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1 in dipalmitate and 2-O-beta-d-glucopyranosyl-l-ascorbic acid.
2 ulated 2.2-fold by the addition of 50 microm l-ascorbic acid.
3 the de novo synthesis of GDP-L-galactose and L-ascorbic acid.
4 support the interactions between solutes and L-ascorbic acid.
5 rectly oxidized the major plant antioxidant, l-ascorbic acid.
6 such as rutin and 2-O-beta-d-glucopyranosyl-l-ascorbic acid.
7 of any trend supporting a protective role of L-ascorbic acid.
8 correlates of circulating concentrations of L-ascorbic acid.
9 gene locus and circulating concentrations of L-ascorbic acid.
10 arable for ascorbic acid and 6-bromo-6-deoxy-L-ascorbic acid.
11 served for ascorbic acid and 6-bromo-6-deoxy-L-ascorbic acid.
12 l beverages (citric acid, pH 3.0) containing l-ascorbic acid (0.050%) degraded with a first-order rea
14 hree components: the basal medium RPMI 1640, L-ascorbic acid 2-phosphate and rice-derived recombinant
15 toxicant injury and stimulation of repair by L-ascorbic acid-2-phosphate (AscP), exogenous collagen I
16 a reduction in circulating concentrations of L-ascorbic acid (-4.15 micromol/L; 95% CI: -0.49, -7.81
17 drogenase was activated by either 150 microm L-ascorbic acid (56%) or 300 microm iron (Fe(2+) or Fe(3
18 hlorogenic acid (CG), (-) epicatechin (EPI), L-ascorbic acid (AA) and polyphenoloxidase (PPO) activit
21 y DHA and GLUT transporters, 6-bromo-6-deoxy-L-ascorbic acid accumulation was <1% of accumulation whe
22 relation between rs33972313 and circulating L-ascorbic acid across all studies confirmed this and sh
24 incorporation into a topical solution of 15%l-ascorbic acid and 1%alpha-tocopherol improved chemical
27 gestion that observational relations between L-ascorbic acid and cardiometabolic health may be attrib
29 ons of haemoglobin and natural antioxidants (l-ascorbic acid and Fuscus vesiculosus extract), were hy
33 ared with that expected given the rs33972313-L-ascorbic acid and L-ascorbic acid-outcome associations
34 tes, inverse associations were shown between L-ascorbic acid and systolic blood pressure, triglycerid
35 utritional compounds (phenolic compounds and L-ascorbic acid), and antioxidant capacity of 4 Spanish
36 phenolic compounds, carotenoid, anthocyanin, L-ascorbic acid, and fibre content), antioxidant potenti
37 noreactor (NR) that comprises chlorophyll a, l-ascorbic acid, and gold nanoparticles that are encapsu
38 07-SD change; P = 0.0001) per SD increase in L-ascorbic acid], and a positive association was shown w
43 is mutant vitamin c-1 (vtc1) is deficient in l-ascorbic acid (AsA) due to a mutation in GDP-Man pyrop
46 nderlying the regulation of fruit vitamin C (L-ascorbic acid [AsA]) concentrations, quantitative trai
47 Two biosynthetic pathways for ascorbate (l-ascorbic acid [AsA]; vitamin C) in plants are presentl
49 idue (Cys32) located close to the substrate (L-ascorbic acid) binding site has been modified to precl
50 alized using the Hill equation; oxidation of L-ascorbic acid by Cys32-modified rsAPX showed no eviden
52 his study, nine different food constituents (l-ascorbic acid, caffeic acid, caffeine, curcumin, (-)-e
53 hocyanins (0.025%) in model beverages (0.05% l-ascorbic acid, citric acid, pH 3.0) stored at elevated
57 ultivar on the antioxidant capacity, TP, and l-ascorbic acid content, whilst the breeding strategy an
59 c transients for reduction of Compound II by L-ascorbic acid for Cys32-modified rsAPX are monophasic
61 ated on carbon electrodes working area using L(+) ascorbic acid, gold chroloauric acid and poly-l-lys
64 25 and 0.35) mol kg(-1) aqueous solutions of l-ascorbic acid have been determined from density data m
68 r is capable of measuring d-ascorbic acid or l-ascorbic acid in aqueous as well as in real and commer
70 anches on a Pd core by reducing K2PtCl4 with L-ascorbic acid in the presence of uniform Pd nanocrysta
71 he combined data support the conclusion that l-ascorbic acid is a physiological activator of mitochon
74 activity of PANI/SBA-15 toward oxidation of L-ascorbic acid is comparable to that obtained from a co
75 This shows that the addition sequence of l-ascorbic acid is crucial in determining the onset of d
82 a reduction in circulating concentrations of L-ascorbic acid of -5.98 micromol/L (95% CI: -8.23, -3.7
84 glycerol-3-phosphate dehydrogenase by either L-ascorbic acid or iron or its combination could be tota
88 n model beverage systems (pH 3.0) containing L-ascorbic acid proceeded with a first-order reaction ra
89 ta-induced paralysis because the antioxidant L-ascorbic acid reduced intracellular levels of hydrogen
90 -O-allyl derivatives of 5,6-O-isopropylidene-l-ascorbic acid, respectively, followed by stereospecifi
92 the growth of the mutant mice, which require L-ascorbic acid supplemented in their drinking water (33
93 irst step in investigating the importance of L-ascorbic acid transport in regulating the supply and m
94 T2 each mediate concentrative, high-affinity L-ascorbic acid transport that is stereospecific and is
95 erequisite for investigating 6-bromo-6-deoxy-L-ascorbic acid transported by SVCTs, SVCT2 transport ac
98 eeler) pathway represents the major route to L-ascorbic acid (vitamin C) biosynthesis in higher plant
100 bservational studies showed that circulating L-ascorbic acid (vitamin C) is inversely associated with
102 cultured endothelial cells, the antioxidant, L-ascorbic acid (vitamin C), increases nitric oxide synt
107 The reduction of 2,6-dichloroindophenol by l-ascorbic acid was performed using the theta-glass emit
111 yme activity, dependent upon the presence of l-ascorbic acid, was inhibited by lauryl gallate, propyl
113 ore and after the de-doping of chiral d- and l-ascorbic acid were characterized by scanning electron
115 ch migrates for the alcohol-rich phase, from l-ascorbic acid, which preferentially partitions for the
117 aldono-1,4-lactone derivatives starting from l-ascorbic acid, which would be valuable in the synthesi
118 h electrocatalytic activity for oxidation of L-ascorbic acid, with very low overpotential and high cu
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