ライフサイエンスコーパス: L cell

1 ls producing the viral receptor ICAM-1 (ICAM-L cells).

2 n (D-cells) and peptide YY-expressing cells (L-cells).

3 as diminished to the level of vector control L cells.

4 le to establish persistent infection in HeLa-L cells.

5 ily established persistent infection in HeLa-L cells.

6 ible p150 proteins were found in IFN-treated L cells.

7 l/GTP-stabilized cytosol prepared from GR(-) L cells.

8 age of rhinovirus 16 between HeLa and ICAM-1 L cells.

9 erified 22:4,n6 did not accumulate in HEK293-L cells.

10 expressed at the cell surface of transfected L cells.

11 had no effect on LXRalpha activity in HEK293-L cells.

12 h levels of apoptosis in both HeLa cells and L cells.

13 induced detectable Pkr mRNA accumulation in L cells.

14 in reticulocyte lysate remained unstable in L cells.

15 in assays using extracts prepared from mouse L cells.

16 , we failed to express the truncated form in L cells.

17 ncreased free fatty acids reaching the ileal L cells.

18 cose-stimulated GLP-1 release in human ileal L cells.

19 xin-1a (syn1a) was expressed by murine ileal L cells.

20 erived peptide secreted from enteroendocrine L cells.

21 utics aimed at targeting the GLP-1 producing L-cells.

22 o two HSPG-deficient cell lines derived from L-cells.

23 and cis-parinaric acids was undetectable in L-cells.

24 26 A between the two fluorescent sterols in L-cells.

25 nd control cells and was enriched in colonic L-cells.

26 als of prandial GLP-1 secreted by intestinal L-cells.

27 the development of the first rapid injection LD cell.

28 nmol/L), RKO (30 nmol/L), and SW620 (30 nmol/L) cells.

29 endent increase in oxidative DNA damage in A(L) cells.

30 ramidal neurons provide excitatory inputs to LS cells.

31 ediated, feedforward mechanism that inhibits LS cells.

32 hibit Ca2+ influx into carrot (Daucus carota L.) cells.

33 of cells within layer 1 called late-spiking (LS) cells.

34 ower, estimated in the intact cell (85 mmol (l cell)(-1) (pH unit)(-1) at resting pH(i)), was somewha

35 ous estimates from cell lysates (50-70 mmol (l cell)(-1) (pH unit)(-1)).

36 centration factors were calculated (>10(3) L L(cell)(-1)).

37 concentrations between 10(-5) and 10(-3) mol L(cell)(-1).

38 HT-29 cells (58%), and SCP-2 overexpressing L-cells (37%).

39 is using human breast carcinoma MDA-MB-435-F-L cells, a highly metastatic variant of human breast can

40 In transiently transfected mouse L cells, a promoter fragment in which the NF-I site was

41 epG2, a human hepatic derived cell line, and L cells, a subline of NCTC clone 929 mouse fibroblasts.

42 e as well as in null versus AQP4-transfected L-cells, a cell type lacking endogenous adhesion molecul

43 GLUT2 and CasR regulate K- and L-cell activity in response to nutrient and non-nutrient

44 ceptor agonist, are cosecreted by intestinal L-cells after each meal.

45 We here measure the surface tensions of L cell aggregates transfected to express N-, P- or E-cad

46 PECAM-1-dependent homophilic adhesion in an L cell aggregation assay also blocked TNF-alpha-induced

47 rcISVPs retained full infectivity in murine L cells, allowing their use to study sigma3 functions in

48 Mac-l+ cells alone gave no appreciable change in responsive

49 Mouse intestinal L cells also express alpha-gustducin.

50 oduction of glucagon-like peptide (GLP)-1 by L cells and alpha cells, leading to secretion of insulin

51 uction assays and inhibited T1L adherence to L cells and Caco-2(BBe) intestinal epithelial cells in v

52 Whether FXR is expressed in L cells and controls GLP-1 production is unknown.

53 ed buds of the trans-Golgi networks of mouse L cells and human HeLa cells.

