ライフサイエンスコーパス: Kruppel-like factor

1 The Kruppel-like factor 1 (KLF1) and KLF2 positively regulat

2 l-like factor (EKLF), which is also known as Kruppel-like factor 1 (KLF1), play a coordinating role i

3 ns in the key erythroid transcription factor Kruppel-like factor 1 in patients who presented with sev

4 Kruppel-like factor 1(KLF1) is a hematopoietic-specific

5 aused by mutations in a trans-acting factor (Kruppel-like factor 1) and reveal an important pathway r

6 rs GATA1 (GATA1 binding protein 1) and KLF1 (Kruppel-like factor 1) cause benign and disease phenotyp

7 They find that mutations in Kruppel-like factor-1 (KLF1) are significantly more prev

8 ssense mutation in the second zinc finger of Kruppel-like factor-1 (KLF1) leads to degenerate DNA-bin

9 t findings have supported a primary role for Kruppel-like factor 10 (KLF10) as an important transcrip

10 Herein, we provide evidence that Kruppel-like factor 10 (KLF10/TIEG1) constitutes an impo

11 , NAB2 (Ngfi-A-binding protein-2) and KLF10 (Kruppel-like factor-10) that, when individually silenced

12 levels of a MAO A-transcriptional activator, Kruppel-like factor 11 (KLF11 , also recognized as trans

13 The function of Kruppel-like factor 11 (KLF11) in the regulation of meta

14 volutionarily conserved metabolic regulator, Kruppel-like factor 11 (KLF11), as a novel browning tran

15 s-induced MAO A and the transcription factor Kruppel-like factor 11 (KLF11, also called TIEG2, a memb

16 viously showed that the transcription factor Kruppel-like factor 13 (KLF13) controls the late (3-5 da

17 Here we show that the transcription factor Kruppel-like factor 13 (KLF13) has a critical role in th

18 ES) and its major transcriptional regulator, Kruppel-like factor 13 (KLF13), are expressed late (3-5

19 gene locus encoding the transcription factor Kruppel-like factor 14 (KLF14) are strongly associated w

20 Here we report that disruption of the Kruppel-like factor 14 (KLF14) gene in mice causes centr

21 A Kruppel-like factor 15 (dKlf15) loss-of-function strateg

22 Although previous studies have defined Kruppel-like factor 15 (KLF15) as a transcriptional repr

23 In this study, we identified Kruppel-like factor 15 (KLF15) as an essential regulator

24 Although the transcription factor Kruppel-like factor 15 (KLF15) is an important regulator

25 y induced the ergogenic transcription factor Kruppel-like factor 15 (Klf15) while decreasing Fbxo32.

26 with known CREB target genes, we identified Kruppel-like factor 15 (KLF15), a kidney-enriched nuclea

27 We previously determined that Kruppel-like factor 15 (KLF15), a kidney-enriched zinc f

28 Here, we demonstrate that Kruppel-like factor 15 (KLF15), a liver-enriched transcr

29 ined that dual transcriptional regulation of Kruppel-like factor 15 (Klf15), by both the transforming

30 we established that PR-mediated induction of Kruppel-like factor 15 (KLF15), which can bind to GC-ric

31 the control of a clock-dependent oscillator, kruppel-like factor 15 (Klf15).

32 dexamethasone-induced transcription factors, Kruppel-like factor 15, Slug, and SPDEF, stimulated the

33 TWEAK represses the levels of Kruppel-like factor 15; myocyte enhancer factor 2, and p

34 Here, we report that Kruppel-like factor 17 (Klf17), like Grhl3, acts downstr

35 y up-regulation of its transcription factor, Kruppel-like factor 2 (KLF-2).

36 othelial nitric oxide synthase (P< .005) and Kruppel-like factor 2 (KLF2) (P< .005).

