ライフサイエンスコーパス: LDH

1 LDH and IL-18 concentrations were associated with PNF (o

2 LDH and MTT assays demonstrate that peptide 1a is toxic

3 LDH can be chemically immobilized on those surfaces afte

4 LDH clearly colocalized with mitochondria in intact, as

5 LDH is likely localized inside the outer mitochondrial m

6 LDH release temporally correlated with arachidonic acid

7 LDH, which is a non-limiting enzyme of glycolysis in dif

8 LDHs have found widespread application as catalysts, ani

9 cess, human lactate dehydrogenase isozyme 1 (LDH-1) microcrystals were separately dissolved and subse

10 oma, comprising: age >/= 60 years, PS >/= 2, LDH > normal, and bone marrow involvement; and the alter

11 Lactate dehydrogenase A (LDH-A) catalyzes the interconversion of lactate and pyru

12 acrophage-expressed lactate dehydrogenase-A (LDH-A) to tumor formation in a K-Ras murine model of lun

13 ctate by the enzyme lactate dehydrogenase-A (LDH-A), suggesting a possible vulnerability at this targ

14 ls of hepatobiliary injury markers AST, ALT, LDH, and gamma-GT after 6 hours of NMP.

15 PP) was significantly enhanced by adding AMO-LDH-OCNT hybrids.

16 egree of dispersion (mixability) between AMO-LDH and OCNT has a significant effect on the flame retar

17 3)0.5.yH2O LDH-oxidized carbon nanotube (AMO-LDH-OCNT) hybrids are shown to perform better than the e

18 Further, a system with 10 wt% of AMO-LDH and 1 wt% OCNT showed a peak heat release rate (PHRR

19 In addition, the incorporation of AMO-LDH-OCNT hybrids led to better mechanical properties, su

20 ), than those composites containing only AMO-LDH (25.6 MPa and 7.5%, respectively).

21 perform better than the equivalent pure AMO-LDH.

22 A synergistic effect between the AMO-LDH and OCNT was observed; this endows the hybrid materi

23 For PP mixed with AMO-LDH-OCNT hybrids to produce a composite with 10 wt% LDH

24 r than the PHRR reduction with PP/20 wt% AMO-LDH (31%).

25 cible organic layered double hydroxides (AMO-LDHs) can act as organophilic inorganic flame retardant

26 controlled their fate through the AMPKalpha1/LDH pathway, emphasizing the importance of metabolism in

27 dian age, 15.7 years (range, 12.5-19.7); and LDH < 2 versus >/= 2 x the upper limit of normal, 23:19.

28 urprisingly, release of arachidonic acid and LDH from cPLA2alpha-deficient fibroblasts was inhibited

29 ts with CTCs >/= 5 cells/7.5 mL of blood and LDH > 250 U/L (high risk) at 12 weeks was 46% and 2%, re

30 ISS, CA, and LDH data were simultaneously available in 3,060 of 4,445

31 eriment, the immobilized protein content and LDH activity on each modified surface was used as an ind

32 ny corelation of the Wells rule, D-dimer and LDH values with computerized tomography pulmonary angiog

33 l only occurs for some genes (e.g. GAPDH and LDH) but not others (e.g. Hsp90 and cyclophilin A).

34 TSLP, IFN-lambda and LDH were not increased by allergen or dsRNA challenges a

35 ervous system involvement, leukocytosis, and LDH >3 times the upper limit of normal were associated w

36 Malate and lactate dehydrogenases (MDH and LDH) are homologous, core metabolic enzymes that share a

37 at proliferating cells rely on both MDH1 and LDH to replenish cytosolic NAD, and that therapies desig

38 MTT and LDH assays confirmed cytotoxicity of perfluorooctanoic a

39 0.25 mg/ml extract as measured with MTT and LDH-leakage assays.

40 A biomarker panel containing CTC number and LDH level was shown to be a surrogate for survival at th

41 crease in bronchoalveolar lavage protein and LDH, respectively.

42 Both cellular viability and LDH level remained unchanged with up to 100 microg/mL of

43 In canonical MDHs and LDHs, a single residue in the active-site loop governs s

44 The composite AO/LDH reaches the highest fluorescence intensity when the

45 nce intensity was achieved with a pulp to AO/LDH ratio of 1:5 which can be used to detect Hg(2+) in w

46 During the evolution of the apicomplexan LDH, however, specificity switched via an insertion that

47 ind that specificity evolved in apicomplexan LDHs by classic neofunctionalization characterized by lo

48 Lactate dehydrogenase assay (LDH), electronic microscopy analysis, collagen I product

49 Targeting HER2 or Src attenuated LDH activity as well as invasive potential in head and n

50 is review we summarize the current available LDH exfoliation methods.

51 combination in patients with normal baseline LDH.