54 glucagon-like peptides (GLP)-1 and -2 in the L cells and neurons.

55 s a pronounced antimitogenic effect in mouse L cells and NIH 3T3 cells.

56 genous chromosome from revertants into mouse L cells and performing allelic deletion mapping studies

57 eptide hormone secreted from enteroendocrine L cells and potentiates glucose-dependent insulin secret

58 specific hormones produced by the endocrine L cells and serotonin-producing cells.

59 itated from pervanadate-treated MC38/CEACAM1-L cells and the associated proteins were analyzed by two

60 Calcium imaging of primary L cells and the model cell line GLUTag revealed response

61 00-fold more infectious than cores in murine L cells and, like virions but not ISVPs or cores, were i

62 Examining translation in RNase L(-/-) cells and mice confirmed that RNase L activity re

63 DISC of both MCF7-Fas-Vec and MCF7-Fas-Bcl-x(L) cells and that the early steps of CD95 signaling such

64 types, specifically in intestinal endocrine L-cells and brown adipose tissue, has made it a promisin

65 ptor 1B, which is highly enriched in colonic L-cells and linked to the elevation of L-cell calcium an

66 veolin-1 primarily at the plasma membrane of L-cells and more so within intracellular punctuate struc

67 racterise the electrical activity of primary L-cells and the importance of voltage gated sodium and c

68 beta-locus YAC was first microinjected into L-cells and then transferred into MEL cells by fusion, t

69 Cell fractionation of SCP-2 overexpressing L-cells and Western blotting detected SCP-2 in purified

70 in suspension cultured carrot (Daucas carota L.) cells and tobacco (Nicotiana tabacum L.) leaf tissue

71 g (83%), whereas electrical coupling between LS cells and non-LS cells was infrequent (2%).

72 e peptide-1- and peptide-2-expressing cells (L-cells), and have reduced numbers of somatostatin (D-ce

73 (grp43) and ffar3 (gpr41) in GLP-1-secreting L cells, and consistent with the reported coupling of GP

74 in three assays, with Xenopus oocytes, mouse L cells, and human embryonic stem cells, that secreted X

75 ressed in pancreatic alpha cells, intestinal L cells, and some hypothalamic and brainstem neurons.

76 a potent mutagen in human-hamster hybrid (A(L)) cells, and that it induces predominantly multilocus

77 dation, attention is now turning towards the L-cell, and addressing whether it would be both possible

78 urface of Vero, A431, HeLa, and BSC-1 cells, L-cells, and a mouse melanoma cell line expressing the g

79 peptide 1 (GLP-1) is secreted by intestinal L-cells, and augments glucose-induced insulin secretion,

80 Interestingly, DeltaPsi(m)L cells are highly efficient at in vitro mesodermal diff

81 JT/Bcl-2 and JT/Bcl-X(L) cells are susceptible to NO-mediated apoptosis, but B

82 Our data suggest L-cells are active players in the hypothalamic control o

83 and application of the micro-volume Couette LD cell are illustrated by the collection of data for DN

84 n yeast which can each be abrogated by BCL-X(L): cell arrest, which does not require aspects of mitoc

85 measurements indicated that Bcl-2 and Bcl-x(L) cells arrested more effectively in G(0) than controls

86 elevated calcium and cAMP concentrations in L-cells as measured in cultures from GLU-Cre/ROSA26-GCaM

87 ells formed GABAergic connections with other LS cells as well as with non-LS cells in layer 1 and wit

88 to different secretagogues in murine GLUTag L cells, as well as in the mRNA levels of several canoni

89 ut still detectable relative to the parental L-cells, as previously reported.

