ライフサイエンスコーパス: J

1 J-couplings in methyl alpha-l-[6-(13)C]idopyranosiduroni

2 J. spicigera showed colors (green-iridescent, green-yell

3 ecreased from 4.3 (3.5-6.3) to 2.1 (1.5-5.0) J/min, respectively (p = 0.031).

4 UVB irradiation and healing (UVB doses 1.01 J/cm(2) once daily for four days).

5 order down to 1.8 K (T/J approximately 0.02; J is the dominant exchange constant) renders the compoun

6 hanical work close to zero (-0.012 +/- 0.054 J/kg).

7 nditions at low laser excitation energy (0.1 J/cm(2) at 800 nm) without the use of photoinitiators, w

8 ) for 1000 seconds for a total fluence of 10 J/cm2.

9 in the range of 52-55 degrees C and 100-115 J/g, respectively.

10 capable of generating entropy changes of 14 J kg(-1) K(-1) or 22 J kg (-1) K(-1), and corresponding

11 ed into the seed at an oblique angle with 14 J sr(-1), and net gains of more than eight orders of mag

12 ich display high-energy densities (up to 166 J cm(-3)) and efficiencies (up to 96%).

13 can detect a single photon (10(-18)-10(-19) J) of visible light.

14 delivering high energy densities, i.e., 1.2 J cm(-3) , even at a temperature close to the glass tran

15 e aggregates, whereas the Er:YAG laser (38.2 J/cm(2)) completely stripped away the plaque and TiO2 la

16 er (metal tip) or Er:YAG laser (20.3 or 38.2 J/cm(2)) in non-contact mode.

17 ler and </=0.03% with the Er:YAG laser (38.2 J/cm(2)).

18 tant was derived for the soot (1.4 x 10(-20) J) using the colloidal chemistry approach.

19 s the retention of thermal energy (about 200 J g(-1)) in the materials for at least 10 h at temperatu

20 cing, nonbarotraumatic, cathodal 100 and 200 J applications were delivered at 2 different sites on th

21 A single 200 J application can create deep myocardial lesions.

22 Male FVB/N-Tg(MMTVneu)202Mul/J (Her2) transgenic mice were bred to female MNX mice ha

23 g entropy changes of 14 J kg(-1) K(-1) or 22 J kg (-1) K(-1), and corresponding magnetocaloric coolin

24 s 11-20, mice (n = 120) were exposed to 2240 J/m(2) UV-B light, 3x/week, and tumors were tracked week

25 ture energy ([Formula: see text]350 to 1,240 J/m(2)).

26 L/6-Tg (Csf1r-EGFP-NGFR/FKBP1A/TNFRSF6) 2Bck/J mice received rhodamine-conjugated ferumoxytol.

27 that were determined from 1D (1)H NMR and 2D J-resolved HMBC spectra in various solvents.

28 ved from measured proton chemical shifts, (3)J-values, and (1)H-(1)H-NOESY contacts.

29 iciency was demonstrated at doses of 150-300 J/cm(2) against two different in vivo tumor models, achi

30 G c to decrease from 0.424 J m(-2) to 0.365 J m(-2).

31 y for CO2 adsorption (Deltasads = -204 +/- 4 J/mol.K) positioning the step in the optimal range for c

32 -66B.U.), alveograph W (193x10(-4)-223x10(-4)J) and P/L (2.2-2.7).

33 For a typical UV disinfection dose (400 J/m(2)), various extents of phototransformations (10-90%

34 dhesion toughness G c to decrease from 0.424 J m(-2) to 0.365 J m(-2).

35 ss G c decreases from 0.543 J m(-2) to 0.438 J m(-2).

36 the maximal energy storage density of 15.48 J/cm(3) due to its higher breakdown strength.

37 howed low phototoxicity (IC50 > 100 muM, 1.5 J/cm(2)) toward human HEp2 cells.