37 cific deficiency of the transcription factor Kruppel-like factor 2 (KLF2) accelerates atherosclerosis

38 in expression of the atheroprotective factor Kruppel-like factor 2 (KLF2) and cytokines were also stu

39 Kruppel-like factor 2 (KLF2) and endothelial nitric oxid

40 The expression of the transcription factor Kruppel-like factor 2 (KLF2) and its vasoprotective targ

41 Recent studies have identified Kruppel-like factor 2 (KLF2) as a potent inhibitor of an

42 icoagulant protein C system, is regulated by Kruppel-like factor 2 (KLF2) at the transcriptional leve

43 ndent upon the vascular transcription factor Kruppel-like factor 2 (KLF2) binding near the transcript

44 We now report that transcription factor Kruppel-like factor 2 (KLF2) controls naive Treg migrati

45 The transcription factor Kruppel-like factor 2 (KLF2) controls the emigration of

46 es are regulated by the transcription factor Kruppel-like factor 2 (KLF2) in conventional alphabeta T

47 eases expression of the transcription factor Kruppel-like factor 2 (KLF2) in multiple cell types.

48 flammatory role for the transcription factor Kruppel-like factor 2 (KLF2) in regulating activation of

49 The transcription factor Kruppel-like factor 2 (KLF2) is a critical anti-inflamma

50 l expansion and survival, we now report that Kruppel-like factor 2 (KLF2) is a key transcription fact

51 though mechanosensitive transcription factor Kruppel-like factor 2 (KLF2) is a major regulator of end

52 Kruppel-like factor 2 (KLF2) is a member of zinc-finger

53 Kruppel-like factor 2 (KLF2) is a potent regulator of my

54 Kruppel-like Factor 2 (KLF2) is a shear stress-sensitive

55 Kruppel-like factor 2 (KLF2) is a transcription factor t

56 The transcription factor Kruppel-like factor 2 (KLF2) is critical for normal traf

57 eroprotective flow, the transcription factor Kruppel-like factor 2 (KLF2) is crucial for maintaining

58 We now report that transcription factor Kruppel-like factor 2 (KLF2) is necessary for the genera

59 In this study, we show that Kruppel-like factor 2 (Klf2) is phosphorylated by Erk2 a

60 The transcription factor Kruppel-like factor 2 (KLF2) is required for the quiesce

61 y isolated HUAECs expressed higher levels of Kruppel-like factor 2 (KLF2) than HUVECs, consistent wit

62 surface S1P-R1 expression, as well S1PR1 and Kruppel-like factor 2 (KLF2) transcripts, were significa

63 The transcription factor Kruppel-like factor 2 (KLF2) was proposed to regulate ge

64 gulates other stress-sensitive genes such as Kruppel-like factor 2 (Klf2), endothelial nitric oxide s

65 l cell homeostasis as shown by activation of Kruppel-like factor 2 (KLF2), increased endothelial nitr

66 ons, which was associated with modulation of Kruppel-like factor 2 (KLF2), Kruppel-like factor 4 (KLF

67 shear stress-sensitive transcription factor, Kruppel-like Factor 2 (KLF2), mediates increased CBC sen

68 to facilitate a physical interaction between Kruppel-like factor 2 (KLF2), the major regulator of she

69 h the regulation of the transcription factor Kruppel-like factor 2 (Klf2).

70 n of the vasoprotective transcription factor Kruppel-like Factor 2 (KLF2).

71 he adipogenesis inhibitors Pref-1, Sox9, and Kruppel-like factor 2 (KLF2).

72 activation and the consequent regulation of Kruppel-like factor 2 and endothelial nitric oxide synth

73 y effect of H(2)O(2) in shear stress-induced Kruppel-like factor 2 and endothelial nitric oxide synth

74 This activation is instrumental in inducing Kruppel-like factor 2 and several immediate early genes

75 elial anti-inflammatory transcription factor Kruppel-like factor 2 as well as endothelial nitric oxid

76 S1P-R1 and Kruppel-like factor 2 expression were monitored after S1

77 Kruppel-like factor 2 expression, a flow sensitive trans

78 Importantly, restoring Kruppel-like factor 2 in IRF2BP2-deficient macrophages a

79 s present within a repressing complex on the Kruppel-like factor 2 promoter.

80 f the anti-inflammatory transcription factor Kruppel-like factor 2 was markedly reduced.