90 roductive HRV39 infection is blocked in ICAM-L cells at a step posttranslation and prior to the forma

91 In conclusion, we propose that AVP activates L-cell AVPR1B, causing GLP-1 and PYY secretion.

92 test whether passage of the beta-YAC through L-cells before transfer into MEL cells was the reason fo

93 fied as a product of colonic enteroendocrine L-cells, better known for their secretion of glucagon-li

94 Here, we review the current knowledge of LD cell biology and its translation to physiology.

95 le to establish persistent infection in HeLa-L cells but that recombinant viruses with combined subst

96 h Cad11-mediated cell-cell adhesion of mouse L cells, but significantly reduced Cad11-mediated cell m

97 ain with a wild-type alpha chain in a murine L cell by gene transfection.

98 these AD-related endocytic changes in murine L cells by overexpressing Rab5, a positive regulator of

99 stablishment of persistent infection in HeLa-L cells by the HeLa-H-EAVP80 virus.

100 EL hybrid cells, we transfected the YAC into L-cells by lipofection.

101 irus, KgBpK- showed reduced binding to mouse L cells (ca. 20%), while a gC null mutant virus in which

102 lonic L-cells and linked to the elevation of L-cell calcium and cAMP concentrations.

103 s; thus, biological effects on neighboring A(L) cells can be attributed to the bystander response.

104 f fibroblast-like synoviocytes, we generated L cell clones expressing wild-type cadherin 11, mutant c

105 invasive capacity of cadherin 11-transfected L cells compared with L cells transfected with E-cadheri

106 ander mutation incidence among neighboring A(L) cells compared with controls.

107 s were produced by susceptible C57BL/6 RNase L-/- cells compared to RNase L+/+ cells that were either

108 Transfection of Xbves into nonadherent mouse L cells confers cell/cell adhesion.

109 ly inactive PTP1B with the Y152F mutation in L-cells constitutively expressing N-cadherin has no effe

110 ceptor protein-tyrosine phosphatase PTP1B in L-cells constitutively expressing N-cadherin results in

111 The K289M protein was expressed in mouse L cells containing the lambda cII mutational target.

112 eased expression of exogenous genes in RNase L(-/-) cells correlated with elevated levels of mRNA and

113 a novel sensory mechanism in enteroendocrine L cells, coupled to the facilitation of GLP-1 release, w

114 The purified enzyme is abundant (55 mg/L cell culture), highly active (approximately 4.7 x 10(3

115 Non-lignifying maize (Zea mays L.) cell cultures were incubated with l-[1-(3)H]arabinos

116 GR.hsp90 heterocomplexes immunoadsorbed from L cell cytosol contain dynein and that GR.hsp90 heteroco

117 Here, we show that FXR activation in L cells decreases proglucagon expression by interfering

118 P) stimulates the release of enteroendocrine L-cell derived hormones glucagon-like peptide-1 (GLP-1)

119 B82 mouse L cells devoid of EGFR were used to further investigate

120 In addition to Bcl-X(L), cells devoid of mitochondrial DNA (rho degrees cells

121 ndogenous GLP-1 production include promoting L-cell differentiation and increasing L-cell number.

122 that an independent peripheral clock in the L cell drives a circadian rhythm in GLP-1 secretory resp

123 tive PCR analysis, we identified trpa1 as an L cell-enriched transcript in the small intestine.

124 R when the YAC is first transferred into the L-cell environment raises the possibility that normal ac

125 Duodenal L cells express sweet taste receptors, the taste G prote

126 We show that human duodenal L cells express sweet taste receptors, the taste G prote

127 erent, as compared with approximately 10% on L cells expressing E-selectin or ICAM-1 alone.

128 When transfected L cells expressing I-A(d) (AalphaAbeta(d)), I-E(d) (Ealp

129 In various combinations of L cells expressing NCAM, E-, P-, N-, R-, or B-cadherin,

130 ted to transfected Chinese hamster ovary and L cells expressing the GP Ib-IX-V complex.

