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1 d herpesvirus (KSHV), the etiologic agent of Kaposi's sarcoma.
2 homas, multicentric Castleman's disease, and Kaposi's sarcoma.
3 ovarian, breast, and lung cancer, as well as Kaposi's sarcoma.
4 ly induces angiogenic tumor resembling human Kaposi's sarcoma.
5 elated malignancies, including lymphomas and Kaposi's sarcoma.
6 of vGPCR-induced lesions that resemble human Kaposi's sarcoma.
7 Castleman's disease, as well as its namesake Kaposi's sarcoma.
8 9, 95% CI 1.67-1.72), AIDS-defining cancers (Kaposi's sarcoma [498.11, 477.82-519.03], non-Hodgkin ly
9 mmaherpesvirus etiologically associated with Kaposi's sarcoma, a vascular tumor of endothelial cells.
10 SHV) is the cause of aggressive AIDS-related Kaposi's sarcoma (AIDS-KS) characterized by abnormal ang
11 erpesvirus 8 (HHV-8), the causative agent of Kaposi's sarcoma and B cell neoplasms in AIDS patients,
13 s tightly associated with the development of Kaposi's sarcoma and multicentric Castleman's disease, a
14 tients at the Haitian Group for the Study of Kaposi's Sarcoma and Opportunistic infections (GHESKIO)
15 several AIDS-related malignancies, including Kaposi's sarcoma and primary effusion lymphoma (PEL).
16 herpesvirus (KSHV), the etiological agent of Kaposi's sarcoma and primary effusion lymphoma, has deve
18 a beneficial component for the treatment of Kaposi's sarcoma and the role of antivirals for HHV-8 in
19 herpesvirus (KSHV) is the causative agent of Kaposi's sarcoma, and a constitutively active viral G pr
21 Human herpes virus 8 (HHV-8), also known as Kaposi's sarcoma associated herpesvirus (KSHV), is an on
22 G, and panel), Epstein-Barr Virus (EBV), and Kaposi's Sarcoma-associated Herpes Virus (KSHV) in a fin
27 r maturation of RRV, the closest relative of Kaposi's sarcoma-associated herpesvirus (KSHV) (a human
28 protein (SCP) of human tumor herpesviruses--Kaposi's sarcoma-associated herpesvirus (KSHV) and Epste
30 ame 50 (ORF50) DNA template in the genome of Kaposi's sarcoma-associated herpesvirus (KSHV) and has b
34 lytic cycles of Epstein-Barr virus (EBV) and Kaposi's sarcoma-associated herpesvirus (KSHV) are induc
37 maherpesviruses Epstein-Barr virus (EBV) and Kaposi's sarcoma-associated herpesvirus (KSHV) cause lif
38 ive stages of the KSHV life cycle.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV) causes AI
41 ent SCFAs stimulate lytic gene expression of Kaposi's sarcoma-associated herpesvirus (KSHV) dose depe
49 encoded by open reading frame 36 (ORF36) of Kaposi's sarcoma-associated herpesvirus (KSHV) enhances
58 inity tagged and purified all 89 proteins of Kaposi's sarcoma-associated herpesvirus (KSHV) from huma
60 ous transcription across the entirety of the Kaposi's sarcoma-associated herpesvirus (KSHV) genome an
61 e found that most transcripts encoded by the Kaposi's sarcoma-associated herpesvirus (KSHV) genome un
63 y focal assembly of RNA polymerase II around Kaposi's sarcoma-associated herpesvirus (KSHV) genomes i
65 ected cells.IMPORTANCE B cells infected with Kaposi's sarcoma-associated herpesvirus (KSHV) harbor mu
73 we present an innovative approach to culture Kaposi's sarcoma-associated herpesvirus (KSHV) infected
74 d levels of viral lytic gene expression when Kaposi's sarcoma-associated herpesvirus (KSHV) infected
76 was undertaken to determine whether de novo Kaposi's sarcoma-associated herpesvirus (KSHV) infection
77 an effective small-animal model to study the Kaposi's sarcoma-associated herpesvirus (KSHV) infection
78 L) is a lymphogenic disorder associated with Kaposi's sarcoma-associated herpesvirus (KSHV) infection
81 lar RNA degradation enzyme Xrn1 during lytic Kaposi's sarcoma-associated herpesvirus (KSHV) infection
82 mined the expression of the DDR genes during Kaposi's sarcoma-associated herpesvirus (KSHV) infection
131 lytic phases.IMPORTANCE The K15P protein of Kaposi's sarcoma-associated herpesvirus (KSHV) is though
138 HV68) ORF73 (mLANA) has sequence homology to Kaposi's sarcoma-associated herpesvirus (KSHV) latency-a
141 at the constitutive AMPK activity restricted Kaposi's sarcoma-associated herpesvirus (KSHV) lytic rep
144 DSBs) in DNA, although detail concerning how Kaposi's sarcoma-associated herpesvirus (KSHV) modulates