81 065], P=1.73E-03) and had lower IRF2BP2 (and Kruppel-like factor 2) protein levels in peripheral bloo

82 Furthermore, we found that the expression of Kruppel-like factor 2, a key anti-inflammatory transcrip

83 f the sphingosine 1-phosphate 1 receptor and Kruppel-like factor 2, both of which regulate thymic and

84 as a transcriptional corepressor to regulate Kruppel-like factor 2-dependent gene expression.

85 ancer factor 2 (MEF2) transcriptional target Kruppel-like factor 2.

86 al activity, as well as promoter activity of Kruppel-like factor 2.

87 is required for MEF2-dependent activation of Kruppel-like factor 2.

88 PRCP(gt/gt) aortas express reduced levels of Kruppel-like factors 2 and 4, thrombomodulin, and eNOS m

89 hanistic relationship between p66shc and the kruppel like factor-2 (KLF2) transcription factor and sh

90 n of the zinc finger transcriptional factor, Kruppel-like factor-2 (KLF2) in tumor endothelial cells.

91 regulating NF-E2-related factor-2 (Nrf2) and Kruppel-like factor-2 (KLF2), two transcription factors

92 ed antiinflammatory tissue expression of von Kruppel-like Factor-2.

93 ow that the zinc finger transcription factor Kruppel-like factor 3 (KLF3) directly represses galectin

94 Kruppel-like factor 3 (KLF3) is a transcriptional regula

95 Constitutive expression of Kruppel-like factor 3 (KLF3, BKLF) increases marginal zo

96 tracellular domain) transactivated the mouse Kruppel-like factor 4 (Klf) promoter and that Klf4 direc

97 mall interfering RNA-mediated suppression of Kruppel-like factor 4 (Klf4) and Sp1, and was abolished

98 Here we report our studies that identify Kruppel-like factor 4 (KLF4) as a critical regulator of

99 ed that the zinc finger transcription factor Kruppel-like factor 4 (KLF4) can promote RAS-ERK signali

100 thickness and PU mouse models, we found that Kruppel-like factor 4 (KLF4) deficiency resulted in decr

101 Here, we explore the mechanisms that loss of Kruppel-like factor 4 (KLF4) exacerbates oncogenic TGF-b

102 Decreased Kruppel-like factor 4 (KLF4) expression has been observe

103 omplex long-range enhancer cluster governing Kruppel-like factor 4 (Klf4) expression in naive pluripo

104 we have shown that the transcription factor Kruppel-like factor 4 (KLF4) governs mitochondrial bioge

105 The zinc finger transcription factor Kruppel-like factor 4 (KLF4) has been implicated in both

106 The transcription factor Kruppel-like factor 4 (KLF4) has the ability, along with

107 s death-associated protein kinase (DAPK) and Kruppel-like factor 4 (KLF4) in CRC cells, resulting in

108 to overexpress the transcriptional regulator Kruppel-like factor 4 (Klf4) in esophageal epithelia of

109 ole for the zinc finger transcription factor Kruppel-like factor 4 (KLF4) in the modulation of the in

110 Here, we show that Kruppel-like factor 4 (KLF4) is a central regulator of s

111 We found that the Kruppel-like factor 4 (Klf4) is a potent activator of Ag

112 Kruppel-like factor 4 (Klf4) is a putative gastric tumor

113 Kruppel-like factor 4 (KLF4) is a transcription factor a

114 Kruppel-like factor 4 (Klf4) is a transcription factor i

115 Kruppel-like factor 4 (KLF4) is a transcription factor i

116 The Kruppel-like factor 4 (KLF4) is a transcriptional regula

117 Kruppel-like factor 4 (KLF4) is a zinc finger protein hi

118 Kruppel-like factor 4 (KLF4) is a zinc-finger transcript

119 Endothelial Kruppel-Like Factor 4 (KLF4) is an important anti-inflam

120 The transcription factor Kruppel-like factor 4 (KLF4) is an important regulator o

121 Transcription factor Kruppel-like factor 4 (Klf4) is essential for somatic ce

122 Kruppel-like factor 4 (KLF4) is expressed in neural stem

123 Kruppel-like factor 4 (KLF4) is highly expressed in more

124 Kruppel-like factor 4 (KLF4) is involved in self-renewal

125 Here, we have provided evidence that Kruppel-like factor 4 (Klf4) is required in IRF4-express

126 Effect of Kruppel-like factor 4 (Klf4) on Slurp1 promoter was eval

127 Kruppel-like factor 4 (Klf4) plays a crucial role in the

128 SMC-specific conditional knockout of Kruppel-like factor 4 (Klf4) resulted in reduced numbers

129 We previously showed that Kruppel-like factor 4 (Klf4) suppressed SMC differentiat

130 ophage lineage identity transcription factor Kruppel-like factor 4 (KLF4) to be inhibited during LC d

