ライフサイエンスコーパス: L-cysteine

1 tive oxygen species (ROS) scavenger N-acetyl-L-cysteine.

2 vity by using the antioxidant agent N-acetyl-L-cysteine.

3 ctrocatalytic oxidation and determination of L-cysteine.

4 lls with the free radical scavenger N-acetyl-L-cysteine.

5 feature of SAT is its feedback inhibition by L-cysteine.

6 nyleneiodonium, or the antioxidant, N-acetyl-L-cysteine.

7 f O-acetyl-L-serine (OAS) by a thiol to give L-cysteine.

8 e and upon changing the protonation state of l-cysteine.

9 roup of O-acetyl-l-serine by a thiol to give l-cysteine.

10 p.o. administration of antioxidant N-acetyl-L-cysteine.

11 cells treated with the antioxidant N-acetyl-L-cysteine.

12 after partial removal of the stabilizers of L-cysteine.

13 esence of hydrogen sulfide, L-serine yielded L-cysteine.

14 as S-ethyl-l-cysteine and S-(o-nitrophenyl)-l-cysteine.

15 eas an NRPS EpoB carrier protein contributes l-cysteine.

16 ctions: the adenylation and S-methylation of l-cysteine.

17 NaHS (0.1-100 microM) or l-cysteine (0.1-100 microM) significantly increased plat

18 ietary protein content (14% protein and 0.3% L-cysteine-0.9% L-arginine, or 24% protein and 0.5% L-cy

19 The ACV analogue delta-L-alpha-aminoadipoyl-L-cysteine (1-(S)-carboxy-2-thiomethyl)ethyl ester (ACOm

20 on was inhibited by endogenous H2S precursor l-cysteine (10 mm) but not by l-serine (10 mm) or either

21 arboxypropyl]carbamoyl]-4-(18)F-fluorobenzyl-L-cysteine ((18)F-DCFBC) could image an experimental mod

22 arboxypropyl]carbamoyl]-4-(18)F-fluorobenzyl-L-cysteine ((18)F-DCFBC), a low-molecular-weight radiotr

23 arboxypropyl]carbamoyl]-4-(18)F-fluorobenzyl-l-cysteine ((18)F-DCFBC), to detect metastatic hormone-n

24 ine-0.9% L-arginine, or 24% protein and 0.5% L-cysteine-2.0% L-arginine), dietary antioxidant content

25 lites in urine--N-acetyl-S-(3-hydroxypropyl)-L-cysteine (3HPMA) and N-acetyl-S-(2-carboxyethyl)-L-cys

26 N-(Acyl)-1H-benzotriazoles 6a-f react with l-cysteine 5 at 20 degrees C to give exclusively (i) N-a

27 ginase-inhibited (5.0 mM (S)-(2-boronoethyl)-L-cysteine + 5.0 mM Nomega-hydroxy-nor-L-arginine), L-ar

28 mine in CH(3)CN-H(2)O (3:1), but (ii) S-acyl-l-cysteines 7a-e in CH(3)CN-H(2)O (5:1) in the absence o

29 20 degrees C to give exclusively (i) N-acyl-l-cysteines 8a-e in the presence of triethylamine in CH(

30 nts intracellular acidification) and N-acety-l-cysteine (a scavenger of ROS).

31 Inclusion of N-acetyl-L-cysteine, a known antioxidant and NF-kappaB inhibitor,

32 hylated) to that observed in the presence of l-cysteine, a ligand facilitating rapid Hg(II) biouptake

33 on of NO to CDO in the presence of substrate l-cysteine, a low-spin {FeNO}7 (S = 1/2) signal that acc

34 Treatment with N-acetyl-l-cysteine, a potent antioxidant, abolished lipid peroxi

35 ffective CysSSH synthesis from the substrate L-cysteine, a reaction catalyzed by prokaryotic and mamm

36 N-acetyl-S-farnesyl-L-cysteine (AFC), a modulator of G protein and G-protein

37 QDs emission was observed in the presence of L-cysteine after 2 hours.

38 ls with intracellular ROS scavenger N-acetyl-l-cysteine also inhibits AGP-induced activation of ASK1,

39 first time that in the presence of iron and L-cysteine, an [Fe(4)S(4)] cluster is formed within the

40 ic acid, via a condensation reaction between L-cysteine and 5-hydroxyindole acetaldehyde.

