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1                                              J-couplings in methyl alpha-l-[6-(13)C]idopyranosiduroni
2                                              J. spicigera showed colors (green-iridescent, green-yell
3 ecreased from 4.3 (3.5-6.3) to 2.1 (1.5-5.0) J/min, respectively (p = 0.031).
4  UVB irradiation and healing (UVB doses 1.01 J/cm(2) once daily for four days).
5 order down to 1.8 K (T/J approximately 0.02; J is the dominant exchange constant) renders the compoun
6 hanical work close to zero (-0.012 +/- 0.054 J/kg).
7 nditions at low laser excitation energy (0.1 J/cm(2) at 800 nm) without the use of photoinitiators, w
8 ) for 1000 seconds for a total fluence of 10 J/cm2.
9  in the range of 52-55 degrees C and 100-115 J/g, respectively.
10  capable of generating entropy changes of 14 J kg(-1) K(-1) or 22 J kg (-1) K(-1), and corresponding
11 ed into the seed at an oblique angle with 14 J sr(-1), and net gains of more than eight orders of mag
12 ich display high-energy densities (up to 166 J cm(-3)) and efficiencies (up to 96%).
13  can detect a single photon (10(-18)-10(-19) J) of visible light.
14  delivering high energy densities, i.e., 1.2 J cm(-3) , even at a temperature close to the glass tran
15 e aggregates, whereas the Er:YAG laser (38.2 J/cm(2)) completely stripped away the plaque and TiO2 la
16 er (metal tip) or Er:YAG laser (20.3 or 38.2 J/cm(2)) in non-contact mode.
17 ler and </=0.03% with the Er:YAG laser (38.2 J/cm(2)).
18 tant was derived for the soot (1.4 x 10(-20) J) using the colloidal chemistry approach.
19 s the retention of thermal energy (about 200 J g(-1)) in the materials for at least 10 h at temperatu
20 cing, nonbarotraumatic, cathodal 100 and 200 J applications were delivered at 2 different sites on th
21                                 A single 200 J application can create deep myocardial lesions.
22                 Male FVB/N-Tg(MMTVneu)202Mul/J (Her2) transgenic mice were bred to female MNX mice ha
23 g entropy changes of 14 J kg(-1) K(-1) or 22 J kg (-1) K(-1), and corresponding magnetocaloric coolin
24 s 11-20, mice (n = 120) were exposed to 2240 J/m(2) UV-B light, 3x/week, and tumors were tracked week
25 ture energy ([Formula: see text]350 to 1,240 J/m(2)).
26 L/6-Tg (Csf1r-EGFP-NGFR/FKBP1A/TNFRSF6) 2Bck/J mice received rhodamine-conjugated ferumoxytol.
27 that were determined from 1D (1)H NMR and 2D J-resolved HMBC spectra in various solvents.
28 ved from measured proton chemical shifts, (3)J-values, and (1)H-(1)H-NOESY contacts.
29 iciency was demonstrated at doses of 150-300 J/cm(2) against two different in vivo tumor models, achi
30  G c to decrease from 0.424 J m(-2) to 0.365 J m(-2).
31 y for CO2 adsorption (Deltasads = -204 +/- 4 J/mol.K) positioning the step in the optimal range for c
32 -66B.U.), alveograph W (193x10(-4)-223x10(-4)J) and P/L (2.2-2.7).
33      For a typical UV disinfection dose (400 J/m(2)), various extents of phototransformations (10-90%
34 dhesion toughness G c to decrease from 0.424 J m(-2) to 0.365 J m(-2).
35 ss G c decreases from 0.543 J m(-2) to 0.438 J m(-2).
36  the maximal energy storage density of 15.48 J/cm(3) due to its higher breakdown strength.
37 howed low phototoxicity (IC50 > 100 muM, 1.5 J/cm(2)) toward human HEp2 cells.
