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1 xenodiagnosis using the natural tick vector (Ixodes scapularis).
2 cks, including the Lyme disease vector tick, Ixodes scapularis.
3 ocytophila) are both transmitted by the tick Ixodes scapularis.
4 th a cell line isolated from the vector tick Ixodes scapularis.
5 tick cell line, IDE8, derived from embryonic Ixodes scapularis.
6 rickettsial symbiont isolated from the tick Ixodes scapularis.
7 E8, derived from a putative vector, the tick Ixodes scapularis.
8 of Lyme disease, is transmitted by the tick Ixodes scapularis.
9 bitor present in the saliva of the hard tick Ixodes scapularis.
10 ogens known to infect the black-legged tick, Ixodes scapularis.
11 y louse Pediculus humanus and a tick species Ixodes scapularis.
12 is required for spirochetal colonization of Ixodes scapularis.
13 of Lyme disease, is transmitted by the tick Ixodes scapularis.
14 irochete carried in the "black-legged" tick, Ixodes scapularis.
16 mine whether impaired TLR signaling enhances Ixodes scapularis acquisition of B. burgdorferi, we fed
17 quitination machinery is present in the tick Ixodes scapularis and demonstrate that the E3 ubiquitin
18 st southern strains, isolated from the ticks Ixodes scapularis and Ixodes affinis, the cotton rat (Si
21 tect remnants of blood in blacklegged ticks (Ixodes scapularis) and correctly determine the vertebrat
22 , infecting local populations of deer ticks (Ixodes scapularis) and to test the fit to a neutral IAM.
23 ds to the gut of the intermediate tick host (Ixodes scapularis), and that this interaction is mediate
25 ivary protein 20 (Salp20) is a member of the Ixodes scapularis anti-complement protein-like family of
26 A. phagocytophilum induces ticks to express Ixodes scapularis antifreeze glycoprotein (iafgp), which
27 omma americanum, Dermacentor variabilis, and Ixodes scapularis are among the principal tick species a
28 rved protein that was discovered recently in Ixodes scapularis as a tick protective antigen and has a
30 atural antibodies when its arthropod vector, Ixodes scapularis, begins feeding on a mammalian host.
31 s aegypti and Culex quinquefasciatus), tick (Ixodes scapularis), body louse (Pediculus humanus), kiss
32 and that the typical vector of Lyme disease, Ixodes scapularis, can acquire the spirochetes from infe
33 B. burgdorferi in nymphal blacklegged ticks (Ixodes scapularis) collected at the sites the following
34 ber 1994 through December 1995 from the tick Ixodes scapularis, collected from vegetation, and from t
37 nhanced by the presence of B. burgdorferi in Ixodes scapularis, first indicating that spirochaetes mi
38 Out of three encoding genes representing Ixodes scapularis genome paralogs, IrCD1 is the most dis
41 va of the hard tick and Lyme disease vector, Ixodes scapularis, has a repertoire of compounds that co
42 es (PMN) treated with the saliva of the tick Ixodes scapularis have reduced expression of beta(2) int
43 ding the role that nymphal and female ticks, Ixodes scapularis, have in the epidemiology of Lyme dise
44 rtant than the human biting "bridge" vector, Ixodes scapularis, in maintaining the enzootic spirochet
48 ent (WI-1) and nine of nine mice infected by Ixodes scapularis (Ixodes dammini) tick inoculation of a
54 ly, we show that a dae gene in the deer tick Ixodes scapularis limits proliferation of Borrelia burgd
56 etes to ticks after capillary inoculation of Ixodes scapularis nymphs and the subsequent spirochetal
57 spC expression by B. burgdorferi in infected Ixodes scapularis nymphs as they fed on uninfected mice
58 lp lipoproteins in mice after challenge with Ixodes scapularis nymphs harboring B. burgdorferi 297.
59 protected from infection when infested with Ixodes scapularis nymphs harboring virulent B. burgdorfe
61 a tick bite and challenged 16 weeks later by Ixodes scapularis nymphs infected with either the same o
63 nd challenged either 12 or 16 weeks later by Ixodes scapularis nymphs infected with the same agent.