41 at the HydG auxiliary cluster must bind both l-cysteine and a dangler Fe in order to generate the [Fe

42 Also, the L-cysteine and Amplex Red system was implemented as an a

43 In the presence of L-cysteine and ascorbic acid, this compound forms nitric

44 l-L-arginine or by the antioxidants N-acetyl-L-cysteine and ascorbic acid.

45 mine as well as sulfhydryl compounds such as l-cysteine and beta-mercaptoethanol as cosubstrates.

46 Pretreatment with N-acetyl-l-cysteine and catalase expression ameliorated cell deat

47 this, combinatorial treatment with N-acetyl-l-cysteine and catalase substantially inhibited the ROS

48 Similarly, combined N-acetyl-l-cysteine and catalase treatment also suppressed VDAC1-

49 te solution via reactive H2S generation from l-cysteine and controls nanocrystal growth within the qu

50 N-acetyl-L-cysteine and cyclosporine A, blocked ethanol and aceta

51 The thiol-containing molecules l-cysteine and d-cysteine both mimicked the protective e

52 unt of ferrous iron in the presence of IscS, L-cysteine and dithiothreitol, iron-sulfur clusters are

53 reduced glutathione, L-cysteine or N-acetyl-L-cysteine and fully reduced by dithiothreitol or the E.

54 attenuated by thiol-containing nucleophiles, l-cysteine and glutathione.

55 mmon antioxidants alpha-tocopherol, N-acetyl-l-cysteine and GSH, but not by the nonspecific caspase i

56 The levels of L-cysteine and homocysteine in rat brain mitochondria ar

57 Preincubation with S-allyl-l-cysteine and isoliquiritigenin increased MMP in both P

58 rivatives as well as the beta-elimination of l-cysteine and its analogs.

59 domain catalyzes C-S bond cleavage by using l-cysteine and l-cysteine S-modified analogs as substrat

60 can generate these metabolites from N-acetyl-l-cysteine and l-cysteine, respectively, and that the sy

61 l-Cysteine and l-Cystine were determined with detection

62 When H(2)S-releasing compounds L-cysteine and N-acetylcysteine were added to the cell c

63 Similarly, l-cysteine and N-ethylmaleimide significantly attenuated

64 articles protected by monolayers of N-acetyl-l-cysteine and of tiopronin ligands.

65 SE) produces H2S via enzymatic conversion of L-cysteine and plays a critical role in cardiovascular h

66 ipid Mixture 1, Gelatin Peptone N3, N-Acetyl-L-Cysteine and Pluronic F-68) were assayed in order to i

67 or was found to be ascorbic acid followed by L-cysteine and quercetin.

68 yl maleate) nor reducing compounds (N-acetyl-l-cysteine and reducing glutathione) could disrupt the c

69 h other in vitro substrates, such as S-ethyl-l-cysteine and S-(o-nitrophenyl)-l-cysteine.

70 Two main precursors (S-3-(hexan-1-ol)-l-cysteine and S-3-(hexan-1-ol)-l-glutathione) of 3-sulf

71 amino acid-based S-nitrosothiols (S-nitroso-L-cysteine and S-nitrosohomocysteine) occurs via amino a

72 n module, involving the aerobic oxidation of l-cysteine and the concomitant formation of the fluoresc

73 The ROS scavenger N-acetyl L-cysteine and the mitochondrial antioxidant Mito-TEMPO

74 ation is blocked by the antioxidant N-acetyl-L-cysteine and the NADPH oxidase inhibitor, DPI.