38 205 MeV (0.25% spread) using 250 mJ and 2.5 J lasers respectively.

39 Energy densities of 510 J/g (max: 698 J/g) have been achieved in azobenzene-base

40 as maximized of 63.91% at energy input of 54 J/g and pH of 12.0, and variance analysis indicated that

41 adhesion toughness G c decreases from 0.543 J m(-2) to 0.438 J m(-2).

42 erial capability under a low light dose (0.6 J cm(-2) ) toward Gram-negative bacteria E. coli, making

43 deficient mouse (Casp1(Null)) on the C57BL/6 J background that expressed caspase-11.

44 uch larger region of infarction than C57BL/6 J mice after tMCAO challenge.

45 established the PSD model using male C57BL/6 J mice by photothrombosis of the left anterior cortex co

46 Female C57BL/6 J mice were fed a control diet (CON) or a high-fat diet

47 Male C57BL/6 J mice were fed either a low-fat (10% kcal) or one of th

48 e and neurological deficit scores in C57BL/6 J mice, these effect were reduced in TASK(-/-) mice.

49 izukaol F reduced gluconeogenesis in C57BL/6 J mice.

50 weight and blood cell populations in C57BL/6 J mice.

51 Allergic asthma was induced in C57BL/6 J wild-type mice, Toll-like receptor (TLR) 4 knockout (T

52 nduced animal (an isogenic strain of C57BL/6 J:129S1/SvImJ mice) model of NAFLD that closely mimics m

53 The observation of a minimum of 640 J sr(-1) directly backscattered from noise, correspondin

54 Energy densities of 510 J/g (max: 698 J/g) have been achieved in azobenzene-based syndiotactic

55 roximately 600%, and fracture energy of 7300 J m(-2) ).

56 enditure compared with vehicle (17.5 +/- 4.9 J .

57 e toughness of as high as approximately 9200 J m(-2) , their mechanical properties match or exceed th

58 Conversely, creatinine demonstrated a J-shaped relationship with mortality, so that in the maj

59 f the carboxylates bound to the capsule in a J-shaped motif with the carboxylate at the equatorial re

60 Thus, the esters were observed to bind in a J-shaped, U-shaped (the turn in the guest occupying a po

61 ent KPro implantation by a single surgeon (A.J.A.) with or without 1 concurrent Ahmed GDD (New World

62 expression of Venus-dysferlin chimeras in A/J fibres restored the full amplitude of the Ca(2+) trans

63 m adenoma formation in the lung tissues in A/J mice.

64 (2+) transients elicited in dysferlin-null A/J myofibres were smaller than control A/WySnJ fibres.

65 ansients by approximately 20% in uninjured A/J fibres, restoring them to control values.

66 mary, we illustrate the benefit of acquiring J-resolved experiments alongside conventional 1D (1)H NM

67 mediating the binding between chNHE1 and ALV-J gp85.

68 es 28 to 39 both could effectively block ALV-J infection.

69 1 residues 28 to 39, effectively blocked ALV-J infection.

70 insights into the control strategies for ALV-J infection.

71 ith ALV-J gp85; ECL3 is also involved in ALV-J gp85 binding.

72 itical for receptor function and mediate ALV-J entry.IMPORTANCE chNHE1 is a cellular receptor of ALV-

73 of ALV-J gp85 and efficiently mediating ALV-J cell entry.

74 ding and entry assays to map the minimal ALV-J gp85-binding domain of chNHE1.

75 HE1 residues converted the nonfunctional ALV-J receptor huNHE1 to a functional one.

76 domain responsible for chNHE1 binding of ALV-J gp85 and efficiently mediating ALV-J cell entry.

77 CL1, suggesting that the binding site of ALV-J gp85 on chNHE1 is probably located on the apex of the

78 al domain required for chNHE1 binding of ALV-J gp85.