145 egy of selectively isolating VLVs by using a Kaposi's sarcoma-associated herpesvirus (KSHV) mutant th
149 ere, we identify a viral trimeric complex of Kaposi's sarcoma-associated herpesvirus (KSHV) open read
156 report that PABPC1 forms a complex with the Kaposi's sarcoma-associated herpesvirus (KSHV) ORF57 pro
159 ncy-associated nuclear antigen (LANA) of the Kaposi's sarcoma-associated herpesvirus (KSHV) performs
161 nprecedented mechanism of viral replication: Kaposi's sarcoma-associated herpesvirus (KSHV) stably cl
162 recently showed that the interaction between Kaposi's sarcoma-associated herpesvirus (KSHV) tegument
163 ed to murine gammaherpesvirus 68 (MHV68) and Kaposi's sarcoma-associated herpesvirus (KSHV) that esta
164 e kinase A2 (EphA2) is an entry receptor for Kaposi's sarcoma-associated herpesvirus (KSHV) that is e
165 sociated substrate) as a platform to promote Kaposi's sarcoma-associated herpesvirus (KSHV) trafficki
167 o-electron microscopy (cryo-EM) structure of Kaposi's sarcoma-associated herpesvirus (KSHV) virion, a
168 rophoretic response of B cells infected with Kaposi's sarcoma-associated herpesvirus (KSHV) were inve
169 e expression occurs upon superinfection with Kaposi's sarcoma-associated herpesvirus (KSHV), a common
172 iral FLICE-like inhibitor protein (vFLIP) of Kaposi's sarcoma-associated herpesvirus (KSHV), against
174 ncy is an integral part of the life cycle of Kaposi's sarcoma-associated herpesvirus (KSHV), an etiol
175 V1) lineage consists of human herpesvirus 8, Kaposi's sarcoma-associated herpesvirus (KSHV), and clos
176 osely related to human herpesvirus 8 (HHV-8)/Kaposi's Sarcoma-associated herpesvirus (KSHV), and it i
177 the replication of the oncogenic herpesvirus Kaposi's sarcoma-associated herpesvirus (KSHV), by targe
178 , an immediate-early and tegument protein of Kaposi's sarcoma-associated herpesvirus (KSHV), causes s
179 n adaptive immunity, many viruses, including Kaposi's sarcoma-associated herpesvirus (KSHV), have evo
180 othelial cells during de novo infection with Kaposi's sarcoma-associated herpesvirus (KSHV), in cells
181 aherpesviruses, Epstein-Barr virus (EBV) and Kaposi's sarcoma-associated herpesvirus (KSHV), is assoc
183 egument proteins of herpesviruses, including Kaposi's sarcoma-associated herpesvirus (KSHV), play key
184 50 promoters of Epstein-Barr virus (EBV) and Kaposi's sarcoma-associated herpesvirus (KSHV), respecti
186 rld primates and are distinct but related to Kaposi's sarcoma-associated herpesvirus (KSHV), the etio
189 on lymphoma (PEL) cells infected with latent Kaposi's sarcoma-associated herpesvirus (KSHV), the prom
190 (RFHVMn), the pig-tailed macaque homolog of Kaposi's sarcoma-associated herpesvirus (KSHV), was dete
191 viral OX2 (vOX2) immunoregulator encoded by Kaposi's sarcoma-associated herpesvirus (KSHV), we have
192 maherpesviruses Epstein-Barr virus (EBV) and Kaposi's sarcoma-associated herpesvirus (KSHV), which ar
194 factor in the development and progression of Kaposi's sarcoma-associated herpesvirus (KSHV)-induced p
196 n by HIV-1 promotes the aggressive growth of Kaposi's sarcoma-associated herpesvirus (KSHV)-related m
206 16) recognizes nuclear episomal herpesvirus (Kaposi's sarcoma-associated herpesvirus [KSHV], Epstein-
210 ich tumor cells are consistently infected by Kaposi's sarcoma-associated herpesvirus and usually grow
211 view the evidence for Epstein-Barr virus and Kaposi's sarcoma-associated herpesvirus as causal lympho
212 c mechanisms by which Epstein-Barr virus and Kaposi's sarcoma-associated herpesvirus cause B-cell lym
213 tors (e.g., herpes simplex virus nectin1 and Kaposi's sarcoma-associated herpesvirus EphA2), or assoc
215 Microbe, Chang and Ganem (2013) report that Kaposi's sarcoma-associated herpesvirus infection of lym
218 s, the cofactor activity (CFA), by using the Kaposi's sarcoma-associated herpesvirus RCA homolog Kapo
219 t HIV, together with a transient increase in Kaposi's sarcoma-associated herpesvirus viral load at we
221 ciated polyomavirus, Epstein-Barr virus, and Kaposi's sarcoma-associated herpesvirus, because GEMs ha
222 ovirus (HCMV), Epstein-Barr virus (EBV), and Kaposi's sarcoma-associated herpesvirus, establish laten
223 ose relative of the human oncogenic pathogen Kaposi's sarcoma-associated herpesvirus, ORF52 is a high