131 re confirmed for endogenous mouse Abeta40 in L cells expressing these CD-MPR constructs but not overe

132 nd C/EBPgamma to activate a reporter gene in L cell fibroblasts but did not inhibit a chimeric C/EBPb

133 from purified plasma membranes isolated from L-cell fibroblasts and MDCK cells by detergent-free affi

134 laser scanning microscopy (MPLSM) of living L-cell fibroblasts cultured with dehydroergosterol for t

135 on in this role was addressed in transfected L-cell fibroblasts overexpressing L-FABP using a series

136 rst time, expression of SCP-2 in transfected L-cell fibroblasts reduced the plasma membrane levels of

137 Overall, HDL-mediated sterol efflux from L-cell fibroblasts reflected that of the cytoplasmic rat

138 scent sterol, was incorporated into cultured L-cell fibroblasts.

139 ized with PPARalpha in nuclei of transfected L-cell fibroblasts.

140 nlipid raft domains from plasma membranes of L-cell fibroblasts.

141 sterol uptake and intracellular targeting in L-cell fibroblasts.

142 eptide-1 (GLP-1) is released from intestinal L-cells following food ingestion.

143 r 12 hours and subsequently incubated with A(L) cells for 24 hours at 11 degrees C.

144 relin plays a role in priming the intestinal L-cell for nutrient-induced GLP-1 release.

145 Using paired recording, we showed that LS cells formed GABAergic connections with other LS cell

146 te of fall of specific activities of labeled L-cell fractions have been observed.

147 L cells from intestinal-epithelial syn1a-deficient mice

148 vel in vitro platform to generate functional L cells from three-dimensional cultures of mouse and hum

149 peptide 1 (GLP-1), secreted from intestinal L cells, glucose dependently stimulates insulin secretio

150 GLP-1 secretion is metabolically coupled in L cells (GLUTag) and in vivo in mice using the insulin-s

151 s and (b) mice inoculated i.m. with the RH30-L cells had more rhabdomyosarcoma cells in the bone marr

152 was fully functional in stable, transfected L-cells harboring a single copy of this transgene, inclu

153 GLP-1 is a gastrointestinal L-cell hormone that enhances glucose-stimulated insulin

154 Moreover, in Chinese hamster ovary and L-cells, HPV E7 and the adenovirus E1A protein repressed

155 cose infusion activated half of all duodenal L cells in humans.

156 ibitor dibenzazepine increased the number of L cells in intestinal organoid-based mouse and human cul

157 us to study and modulate the development of L cells in mouse and human crypts as a potential basis f

158 ing pathways, SCFAs raised cytosolic Ca2+ in L cells in primary culture.

159 ptide-1 (GLP-1), released from gut endocrine L cells in response to glucose, regulates appetite, insu

160 intestinal epithelium, and reduced number of L cells in the distal ileum.

161 ermeabilize membranes in vitro and to infect L cells in the presence of NH4Cl, providing new evidence

162 e membrane and of cell particulate matter of L cells in tissue culture in logarithmic and plateau pha

163 CHO cells were mixed with A(L) cells in a 1:5 ratio and briefly centrifuged to produ

164 te fiber-induced DNA damage mutagenesis in A(L) cells in a concentration-dependent manner.

165 V) into xenografts derived from DU-145-Bcl-x(L) cells in athymic nude mice completely eradicated not

166 gs describe, for the first time, the role of L-cells in AVP regulated intestinal fluid secretion, pot

167 eptide-1 (GLP-1) is released from intestinal L-cells in response to a range of nutrients, hormones, a

168 eptide-1 (GLP-1) is released from intestinal L-cells in response to nutrient ingestion.

169 ucagon-like peptide-1 (GLP-1), released from L-cells in the intestinal epithelium, plays an important

170 ions with other LS cells as well as with non-LS cells in layer 1 and with pyramidal cells in layer 2/

171 nd virions used similar routes of entry into L cells, including processing by lysosomal cysteine prot

172 terfering RNA inhibition of hERG in beta and L cells increased insulin and GLP-1 secretion up to 50%.