131 Previous work has shown that the Kruppel-like factor 4 (KLF4) transcription factor repres

132 d that gemfibrozil induced the activation of Kruppel-like factor 4 (KLF4) via the PI 3-kinase-AKT pat

133 ng cascade involving sustained expression of Kruppel-like factor 4 (KLF4), a regulator of stem cell m

134 We demonstrate that miR-29 directly targets Kruppel-like factor 4 (KLF4), a transcription factor req

135 Kruppel-like factor 4 (KLF4), a transcription factor tha

136 Kruppel-like factor 4 (KLF4), a transcription factor, pl

137 Kruppel-like factor 4 (KLF4), a zinc finger-containing t

138 Kruppel-like factor 4 (KLF4), a zinc finger-containing t

139 date the underlying mechanism, we focused on Kruppel-like factor 4 (KLF4), and decoupled its mCpG- an

140 modulation of Kruppel-like factor 2 (KLF2), Kruppel-like factor 4 (KLF4), and suppressor of cytokine

141 many human transcription factors, including Kruppel-like factor 4 (KLF4), as sequence-specific DNA m

142 The zinc finger transcription factor, Kruppel-like factor 4 (KLF4), is expressed in the post-m

143 Transcription factor Kruppel-like factor 4 (Klf4), one of the factors directi

144 The zinc finger transcription factor, Kruppel-like factor 4 (KLF4), regulates numerous biologi

145 polarization involves induction of STAT6 and Kruppel-like factor 4 (KLF4), which induce each other an

146 s, a pathological mechanism also governed by Kruppel-like factor 4 (KLF4).

147 ed that kallistatin-binding protein is human Kruppel-like factor 4 (KLF4).

148 Kruppel-like factor 4 (KLF4, GKLF) is a zinc-finger tran

149 Kruppel-like factor 4 (Klf; previously known a gut-enric

150 man gastric tumors had reduced expression of Kruppel-like factor 4 and increased expression of FoxM1

151 arget of rapamycin pathway inhibits ELF4 and Kruppel-like factor 4 expression downstream of ERK and P

152 Kruppel-like factor 4 expression has been documented in

153 xpression of the E74-like factor (ELF) 4 and Kruppel-like factor 4 genes to release naive CD8(+) T ce

154 cribe the expression in vitro and in vivo of Kruppel-like factor 4 in human and mouse endothelial cel

155 Expression of Kruppel-like factor 4 suppressed transcription of FoxM1.

156 ssion of transcription factors such as KLF4 (Kruppel-like factor 4) and EGR1 (early-growth-response 1

157 rk of transcription factors, including Klf4 (Kruppel-like factor 4), myocardin and Elk-1 (ELK1, membe

158 ophage polarization, including STAT3, STAT6, Kruppel-like factor 4, and peroxisome proliferator-activ

159 eased expression of the transcription factor Kruppel-like factor 4.

160 ntrol of expression of transcription factors Kruppel-like factors 4 or 5 (Klf4 or Klf5) which in turn

161 retinal ganglion cells (RGCs), we identified Kruppel-like factor-4 (KLF4) as a transcriptional repres

162 Kruppel-like factor-4 (KLF4) is known to inhibit activat

163 f the gene encoding the transcription factor Kruppel-like factor-4 (Klf4), a negative regulator of th

164 or-1alpha (HIF-1alpha, encoded by HIF1A) and Kruppel-like factor-4 (KLF4)-dependent.

165 eated mice also showed reduced expression of Kruppel-like factor-4.

166 Kruppel-like factor 5 (Klf5) encodes a zinc-finger trans

167 ociated with increased ERK1/2 activation and Kruppel-like factor 5 (KLF5) expression in IEC.