38  205 MeV (0.25% spread) using 250 mJ and 2.5 J lasers respectively.
39                     Energy densities of 510 J/g (max: 698 J/g) have been achieved in azobenzene-base
40 as maximized of 63.91% at energy input of 54 J/g and pH of 12.0, and variance analysis indicated that
41  adhesion toughness G c decreases from 0.543 J m(-2) to 0.438 J m(-2).
42 erial capability under a low light dose (0.6 J cm(-2) ) toward Gram-negative bacteria E. coli, making
43 deficient mouse (Casp1(Null)) on the C57BL/6 J background that expressed caspase-11.
44 uch larger region of infarction than C57BL/6 J mice after tMCAO challenge.
45 established the PSD model using male C57BL/6 J mice by photothrombosis of the left anterior cortex co
46                               Female C57BL/6 J mice were fed a control diet (CON) or a high-fat diet
47                                 Male C57BL/6 J mice were fed either a low-fat (10% kcal) or one of th
48 e and neurological deficit scores in C57BL/6 J mice, these effect were reduced in TASK(-/-) mice.
49 izukaol F reduced gluconeogenesis in C57BL/6 J mice.
50 weight and blood cell populations in C57BL/6 J mice.
51       Allergic asthma was induced in C57BL/6 J wild-type mice, Toll-like receptor (TLR) 4 knockout (T
52 nduced animal (an isogenic strain of C57BL/6 J:129S1/SvImJ mice) model of NAFLD that closely mimics m
53          The observation of a minimum of 640 J sr(-1) directly backscattered from noise, correspondin
54       Energy densities of 510 J/g (max: 698 J/g) have been achieved in azobenzene-based syndiotactic
55 roximately 600%, and fracture energy of 7300 J m(-2) ).
56 enditure compared with vehicle (17.5 +/- 4.9 J .
57 e toughness of as high as approximately 9200 J m(-2) , their mechanical properties match or exceed th
58        Conversely, creatinine demonstrated a J-shaped relationship with mortality, so that in the maj
59 f the carboxylates bound to the capsule in a J-shaped motif with the carboxylate at the equatorial re
60  Thus, the esters were observed to bind in a J-shaped, U-shaped (the turn in the guest occupying a po
61 ent KPro implantation by a single surgeon (A.J.A.) with or without 1 concurrent Ahmed GDD (New World
62  expression of Venus-dysferlin chimeras in A/J fibres restored the full amplitude of the Ca(2+) trans
63 m adenoma formation in the lung tissues in A/J mice.
64 (2+) transients elicited in dysferlin-null A/J myofibres were smaller than control A/WySnJ fibres.
65 ansients by approximately 20% in uninjured A/J fibres, restoring them to control values.
66 mary, we illustrate the benefit of acquiring J-resolved experiments alongside conventional 1D (1)H NM
67 mediating the binding between chNHE1 and ALV-J gp85.
68 es 28 to 39 both could effectively block ALV-J infection.
69 1 residues 28 to 39, effectively blocked ALV-J infection.
70 insights into the control strategies for ALV-J infection.
71 ith ALV-J gp85; ECL3 is also involved in ALV-J gp85 binding.
72 itical for receptor function and mediate ALV-J entry.IMPORTANCE chNHE1 is a cellular receptor of ALV-
73  of ALV-J gp85 and efficiently mediating ALV-J cell entry.
74 ding and entry assays to map the minimal ALV-J gp85-binding domain of chNHE1.
75 HE1 residues converted the nonfunctional ALV-J receptor huNHE1 to a functional one.
76 domain responsible for chNHE1 binding of ALV-J gp85 and efficiently mediating ALV-J cell entry.
77 CL1, suggesting that the binding site of ALV-J gp85 on chNHE1 is probably located on the apex of the
78 al domain required for chNHE1 binding of ALV-J gp85.