173 heterotrophic sycamore (Acer pseudoplatanus L.) cells incubated in various nutrient media contain lo

174 uanosine (8-OHdG) in human-hamster hybrid (A(L)) cells induced by crocidolite fibers in an attempt to

175 ination of purified T cells and purified Mac-l+ cells induced massive and permanent deletion of the h

176 Nutrient stimulation of enteroendocrine L cells induces the release of the incretin and satiatin

177 gon-like peptide 1 (GLP-1) by the intestinal L cell is essential for the incretin effect after nutrie

178 the mechanisms underlying GLP-1 release from L-cells is key to this type of approach, and the use of

179 GLP-1 release from primary L-cells is linked to electrical activity and activation

180 The stimulus-secretion pathway in L-cells is still incompletely understood and a topic of

181 gon in pancreatic alpha-cells and intestinal L cells leads to production of distinct hormonal product

182 At the single L-cell level, first-phase forskolin-induced exocytosis w

183 2, we analyzed splicing responses on the MSD-L cell line and found that the missense mutation of prol

184 The human L cell line NCI-H716 expresses alpha-gustducin, taste re

185 e compared after expression in a mouse RNase L(-/-) cell line.

186 This revealed that every heteroplasmic L-cell line harbored a mtDNA that had been generated by

187 and alpha7-nAChR transcription in human SCC-L cell lines and SCC-L tumors.

188 er388Tyr and Thr389Pro, we created stable A9 L cell lines expressing HM(1)(S388Y) or HM(1)(T389P) rec

189 loss of gene expression; (2) treatment of L/L cell lines with a demethylating agent resulted in re-e

190 While analyzing the mtDNAs of several mouse L cell lines, we discovered that every cell line harbore

191 ) increased alpha7-nAChR levels in human SCC-L cell lines.

192 downregulation of phosphorylated STAT3 in L/L cell lines; (3) all 55 control specimens and the lymph

193 -2(-)/Bcl-x(L)(+)) and Daudi (Bcl-2(+)/Bcl-x(L)(+)) cell lines.

194 relin directly stimulated GLP-1 release from L-cell lines (murine GLUTag, human NCI-H716) through an

195 We found previously that in gut endocrine L-cell lines, TCF7L2 controls transcription of the progl

196 s were then targeted to the lox sites of the LES cell lines.

197 d that both fluorescent sterols localized to L-cell lipid droplets, the surface of which contained ad

198 ) Priming: After H2O2 exposure (100 micromol/L), cells maintain a constant DeltaPsi(m) for the cell-t

199 glutamine and related analogs by GDH in the L cell may explain why GLP-1 secretion, but not that of

200 ectrical activity and GLP-1 release from the L-cell model line GLUTag.

201 plate flow chamber having as a substrate an L cell monolayer coexpressing E-selectin and ICAM-1 (E/I

202 e of neutrophils rolling on HUVECs and on an L-cell monolayer coexpressing E-selectin and ICAM-1.

203 phosphorylation was not diminished in Y325F L-cell mutants suggesting Tyr-325 was not required for t

204 In HEK293-L cells, non-esterified 20:4,n6 also increased (5-fold)

205 moting L-cell differentiation and increasing L-cell number.

206 etes, dibenzazepine administration increased L cell numbers in the intestine, improved the early insu

207 g of the signalling mechanisms that underlie L-cell nutrient responsiveness.

208 We conclude that L cells of the gut "taste" glucose through the same mech

209 naturally occurring peptide secreted by the L cells of the small intestine.

210 When control and Bcl-x(L) cells of equivalent size and pyronin Y fluorescence w

211 Peptide-YY (PYY) is secreted from endocrine L-cells of the gastrointestinal tract in response to cal

212 (SS), fast-adapting (FA), and late-spiking (LS) cells, of which RS and IB cells were common to LII a

213 ecombinant insulin secretion from intestinal L-cells on both a per-cell and per-graft basis.

214 When expressed in thymidine kinase-deficient L cells or 3T3 cells, C/EBPalpha is detected in a protei

215 th aggregation experiments using transfected L cells or primary chick brain cells.

216 ipped steroid receptors isolated from either L cells or WCL2 cells.

