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1 Intact porcine lenses were incubated in Krebs solution.
2 either a high-K+ (7 mM) or Cd2+ (100 microM) Krebs solution.
3 tial shifted to -90 mV in a low-Na+, high-K+ Krebs solution.
4 guinea pigs and kept alive in oxygen bubbled Krebs' solution.
5 - 2.3% with an IC50 of 19.9 microM in normal Krebs solution (2.5 mM Ca2+, 1.2 mM Mg2+) without effect
6 ally before and after incubation with either Krebs solution alone or with the NO-inhibitor, NG-monome
8 trode in an airtight stirred bath containing Krebs solution buffered with HEPES at 37 degrees C (pH 7
9 - 0.8 ml min-1 in hearts perfused with plain Krebs solution, by 3.8 +/- 0.8 ml min-1 in hearts to whi
10 s (human, rabbit, and rat) were incubated in Krebs solution containing [3H]-norepinephrine ([3H]NE) f
11 hed by low Ca2+, high Mg2+ Krebs solution or Krebs solution containing Co2+ (2 mM) and Cd2+ (400 micr
16 on, were reduced by 70% in a low-Na+ (26 mM) Krebs solution, indicating the involvement of Na+ ions.
17 its were suspended in organ baths containing Krebs solution; isometric tension was then measured.
18 This current was inhibited in chloride-free Krebs solution or by inhibiting basolateral chloride upt
19 completely abolished by low Ca2+, high Mg2+ Krebs solution or Krebs solution containing Co2+ (2 mM)
21 re randomly assigned to perfusion with plain Krebs solution, or with Krebs solution to which L-NAME a
23 sted even in perfusions of zero calcium-EGTA Krebs solution suggesting that the calcium oscillation i
24 perfusion with plain Krebs solution, or with Krebs solution to which L-NAME and/or indomethacin had b
25 ich persisted in TTX (0.5 microM)-containing Krebs solution, were reduced by 70% in a low-Na+ (26 mM)
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