79 ORTANCE chNHE1 is a cellular receptor of ALV-J, a retrovirus that causes infections in chickens and s
80 tribute to a better understanding of the ALV-J infection mechanism and also provide new insights into
81 terminants of chNHE1 responsible for the ALV-J receptor activity, a series of chimeric receptors was
82  of the subgroup J avian leukosis virus (ALV-J).
83 ctional ECL that interacts directly with ALV-J gp85; ECL3 is also involved in ALV-J gp85 binding.
84  (SPF) layer chickens were infected with ALV-J or maintained as non-injected controls.
85 ies analysis proposed by Flanders et al. (Am J Epidemiol.
86 men's Health Initiative, Prentice et al. (Am J Epidemiol.
87              In the accompanying article (Am J Epidemiol.
88 nts on Epidemiology in an Age of Change" (Am J Epidemiol.
89 d using Rothman's sufficient cause model (Am J Epidemiol.
90 e related to the risk of early mortality (Am J Epidemiol.
91    Kuller and Reisler's 1971 publication (Am J Epidemiol.
92 and Estimation in Case-Referent Studies" (Am J Epidemiol.
93  = 2), D (n = 1), F1 (n = 1), H (n = 3), and J (n = 1) genomes.
94 orously quantifying differences in V, D, and J gene segment utilization.
95  type II implanted by 2 surgeons (C.H.D. and J.C.).
96    Additionally, three shearinines D, F, and J produced by the fungal pathogen Escovopsis TZ49 were d
97 in cavity mediated hybridization of GaAs and J-aggregate excitons in the strong coupling regime under
98 oup M subtypes (i.e., subtypes A-D, F-H, and J-K).
99 hat structural revisions of curcusones I and J are needed.
100  diterpene natural products curcusones I and J in racemic form were achieved.
101 in pairing, functional network integrity and J-protein specialization.
102 ican Y chromosome markers (E-M81, E-M78, and J-M267) and mitochondrial lineages such as U6b, in addit
103                     Rates of S. palmetto and J. virginiana mortality increased nonlinearly over time
104  determined that F1 female offspring (B6D2F1/J) from HFD-fed dams have decreased motivation (decrease
105 idate the relationship between 5hmU and base J, we also map base J loci by introducing a chemical tag
106 ta-glucopyranosyl) hydroxymethyluracil (base J), plays a key role during transcription.
107 ip between 5hmU and base J, we also map base J loci by introducing a chemical tagging strategy for th
108             No correlation was found between J-ST point elevation and activation recovery intervals p
109 ry (S-shaped, p<0.0001) and low birthweight (J-shaped, p=0.0001); the adjusted odds ratios (ORs) for
110 in-labeled monocytes into Dysf-deficient BLA/J mice with age-related (2 to 10 months) muscle disease
111 ution of Zn soaps in the painting Alchemy by J.
112 integrity ..." Read more in the Editorial by J.
113 ng the classification originally proposed by J.
114 4 expression in the bone marrow of BALB/c/By/J mice prior to L. monocytogenes exposure in the gut did
115  membrane depolarization in order to open (C.J.P. et al., manuscript submitted).
116             We performed studies with C57Bl6-J (control) and interleukin 6-knockout mice.
117                                Female C57BL6/J mice were fed ethanol or pair-fed control diets and tr
118   Five week old lean and obese female C57BL6/J mice were mated with chow fed males.
119  mechanical measurements were made in C57BL6/J mice and congenic Sftpd-/- mice at 8, 27 and 80 weeks
120 s for opportunities to attack younger C57BL6/J mice.
121 nal ganglion cell (RGC) type in mouse called J-RGC.
122 ascular events, giving rise to the so-called J-curve phenomenon.
123 ure for interclass complexation of canonical J-proteins.
124 ially influenced by changes in TA -Do Carmo, J.
125 or pulmonary fibrosis.-Gu, L., Larson-Casey, J.