168 The transcription factor Kruppel-like Factor 5 (KLF5) has been shown to mediate c

169 tigated the role of the transcription factor Kruppel-like factor 5 (KLF5) in ADM and KRAS-mediated fo

170 Kruppel-like factor 5 (KLF5) is a pro-proliferative tran

171 Kruppel-like factor 5 (Klf5) is a transcription factor e

172 Kruppel-like factor 5 (KLF5) is a transcription factor t

173 Kruppel-like factor 5 (KLF5) is a transcription factor t

174 Kruppel-like factor 5 (KLF5) is a zinc finger transcript

175 The Kruppel-like factor 5 (KLF5) is a zinc-finger transcript

176 Kruppel-like factor 5 (KLF5) is an effector of LPA-induc

177 Kruppel-like factor 5 (KLF5) is an important mediator of

178 The zinc finger transcription factor Kruppel-like factor 5 (KLF5) is regulated posttranslatio

179 Kruppel-like factor 5 (KLF5) is transcription factor tha

180 ow that the zinc finger transcription factor Kruppel-like factor 5 (KLF5) transactivates NOTCH1 in th

181 Kruppel-like factor 5 (KLF5), a proproliferative transcr

182 are primarily regulated by ovarian hormones, Kruppel-like factor 5 (KLF5), a zinc finger-containing t

183 s, Friend leukaemia integration 1 (Fli1) and Kruppel-like factor 5 (KLF5).

184 Kruppel-like factor 5 binds directly to the promoter of

185 rian cancer cells was mediated by C3a and is Kruppel-like factor 5 dependent.

186 transgenic mice were generated in which the Kruppel-like factor 5 gene (Klf5) was conditionally dele

187 In vivo-inducible gain-of-function of Kruppel-like factor 5 in haematopoietic stem cells incre

188 Altogether, these data indicate that Kruppel-like factor 5 is indispensable for adhesion, hom

189 Kruppel-like factor 5 regulates pluripotent stem cell se

190 proliferation and decreases in beta-catenin, Kruppel-like factor 5, and cyclooxygenase-2 expression w

191 Here we show that Kruppel-like factor 5-deficient haematopoietic stem cell

192 onal network involving the tumor suppressors Kruppel-like factor 6 (KLF6) and forkhead box O1 (FOXO1)

193 In the current study, we identified Kruppel-like factor 6 (KLF6) as a critical transcription

194 programmed cell death protein 4 (PDCD4) and Kruppel-like factor 6 (KLF6) as critical regulators and

195 Kruppel-like factor 6 (Klf6) belongs to a family of zinc

196 The Kruppel-like factor 6 (KLF6) gene is a zinc finger trans

197 of the transcription factor/tumor suppressor Kruppel-like factor 6 (KLF6) has been described in prost

198 Kruppel-like factor 6 (KLF6) is a transcription factor a

199 Kruppel-like factor 6 (KLF6) is a tumor suppressor gene

200 etic activation of the tumor suppressor gene Kruppel-like factor 6 (KLF6) plays a role in ERMA-induce

201 The polymorphism, KLF6-IVS1-27A, in the Kruppel-like factor 6 (KLF6) transcription factor gene e

202 estingly, a key factor in tissue remodeling, Kruppel-like factor 6 (KLF6) translocates to the cell nu

203 reased expression of a splice variant of the Kruppel-like factor 6 (KLF6) tumor suppressor gene, know

204 Kruppel-like factor 6 (KLF6) was identified as a key tra

205 Among these, Kruppel-like factor 6 (KLF6) was reduced in DDLPS, with

206 domain of LCoR and the transcription factor Kruppel-like factor 6 (KLF6), a putative tumor suppresso

207 Here, we identified Kruppel-like factor 6 (KLF6), a zinc finger domain trans

208 Kruppel-like factor 6 (KLF6), a zinc finger transcriptio

209 Transcription factor Kruppel-like factor 6 (KLF6)-myeloid-specific conditiona

210 Kruppel-like factor 6 (Klf6; copeb in zebrafish) is a zi

211 manner dependent on the transcription factor Kruppel-like factor 6 , suggesting that this pathway und

212 onjunction with the tumor suppressor protein Kruppel-like factor 6 functioning as an AhR binding part

213 KLF6 (Kruppel-like factor 6) is a zinc finger transcription fa

214 nding protein 4, GATA-binding protein 6, and Kruppel-like factor 6.