52 surfaces modified with APTES-GA gave better LDH immobilizing efficiency than APTES, especially the S

53 rrelation test showed no correlation between LDH and D-dimer levels.

54 hnology was validated using five biomarkers; LDH, CRP, PSA, MMP-7, and C3a.

55 dicating that metabolism of pyruvate by both LDH and pyruvate dehydrogenase is subject to the acute e

56 Cell death was assayed by LDH and MTT methods.

57 n Caco-2 and macrophage cells, as assayed by LDH release, and escape by filamentation.

58 damage or major inflammation as assessed by LDH release or IL-8 secretion, respectively, compared wi

59 plex interplay of anion uptake mechanisms by LDH phases, by using changes in Mo geometry as powerful

60 Thrombosis was presaged by LDH levels that more than doubled, from 540 IU per liter

61 Cell death was proven by LDH assay and cell viability by IL-8 ELISA.

62 over, an anion exchange process on both CaAl LDHs was followed by in situ time-resolved synchrotron-b

63 bdate (MoO4(2-)) sorption mechanisms on CaAl LDHs with increasing loadings of molybdate.

64 aluminum layered double hydroxide chloride (LDH), is synthesized and characterized with X-ray powder

65 tallic Zn-Co layered double hydroxide (Zn-Co-LDH) can serve as an efficient electrocatalyst and catal

66 can be directly synthesized on the ZnAl-CO3 LDH buffer layer-modified substrates, owing to the speci

67 metal-imidazole interaction between ZnAl-CO3 LDHs and ZIF-8.

68 Compared to pristine CoFe LDHs, the as-exfoliated ultrathin CoFe LDHs nanosheets e

69 CoFe LDHs, the as-exfoliated ultrathin CoFe LDHs nanosheets exhibit excellent catalytic activity for

70 rst time, this study prepares ultrathin CoFe LDHs nanosheets with multivacancies as OER electrocataly

71 flow cytometry method (FCM), a colorimetric LDH-based ELISA : DELI), and standard microscopic slide

72 alt-manganese layered double hydroxide (CoMn LDH) are a highly active and stable oxygen evolution cat

73 as achieved in 4T1 breast cancer with (64)Cu-LDH-BSA via passive targeting alone (7.7 +/- 0.1%ID/g at

74 ide (LDH) photocatalysts, in particular CuCr-LDH nanosheets, possess remarkable photocatalytic activi

75 d-Lactate dehydrogenase (d-LDH) is encoded by a single gene in Arabidopsis (Arabido

76 In addition, overexpression of d-LDH and CYTc increased tolerance to d-lactate and MGO To

77 icture of the organization and function of d-LDH in the plant cell and exemplify how the plant mitoch

78 and MGO Together with fine-localization of d-LDH, the functional interaction with CYTc in vivo strong

79 c acts as the in vivo electron acceptor of d-LDH.

80 rometry allowed us to definitely show that d-LDH acts specifically on d-lactate, is active as a dimer

81 c loss-of-function mutants, as well as the d-LDH mutants, were more sensitive to d-lactate and MGO, i

82 cent mice in hyperoxia by 24 h and decreased LDH release and lung cell apoptosis after 72 h of exposu

83 ratio following PI103 treatment or decreased LDH activity and unchanged NAD+/NADH ratio following sta

84 lar protein (48%) and lactate dehydrogenase (LDH) (68%) following 72 hours of hyperoxia.

85 cases, and unchanged lactate dehydrogenase (LDH) activity and increased NAD+/NADH ratio following PI

86 Via immobilization of lactate dehydrogenase (LDH) as a model dehydrogenase enzyme onto the Fe3O4/r-GO

87 We identified lactate dehydrogenase (LDH) as a new functional target of AMPKalpha1.

88 viability, autophagy, lactate dehydrogenase (LDH) assay, and mammalian target of rapamycin (mTOR) pat

89 ves the expression of lactate dehydrogenase (LDH) B in an estrogen-related receptor-alpha-dependent m

90 Patients with normal lactate dehydrogenase (LDH) concentration and fewer than three organ sites cont

91 We measured lactate dehydrogenase (LDH) concentration as a marker of cellular cytotoxicity,

92 The release of lactate dehydrogenase (LDH) during brain death was reduced in the NOD group.

93 osed that human heart lactate dehydrogenase (LDH) employs protein promoting vibrations (PPVs) on the

94 prognostic variables: lactate dehydrogenase (LDH) higher than than normal, International Staging Syst

95 ON) were modified for lactate dehydrogenase (LDH) immobilization using (3-aminopropyl)triethoxysilane

96 We demonstrate that lactate dehydrogenase (LDH) isoform 5 secreted by glioblastoma cells induces NK

97 n, FBSH-III inhibited lactate dehydrogenase (LDH) leakage and intracellular reactive species (ROS) pr

98 y: cell viability and lactate dehydrogenase (LDH) leakage.