75 nhancer solution (FES) (d-glucose, d-ribose, l-cysteine and thiamin) and of sous-vide cooking or roas

76 cysteine (PPC) from (R)-phosphopantothenate, l-cysteine, and cytidine-5'-triphosphate (CTP) and has b

77 ubstrates, S-methyl-L-cysteine sulfoxide and L-cysteine, and had both cysteine sulfoxide lyase and cy

78 lease, further enhanced by the IAP inhibitor l-cysteine, and reduced by the exogenous ATPase apyrase.

79 bic acid, sodium diethyldithiocarbamic acid, L-cysteine, and sodium metabisulfite were effective inhi

80 the quantitative reaction of maleimide with l-cysteine, and the subsequent determination of the unre

81 Pretreatment with antioxidant N-acetyl-l-cysteine, apoptosis signal-regulating kinase 1 (ASK1)

82 CdTe nanoparticles stabilized by l-cysteine are chemically transformed into CdS nanoparti

83 In Turkey, usage of l-Cysteine as a food additive isn't allowed in wheat flo

84 Previously, we identified S-trityl-L-cysteine as a selective inhibitor of Eg5 and developed

85 dent beta-elimination reaction, establishing l-cysteine as the origin of sulfur at C-3 of LNM; and (i

86 including glycine, L-serine, L-alanine, and L-cysteine, as well as their D-enantiomers.

87 allic acid, glycine, reduced glutathione and l-cysteine at 0.1, 0.5, 1.0 and 2.0% levels to inhibit b

88 The electrochemical behavior of L-cysteine at the Au-SH-SiO(2)@Cu-MOF was investigated b

89 ddition, the free radical scavenger N-acetyl-L-cysteine attenuated ROS generation and apoptosis media

90 ants pyrrolidinedithiocarbamate and N-acetyl-l-cysteine attenuated this response, as well as the hypo

91 We report the development of a novel L-cysteine-based poly (disulfide amide) (Cys-PDSA) famil

92 Arg inhibition with S-(2-boronoethyl)-l-cysteine (BEC) augmented Ca(2+)-dependent NOS activity

93 nhibition of arginase with S-(2-boronoethyl)-l-cysteine (BEC) significantly enhanced NO generation in

94 cific arginase inhibitor (S)-(2-boronoethyl)-l-cysteine (BEC).

95 N-acetyl-L-cysteine blocked the EGFR activation and reduced the N

96 OS was prevented by the antioxidant N-acetyl-l-cysteine but not by the NADPH oxidase inhibitor diphen

97 emonstrated by sensitivity of acute 1-NCA to l-cysteine but not to hydroxocobalamin in the normal rat

98 transferred to IscS only in the presence of l-cysteine, but not to the C328S variant.

99 We show here that L-cysteine can be used to transform a highly reactive Si

100 ve composite material consisting of N-acetyl-L-cysteine capped CdAgTe quantum dots (NAC-CdAgTe QDs) a

101 ce sensor is developed to detect DA based on l-cysteine capped Mn doped ZnS quantum dots (l-cys ZnS:M

102 L-cysteine capped Mn doped ZnS quantum dots/ Idarubicin

103 by immobilizing the aptamer on water soluble l-cysteine capped ZnS quantum dots (QDs).

104 ated to the in situ synthesis of amino acid (L-Cysteine) capped lanthanum hydroxide nanoparticles [Cy

105 system using resonance energy transfer with l-cysteine-capped cadmium telluride quantum dots (CdTe-Q

106 The l-cysteine-capped CdTe-QDs were synthesized and added in

107 s achieved due to the energy transfer by the l-cysteine-capped CdTe-QDs.

108 addition, Na2S, the H2S donor GYY-4137, and l-cysteine caused relaxation of airways pre-contracted w

109 eine (3HPMA) and N-acetyl-S-(2-carboxyethyl)-L-cysteine (CEMA)--as biomarkers of exposure to acrolein

110 atalytic current increased linearly with the L-cysteine concentration in the range of 0.02-300 muM an

111 plying it to the analytical determination of L-cysteine concentration.