215 d, in part, by the transcriptional activator Kruppel-like factor-6 (Klf6).

216 Increased expression of Kruppel-like factor 7 (KLF7) is an independent predictor

217 nown coactivator of the transcription factor Kruppel-like factor 7 (KLF7), which regulates genes requ

218 We have recently identified Kruppel-like factor 8 (KLF8) as a critical inducer of EM

219 Kruppel-like factor 8 (KLF8) is a transcriptional factor

220 We have previously demonstrated that Kruppel-like factor 8 (KLF8) participates in oncogenic t

221 Kruppel-like factor 8 (KLF8) plays a key role in oncogen

222 Kruppel-like factor 8 (KLF8) regulates critical gene tra

223 Kruppel-like factor 8 (KLF8) regulates critical gene tra

224 Kruppel-like factor 8 (KLF8) regulates critical gene tra

225 KLF8 (Kruppel-like factor 8) is a transcription factor downstr

226 -Myc, Cyclin D1, Bcl-xL, Survivin, VEGF, and Kruppel-like factor 8, which is identified as a Stat3 ta

227 ulator of dentate granule neuron maturation, Kruppel-like factor 9 (Klf-9).

228 of a negative regulator of dendritic spines, Kruppel-like factor 9 (Klf9), in mature DGCs enhanced in

229 GR1), forkhead box protein A3 (FOXA3), JUNB, Kruppel-like factor 9 (KLF9), KLF10, and REV-ERBalpha-we

230 Nrf2 stimulates expression of transcription Kruppel-like factor 9 (Klf9), resulting in further Klf9-

231 o3), thyroid receptors (tralpha, trbeta) and Kruppel-like factor 9 (klf9), suggesting regulation by t

232 identified a circadian transcription factor, Kruppel-like factor 9 (Klf9), that is substantially up-r

233 -of-function approaches, we also showed that Kruppel-like factor 9 was a key mediator of this effect

234 Erythroid Kruppel-like factor, a pivotal regulator of erythropoies

235 Although Kruppel-like factors are unequivocally implicated in thi

236 The Kruppel-like factor, BTEB1/KLF9, was expressed in both p

237 Erythroid Kruppel-like factor (EKLF [KLF1]) is a transcriptional r

238 Erythroid Kruppel-like factor (EKLF or KLF1) is a transcription fa

239 Erythroid Kruppel-like factor (EKLF or KLF1) is a transcriptional

240 Mutations in the human erythroid Kruppel-like factor (EKLF) can lead to either anemia or

241 Erythroid Kruppel-like factor (EKLF) is a hematopoietic-specific t

242 Erythroid Kruppel-like factor (EKLF) is a Kruppel-like transcripti

243 man erythroid cells, expression of erythroid Kruppel-like factor (EKLF) precedes PlGF, and its enforc

244 thin the second zinc finger of the erythroid Kruppel-like factor (EKLF), a critical erythroid regulat

245 here transcription factors such as erythroid Kruppel-like factor (EKLF), which is also known as Krupp

246 Because Erythroid Kruppel-like Factor (EKLF)-knockout mice showed similar

247 , including the erythroid-specific erythroid Kruppel-like factor (EKLF).

248 Erythroid Kruppel-like Factor (EKLF/KLF1) is a master transcriptio

249 Erythroid Kruppel-like factor (EKLF/KLF1) is one of a very small n

250 tic leukemia 1 protein (Tal1), and Erythroid Kruppel-like factor (EKLF; henceforth referred to as Klf

251 Erythroid Kruppel-like factor (EKLF; KLF1) is an erythroid-specifi

252 Erythroid Kruppel-like factor (EKLF; KLF1), a crucial zinc finger

253 tion of relevant heat shock elements and SP1/Kruppel-like factor element completely prevents statin-i

254 used to demonstrate that mutation of the SP1/Kruppel-like factor element in the TM promoter only part

255 Our group and others have implicated the Kruppel-like factor family of transcription factors as c

256 The Kruppel-like factor family of transcription factors play

257 KLF4 is a member of the Kruppel-like factor family of transcriptional regulators

258 codes a novel transcription regulator in the Kruppel-like factor family.