79 ORTANCE chNHE1 is a cellular receptor of ALV-J, a retrovirus that causes infections in chickens and s

80 tribute to a better understanding of the ALV-J infection mechanism and also provide new insights into

81 terminants of chNHE1 responsible for the ALV-J receptor activity, a series of chimeric receptors was

82 of the subgroup J avian leukosis virus (ALV-J).

83 ctional ECL that interacts directly with ALV-J gp85; ECL3 is also involved in ALV-J gp85 binding.

84 (SPF) layer chickens were infected with ALV-J or maintained as non-injected controls.

85 ies analysis proposed by Flanders et al. (Am J Epidemiol.

86 men's Health Initiative, Prentice et al. (Am J Epidemiol.

87 In the accompanying article (Am J Epidemiol.

88 nts on Epidemiology in an Age of Change" (Am J Epidemiol.

89 d using Rothman's sufficient cause model (Am J Epidemiol.

90 e related to the risk of early mortality (Am J Epidemiol.

91 Kuller and Reisler's 1971 publication (Am J Epidemiol.

92 and Estimation in Case-Referent Studies" (Am J Epidemiol.

93 = 2), D (n = 1), F1 (n = 1), H (n = 3), and J (n = 1) genomes.

94 orously quantifying differences in V, D, and J gene segment utilization.

95 type II implanted by 2 surgeons (C.H.D. and J.C.).

96 Additionally, three shearinines D, F, and J produced by the fungal pathogen Escovopsis TZ49 were d

97 in cavity mediated hybridization of GaAs and J-aggregate excitons in the strong coupling regime under

98 oup M subtypes (i.e., subtypes A-D, F-H, and J-K).

99 hat structural revisions of curcusones I and J are needed.

100 diterpene natural products curcusones I and J in racemic form were achieved.

101 in pairing, functional network integrity and J-protein specialization.

102 ican Y chromosome markers (E-M81, E-M78, and J-M267) and mitochondrial lineages such as U6b, in addit

103 Rates of S. palmetto and J. virginiana mortality increased nonlinearly over time

104 determined that F1 female offspring (B6D2F1/J) from HFD-fed dams have decreased motivation (decrease

105 idate the relationship between 5hmU and base J, we also map base J loci by introducing a chemical tag

106 ta-glucopyranosyl) hydroxymethyluracil (base J), plays a key role during transcription.

107 ip between 5hmU and base J, we also map base J loci by introducing a chemical tagging strategy for th

108 No correlation was found between J-ST point elevation and activation recovery intervals p

109 ry (S-shaped, p<0.0001) and low birthweight (J-shaped, p=0.0001); the adjusted odds ratios (ORs) for

110 in-labeled monocytes into Dysf-deficient BLA/J mice with age-related (2 to 10 months) muscle disease

111 ution of Zn soaps in the painting Alchemy by J.

112 integrity ..." Read more in the Editorial by J.

113 ng the classification originally proposed by J.

114 4 expression in the bone marrow of BALB/c/By/J mice prior to L. monocytogenes exposure in the gut did

115 membrane depolarization in order to open (C.J.P. et al., manuscript submitted).

116 We performed studies with C57Bl6-J (control) and interleukin 6-knockout mice.

117 Female C57BL6/J mice were fed ethanol or pair-fed control diets and tr

118 Five week old lean and obese female C57BL6/J mice were mated with chow fed males.

119 mechanical measurements were made in C57BL6/J mice and congenic Sftpd-/- mice at 8, 27 and 80 weeks

120 s for opportunities to attack younger C57BL6/J mice.

121 nal ganglion cell (RGC) type in mouse called J-RGC.

122 ascular events, giving rise to the so-called J-curve phenomenon.

123 ure for interclass complexation of canonical J-proteins.

124 ially influenced by changes in TA -Do Carmo, J.

125 or pulmonary fibrosis.-Gu, L., Larson-Casey, J.