224 genomes, those of human cytomegalovirus and Kaposi's sarcoma-associated herpesvirus, revealing trans
225 be infected with the human pathogens, EBV or Kaposi's sarcoma-associated herpesvirus, we have used a
226 d from a newly established and characterized Kaposi's sarcoma-associated herpesvirus- and Epstein-Bar
229 EL clinical samples, and three extracavitary Kaposi's sarcoma-associated herpesvirus-positive solid l
233 a 50 years ago, only one other virus, namely Kaposi's sarcoma-associated herpesvirus/human herpesviru
234 omolog, the oncogenic human gammaherpesvirus Kaposi's sarcoma-associated herpesvirus/human herpesviru
235 s an oncogenic gamma-herpesvirus that causes Kaposi's sarcoma, Castleman's disease and primary effusi
236 sal agent of several malignancies, including Kaposi's sarcoma, commonly found in immunocompromised pa
239 In lymphomas the Epstein-Barr virus (EBV), Kaposi's sarcoma herpesvirus (KSHV) and human T-lymphotr
240 mutant with prototype foamy virus (PFV) and Kaposi's sarcoma herpesvirus (KSHV) tethering sequences
241 fied infections by Epstein-Barr virus (EBV), Kaposi's sarcoma herpesvirus (KSHV), and human T-lymphot
244 ttings and the significant burden exacted by Kaposi's sarcoma in these areas, we reviewed data regard
247 ow that HHV-8, a DNA tumor virus that causes Kaposi's sarcoma, infects three types of dendritic cells
251 Therefore, an understanding of the burden of Kaposi's sarcoma (KS) and non-Hodgkin lymphoma (NHL) rel
252 al and B cells are etiologically linked with Kaposi's sarcoma (KS) and primary effusion B-cell lympho
253 ked to several human malignancies, including Kaposi's sarcoma (KS) and primary effusion lymphoma (PEL
254 irus (KSHV) is etiologically associated with Kaposi's sarcoma (KS) and primary effusion lymphoma (PEL
255 virus (KSHV)-related malignancies, including Kaposi's sarcoma (KS) and primary effusion lymphoma (PEL
257 several AIDS-related malignancies, including Kaposi's sarcoma (KS) and primary effusion lymphoma (PEL
258 erpesvirus (KSHV) is the causative agent for Kaposi's sarcoma (KS) and two other lymphoproliferative
265 m multicentric Castleman's disease (MCD) and Kaposi's sarcoma (KS) lesions, suggesting that pERP1 may
267 herpesvirus (KSHV) is the etiologic agent of Kaposi's sarcoma (KS), a vascular tumor frequently found
268 of care for patients with HIV diagnosed with Kaposi's sarcoma (KS), but the current role of systemic
269 d herpesvirus (KSHV), the etiologic agent of Kaposi's sarcoma (KS), is present in the predominant tum
270 a human gammaherpesvirus casually linked to Kaposi's sarcoma (KS), multicentric Castleman's disease
271 irus (KSHV) is etiologically associated with Kaposi's sarcoma (KS), primary effusion lymphoma (PEL),
272 rpesvirus 8) is linked to the development of Kaposi's sarcoma (KS), primary effusion lymphoma (PEL),
273 neoplastic or lymphoproliferative diseases: Kaposi's sarcoma (KS), primary effusion lymphoma (PEL),
274 herpesvirus (KSHV) is the etiologic agent of Kaposi's sarcoma (KS), the most common tumor of AIDS pat
275 ssociated herpesvirus (KSHV) is the cause of Kaposi's sarcoma (KS), which is the most common AIDS-ass
289 e infected with human herpesvirus 8 (HHV-8) (Kaposi's sarcoma [KS]-associated herpesvirus) and have a
290 ual men were over-represented among men with Kaposi's sarcoma (OR, 48.2; 95% CI, 22.0 to 105.6), anal
291 ting it as a contributor to virus-associated Kaposi's sarcoma, primary effusion lymphoma (PEL), and m
292 virus 8 (HHV-8) infection is associated with Kaposi's sarcoma, primary effusion lymphoma (PEL), and m
293 nd angiogenic activities to HHV-8-associated Kaposi's sarcoma, primary effusion lymphoma (PEL), and m
294 (KSHV) has been linked to the development of Kaposi's sarcoma, primary effusion lymphoma, and multice
295 ant lymphoproliferative disorders (PTLD) and Kaposi's sarcoma remain important complications of immun
296 lated diagnoses, oesophageal candidiasis and Kaposi's sarcoma, rose from almost zero to very high lev
297 rs.IMPORTANCE KSHV is the etiologic agent of Kaposi's sarcoma, the most common tumor of AIDS patients
299 decreased significantly across 1996-2012 for Kaposi's sarcoma, two subtypes of non-Hodgkin lymphoma,
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