126                         Using a murine CB6F1/J hybrid model of respiratory syncytial virus (RSV) infe
127 ic potentials in CD-1Cx30(A88V/A88V) and CBA/J mice are comparable.
128 hleae of CD-1Cx30(A88V/A88V) compared to CBA/J mice with sensitive high-frequency hearing, suggesting
129       Here we report the detection of CH(+) (J = 1-0) emission and absorption lines in the spectra of
130 ery in 1961 of dibenzo-18-crown-6 by Charles J.
131 nduced bone loss in adult female rats.-Chen, J.-R., Lazarenko, O.
132 e work.-Liu, C.-H., Wang, Z., Sun, Y., Chen, J.
133 wo three-way junctions with a closed (Closed-J) or open (Open-J) junction and their appropriate contr
134                         For instance, Closed-J sacrifices thermal stability of the Dumbbell structure
135 arison of the experimental results of Closed-J and Open-J with those of their component stem-loop mot
136 her number of basepair stacks whereas Closed-J has a defined structure and retains the basepair stack
137 ity of the Japan Proton Accelerator Complex (J-PARC) for an iron sample.
138 teins from networking, which ensures correct J-protein pairing, functional network integrity and J-pr
139 quantitative estimate of the Hund's coupling J=0.4 eV.
140 in the present work, we verify the couplings J = 5.13 K, alpha = 0.23(1) and Lande factors g parallel
141 aM-binding domain to a site within the CPK28 J domain that overlaps with the known site of intramolec
142 CMB-M0042F), yielded the known cytochalasins J (1) and H (2), together with five new analogues, cytoc
143 s undergo somatic hypermutation (SHM) of V(D)J exons followed by selection for SHMs that increase ant
144  machine that was developed to visualize V(D)J gene usage.
145 by the V segment and CDR3 encoded by the V(D)J junction region.
146 o elucidate such requirements, we used a V(D)J passenger allele system to assay, in mouse GC B cells,
147  of a B-cell clonal lineage, the initial V(D)J rearrangement.
148 b KI models expressing deduced precursor V(D)J rearrangements of mature bnAbs or unrearranged germlin
149 n MULE, hAT and Transib elements and the V(D)J recombinase.
150 S C terminus is dispensable for cellular V(D)J recombination and in vitro nuclease assays with C-term
151 fferent recombination reactions, such as V(D)J recombination and transposition.
152 n increased ARTEMIS activity in cellular V(D)J recombination assays.
153 l role in adaptive immunity by mediating V(D)J recombination in developing lymphocytes.
154                                          V(D)J recombination is initiated by the recombination-activa
155 nsposition pathway resembles that of the V(D)J recombination reaction and the mechanism of hAT and Tr
156 istinct from DNA transposition and trans-V(D)J recombination that destabilizes the genome and shares
157  (D), and joining (J) subgenic elements (V(D)J recombination).
158 NA insertions by transposition and trans-V(D)J recombination, but only few such putative events have
159 the germline TCR locus, a process termed V(D)J recombination, that has the potential to generate mark
160 consequences of BCL11A/RAG modulation on V(D)J recombination.
161 d caused by defects in lymphoid-specific V(D)J recombination.
162 s that the variable (diversity) joining [V(D)J] recombination-mediated assembly of diverse B and T ly
163 r initiating variable-diversity-joining [V(D)J] segment recombination, an essential process for antig
164 ons are assembled developmentally from V, D, J gene segments.
165  mature bnAbs or unrearranged germline V, D, J segments (that can be assembled into variable region e
166 he expertise in these trials of one of us (D.J.S.).
167                           M., Lam, L., Dang, J., Jiang, W., Rodriguez, F., Rigali, E., Weitzman, S.,
168  be applied to treat traumatic injury, David J Lockey calls for research to determine which of these
169 , Tian, S., Zhu, R., Bai, S., Fu, K., Davis, J.
170           In intervention studies using DBA2/J and Nos3 (eNos) KO mouse models of diabetes, TEPP-46 t
171 ntribute to a short-circuit current density (J sc ) of 17.07 mA cm(-2) .