112 estational day 20, maternal plasma urate and L-cysteine concentrations, and placental levels of 4-hyd

113 of new biosensor for the electrooxidation of l-cysteine (CSH) in aqueous media.

114 address these questions, we herein show that L-cysteine (Cys) binds the auxiliary [4Fe-4S] cluster of

115 Gold disc electrodes functionalised with a l-Cysteine (Cys) self-assembled monolayer (SAM) were use

116 d HClO4 media, in the presence or absence of L-cysteine (Cys).

117 be used in the electrocatalytic oxidation of L-cysteine (Cys).

118 isted amino acids such as L-Lysine (lysine), L-Cysteine (cysteine) and L-Arginine (arginine) as bifun

119 Au nanoparticles modified with l-cysteine (d-cysteine) selectively adsorb the (R)-propy

120 The oxidation overpotentials of L-cysteine decreased significantly and their oxidation p

121 he arginase inhibitor BEC (S-(2-boronoethyl)-l-cysteine) decreased arginase activity and caused alter

122 E were markedly abolished by the antioxidant L-cysteine derivative N-acetylcysteine (NAC).

123 -MS coupled with o-phthaldialdehyde/N-acetyl-L-cysteine derivatization.

124 duced in the cytosol through the activity of l-cysteine desulfhydrase (DES1).

125 dopsis thaliana, DES1 is the only identified L-Cysteine desulfhydrase located in the cytosol, and it

126 The Escherichia coli L-cysteine desulfurase IscS mobilizes sulfur from L-cyst

127 studies showed that TUM1 interacts with the l-cysteine desulfurase NFS1 and the rhodanese-like prote

128 l-Cysteine desulfurases provide sulfur to several metabo

129 IscS is one of the three Escherichia coli l-cysteine desulfurases.

130 Gallic acid and l-cysteine did not exhibit browning inhibition effect at

131 Subsequent addition of L-cysteine efficiently mobilizes the iron center in IscA

132 Using S-nitroso-L-cysteine-ethyl ester (SNCEE) and S-nitrosoglutathione

133 By using S-nitroso-l-cysteine-ethyl-ester, an intracellular NO donor and in

134 the thiol-lacking molecule N-acetyl-S-methyl-l-cysteine failed to exert protection or blunt the rise

135 on paste electrode modified with ds-DNA/poly(L-cysteine)/Fe3O4 nanoparticles-graphene oxide (ds-DNA/p

136 he formation of the CS-conjugates S-furfuryl-l-cysteine (FFT-S-Cys) and S-(2-methyl-3-furyl)-l-cystei

137 Blocking arginases using S-(2-boronoethyl)-L-cysteine for 9 weeks in Ins2(Akita) mice or 6 weeks in

138 teine desulfurase IscS mobilizes sulfur from L-cysteine for the synthesis of several biomolecules suc

139 fhydrylase, which catalyzes the formation of l-cysteine from O-acetyl serine and hydrogen sulfide.

140 To break the stalemate, N-acetyl-L-cysteine functionalized chitosan copolymer (CS-NAC), w

141 to-4-methylpentan-2-one-N-(l-gamma-glutamyl)-l-cysteine (gammaGluCys-4MMP) but at too low concentrati

142 rsatile sensing method for analyzing thiols (L-cysteine, glutathione) using the H2O2-mediated DNAzyme

143 iberately added to various flour types since l-Cysteine has enabled favorable baking conditions such

144 ectrocatalytic activity for the oxidation of L-cysteine in 0.1 M phosphate buffer solution (pH 5.0).

145 rkedly increased in response to both NaHS or l-cysteine in platelets of healthy volunteers.

146 The L-cysteine in situ capped cadmium sulfide (CdS; size < 7

147 oylcysteine from (R)-phosphopantothenate and L-cysteine in the biosynthetic pathway leading to the fo

148 of the bidentate thiol/amine coordination of l-cysteine in the NO-bound CDO active site.