126 Using a murine CB6F1/J hybrid model of respiratory syncytial virus (RSV) infe

127 ic potentials in CD-1Cx30(A88V/A88V) and CBA/J mice are comparable.

128 hleae of CD-1Cx30(A88V/A88V) compared to CBA/J mice with sensitive high-frequency hearing, suggesting

129 Here we report the detection of CH(+) (J = 1-0) emission and absorption lines in the spectra of

130 ery in 1961 of dibenzo-18-crown-6 by Charles J.

131 nduced bone loss in adult female rats.-Chen, J.-R., Lazarenko, O.

132 e work.-Liu, C.-H., Wang, Z., Sun, Y., Chen, J.

133 wo three-way junctions with a closed (Closed-J) or open (Open-J) junction and their appropriate contr

134 For instance, Closed-J sacrifices thermal stability of the Dumbbell structure

135 arison of the experimental results of Closed-J and Open-J with those of their component stem-loop mot

136 her number of basepair stacks whereas Closed-J has a defined structure and retains the basepair stack

137 ity of the Japan Proton Accelerator Complex (J-PARC) for an iron sample.

138 teins from networking, which ensures correct J-protein pairing, functional network integrity and J-pr

139 quantitative estimate of the Hund's coupling J=0.4 eV.

140 in the present work, we verify the couplings J = 5.13 K, alpha = 0.23(1) and Lande factors g parallel

141 aM-binding domain to a site within the CPK28 J domain that overlaps with the known site of intramolec

142 CMB-M0042F), yielded the known cytochalasins J (1) and H (2), together with five new analogues, cytoc

143 s undergo somatic hypermutation (SHM) of V(D)J exons followed by selection for SHMs that increase ant

144 machine that was developed to visualize V(D)J gene usage.

145 by the V segment and CDR3 encoded by the V(D)J junction region.

146 o elucidate such requirements, we used a V(D)J passenger allele system to assay, in mouse GC B cells,

147 of a B-cell clonal lineage, the initial V(D)J rearrangement.

148 b KI models expressing deduced precursor V(D)J rearrangements of mature bnAbs or unrearranged germlin

149 n MULE, hAT and Transib elements and the V(D)J recombinase.

150 S C terminus is dispensable for cellular V(D)J recombination and in vitro nuclease assays with C-term

151 fferent recombination reactions, such as V(D)J recombination and transposition.

152 n increased ARTEMIS activity in cellular V(D)J recombination assays.

153 l role in adaptive immunity by mediating V(D)J recombination in developing lymphocytes.

154 V(D)J recombination is initiated by the recombination-activa

155 nsposition pathway resembles that of the V(D)J recombination reaction and the mechanism of hAT and Tr

156 istinct from DNA transposition and trans-V(D)J recombination that destabilizes the genome and shares

157 (D), and joining (J) subgenic elements (V(D)J recombination).

158 NA insertions by transposition and trans-V(D)J recombination, but only few such putative events have

159 the germline TCR locus, a process termed V(D)J recombination, that has the potential to generate mark

160 consequences of BCL11A/RAG modulation on V(D)J recombination.

161 d caused by defects in lymphoid-specific V(D)J recombination.

162 s that the variable (diversity) joining [V(D)J] recombination-mediated assembly of diverse B and T ly

163 r initiating variable-diversity-joining [V(D)J] segment recombination, an essential process for antig

164 ons are assembled developmentally from V, D, J gene segments.

165 mature bnAbs or unrearranged germline V, D, J segments (that can be assembled into variable region e

166 he expertise in these trials of one of us (D.J.S.).

167 M., Lam, L., Dang, J., Jiang, W., Rodriguez, F., Rigali, E., Weitzman, S.,

168 be applied to treat traumatic injury, David J Lockey calls for research to determine which of these

169 , Tian, S., Zhu, R., Bai, S., Fu, K., Davis, J.