99 l using CTC count and lactate dehydrogenase (LDH) level was shown to satisfy the four Prentice criter

100 mer level or elevated lactate dehydrogenase (LDH) level were suspected of embolism and underwent tomo

101 ansaminase (SGPT) and lactate dehydrogenase (LDH) levels along with reduction in liver superoxide dis

102 rombosis and elevated lactate dehydrogenase (LDH) levels, LDH levels presaging thrombosis (and associ

103 ubclass, and baseline lactate dehydrogenase (LDH) levels.

104 ized by mitochondrial lactate dehydrogenase (LDH) of the same cell.

105 bromide (MTT) assay, lactate dehydrogenase (LDH) release assay, Hoechst 33342 staining, annexin V/PI

106 induce a significant lactate dehydrogenase (LDH) release from Calu-3 cells after a 20 h exposure.

107 r, and 8i in lowering lactate dehydrogenase (LDH) release induced by ischemia-like conditions in rat

108 h was quantified with lactate dehydrogenase (LDH) release measurements and Nissl-stained neuron count

109 ore (MPTP) formation, lactate dehydrogenase (LDH) release, and necrotic cell death that were blocked

110 were evaluated using lactate dehydrogenase (LDH) release, the fluorescent probe DCF, and Bax express

111 ition of hypothalamic lactate dehydrogenase (LDH) suggesting that metabolic flux through LDH was requ

112 urification and serum lactate dehydrogenase (LDH) to evaluate their prognostic value in newly diagnos

113 ide (RGO-AuNPs) and l-lactate dehydrogenase (LDH) was developed for the sensing of l-lactate.

114 Serum containing lactate dehydrogenase (LDH) was directly spotted on to the pullulan-coated bioa

115 (LR range, 7.1-250), lactate dehydrogenase (LDH) was greater than 200 U/L (LR, 18; 95% CI, 6.8-46),

116 roglobulin, 5.7 mg/L; lactate dehydrogenase (LDH), 397 IU/L; and normal liver function tests.

117 he up-regulation of L-lactate dehydrogenase (LDH), an intracellular enzyme present in most of all bod

118 ility assays, MTT and lactate dehydrogenase (LDH), and an assay measuring cytochrome P4501A (CYP1A) e

119 erential cell counts, lactate dehydrogenase (LDH), and protein.

120 ed for total protein, lactate dehydrogenase (LDH), CXCL1/KC, CCL2/MCP-1 and differential cell counts.

121 thione S-transferase, lactate dehydrogenase (LDH), heart-type fatty acid binding protein, redox-activ

122 Five predictors (age, lactate dehydrogenase (LDH), sites of involvement, Ann Arbor stage, ECOG perfor

123 significant change in lactate dehydrogenase (LDH), total protein, and total cell counts in the BAL, a

124 estigated the role of lactate dehydrogenase (LDH), which converts pyruvate to lactate and is an essen

125 al glycolytic enzyme, lactate dehydrogenase (LDH).

126 d inhibitors of human lactate dehydrogenase (LDH).

127 ges III to IV disease, lactic dehydrogenase (LDH) > normal, extranodal sites (ENSs) > one, and perfor

128 biomarkers (S100 and lactate dehydrogenase [LDH]) perform poorly in patients with uveal melanoma, an

129 and substrate-level lactate dehydrogenases (LDHs) from the obligate human pathogen Neisseria gonorrh

130 ic properties, the l-lactate dehydrogenases (LDHs) in lactic acid bacteria (LAB) display differences

131 Herein, we demonstrate LDH detection using porous silicon (pSi) microcavities a

132 NiFe-LDH due to (i) amorphous and distorted LDH structure, (ii) enhanced active surface area, and (i

133 The occurrence of elevated LDH levels within 3 months after implantation mirrored t

134 ms in the patient group in terms of elevated LDH or/and D-dimer levels (P=0.263 and P=1.000, respecti

135 ignificant reduction of serum liver enzymes (LDH (47,147 +/- 12,726 IU/l vs. 15,822 +/- 10,629 IU/l,

136 work provides a novel strategy to exfoliate LDHs and to produce multivacancies simultaneously as hig

137 The ability to exfoliate LDHs into ultrathin nanosheets enables a range of new op

138 In functional experiments, LDH overexpression phenocopied AMPKalpha1(-/-) phenotype

139 cious pCNT@Fe@GL/CNF ORR electrode and Ni-Fe LDH/NiF oxygen evolution reaction (OER) electrode exhibi

140 On the other hand, the spent Cu/Fe-LDH could be employed to produce porous carbon materials

141 spent Cu/Fe layered double hydroxide (Cu/Fe-LDH) which is generated from the adsorption of dyes by c

142 luid LDH to serum LDH >0.6, or pleural fluid LDH >two-thirds the upper limit of normal for serum LDH)

143 ht's criteria, cholesterol and pleural fluid LDH levels, and the pleural fluid cholesterol-to-serum r

144 serum protein >0.5, a ratio of pleural fluid LDH to serum LDH >0.6, or pleural fluid LDH >two-thirds

145 pacity (+25% for cell viability and +30% for LDH leakage) were observed in grape juices following PEF

146 an 0.7, these two high-throughput assays for LDH are both label free and complementary to each other

147 e metals solution herein could be reused for LDH synthesis.