149 In the reaction of S-ethyl-l-cysteine in the presence of 4-hydroxypyridine, a subse

150 5-HITCA can be formed using available, free L-cysteine in these tissues.

151 Finally, the sensor was applied to determine L-cysteine in water and biological samples.

152 ective method was developed for detection of l-Cysteine in wheat flour using Raman microscopy.

153 Detection of l-Cysteine in wheat flour was accomplished successfully

154 urgent need for effective methods to detect l-Cysteine in wheat flour.

155 N-acetyl-L-cysteine inhibited BPEx-induced eosinophilic airway in

156 cluding cell permeable catalase and N-acetyl-L-cysteine, inhibited glucose-stimulated H2O2 accumulati

157 ures catalyze the oxidation of thiols (e.g., l-cysteine) into disulfides (e.g., cystine).

158 l-Cysteine is deliberately added to various flour types

159 l-Cysteine is effective in mediating release of free Fe(

160 Thus, l-cysteine is not removed from the system and the fluore

161 ence response of the dyes in the presence of L-cysteine is slow, but a ratiometric detection process

162 L-cysteine (L-cys) increases the amplitude of T-type Ca(

163 o the surface of GC/PPy-AuNPs-rGO film using L-cysteine (L-Cys) linker agent and trifluoromethanesulf

164 Under conditions of elevated ROS, endogenous L-cysteine (L-Cys) production is insufficient for GSH sy

165 celerated rate of reduction of aquometHbE by L-cysteine (L-Cys).

166 e, L-leucine, glycine, L-lysine, L-arginine, L-cysteine, L-alanine, and L-proline--in aqueous solutio

167 selenium: seleno-l-methionine, methyl-seleno-l-cysteine, l-selenocystine, methaneseleninic acid, sele

168 termediates in reactions of TPL with S-alkyl-l-cysteines, l-tyrosine, and 3-fluoro-l-tyrosine.

169 racellular thiol species (thioglycolic acid, l-cysteine-l-glycine, cysteine, and glutathione) were up

170 ls)) on monolayers of l-cysteine-l-tyrosine, l-cysteine-l-phenylalanine, or l-cysteine-l-phosphotyros

171 e-l-tyrosine, l-cysteine-l-phenylalanine, or l-cysteine-l-phosphotyrosine formed on crystallographica

172 nd fluorinated terphenyls)) on monolayers of l-cysteine-l-tyrosine, l-cysteine-l-phenylalanine, or l-

173 ar retention of NS mutants, whereas N-acetyl-L-cysteine largely prevented the effects of MG132.

174 l hypothesis that reduced levels of H(2)S or L-cysteine (LC) play a role in the impaired glucose meta

175 e oxygen species, up-regulation of glutamate-l-cysteine ligase activity, increased glutathione level,

176 omocysteine (100 to 500 micromol/L), but not l-cysteine, maintained the Ly-6C(hi) subset and induced

177 Interestingly, the antioxidant N-acetyl-L-cysteine markedly reversed these changes.

178 Additional studies indicate that L-cysteine may have an important role in repairing the N

179 c hemin/G-quadruplex chains that control the l-cysteine-mediated aggregation of Au NPs.

180 ose a model to explain the interplay between l-cysteine metabolism, H2S production, and oxidative str

181 l-5'-phosphate-dependent enzymes involved in L-cysteine metabolism: cystathionine-ss-synthase (CBS) a

182 ysteine (FFT-S-Cys) and S-(2-methyl-3-furyl)-l-cysteine (MFT-S-Cys) in the Maillard reaction of xylos

183 ulation of GSH in these cells using N-acetyl-L-cysteine mimicked the effect of ATRA on MDSC different

184 1.7 x 10(6) was estimated for a monolayer of L-cysteine molecules adsorbed to gold via a thiol linkag

185 owed by linking of monomeric (N-methacryloyl-l-cysteine) molecules through S-Au bonds.

186 by the living polymerization of glycosylated L-cysteine-N-carboxyanhydride (glyco-C NCA) monomers.

