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1 evidence of splice motifs in the sequence of L form.
2  to switch into a wall-free state called the L-form.
3 ific MSC inhibitor GsMTx4, in both the D and L forms.
4 or studying the genetics and cell biology of L-forms.
5 proliferation of wall-less bacteria known as L-forms.
6 the pentoses are better competitors than the l-forms.
7 and the potential threat posed by pathogenic L-forms.
8 iral life emerged using amino acids in their l-form?
9 nto a cell-wall-deficient state, called the "L-form" [1-3].
10                                        RNase L forms a crossed homodimer stabilized by ankyrin (ANK)
11 et cells are absent, the other subunit (CylL(L)'') forms a complex with CylL(S)'', blocking it from a
12  and gp120 than their respective native, all-l forms, although activity differences were modest, part
13  mutant strains indicated that exsD and pscB-L form an operon.
14                                       In the L-form analogues, the L-Tyr analogue has the highest ove
15 an MAIL protein detected was the 80 kDa MAIL-L form and human MAIL showed nuclear localization.
16 ere sufficient to support the import of both L-form and D-form conjugates in permeabilized cells.
17  investigated the functional significance of L-form and S-form C-CAM in rat prostate by examining the
18  (Phaseolus vulgaris agglutinin; PHA; E- and L-forms) and snowdrop lectin (Galanthus nivalis agglutin
19  (2) RPA showed the 3' termini of both S and L forms, and there are putative polyadenylation signals
20 i to generate cell-wall-deficient, spherical L-forms, and found that they robustly reverted to a rod-
21 known for decades, the general properties of L-forms are poorly understood, largely due to the lack o
22                                              L-forms are variants of common bacteria that can grow an
23                                              L-forms are wall-deficient variants of common bacteria t
24 leucine, whereas their stereoisomers (d- and l-forms) are predicted to be significantly more challeng
25 dorferi, known variably as "cyst forms" and "L-forms," are responsible for the pathogenesis of chroni
26 ely competed only by cationic amino acids in L-form: arginine, lysine, ornithine, and 2,4-diamino-n-b
27  L', the all-trans photoproduct of wild-type L formed at 80 K.
28 es, in the life cycle of cell wall-deficient L-form bacteria.
29                                              L-forms become completely independent of the normally es
30                Subsequent reaction in CCl3Br(l) formed Br-terminated Ge(111), as shown by the disappe
31 the two isoforms, namely short (S) and long (L) forms by northern blot and RNAse protection assay (RP
32 e selectively engineered toward either d- or l-forms by changing the gelation conditions.
33 cent discovery that cell-wall-less bacteria (L-forms) can grow and divide independently of FtsZ(4,5),
34 cases, there were 82 S-form cases (37%), 121 L-form cases (55%), and 18 V-form cases (8%).
35 so, an increased percentage of S-form versus L-form cases had the M3 variant phenotype, 24% v 12% (P
36 There were no differences between S-form and L-form cases in either CR rate (79% v 69%; P = .14) or d
37 ng the relative V(max)/K(m) values of D- and L-form dideoxy-CMP, we showed that this kinase lacked st
38 s membrane synthesis are sufficient to drive L-form division in Bacillus subtilis.
39                  We show that propagation of L-forms does not require the normal FtsZ-dependent divis
40 ified an unexpected 'escape' step needed for L-form emergence from the rod.
41 with the 'R' form favoring S strand and the 'L' form favoring AS strand silencing.
42 tions act by facilitating the release of the L-form from its walled parent cell but that they act in
43 ecursor synthesis promotes the generation of L-forms from both Gram-positive and Gram-negative bacter
44                             We show that the L-forms generated have in common a mechanism of prolifer
45 ic culture conditions also worked to promote L-form growth without the class 2 mutations in both Gram
46 ving two stereoisomers d and l, but only the l form has a biological positive effect, thus chiral rec
47 veloped a controllable system for generating L-forms in the highly tractable model bacterium Bacillus
48 ly due to the lack of systematic analysis of L-forms in the molecular biology era.
49                 Our results suggest that the L-form mode of proliferation is conserved across the bac
50              This indicates that both D- and L-form NLS peptides use the same import machinery.
51                                     The D or L form of 2-hydroxyglutarate (2HG) accumulates in certai
52                                          The L form of HBsAg was both necessary and sufficient for vi
53  The transcriptional activities of the J and L forms of PTF1 are independent of Notch signaling, beca
54                Chemically synthesized D- and L- forms of the protein ligand showed reciprocal chiral
55 e all possible dipeptide combinations of the L-form of alanine, valine, and leucine and the achiral a
56 e all possible dipeptide combinations of the L-form of alanine, valine, leucine, and the achiral amin
57 ouble mutant R104M-D133A in complex with the L-form of thymidine supplies a structural explanation fo
58 riers, exhibiting greater preference for the l-forms of arginine, lysine and ornithine.
59               In order to measure the d- and l-forms of aspartate and glutamate, we developed and app
60 yme that catalyzes the degradation of d- and l-forms of DAP to pyruvate and ammonia.
61 motif [CS][CS]-x(0,2)-G-x(1)-C-x(2,3)-S-x(3)-L formed part of an endosome targeting signal for GPI-PL
62                             In addition, the LES formed pathogenic partnerships with AGS in the G. me
63 Bcl-x(L) and DeltaN76Bcl-x(L), but not Bcl-x(L), formed pores large enough to release cytochrome c an
64                         Moreover, the D- and L-forms preferentially bound spleen-derived T-cells over
65 ell wall synthesis is blocked is crucial for L-form proliferation in a wide range of bacteria and als
66                                     Instead, L-form proliferation is driven by a simple biophysical p
67                                              L-form proliferation is remarkable in being independent
68 m Bacillus subtilis and thus how to generate L-forms reliably and reproducibly.
69 uantitatively convert from the walled to the L-form state.
70 ave the ability to switch to a wall-free or 'L-form' state.
71                                              L-form strains are wall-deficient derivatives of common
72                                C-CAM1 (long (L)-form) strongly suppresses the tumorigenicity of human
73                                Using the new L-form system, we show here that we can delete essential
74 because of the higher solubility of the pure L form than of the more stable DL racemic compound cryst
75  only two genetic changes are needed for the L-form transition in Bacillus subtilis [7].
76 ecently determined the genetic basis for the L-form transition in the rod-shaped bacterium Bacillus s
77 e promoters, 11 common SNPs are in extensive LD, forming two 48-kb haplotypes of equal frequency, in
78 m type, PML exon 3 RAR alpha exon 3; a long (L)-form type, PML exon 6 RAR alpha exon 3; or a variable
79 but that the identification of the S-form or L-form type of PML-RAR alpha mRNA, per se, does not pred
80  therapy, the S-form type, compared with the L-form type, was associated with higher values for the w
81 with the second residue (W) in the D- versus L-form was 90 +/- 10%.
82 AEFLa in Aplysia was detected only in an all L-form, we found that both GFFD and GdFFD were present i
83                           The D-form and the L-form were equally effective.
84 re proteolytically resistant in cytosol, the L-forms were rapidly degraded.

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