172 m, and a high short-circuit current density (J SC ) of 17.92 mA cm(-2) .
173 er overall T c and critical-current-density, J c .
174 uce the total scan time in multi-dimensional J-resolved spectroscopic imaging (JRESI) using an echo-p
175 etabolite-edited spectra and two-dimensional J-resolved spectroscopy data were acquired from the pari
176 ction surface formed by helix II of the DnaJ J-domain and a structurally contiguous region of DnaK, i
177 eration when interpreting the results of DQF J-resolved experiments, and conversely, these experiment
178 r results show that the magnitude of the DQF J splitting is influenced by both the crystallographic s
179 ed using (11)B double-quantum-filtered (DQF) J-resolved solid-state nuclear magnetic resonance (SSNMR
180 ds were found, in average, for F, JD, and DS J. spicigera, respectively.
181 -0.94g with the DPPH method for F, JD and DS J. spicigera, respectively.
182                         By analysing the E - J curves for determining Jc, a non-Ohmic linear differen
183                      Using C57BL/6J-Mc1r(e/e)J mice, in which endogenous MC1R is prematurely terminat
184 c arbors and synaptic distribution, enabling J-RGC connectivity and thus, function.
185 y materials is well documented [for example, J.
186 reveals that the spiro-structures facilitate J-stacking in the solid state.
187 nd its potential benefits to society.-Flier, J.
188 EPR spectra, using a developed algorithm for J modulation in flexible organic biradicals, supports th
189 ion in the HPD motif, which is necessary for J-protein-Hsp70 interactions, suggesting the phenomenon
190  diaryl chelate unit have been found to form J-aggregates in methanol-water solvent mixture and brigh
191                       Spodoptera frugiperda (J.
192 omenon observed is independent on functional J-protein-Hsp70 interactions.
193 re subtypes, including subtypes A1, F, G, H, J, and K and unclassified fragments, including one subty
194                                          H2A.J accumulation may thus promote the signalling of senesc
195                                          H2A.J also accumulates in mice with aging in a tissue-specif
196                Here we show that histone H2A.J, a poorly studied H2A variant found only in mammals, a
197 phenotype (SASP), and over expression of H2A.J increases the expression of some of these genes in pro
198                            Knock-down of H2A.J inhibits the expression of inflammatory genes that con
199  (2) dendritic complexity, which affects how J-RGCs sample space; (3) asymmetry, which contributes to
200 ailability of Hsp70 cofactors, such as Hsp40 J-proteins and nucleotide exchange factors.
201  Hsp70 and the Hsp70 cochaperones, an Hsp40 (J-protein) and a nucleotide exchange factor.
202                      Nine haplogroups (H, I, J, K, T, U, V, W, and X) were present in the population.
203                       Because haplogroups I, J, K, O-X, T, and U encompass 55% of the European popula
204 ified from histological sections using Image J skeleton and fractal analysis procedures at three time
205                   This fundamental change in J-protein biology during the prokaryote-to-eukaryote tra
206     In controls, there was minimal change in J-ST point elevation, conduction delay, or activation re
207                 We found dendritic growth in J-RGCs is accompanied by a refinement in dendritic self-
208  includes residues that are also involved in J-protein binding, suggesting a functional interplay amo
209 sts, especially at low speeds, may result in J- or U-shaped metabolism-speed curves; (ii) anaerobic m
210 trnC-trnY is rearranged to trnY-trnW-trnC in J. hyalinus, the first reported gene rearrangement in Me
211 y conducted in 1988-1993 (Jockel et al., Int J Epidemiol.
212                    The exchange interaction, J, between two spin centres is a convenient measure of t
213     These observations establish BiP and its J domain co-chaperones as key regulators of the UPR.
214 om variable (V), diversity (D), and joining (J) subgenic elements (V(D)J recombination).