187 metabolized to N-acetyl-S-1,2-dichlorovinyl-L-cysteine (NA-DCVC) before being excreted in the urine.

188 Neutralization of ROS by N-acetyl-l-cysteine (NAC) abrogated the phosphorylation of JAK2 a

189 n that involves derivatization with N-acetyl-l-cysteine (NAC) and separation by HPLC was developed.

190 N-acetyl-l-cysteine (NAC) exhibits protective properties in brain

191 ROS scavengers edaravone (EDA) and N-acetyl-L-cysteine (NAC) reduced ROS level, downregulated JNK ac

192 -treated mothers, while antioxidant N-acetyl-L-cysteine (NAC) reversed these changes together with im

193 The antioxidant N-acetyl-l-cysteine (NAC) significantly reduces the cytotoxicity

194 In contrast, N-acetyl-L-cysteine (NAC) treatment in H9c2 cells, or overexpress

195 ure, whereas similar treatment with N-acetyl-L-cysteine (NAC) was less effective.

196 In murine embryonic fibroblasts, N-acetyl-L-cysteine (NAC), a GSH generating agent, enhances hypox

197 our reductants, namely, GSH, l-Cys, N-acetyl-l-cysteine (NAC), and ascorbic acid.

198 Thiol antioxidants, including N-acetyl-L-cysteine (NAC), are widely used as modulators of the i

199 tor LY294002, glutathione precursor N-acetyl-L-cysteine (NAC), curcumin, epigallocatechin-3 gallate (

200 eactions of these thiol esters with N-acetyl-l-cysteine (NAC), N-acetylcysteamine, and N(2)-acetyl-L-

201 ficacy of a weak organic acid drug, N-acetyl-L-cysteine (NAC), on the eradication of biofilms formed

202 itor of ROS production, antioxidant N-acetyl-L-cysteine (NAC), or an inhibitor of NO, 1,400W, prevent

203 d by increasing the thiol pool with N-acetyl L-cysteine (NAC), while NAC had little effect on BAA.

204 Thiol antioxidants (N-acetyl-l-cysteine [NAC] and N-acystelyn, carbocysteine, erdoste

205 CD) and oxidative stress inhibitor (N-acetyl-L-cysteine, NAC) slightly rescued the viability of cells

206 USP method to that of the standard N-acetyl-L-cysteine-NaOH (NALC) method for conventional diagnosis

207 sence of IscS and dithiothreitol but without L-cysteine, nearly all iron is bound to IscA.

208 The antioxidant N-acetyl-L-cysteine normalized ROS levels and restored ICaL densi

209 ving activation, methylation, and loading of l-cysteine onto the TioN partner thiolation domain, TioS

210 de of XO activity by pharmacologic (N-acetyl-L-cysteine or allopurinol) or molecular (by small interf

211 and injected with the antioxidants, N-acetyl-L-cysteine or dimethylthiourea, prior to sensitization,

212 d by monothiols such as reduced glutathione, L-cysteine or N-acetyl-L-cysteine and fully reduced by d

213 ed by general antioxidants, such as N-acetyl-l-cysteine or Tiron.

214 was attenuated by the antioxidant, N-acetyl-L-cysteine, or Cu sequestration.

215 rginine, the free radical scavenger N-acetyl-l-cysteine, or the NOS substrate l-arginine partially in

216 dase inhibitor DPI, the antioxidant N-acetyl-L-cysteine, or the superoxide scavenger Tiron, further i

217 l-Cysteine predominantly inhibits priming but not the ac

218 The antioxidant N-acetyl-l-cysteine prevented mitochondrial inhibitors from rescu

219 Pretreatment with the antioxidant N-acetyl-L-cysteine prevented ouabain-stimulated Na/K-ATPase.c-Sr

220 ocyanatostilbene-2,2'-disulfonate or N-acety-l-cysteine prevented the acid-induced increase in phosph

221 verloaded mice with the antioxidant N-acetyl-L-cysteine prevented the development of trabecular but n

222 ngers butylated hydroxyanisole, and N-acetyl-L-cysteine prevented the luteolin-induced suppression of

223 eriments, CDO exhibits an ordered binding of l-cysteine prior to NO (and presumably O2) similar to th

224 e, suggesting an obligate ordered binding of l-cysteine prior to NO.

