichia (IOE), an obligate intracellular
31 uris, which causes persistent infection, and Ixodes ovatus Ehrlichia (IOE), which is either acutely l
33 wild-type (WT) mice with virulent Ehrlichia (Ixodes ovatus Ehrlichia [IOE]) results in CD8+ T-cell-me
34 t mice cleared a primary low-dose (nonfatal) Ixodes ovatus ehrlichia infection, a secondary low-dose
36 in vitro coculture of memory CD8 T cells and Ixodes ovatus ehrlichia-infected peritoneal exudate cell
40 sely related to E. chaffeensis isolated from Ixodes ovatus ticks in Japan, causes fatal infection of
41 duced actin phosphorylation was dependent on Ixodes p21-activated kinase (IPAK1)-mediated signaling.
46 ation in the continuous part of the range of Ixodes pacificus reinforces recent recognition of the di
58 he midgut transcriptome composition in adult Ixodes ricinus females during early and late phase of en
62 m induces the phosphorylation of actin in an Ixodes ricinus tick cell line and Ixodes scapularis tick
64 ed to silencing of virus replication in live Ixodes ricinus ticks and abolished virus neurotropism in
66 conducted on an arthropod species (the tick Ixodes ricinus) on which de novo sequencing was performe
67 protist Trichomonas vaginalis, the hard tick Ixodes ricinus, and the flatworm Schistosoma mansoni.
69 ocytophilum infection on an enclosed area of Ixodes ricinus-infested pasture in North Wales, United K
70 ent (WI-1) and nine of nine mice infected by Ixodes scapularis (Ixodes dammini) tick inoculation of a
72 ft genome for the Rickettsia endosymbiont of Ixodes scapularis (REIS), a symbiont of the deer tick ve
73 es of subjects who presented with a definite Ixodes scapularis (Say) tick bite were measured to deter
74 ribe the 2.1 Gbp nuclear genome of the tick, Ixodes scapularis (Say), which vectors pathogens that ca
77 mine whether impaired TLR signaling enhances Ixodes scapularis acquisition of B. burgdorferi, we fed
78 quitination machinery is present in the tick Ixodes scapularis and demonstrate that the E3 ubiquitin
79 st southern strains, isolated from the ticks Ixodes scapularis and Ixodes affinis, the cotton rat (Si
82 ivary protein 20 (Salp20) is a member of the Ixodes scapularis anti-complement protein-like family of
83 A. phagocytophilum induces ticks to express Ixodes scapularis antifreeze glycoprotein (iafgp), which
84 omma americanum, Dermacentor variabilis, and Ixodes scapularis are among the principal tick species a
85 rved protein that was discovered recently in Ixodes scapularis as a tick protective antigen and has a
88 Out of three encoding genes representing Ixodes scapularis genome paralogs, IrCD1 is the most dis
91 es (PMN) treated with the saliva of the tick Ixodes scapularis have reduced expression of beta(2) int
98 ly, we show that a dae gene in the deer tick Ixodes scapularis limits proliferation of Borrelia burgd
99 etes to ticks after capillary inoculation of Ixodes scapularis nymphs and the subsequent spirochetal
100 spC expression by B. burgdorferi in infected Ixodes scapularis nymphs as they fed on uninfected mice
101 lp lipoproteins in mice after challenge with Ixodes scapularis nymphs harboring B. burgdorferi 297.
102 protected from infection when infested with Ixodes scapularis nymphs harboring virulent B. burgdorfe
104 a tick bite and challenged 16 weeks later by Ixodes scapularis nymphs infected with either the same o
106 nd challenged either 12 or 16 weeks later by Ixodes scapularis nymphs infected with the same agent.
107 were infested four times with pathogen-free Ixodes scapularis nymphs prior to infestation with nymph
112 Previous studies have demonstrated that both Ixodes scapularis saliva and Borrelia burgdorferi antige
114 now describe a feeding-inducible protein in Ixodes scapularis saliva, Salp15, that inhibits CD4(+) T
117 A. phagocytophilum induces expression of the Ixodes scapularis salp16 gene in the arthropod salivary
118 phalitis following a documented bite from an Ixodes scapularis tick and the erythema migrans rash ass
119 ear whether antimicrobial treatment after an Ixodes scapularis tick bite will prevent Lyme disease.
120 nine components to estimate the frequency of Ixodes scapularis tick bites and the resulting incidence
121 agated continuously for over 500 days in the Ixodes scapularis tick cell line IDE8 by using the Garde
123 iftly upregulated when spirochetes leave the Ixodes scapularis tick gut, migrate to the salivary glan
124 agent of human granulocytic anaplasmosis, in Ixodes scapularis tick salivary glands, to detect protei
125 me disease is transmitted by the bite of the Ixodes scapularis tick, which can also transmit Anaplasm
127 infected mice (a reservoir model) to nymphal Ixodes scapularis ticks (a biological vector) and subseq
128 aled that both uninfected larval and nymphal Ixodes scapularis ticks acquired B. burgdorferi as early
129 The mutant cells were able to survive within Ixodes scapularis ticks after a blood meal from naive mi
131 isingly, sigma54 mutants were able to infect Ixodes scapularis ticks and be maintained for at least 2
132 were identified from among 99 isolates from Ixodes scapularis ticks and from white-footed mice (Pero
133 valence of Borrelia burgdorferi infection in Ixodes scapularis ticks and Peromyscus sp. mice captured
134 The assay was tested on field-collected Ixodes scapularis ticks and shown to have 100% concordan
135 urgdorferi bbk32 and bbk50 expression within Ixodes scapularis ticks and the murine host, and the eff
136 ssible from inoculated C.B-17 mice to larval Ixodes scapularis ticks and, subsequently, from infected
139 s amplified from template DNA extracted from Ixodes scapularis ticks collected in Rhode Island and fr
140 hether the salivary gland extract (SGE) from Ixodes scapularis ticks facilitates the transmission and
141 d to show that spirochetes could be found in Ixodes scapularis ticks feeding on 4 of 10 antibiotic-tr
143 o 2 strains of DTV that had been detected in Ixodes scapularis ticks from Massachusetts in 1996 and i
146 . phagocytophilum strain HZ in SCID mice and Ixodes scapularis ticks infected with strain NTN-1.