170 In intervention studies using DBA2/J and Nos3 (eNos) KO mouse models of diabetes, TEPP-46 t

171 ntribute to a short-circuit current density (J sc ) of 17.07 mA cm(-2) .

172 m, and a high short-circuit current density (J SC ) of 17.92 mA cm(-2) .

173 er overall T c and critical-current-density, J c .

174 uce the total scan time in multi-dimensional J-resolved spectroscopic imaging (JRESI) using an echo-p

175 etabolite-edited spectra and two-dimensional J-resolved spectroscopy data were acquired from the pari

176 ction surface formed by helix II of the DnaJ J-domain and a structurally contiguous region of DnaK, i

177 eration when interpreting the results of DQF J-resolved experiments, and conversely, these experiment

178 r results show that the magnitude of the DQF J splitting is influenced by both the crystallographic s

179 ed using (11)B double-quantum-filtered (DQF) J-resolved solid-state nuclear magnetic resonance (SSNMR

180 ds were found, in average, for F, JD, and DS J. spicigera, respectively.

181 -0.94g with the DPPH method for F, JD and DS J. spicigera, respectively.

182 By analysing the E - J curves for determining Jc, a non-Ohmic linear differen

183 Using C57BL/6J-Mc1r(e/e)J mice, in which endogenous MC1R is prematurely terminat

184 c arbors and synaptic distribution, enabling J-RGC connectivity and thus, function.

185 y materials is well documented [for example, J.

186 reveals that the spiro-structures facilitate J-stacking in the solid state.

187 nd its potential benefits to society.-Flier, J.

188 EPR spectra, using a developed algorithm for J modulation in flexible organic biradicals, supports th

189 ion in the HPD motif, which is necessary for J-protein-Hsp70 interactions, suggesting the phenomenon

190 diaryl chelate unit have been found to form J-aggregates in methanol-water solvent mixture and brigh

191 Spodoptera frugiperda (J.

192 omenon observed is independent on functional J-protein-Hsp70 interactions.

193 re subtypes, including subtypes A1, F, G, H, J, and K and unclassified fragments, including one subty

194 H2A.J accumulation may thus promote the signalling of senesc

195 H2A.J also accumulates in mice with aging in a tissue-specif

196 Here we show that histone H2A.J, a poorly studied H2A variant found only in mammals, a

197 phenotype (SASP), and over expression of H2A.J increases the expression of some of these genes in pro

198 Knock-down of H2A.J inhibits the expression of inflammatory genes that con

199 (2) dendritic complexity, which affects how J-RGCs sample space; (3) asymmetry, which contributes to

200 ailability of Hsp70 cofactors, such as Hsp40 J-proteins and nucleotide exchange factors.

201 Hsp70 and the Hsp70 cochaperones, an Hsp40 (J-protein) and a nucleotide exchange factor.

202 Nine haplogroups (H, I, J, K, T, U, V, W, and X) were present in the population.

203 Because haplogroups I, J, K, O-X, T, and U encompass 55% of the European popula

204 ified from histological sections using Image J skeleton and fractal analysis procedures at three time

205 This fundamental change in J-protein biology during the prokaryote-to-eukaryote tra

206 In controls, there was minimal change in J-ST point elevation, conduction delay, or activation re

207 We found dendritic growth in J-RGCs is accompanied by a refinement in dendritic self-

208 includes residues that are also involved in J-protein binding, suggesting a functional interplay amo

209 sts, especially at low speeds, may result in J- or U-shaped metabolism-speed curves; (ii) anaerobic m

210 trnC-trnY is rearranged to trnY-trnW-trnC in J. hyalinus, the first reported gene rearrangement in Me

211 y conducted in 1988-1993 (Jockel et al., Int J Epidemiol.

212 The exchange interaction, J, between two spin centres is a convenient measure of t

213 These observations establish BiP and its J domain co-chaperones as key regulators of the UPR.

214 om variable (V), diversity (D), and joining (J) subgenic elements (V(D)J recombination).