225 nt inhibitor of the major secreted cathepsin L cysteine proteases of F. hepatica, FhCL1 and FhCL2, an

226 Pretreatment with the antioxidant N-acetyl-L-cysteine protected embryos with activated hypoxia sign

227 m and to obtain the stoichiometry factor for l-cysteine quantification.

228 centration range and drastically different d/l-cysteine ratios in simulated body fluids.

229 N-Acetyl-l-cysteine reduced oxidative stress, prevented CCTalpha

230 e reactive oxygen species scavenger N-acetyl-l-cysteine reduced the levels of interleukin-6, interleu

231 iated by peroxidase activity (oxidation) and l-cysteine (reduction).

232 -deficient T cells with metabolically active L-cysteine rescued mTOR activation and proliferation but

233 and show that the IgG2 C H1 and C-terminal C L cysteine residues are either linked to each other or t

234 oxidation of L-methionine, L-tryptophan, and L-cysteine residues in its active site.

235 ns in its peptidic backbone two S-methylated l-cysteine residues.

236 ese metabolites from N-acetyl-l-cysteine and l-cysteine, respectively, and that the synthesis of stea

237 microM for CTP, (R)-phosphopantothenate, and L-cysteine, respectively, and the k(cat) was 2.9 s(-1).

238 K inhibitor PD98059 and antioxidant N-acetyl-l-cysteine restored normal proliferation of Atm(-/-) ast

239 atment with the antioxidative agent N-acetyl-L-cysteine resulted in reversion of the FoxO-deficient H

240 3COOH or TRIS buffer after prereduction with l-cysteine resulted in the formation of only the corresp

241 gly, treatment with the antioxidant N-acetyl-l-cysteine reversed the phenotype, normalizing atheroscl

242 Also, the cell-permeable thiol N-acetyl l-cysteine, reverses DMF inhibition of the NFkappaB path

243 es C-S bond cleavage by using l-cysteine and l-cysteine S-modified analogs as substrates through a PL

244 igated the neuroprotective effect of S-allyl L-cysteine (SAC), a water soluble compound from garlic,

245 Au nanoparticles modified with either d- or l-cysteine selectively adsorb one enantiomer of propylen

246 ding endogenous sulfur-containing amino acid l-cysteine, selectively enhance native T-type currents i

247 Among the chemical inhibitors tested, l-cysteine showed the best inhibitory effect, with an IC

248 addition of a reducing agent DTT or N-acetyl-L-cysteine, showing that process of oxidation is require

249 tem is dietary supplementation with S-methyl-L-cysteine (SMLC), found abundantly in garlic, cabbage,

250 luded chemical decontamination with N-acetyl-l-cysteine-sodium hydroxide (NALC-NaOH), alone and in co

251 ork, water-dispersible N-acetylcysteine- and l-cysteine-stabilized palladium nanoparticles are introd

252 For example, S-trityl-l-cysteine (STC) and monastrol are HsEg5 inhibitors that

253 As l-cysteine stimulates the aggregation of Au nanoparticle

254 We previously identified S-trityl-l-cysteine (STLC) and related analogues as selective pot

255 We previously identified S-trityl-L-cysteine (STLC) as a potent allosteric inhibitor of Eg

256 ed by deletion of L5 or addition of S-trityl-l-cysteine (STLC), an allosteric inhibitor that binds to

257 ated with the kinesin Eg5 inhibitor S-Trityl-l-cysteine (STLC).

258 SufE enhances the initial l-cysteine substrate binding to SufS and formation of th

259 t by growth with exogenous dithiothreitol or L-cysteine, suggesting that in the absence of CccA, apoc

260 potently inhibited by nickel and enhanced by L-cysteine, suggesting that the Ca(V)3.2 T-type channel

261 ding kinetics of two alternative substrates: L-cysteine sulfinic acid and d-aspartate.