149 In this paper we explore the contribution of Ixodes scapularis ticks to the pathogenicity of Borrelia
154 gene expression within the guts of engorging Ixodes scapularis ticks was examined by use of different
157 burgdorferi produce OspA in the gut of unfed Ixodes scapularis ticks, and many spirochetes repress Os
158 the level of thiamin and its derivatives in Ixodes scapularis ticks, the enzootic vector of Bb, is e
159 ctin in an Ixodes ricinus tick cell line and Ixodes scapularis ticks, to alter the ratio of monomeric
169 ized Lyme disease focus with an abundance of Ixodes scapularis vector ticks and the first documentati
171 tect remnants of blood in blacklegged ticks (Ixodes scapularis) and correctly determine the vertebrat
172 , infecting local populations of deer ticks (Ixodes scapularis) and to test the fit to a neutral IAM.
173 B. burgdorferi in nymphal blacklegged ticks (Ixodes scapularis) collected at the sites the following
175 ain 297 naturally colonized their arthropod (Ixodes scapularis) vector, were maintained in ticks thro
176 relia burgdorferi) and its main tick vector (Ixodes scapularis) was studied concurrently and comparat
177 ds to the gut of the intermediate tick host (Ixodes scapularis), and that this interaction is mediate
178 s aegypti and Culex quinquefasciatus), tick (Ixodes scapularis), body louse (Pediculus humanus), kiss
181 atural antibodies when its arthropod vector, Ixodes scapularis, begins feeding on a mammalian host.
182 and that the typical vector of Lyme disease, Ixodes scapularis, can acquire the spirochetes from infe
183 ber 1994 through December 1995 from the tick Ixodes scapularis, collected from vegetation, and from t
184 nhanced by the presence of B. burgdorferi in Ixodes scapularis, first indicating that spirochaetes mi
185 va of the hard tick and Lyme disease vector, Ixodes scapularis, has a repertoire of compounds that co
186 ding the role that nymphal and female ticks, Ixodes scapularis, have in the epidemiology of Lyme dise
187 rtant than the human biting "bridge" vector, Ixodes scapularis, in maintaining the enzootic spirochet
191 eeking larvae uninfected with the spirochete Ixodes scapularis, thereby perpetuating the agent throug
192 e to the range expansion of the tick vector, Ixodes scapularis, upon which the causative agent, Borre
193 itor (TFPI), Ixolaris, from the ixodid tick, Ixodes scapularis, which has 10 cysteines, and a thrombi
195 a burgdorferi, colonizes the gut of the tick Ixodes scapularis, which transmits the pathogen to verte
212 human anaplasmosis, are both transmitted by Ixodes sp. ticks and may occasionally coinfect a host.
214 nfects mostly neutrophils, it transmitted by Ixodes species ticks, and occurs mostly in the upper mid
218 , is maintained in an enzootic cycle between Ixodes spinipalpis ticks and Neotoma mexicana rats (27).
221 philum is a bacterium that is transmitted by Ixodes spp. ticks, in which it resides in salivary gland
222 urgdorferi sensu lato (s.l.), transmitted by Ixodes spp. ticks, is the causative agent of Lyme diseas
224 wave morphology and borrelial DNA in endemic Ixodes stilesi ticks collected in Chile from environment
225 cits (HDs) at the CP scale in raccoon ticks (Ixodes texanus; n=718) collected from raccoons (Procyon
226 ughout the Northern Hemisphere, and the same Ixodes tick species transmitting the etiologic agents of
227 he Lyme disease pathogen, cycles between its Ixodes tick vector and vertebrate hosts, adapting to vas
230 gression through the enzootic cycle using an Ixodes tick/C3H-HeJ mouse model and tick immersion feedi
232 nking H14 stem varies between six species of Ixodes ticks (Acari: Ixodidae) where only 33 invariant s
243 miyamotoi is a relapsing fever spirochete in Ixodes ticks that has been recently identified as a huma
246 elia burgdorferi, is transmitted by bites of Ixodes ticks to mammalian reservoir hosts and humans.
247 ochetal bacterium that depends on arthropod (Ixodes ticks) and mammalian (rodent) hosts for its persi
248 zootic cycle, transiting between its vector, Ixodes ticks, and a diverse range of vertebrate hosts.
250 rochetes are able to survive normally in the Ixodes ticks, mice fed upon by the DeltacheY2-infected t
252 rstanding of the epidemiology and ecology of ixodes ticks, the established vector for transmission of
253 isease is transmitted by the bite of certain Ixodes ticks, which can also transmit Anaplasma phagocyt
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