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1      Intact porcine lenses were incubated in Krebs solution.
2 either a high-K+ (7 mM) or Cd2+ (100 microM) Krebs solution.
3 tial shifted to -90 mV in a low-Na+, high-K+ Krebs solution.
4 guinea pigs and kept alive in oxygen bubbled Krebs' solution.
5 - 2.3% with an IC50 of 19.9 microM in normal Krebs solution (2.5 mM Ca2+, 1.2 mM Mg2+) without effect
6 ally before and after incubation with either Krebs solution alone or with the NO-inhibitor, NG-monome
7                      In hearts perfused with Krebs solution alone, nitric oxide (NO) release into the
8 trode in an airtight stirred bath containing Krebs solution buffered with HEPES at 37 degrees C (pH 7
9 - 0.8 ml min-1 in hearts perfused with plain Krebs solution, by 3.8 +/- 0.8 ml min-1 in hearts to whi
10 s (human, rabbit, and rat) were incubated in Krebs solution containing [3H]-norepinephrine ([3H]NE) f
11 hed by low Ca2+, high Mg2+ Krebs solution or Krebs solution containing Co2+ (2 mM) and Cd2+ (400 micr
12 s with intact endothelium were perfused with Krebs solution containing phenylephrine.
13                    Removal of CaCl2 from the Krebs solution, disruption of the endothelium, and admin
14  were harvested and maintained in oxygenated Krebs solution in an organ bath at 37 degrees C.
15 were perfused (40 mL/min, 37 degrees C) with Krebs' solution in a recirculating system.
16 on, were reduced by 70% in a low-Na+ (26 mM) Krebs solution, indicating the involvement of Na+ ions.
17 its were suspended in organ baths containing Krebs solution; isometric tension was then measured.
18  This current was inhibited in chloride-free Krebs solution or by inhibiting basolateral chloride upt
19  completely abolished by low Ca2+, high Mg2+ Krebs solution or Krebs solution containing Co2+ (2 mM)
20                           Low Ca2+/high Mg2+ Krebs solution or TTX did not change the resting membran
21 re randomly assigned to perfusion with plain Krebs solution, or with Krebs solution to which L-NAME a
22                           Using an isolated, Krebs solution-perfused rat heart we measured the change
23 sted even in perfusions of zero calcium-EGTA Krebs solution suggesting that the calcium oscillation i
24 perfusion with plain Krebs solution, or with Krebs solution to which L-NAME and/or indomethacin had b
25 ich persisted in TTX (0.5 microM)-containing Krebs solution, were reduced by 70% in a low-Na+ (26 mM)

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