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1 KSbcl-2, from human herpesvirus 8 (HHV8) or Kaposi sarcoma-associated herpesvirus.
2 uch as HIV, Epstein-Barr virus, malaria, and Kaposi sarcoma-associated herpesvirus.
3 plasm of lymphatic endothelium infected with Kaposi sarcoma-associated herpesvirus.
4 Little evidence suggests a link with Kaposi sarcoma-associated herpesvirus.
5 -like inhibitory protein (FLIP) protein from Kaposi sarcoma-associated herpesvirus activates the NF-k
6 hundred percent of PELs carry the genome of Kaposi sarcoma-associated herpesvirus and a majority are
7 The oncogenic gamma-herpesviruses EBV and Kaposi sarcoma-associated herpesvirus are ubiquitous hum
8 an gammaherpesviruses Epstein-Barr virus and Kaposi Sarcoma-associated herpesvirus both contain a gly
9 avity-based lymphoma cells were positive for Kaposi sarcoma-associated herpesvirus by PCR and were ne
13 alysis of lymphoma DNA showed high levels of Kaposi sarcoma-associated herpesvirus (> 40 to 80 genome
14 other viruses such as Epstein-Barr virus and Kaposi sarcoma-associated herpesvirus have led to the ge
15 their use as models for human infection with Kaposi sarcoma-associated herpesvirus (human herpesvirus
16 This family includes Epstein-Barr virus and Kaposi sarcoma-associated herpesvirus, human pathogens a
21 ly that the open reading frame 45 (ORF45) of Kaposi sarcoma-associated herpesvirus interacts with p90
22 sion of the viral interleukin-6 homolog from Kaposi sarcoma-associated herpesvirus is also sufficient
24 interleukin-6 (vIL-6), a virokine encoded by Kaposi sarcoma- associated herpesvirus, is an issue of c
28 n 6 (IL-6) is an important growth factor for Kaposi sarcoma- associated herpesvirus (KSHV)-associated
30 invariably associated with infection by the Kaposi sarcoma-associated herpesvirus (KSHV or HHV8).
31 que rhadinovirus (RRV) is closely related to Kaposi sarcoma-associated herpesvirus (KSHV) and is asso
32 Kaposi sarcoma is caused by infection of Kaposi sarcoma-associated herpesvirus (KSHV) and is char
33 A-binding domains (DBDs), LANA homologs from Kaposi sarcoma-associated herpesvirus (KSHV) and MHV68 e
34 ion that is transferable to MHV68.IMPORTANCE Kaposi sarcoma-associated herpesvirus (KSHV) and murine
35 s subfamily of gammaherpesviruses, including Kaposi sarcoma-associated herpesvirus (KSHV) and murine
36 In Sub-Saharan Africa, where HIV and the Kaposi sarcoma-associated herpesvirus (KSHV) are endemic
39 T-cell immunity is important for controlling Kaposi sarcoma-associated herpesvirus (KSHV) diseases su
43 n tumor viruses Epstein-Barr virus (EBV) and Kaposi sarcoma-associated herpesvirus (KSHV) establish p
44 nces were all approximately equidistant from Kaposi sarcoma-associated herpesvirus (KSHV) gB sequence
49 , a multifunctional protein expressed by the Kaposi sarcoma-associated herpesvirus (KSHV) in latently
53 me studies have inferred that an epidemic of Kaposi sarcoma-associated herpesvirus (KSHV) infection i
56 e of the JCI, Ganem and colleagues show that Kaposi sarcoma-associated herpesvirus (KSHV) inhibits CD
61 ically, the thymidine kinase (TK) encoded by Kaposi sarcoma-associated herpesvirus (KSHV) is an extre
75 ng of genes believed to be essential for the Kaposi sarcoma-associated herpesvirus (KSHV) latent life
76 newly recognized gamma herpesvirus known as Kaposi sarcoma-associated herpesvirus (KSHV) or human he
80 show that the lytic replication cycle of the Kaposi sarcoma-associated herpesvirus (KSHV) promotes do
82 In a previous study, it was shown that the Kaposi sarcoma-associated herpesvirus (KSHV) was specifi
83 at treatment of cells latently infected with Kaposi sarcoma-associated herpesvirus (KSHV) with glycyr
86 associated with epigenetic modifications of Kaposi sarcoma-associated herpesvirus (KSHV), an oncogen
91 virus (HIV) type 1-infected subjects without Kaposi sarcoma-associated herpesvirus (KSHV)-associated
104 ) to infection by a novel human herpesvirus (Kaposi sarcoma-associated herpesvirus [KSHV]) is one of
105 tiated blood vascular endothelial cells with Kaposi sarcoma-associated herpesvirus leads to their lym
106 o the sustained activation of ERK and RSK in Kaposi sarcoma-associated herpesvirus lytic replication.
108 lity complex class I in cells expressing the Kaposi sarcoma-associated herpesvirus protein K3, is una
109 n gamma-2 herpesvirus closely related to the Kaposi sarcoma-associated herpesvirus, replicates lytica
111 and endothelial cells latently infected with Kaposi sarcoma-associated herpesvirus, suggesting that a
112 V4), cytomegalovirus (HHV5), HHV6, HHV7, and Kaposi sarcoma-associated herpesvirus (termed KSV or HHV
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