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1 0.01 vs. control mesenteries superfused with Krebs-Henseleit buffer).
2 eys were isolated and perfused with modified Krebs-Henseleit buffer.
3 = 77) later and placed in a tissue bath with Krebs-Henseleit buffer.
4 e hearts were reperfused for 30 minutes with Krebs-Henseleit buffer.
5 ant myocardial washout of (99m)TcN-NOET with Krebs-Henseleit buffer.
6 ir wild-type littermates, were perfused with Krebs-Henseleit buffer and subjected to 20 minutes of is
7 fused alternately with a modified oxygenated Krebs-Henseleit buffer and with buffer containing varied
8                    Hearts were perfused with Krebs-Henseleit buffer at 85 mm Hg.
9  Langendorff method under a constant flow of Krebs-Henseleit buffer containing (18)F-FDG with a rate
10 ts was perfused at physiologic workload with Krebs-Henseleit buffer containing 10 mmol/L glucose; a s
11 We perfused isolated working rat hearts with Krebs-Henseleit buffer containing [2-3H]glucose (5 mmol/
12      Hearts were labeled for 40 minutes with Krebs-Henseleit buffer containing [35S]methionine, and t
13 olated working rat hearts were perfused with Krebs-Henseleit buffer containing only glucose 5 mmol/L
14 oved from the rat and perfused with modified Krebs-Henseleit buffers containing 7.5 or 2.5 g/dL bovin
15 t hearts were perfused at constant flow with Krebs-Henseleit buffer enriched with bovine red blood ce
16 perfused mouse liver model with deoxygenated Krebs-Henseleit buffer followed by oxygenated buffer.
17 used for 10 min in the Langendorff-mode with Krebs-Henseleit buffer in the absence or presence of bri
18 ce, they were presented with OS suspended in Krebs-Henseleit buffer in the presence or absence of car
19              Three groups were studied using Krebs-Henseleit buffer (KH): controls (12 mL/min, n = 6)
20 fused initially by the Langendorff mode with Krebs-Henseleit buffer (KHB) for 15 minutes in the absen
21 abbit hearts were retrogradely perfused with Krebs-Henseleit buffer (KHB).
22 m normal rats were incubated for 1 hr in the Krebs-Henseleit buffer media containing normal rat sera,
23 m normal rats were incubated for 1 hr in the Krebs-Henseleit buffer media containing zymosan-activate
24             Rabbit hearts were perfused with Krebs-Henseleit buffer on a Langendorff apparatus.
25 er at constant flow; perfusates consisted of Krebs-Henseleit buffer or buffer plus washed RBCs at a H
26 uscles were isolated and incubated in normal Krebs-Henseleit buffer (pH 7.4).
27 larographic chamber containing air-saturated Krebs-Henseleit buffer plus 20 mM glucose, PO2 being mon
28 d storage (SCS), livers were reperfused with Krebs-Henseleit buffer solution at 37 degree C for 30 mi
29    After storage, the cells were rewarmed in Krebs-Henseleit buffer with air at 37 degrees C for 1 hr
30 nutes of low-flow (0.5 mL/min) ischemia with Krebs-Henseleit buffer with or without 11 mmol/L glucose

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