262 L-cysteine sulfinic acid bound with higher affinity than

263 lliinase and its natural substrate, S-benzyl-l-cysteine sulfoxide (petiveriin).

264 ed to bind two kinds of substrates, S-methyl-L-cysteine sulfoxide and L-cysteine, and had both cystei

265 lly occurring P. alliacea compounds S-benzyl-l-cysteine sulfoxide and S-2-hydroxyethyl-l-cysteine sul

266 n energy with its natural substrate S-benzyl-l-cysteine sulfoxide is 64.6 kJ mol(-1).

267 yl-l-cysteine sulfoxide and S-2-hydroxyethyl-l-cysteine sulfoxide.

268 Our results show that l-cysteine supplementation is a potential treatment for

269 PYK2 downregulation, l-cysteine supplementation, or CSE overexpression allevi

270 Pretreatment with the ROS scavenger N-acetyl-L-cysteine, the ERK1/2 inhibitor UO126, or ERK1/2 siRNA

271 N-acetyl-L-cysteine therapy has been used in clinical studies; ho

272 , we explore the effect of low dose N-acetyl-L-cysteine therapy, delivered using a targeted, systemic

273 nd either NO or a suitable reductant such as L-cysteine, these ferric heme sites can function as a ge

274 In the presence of L-cysteine, this compound rapidly yields nitrite and rea

275 dioxygenase (CDO) catalyzes the oxidation of l-cysteine to cysteine sulfinic acid.

276 d does not catalyze the aerobic oxidation of l-cysteine to cystine.

277 e ATP-dependent condensation of GlcN-Ins and l-cysteine to form l-Cys-GlcN-Ins, the penultimate step

278 A [4Fe-4S] enzyme that decomposes L-cysteine to hydrogen sulfide, ammonia, and pyruvate ha

279 OM-sulfide solutions amended with sufficient l-cysteine to prevent beta-HgS(s) formation.

280 ta'-position (2b, 2c) have been treated with L-cysteine to provide thiazolidine derivatives.

281 nanoparticles have been modified with d- or l-cysteine to render them chiral and enantioselective fo

282 , administration of the antioxidant N-acetyl-l-cysteine to Ucp2(-/-) pregnant mice alleviated the eff

283 ecting two different analytes: d-glucose and l-cysteine under nonspecific and specific adsorption con

284 The loading capacity determined for l-cysteine using frontal elution is 2.58 mumol/m.

285 ed glycosides of D-galactose or D-glucose to L-cysteine using thiol-ene "click" chemistry, followed b

286 rotects E. coli against oxidative stress via l-cysteine utilization and H2S-mediated sequestration of

287 azole functionality from an acetyl group and l-cysteine via condensation, cyclization, and dehydratio

288 quitous iron-sulfur clusters is derived from L-cysteine via cysteine desulfurases.

289 Mass balances and quantification of l-cysteine via its sulfur concentration using elemental

290 l-Cysteine was the most effective inhibitor.

291 of the PPCS-catalyzed reaction with CTP and L-cysteine, was monitored by (31)P NMR spectroscopy.

292 on systems, the thiol-containing amino acid, L-Cysteine, was self-assembled onto smooth gold electrod

293 x between the enzyme-persulfide and a second l-cysteine, which adds to the cofactor via its sulfhydry

294 -acetamido-2-deoxy-alpha-D-galactopyranosyl)-L-cysteine, which could be considered as a mimic of Tn a

295 Supplementation with l-cysteine, which is required for optimal TRMU function,

296 fE work together to mobilize persulfide from L-cysteine, which is then donated to the SufB Fe-S clust

297 l-Cysteine, which was previously reported to accelerate

298 The reactions of L-serine and L-cysteine with CBS resulted in the formation of a commo

299 Substitution of l-cysteine with isosteric substrate analogues cysteamine

300 The reaction of l-cysteine with the Ellman's reagent was monitored photo