217 , and gammaFULL LENGTH (FL)) co-expressed in L-cells or CHO cells with wild-type (WT) IL-4Ralpha.

218 Transfection of l-cells or McAR7777 hepatoma cells with cDNA encoding pr

219 diated suppression of the ES, but not of the LS cell output synapse.

220 -6-P/IGF-II receptor at 0 degrees C in mouse L cells overexpressing the Man-6-P/IGF-II receptor and i

221 The receptor is found on gut enteroendocrine L-cells, pancreatic beta-cells, and sympathetic neurons,

222 low-passage-number human cervix cells (HeLa-L cells; passages 95 to 115) or in several other cell li

223 )-1 and GLP-2, the two major enteroendocrine L-cell peptides.

224 be facilitated by a better understanding of L-cell physiology.

225 IgA and IgG MAbs neutralized reovirus T1L in L cell plaque reduction assays and inhibited T1L adheren

226 r in Vero monkey kidney cells than in murine L cells, plaquing efficiency of individual isolates did

227 peptide YY (PYY) is released from intestinal L-cells post-prandially in proportion to calorie intake,

228 Upon a nutrient challenge, L cells produce glucagon-like peptide 1 (GLP-1), a power

229 ential (TRP) ion channels in enteroendocrine L cells producing GLP-1.

230 We hypothesized that enteroendocrine L-cells producing glucagon-like peptide 1 (GLP-1) and pe

231 human lung microvascular endothelial (HMVEC-L) cells promoted their apoptosis.

232 The concept of an electrically excitable L-cell provides a basis for understanding how GLP-1 rele

233 L-cells purified by flow cytometry were used to measure

234 adhesion molecule, transfection of Bves into L-cells readily confers adhesive behavior to these cells

235 e peptide 1 (GLP-1) released from intestinal L cells regulate insulin secretion.

236 ptor Y1 receptor antagonist, suggesting that L-cell-released PYY acts locally on the epithelium to mo

237 essed in duodenal enterochromaffin cells and L cells, respectively.

238 o and overexpression of SCP-2 in transfected L-cells resulted in the following: (i) redistribution of

239 tty acids selectively increase the number of L cells, resulting in an elevation of GLP-1 release.

240 In SCP-2 overexpressing L-cells, SCP-2 was detected in close proximity to caveol

241 oinhibitory feedback mechanism that controls L cell secretion; the effect on insulin and glucagon con

242 We found that (a) RH30-L cells seed better to the bone marrow, liver, and lymph

243 Mouse L cells stably expressing CD-MPRs with mutations that en

244 The invasive behavior of L cells stably transfected with a cadherin 11 construct

245 ake and metabolism play an important role in L cell stimulus-secretion coupling.

246 incretin hormone produced by the intestinal L cell that acts as a link between the gut and pancreati

247 irus type 16 was found to replicate in mouse L cells that express the viral receptor, human intercell

248 tor 2 (FFA2) is expressed on enteroendocrine L cells that release glucagon-like peptide 1 (GLP-1) and

249 The use of A(L) cells that are dominant negative for connexin 43 and

250 e C57BL/6 RNase L-/- cells compared to RNase L+/+ cells that were either left untreated or pretreated

251 on that innervates the longitudinal muscles (L-cells) that contributes to a defensive withdrawal refl

252 revealed that, within the nucleus of control L-cells, the nonhydrolyzable fluorescent LCFA-CoA (BODIP

253 mulated the release of GLP-1 from intestinal L-cells, thereby providing a plausible mechanism for VSL

254 Exposure of JT/Bcl-2 and JT/Bcl-X(L) cells to the NO donor, S-nitroso-N-acetylpenacillamin

255 may underlie the physiological responses of L-cells to food ingestion.