215 domain (CLD) via an autoinhibitory junction (J).
216 nal binding protein for immunoglobulin kappa J region (RBPjkappa), key modulators of adipogenesis and
217                        In his Nobel Lecture, J. Stoddart describes how being able to template the for
218 ound in the imaginary worlds of authors like J.K.
219 ntropy per particle s of a magnitude +/-ln2/(J - 1/2) with 2 </= J </= N at low temperatures.
220 s of a magnitude +/-ln2/(J - 1/2) with 2 &lt;/= J </= N at low temperatures.
221 ., Jobichen, C., Swaminathan, K., Mizuguchi, J., Iwanaga, S., Nuttall, P.
222 manifesting the realization of the molecular J eff = 3/2 ground state in GaTa4Se8.The strong interact
223 heoretically predicted to form the molecular J eff = 3/2 ground state.
224 laryngeal C neurons in the nodose/jugular (N/J) ganglia.
225 tors of laryngeal sensory C neurons in the N/J ganglia retrogradely traced by 1,1'-dioctadecyl-3,3,3'
226 eme in Fig.1b is adapted from Fig.1a in Noh, J., Jeong, S.
227 eterozygous (immunocompetent) mice of the NU/J strain progressed to high grade dysplasia and to carci
228 nt complexation between different classes of J-proteins, which expands the range of protein aggregate
229 ersive mode is seen, as the concentration of J-aggregates is increased.
230  prevalence and evolutionary conservation of J-protein complexation and cooperation in disaggregation
231 .radical coupling with coupling constants of J = -67.5 and -66.8 cm(-1) for 1 and 2, respectively.
232  left ventricle, correlated to the degree of J-ST point elevation (Pearson R, 0.81; P<0.001).
233 te in different spatiotemporal dimensions of J-RGC dendritic patterning to generate the substrate for
234                           After exclusion of J and Q codes, the total payments to and the number of c
235  array and molecular excitons in the form of J-aggregates dispersed on the hybrid structure.
236 s consistent with the well-known function of J-domains of transferring unfolded and misfolded protein
237 d quartet with effective angular momentum of J eff = 1/2 and 3/2, respectively.
238 culations of these structures gave values of J and Mossbauer parameters in agreement with experiment.
239  crystal structure gave calculated values of J incompatible with the spectroscopic results.
240 ryl chelate unit has a significant impact on J-aggregates and fluorescence of BODIPYs.
241 he experimental results of Closed-J and Open-J with those of their component stem-loop motifs allowed
242 ees (5), or free energy, value seen for Open-J at low salt.
243                                However, Open-J is more stable due to a higher enthalpy contribution f
244  stability of the Dumbbell structure in Open-J but causes the CTATC stem to fully fold.
245 tions with a closed (Closed-J) or open (Open-J) junction and their appropriate control stem-loop moti
246 g nexin one (SNX-1) and its binding partner, J-domain protein RME-8, sort cargo away from degradation
247                                         Peak J-ST point elevation was calculated from the surface ECG
248 leration is the five-dimensional echo planar J-resolved spectroscopic imaging (5D EP-JRESI) sequence,
249 equence, called Multi-Echo based Echo-Planar J-resolved Spectroscopic Imaging (ME-EP-JRESI), was eval
250  the new computational framework can predict J-factors within an order of magnitude of experimental m
251 entally measured and theoretically predicted J-factors.
252 h spectroscopy evidence obtained previously (J.
253                   The Yersinia outer protein J (YopJ) family of bacterial effectors depends on a nove
254 imaged as a function of the permeation rate, J, droplet radius, R, membrane permeance, k, water visco
255        However, the molecular details of RBP-J/RITA interactions are unclear.
256 cells failed to control the up-regulated RBP-J expression, leading to suppression of OC genes.
257 ence-binding protein at the Jkappa site (RBP-J) protein, a potent OC inhibitor.