215 domain (CLD) via an autoinhibitory junction (J).

216 nal binding protein for immunoglobulin kappa J region (RBPjkappa), key modulators of adipogenesis and

217 In his Nobel Lecture, J. Stoddart describes how being able to template the for

218 ound in the imaginary worlds of authors like J.K.

219 ntropy per particle s of a magnitude +/-ln2/(J - 1/2) with 2 </= J </= N at low temperatures.

220 s of a magnitude +/-ln2/(J - 1/2) with 2 </= J </= N at low temperatures.

221 ., Jobichen, C., Swaminathan, K., Mizuguchi, J., Iwanaga, S., Nuttall, P.

222 manifesting the realization of the molecular J eff = 3/2 ground state in GaTa4Se8.The strong interact

223 heoretically predicted to form the molecular J eff = 3/2 ground state.

224 laryngeal C neurons in the nodose/jugular (N/J) ganglia.

225 tors of laryngeal sensory C neurons in the N/J ganglia retrogradely traced by 1,1'-dioctadecyl-3,3,3'

226 eme in Fig.1b is adapted from Fig.1a in Noh, J., Jeong, S.

227 eterozygous (immunocompetent) mice of the NU/J strain progressed to high grade dysplasia and to carci

228 nt complexation between different classes of J-proteins, which expands the range of protein aggregate

229 ersive mode is seen, as the concentration of J-aggregates is increased.

230 prevalence and evolutionary conservation of J-protein complexation and cooperation in disaggregation

231 .radical coupling with coupling constants of J = -67.5 and -66.8 cm(-1) for 1 and 2, respectively.

232 left ventricle, correlated to the degree of J-ST point elevation (Pearson R, 0.81; P<0.001).

233 te in different spatiotemporal dimensions of J-RGC dendritic patterning to generate the substrate for

234 After exclusion of J and Q codes, the total payments to and the number of c

235 array and molecular excitons in the form of J-aggregates dispersed on the hybrid structure.

236 s consistent with the well-known function of J-domains of transferring unfolded and misfolded protein

237 d quartet with effective angular momentum of J eff = 1/2 and 3/2, respectively.

238 culations of these structures gave values of J and Mossbauer parameters in agreement with experiment.

239 crystal structure gave calculated values of J incompatible with the spectroscopic results.

240 ryl chelate unit has a significant impact on J-aggregates and fluorescence of BODIPYs.

241 he experimental results of Closed-J and Open-J with those of their component stem-loop motifs allowed

242 ees (5), or free energy, value seen for Open-J at low salt.

243 However, Open-J is more stable due to a higher enthalpy contribution f

244 stability of the Dumbbell structure in Open-J but causes the CTATC stem to fully fold.

245 tions with a closed (Closed-J) or open (Open-J) junction and their appropriate control stem-loop moti

246 g nexin one (SNX-1) and its binding partner, J-domain protein RME-8, sort cargo away from degradation

247 Peak J-ST point elevation was calculated from the surface ECG

248 leration is the five-dimensional echo planar J-resolved spectroscopic imaging (5D EP-JRESI) sequence,

249 equence, called Multi-Echo based Echo-Planar J-resolved Spectroscopic Imaging (ME-EP-JRESI), was eval

250 the new computational framework can predict J-factors within an order of magnitude of experimental m

251 entally measured and theoretically predicted J-factors.

252 h spectroscopy evidence obtained previously (J.

253 The Yersinia outer protein J (YopJ) family of bacterial effectors depends on a nove

254 imaged as a function of the permeation rate, J, droplet radius, R, membrane permeance, k, water visco

255 However, the molecular details of RBP-J/RITA interactions are unclear.

256 cells failed to control the up-regulated RBP-J expression, leading to suppression of OC genes.

257 ence-binding protein at the Jkappa site (RBP-J) protein, a potent OC inhibitor.