259 M) by mechanisms that involve members of the Kruppel-like factor family.

260 review focuses on the relevant literature of Kruppel-like factors in leukocyte biology and their impl

261 OXO may represent a broad mechanism by which Kruppel-like factors integrate with insulin signaling to

262 ctor 4 (Klf; previously known a gut-enriched Kruppel-like factor) is a DNA-binding transcriptional re

263 c mice deficient in the transcription factor Kruppel-like factor (KLF) 13 have comparable numbers of

264 Kruppel-like factor (KLF) 13 is a transcription factor t

265 and decreased induction of antiinflammatory kruppel-like factor (KLF) 2 and KLF4.

266 entified a role for the transcription factor Kruppel-like factor (KLF) 4 in the development of IL-17-

267 nomics, we have identified conserved ETS and Kruppel-like factor (KLF) binding sites within the Flk1

268 Several members of the Kruppel-like factor (KLF) family of transcription factor

269 he Sp1 transcription factor, a member of the Kruppel-like factor (KLF) family of transcriptional regu

270 Kruppel-like factor (KLF) motifs were enriched in cornea

271 Kruppel-like factor (KLF) proteins are emerging as key r

272 Kruppel-like factor (KLF) proteins have elicited signifi

273 Module to analyze the binding regions of the Kruppel-like factor (KLF) transcription factor in embryo

274 chanistically, ORAI1 induced upregulation of Kruppel-like factor (KLF)-2 and KLF4 in the flow-activat

275 Regulating these states is the presence of a Kruppel-like factor (KLF)-containing Polycomb response e

276 The importance of Kruppel-like factor (KLF)-mediated transcriptional pathw

277 Several recent studies reported that Kruppel-like factor (KLF)2 controls trafficking, develop

278 Kruppel-like factor (KLF)2 is a central regulator of end

279 leted mice have injured vessels with reduced Kruppel-like factor (KLF)2, KLF4, endothelial nitric oxi

280 duced activation of the transcription factor Kruppel-like factor (KLF)4 may have an important role in

281 dent pathway by serving as a coactivator for Kruppel-like factor (KLF)4-a driver of tissue-resident m

282 croarray and experimental data revealed that Kruppel-like factor (Klf)5 was the most up-regulated tra

283 Here we identify the C. elegans Kruppel-like factor, KLF-1, as an essential and specific

284 erein, we report that a TGF-beta1-responsive Kruppel- like factor, KLF10, is strongly expressed in PA

285 expression of FABP5 is down-regulated by the Kruppel-like factor KLF2, suggesting a tumor suppressor

286 The Kruppel-like factor Klf4 is implicated in tumorigenesis

287 titution of the sole sumoylation site of the Kruppel-like factor (KLF4), a well known reprogramming f

288 TGFbeta/Smad3 up-regulated Kruppel-like factor (KLF5) protein, and SMC de-different

289 Kruppel-like factors (KLFs) are a family of 17 transcrip

290 Kruppel-like factors (KLFs) are a family of zinc-finger

291 Kruppel-like factors (KLFs) are transcription factors th

292 Kruppel-like factors (KLFs) are zinc finger transcriptio

293 While recent insights implicate Kruppel-like factors (KLFs) as important regulators of v

294 Specificity proteins (SPs) and Kruppel-like factors (KLFs) belong to the family of tran

295 Kruppel-like factors (KLFs) control cell differentiation

296 Recent studies have implicated Kruppel-like factors (KLFs) including KLF5 in the renewa

297 Kruppel-like factors (KLFs) play a critical role in regu

298 of enhancer RNAs pointed a central role for Kruppel-like factor, MEF2C, ETS, NFY, ATF, E2F2, and NRF

299 KLF1 (erythroid Kruppel-like factor) plays essential roles in embryonic

300 e isoform 2 (PKM2) and T-helper-inducing POZ-Kruppel-like factor (ThPOK).