258 c steps are three Julia-Kocienski reactions (J-K), for the formation of the C5-C6, C9-C10, and C17-C1
259 e inner plexiform layer (IPL), which renders J-RGCs responsive to light decrements; and (5) distribut
260 r occupancy of the other pole), or a reverse J-shaped motif (ester moiety and turn each occupying a p
261    In the case-mix adjusted model, a reverse-J-shaped association was observed; a small albeit signif
262 e is quantified by the Jacobson-Stockmayer's J-factor, which measures the propensity for DNA loop for
263               DNP-enhanced 2D (29)Si{(29)Si} J-mediated NMR analyses of calcined Si-SSZ-70 at natural
264 60 y (n = 2990) there was a non-significant 'J'-shaped association.
265 pleen of C57BL/6 and peripheral blood of SJL/J mice along with a decreased TH17 phenotype within CD4(
266 order, whereas animals intercrossed with SJL/J mice (F1.Q54) have a severe phenotype.
267           Signature alterations exclude some J-proteins from networking, which ensures correct J-prot
268 growth factor (anti-VEGF) treatment-specific J-codes (J0178, J2778, J9035, J3490, and J3590).
269 cale targets, up to 5 x 10(9) protons/MeV/sr/J with energies up to 45(+/-5) MeV in a collimated ( 6 d
270                             Magnitude of ST (J point) elevation in the type I BrS pattern is attribut
271 tein, is a cellular receptor of the subgroup J avian leukosis virus (ALV-J).
272 f long-range magnetic order down to 1.8 K (T/J approximately 0.02; J is the dominant exchange constan
273             We used male BTBR T(+)Itpr3(tf) /J (BTBR) mice, a model that reproduces most of the core
274 on resonance experiment, we demonstrate that J can be quantified to high precision even in the presen
275                                          The J((11)B,(11)B) coupling constants of various salts of th
276                                          The J-domain sequence or a fragment thereof was conjugated t
277 he attained blood pressure level because the J curve aligns with the SBP target.
278 te of intramolecular interaction between the J domain and the CLD.
279  while continuing to accurately describe the J-factors of longer sequences.
280 graphic symmetry are both accounted for, the J((11)B,(11)B) coupling constants for various [B2(CN)6](
281   We find that the excitations involving the J eff = 1/2 molecular orbital are absent only at the Ta
282  pressure targets, an identical shape of the J curve was present, with a nadir for cardiovascular eve
283 st step to exploring the consequences of the J eff = 3/2 state.
284  glycan sites on the mu chain and one on the J-chain.
285 n a functionally characterized RGC type, the J-RGC, demonstrating distinct mechanisms that operate in
286                                    Using the J-Lat 10.6 model of persistent infection, we demonstrate
287 ment of secretion was observed even when the J-domain had a mutation in the HPD motif, which is neces
288 antly upregulated when co-expressed with the J-domain fusion protein.
289 as compared to the corresponding theoretical J coupling constants for cases where (1) there is an abs
290                                        These J couplings are indicative of the presence of noncovalen
291                           By comparing these J-couplings to those reported previously in the alpha- a
292 ble molecules was determined largely through J-based configurational analysis, but has been found to
293  atherosclerosis in Apob(tm2Sgy)/Ldlr(tm1Her/J) mice.
294 thout MSCs in diabetic B6.129S7-Rag1(tm1Mom)/J mice.
295  ER membrane by binding to the transmembrane J-protein B14.
296 nce of five dendritic features that underlie J-RGC physiology: (1) dendritic field size, which approx
297  was specifically characterized by reduced V-J segment distance in recombined sequences, suggesting d
298 ate levels were determined continuously with J-difference-editing (1)H-MRS, and time curves were anal
299 an a threshold level ( Wirth and co-workers, J.
300 h normal and steatotic liver.-Li, Z., Zhang, J., Mulholland, M., Zhang, W. mTOR activation protects l

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