258 c steps are three Julia-Kocienski reactions (J-K), for the formation of the C5-C6, C9-C10, and C17-C1

259 e inner plexiform layer (IPL), which renders J-RGCs responsive to light decrements; and (5) distribut

260 r occupancy of the other pole), or a reverse J-shaped motif (ester moiety and turn each occupying a p

261 In the case-mix adjusted model, a reverse-J-shaped association was observed; a small albeit signif

262 e is quantified by the Jacobson-Stockmayer's J-factor, which measures the propensity for DNA loop for

263 DNP-enhanced 2D (29)Si{(29)Si} J-mediated NMR analyses of calcined Si-SSZ-70 at natural

264 60 y (n = 2990) there was a non-significant 'J'-shaped association.

265 pleen of C57BL/6 and peripheral blood of SJL/J mice along with a decreased TH17 phenotype within CD4(

266 order, whereas animals intercrossed with SJL/J mice (F1.Q54) have a severe phenotype.

267 Signature alterations exclude some J-proteins from networking, which ensures correct J-prot

268 growth factor (anti-VEGF) treatment-specific J-codes (J0178, J2778, J9035, J3490, and J3590).

269 cale targets, up to 5 x 10(9) protons/MeV/sr/J with energies up to 45(+/-5) MeV in a collimated ( 6 d

270 Magnitude of ST (J point) elevation in the type I BrS pattern is attribut

271 tein, is a cellular receptor of the subgroup J avian leukosis virus (ALV-J).

272 f long-range magnetic order down to 1.8 K (T/J approximately 0.02; J is the dominant exchange constan

273 We used male BTBR T(+)Itpr3(tf) /J (BTBR) mice, a model that reproduces most of the core

274 on resonance experiment, we demonstrate that J can be quantified to high precision even in the presen

275 The J((11)B,(11)B) coupling constants of various salts of th

276 The J-domain sequence or a fragment thereof was conjugated t

277 he attained blood pressure level because the J curve aligns with the SBP target.

278 te of intramolecular interaction between the J domain and the CLD.

279 while continuing to accurately describe the J-factors of longer sequences.

280 graphic symmetry are both accounted for, the J((11)B,(11)B) coupling constants for various [B2(CN)6](

281 We find that the excitations involving the J eff = 1/2 molecular orbital are absent only at the Ta

282 pressure targets, an identical shape of the J curve was present, with a nadir for cardiovascular eve

283 st step to exploring the consequences of the J eff = 3/2 state.

284 glycan sites on the mu chain and one on the J-chain.

285 n a functionally characterized RGC type, the J-RGC, demonstrating distinct mechanisms that operate in

286 Using the J-Lat 10.6 model of persistent infection, we demonstrate

287 ment of secretion was observed even when the J-domain had a mutation in the HPD motif, which is neces

288 antly upregulated when co-expressed with the J-domain fusion protein.

289 as compared to the corresponding theoretical J coupling constants for cases where (1) there is an abs

290 These J couplings are indicative of the presence of noncovalen

291 By comparing these J-couplings to those reported previously in the alpha- a

292 ble molecules was determined largely through J-based configurational analysis, but has been found to

293 atherosclerosis in Apob(tm2Sgy)/Ldlr(tm1Her/J) mice.

294 thout MSCs in diabetic B6.129S7-Rag1(tm1Mom)/J mice.

295 ER membrane by binding to the transmembrane J-protein B14.

296 nce of five dendritic features that underlie J-RGC physiology: (1) dendritic field size, which approx

297 was specifically characterized by reduced V-J segment distance in recombined sequences, suggesting d

298 ate levels were determined continuously with J-difference-editing (1)H-MRS, and time curves were anal

299 an a threshold level ( Wirth and co-workers, J.

300 h normal and steatotic liver.-Li, Z., Zhang, J., Mulholland, M., Zhang, W. mTOR activation protects l