256 cent model of electrical activity in primary L-cells to include spatiotemporal glucose and Ca(2+) dyn

257 ection with an HRG expression construct that led cells to acquire estrogen independence and metastasi

258 Paradoxically, this anabolic program led cells to apoptosis during chronic ER stress in a man

259 ptogenetic activation of the projection from LS cells to the VMHvl terminated ongoing attacks immedia

260 The invasive capacity of L cell transfectants and cultured fibroblast-like synovi

261 We developed L cell transfectants expressing cadherin-11, cadherin-11

262 L cell transfectants expressing SE-positive DR molecules

263 CD16 Fcgamma receptor and studied in stable L cell transfectants.

264 adherin 11-transfected L cells compared with L cells transfected with E-cadherin or control vector.

265 Using L cells transfected with genes for MHC class II, invaria

266 the JAK-STAT pathway in these models and in L cells transfected with STPB-C.

267 In contrast, l-cells transfected with cDNA encoding SCP-2 exhibited 3

268 F-I proteins, NF-IA, -B, -C, or -X, in mouse L cells transiently transfected with an ldhc promoter-re

269 r 1C, expression was increased in fibroblast L cells treated with IFN, and a consensus ISRE element w

270 In murine L cells, treatment with calpeptin or calpain inhibitor I

271 and Kv9.3 alpha subunits expressed in mouse L cells using the whole-cell patch-clamp technique.

272 Likewise, L-cells very poorly esterified these fluorescent fatty a

273 content (79.4+/-5.7 to 115.2+/-6.6 micromol/L cell volume [mean+/-SEM; P:<0.001]), and, after a wave

274 The complete form of cadherin-8 expressed in L cells was about 130 kDa in molecular mass and was loca

275 that mengovirus plaque formation in HeLa or L cells was inhibited nearly 100% by the presence of 80

276 RGS4 that was transiently expressed in mouse L cells was short-lived in these cells.

277 tabilization of p27 in G(0) in BCL2 or BCL-x(L) cells was due to phosphorylation of p27 at Ser(10) by

278 Electrical activity in L-cells was due to large voltage gated sodium currents,

279 electrical coupling between LS cells and non-LS cells was infrequent (2%).

280 e between the human transfectant and a mouse L cell, was used as donor of the Ecogpt-carrying human c

281 Using the human-hamster hybrid A(L) cells, we show here that arsenic alters mitochondrial

282 peptide-1 (GLP-1) secreting enteroendocrine L-cells, we have designed TGR5 agonists with low intesti

283 elicitor-treated tobacco (Nicotiana tabacum L.) cells, we identified a rapid release and accumulatio

284 When 10(4) AW/tmFlt3-L cells were injected i.v. into mice, only 35.7% (5 of 1

285 Nonlabeled bystander A(L) cells were isolated from among labeled CHO cells by u

286 Primary murine L-cells were identified and purified using transgenic mi

287 Fluorescent L-cells were identified within primary colonic cultures

288 The dendrites and axons of layer 1 LS cells were confined primarily to layer 1.

289 greement with that hypothesis, we found that LS cells were interconnected by electrical coupling (83%

290 RS and IB cells were common to LII and LIII, LS cells were specific to LIII, and the remaining types

291 driven synaptically by LOT stimulation (type L cells) were concentrated in the superficial half of th

292 lucagon-like peptide-1 (GLP-1) production by L cells which potentiates beta-cell glucose-induced insu

293 sis that increasing the number of intestinal L cells, which produce GLP-1, is an alternative strategy

294 an orexigenic hormone released from colonic L-cells, which promotes appetite during conditions of en

295 roximately 86%) of neurons are late-spiking (LS) cells, which can delay the onset of their spike trai

296 ed peptide GLP-2, cosecreted from intestinal L cells with GLP-1, has recently been demonstrated to en

297 stably transfected APP-overexpressing murine L cells with human CD-MPR.

298 at irradiation of 20% of randomly selected A(L) cells with 20 alpha particles each results in a mutan

299 rotein sulfhydryls (mainly glutathione) in A(L) cells with buthionine S-R-sulfoximine increases the m

300 Transduction of insulin-expressing GLUTag L-cells with lentivirus carrying an additional human ins