64 were infested four times with pathogen-free Ixodes scapularis nymphs prior to infestation with nymph
70 ft genome for the Rickettsia endosymbiont of Ixodes scapularis (REIS), a symbiont of the deer tick ve
71 Previous studies have demonstrated that both Ixodes scapularis saliva and Borrelia burgdorferi antige
73 now describe a feeding-inducible protein in Ixodes scapularis saliva, Salp15, that inhibits CD4(+) T
76 A. phagocytophilum induces expression of the Ixodes scapularis salp16 gene in the arthropod salivary
77 es of subjects who presented with a definite Ixodes scapularis (Say) tick bite were measured to deter
78 ribe the 2.1 Gbp nuclear genome of the tick, Ixodes scapularis (Say), which vectors pathogens that ca
82 eeking larvae uninfected with the spirochete Ixodes scapularis, thereby perpetuating the agent throug
83 phalitis following a documented bite from an Ixodes scapularis tick and the erythema migrans rash ass
84 ear whether antimicrobial treatment after an Ixodes scapularis tick bite will prevent Lyme disease.
85 nine components to estimate the frequency of Ixodes scapularis tick bites and the resulting incidence
86 agated continuously for over 500 days in the Ixodes scapularis tick cell line IDE8 by using the Garde
88 iftly upregulated when spirochetes leave the Ixodes scapularis tick gut, migrate to the salivary glan
89 agent of human granulocytic anaplasmosis, in Ixodes scapularis tick salivary glands, to detect protei
90 me disease is transmitted by the bite of the Ixodes scapularis tick, which can also transmit Anaplasm
92 infected mice (a reservoir model) to nymphal Ixodes scapularis ticks (a biological vector) and subseq
93 aled that both uninfected larval and nymphal Ixodes scapularis ticks acquired B. burgdorferi as early
94 The mutant cells were able to survive within Ixodes scapularis ticks after a blood meal from naive mi
96 isingly, sigma54 mutants were able to infect Ixodes scapularis ticks and be maintained for at least 2
97 were identified from among 99 isolates from Ixodes scapularis ticks and from white-footed mice (Pero
98 valence of Borrelia burgdorferi infection in Ixodes scapularis ticks and Peromyscus sp. mice captured
100 urgdorferi bbk32 and bbk50 expression within Ixodes scapularis ticks and the murine host, and the eff
101 ssible from inoculated C.B-17 mice to larval Ixodes scapularis ticks and, subsequently, from infected
104 s amplified from template DNA extracted from Ixodes scapularis ticks collected in Rhode Island and fr
105 hether the salivary gland extract (SGE) from Ixodes scapularis ticks facilitates the transmission and
106 d to show that spirochetes could be found in Ixodes scapularis ticks feeding on 4 of 10 antibiotic-tr
108 o 2 strains of DTV that had been detected in Ixodes scapularis ticks from Massachusetts in 1996 and i
111 . phagocytophilum strain HZ in SCID mice and Ixodes scapularis ticks infected with strain NTN-1.
114 In this paper we explore the contribution of Ixodes scapularis ticks to the pathogenicity of Borrelia
119 gene expression within the guts of engorging Ixodes scapularis ticks was examined by use of different
122 burgdorferi produce OspA in the gut of unfed Ixodes scapularis ticks, and many spirochetes repress Os
123 the level of thiamin and its derivatives in Ixodes scapularis ticks, the enzootic vector of Bb, is e
124 ctin in an Ixodes ricinus tick cell line and Ixodes scapularis ticks, to alter the ratio of monomeric
134 e to the range expansion of the tick vector, Ixodes scapularis, upon which the causative agent, Borre
135 ized Lyme disease focus with an abundance of Ixodes scapularis vector ticks and the first documentati
136 ain 297 naturally colonized their arthropod (Ixodes scapularis) vector, were maintained in ticks thro
137 relia burgdorferi) and its main tick vector (Ixodes scapularis) was studied concurrently and comparat
139 itor (TFPI), Ixolaris, from the ixodid tick, Ixodes scapularis, which has 10 cysteines, and a thrombi
141 a burgdorferi, colonizes the gut of the tick Ixodes scapularis, which transmits the pathogen to verte
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