1 ith vagal afferent nerves can evoke a Bezold-Jarisch (B-J) depressor reflex in anaesthetized rats.

2 coronary blood flow by 23+/-3 mL/min (Bezold-Jarisch reflex).

3 ined whether or not the activation of Bezold-Jarisch reflex with administration of phenylbiguanide (P

4 rdiopulmonary afferents mediating the Bezold-Jarisch reflex (BJR) desensitize upon repeated exposure

5 rdiopulmonary afferents mediating the Bezold-Jarisch reflex (BJR) rapidly desensitize upon repeated e

6   The pronounced tachyphylaxis to the Bezold-Jarisch reflex (BJR) responses elicited by systemic inje

7 mediated functional antagonism in the Bezold-Jarisch reflex assay.

8 ed to the transient activation of the Bezold-Jarisch reflex by very high initial plasma concentration

9 adycardia caused by activation of the Bezold-Jarisch reflex is considered.

10 odilation following activation of the Bezold-Jarisch reflex is enhanced in conscious dogs after exerc

11 odilation following activation of the Bezold-Jarisch reflex is most likely due to the increased relea

12 odilation following activation of the Bezold-Jarisch reflex were examined in conscious dogs.

13 ory and hypotensive components of the Bezold-Jarisch reflex.

14  pressure response, consisting of the Bezold-Jarisch-associated depressor response, a pressor action,

15  NO-dependent, coronary vasodilation (Bezold-Jarisch reflex) was reduced by 32% but was restored by s

16 nes behaved as 5-HT3 antagonists (von Bezold-Jarisch assay) whereas the arylbiguanides and arylguanid

17                                          The Jarisch-Herxheimer reaction (J-HR), which follows antibi

18                                          The Jarisch-Herxheimer reaction (JHR) is a well-recognized t

19                                          The Jarisch-Herxheimer reaction (JHR) observed after antibio