148 The biosensor was selective for LDH and did not produce a signal upon incubation with an

149 NK cells from LDH-A-depleted tumors had improved cytolytic function.

150 of two enzyme pathways (G6pDH-MDH and G6pDH-LDH) through the control of NAD(+) substrate channeling

151 with mRNA expression of HIF-mediated genes, LDH and VEGF.

152 Heavy LDH has slowed chemistry in single turnover experiments,

153 prepared the native (light) LDH and a heavy LDH labeled with (13)C, (15)N, and nonexchangeable (2)H

154 ier-crossing probability is reduced in heavy LDH, the concerted mechanism of the hydride-proton trans

155 and proton transfer for both light and heavy LDHs.

156 pattern maintained with both light and heavy LDHs.

157 hogenesis of pain in lumbar disc herniation (LDH) remains poorly understood.

158 n level > 5.5 mg/L) and high-risk CA or high LDH level; and R-ISS II (n = 1,894), including all the o

159 /or del(17p) in addition to ISS3 and/or high LDH, comprised 5% (20 of 387 patients) to 8% (94 of 1,13

160 dition, the SiO2 surface offered the highest LDH immobilization among tested surfaces, with both APTE

161 Hydrotalcite (LDH) is one of the most excellent carrier materials.

162 symmetric ZnAl-CO3 layered double hydroxide (LDH) buffer layers with various stable equilibrium morph

163 toxic, degradable, layered double hydroxide (LDH) clay nanosheets.

164 Layered double hydroxide (LDH) nanomaterial has emerged as a novel delivery agent

165 ary Fe(II)-Al(III)-layered double hydroxide (LDH) phases during reaction with the Al-oxide sorbent, w

166 5.5, whereas Zn-Al layered double hydroxide (LDH) precipitates formed at pH 8.0.

167 Layered double hydroxide (LDH)-based nanomaterials are considered as promising ele

168 tes that ultrathin layered-double-hydroxide (LDH) photocatalysts, in particular CuCr-LDH nanosheets,

169 ized in Mg2Al-NO3 Layered Double Hydroxides (LDH) and the electrochemical detection was achieved with

170 cally homogeneous layered double hydroxides (LDHs) can elicit diverse human dendritic cell responses

171 Layered double hydroxides (LDHs) have been considered as effective phases for the r

172 wastewater using layered double hydroxides (LDHs) through their formation is presented.

173 Layered double hydroxides (LDHs) with their highly flexible and tunable chemical co

174 irpin RNA-mediated knockdown of hypothalamic LDH-A, an astrocytic component of the ANLS, also blunted

175 with the Al supply needed for Fe(II)-Al(III)-LDH precipitation, possibly combined with enhanced surfa

176 (V) hindered the formation of Fe(II)-Al(III)-LDH, slowing down precipitation at low As(V) concentrati

177 including lactate sensors using immobilized LDH on the ISFET surface.

178 ficantly attenuated pain hypersensitivity in LDH rats.

179 te specificity: Arg102 in MDHs and Gln102 in LDHs.

180 5% CI 1.32-5.17, p = 0.006), while increased LDH portended inferior OS (HR 4.16, 95% CI 1.29-13.46, p

181 (140%, p = 0.05), associated with increased LDH activity and total cellular NAD(H).

182 Patients with increased LDH tend to relapse earlier, and individuals in remissio

183 hemolysis, thrombophilia, and inflammation (LDH, bilirubin, D-dimer, C-reactive protein [CRP]) impro

184 sed cell viability, aggravated intracellular LDH release, intracellular Ca(2+), ROS levels, apoptosis

185 etravalent cation (89)Zr(4+) could not label LDH since it does not fit into the LDH crystal structure

186 ation (44)Sc(3+) were found to readily label LDH nanoparticles with excellent labeling efficiency and

187 nts for the direct synthesis of single-layer LDH nanosheets, as well as the emerging applications of

188 Furthermore, RV-B released less LDH than RV-A or RV-C, and induced lower levels of cytok

189 elevated lactate dehydrogenase (LDH) levels, LDH levels presaging thrombosis (and associated hemolysi

190 We prepared the native (light) LDH and a heavy LDH labeled with (13)C, (15)N, and nonex

191 irmed and suspected thrombosis, longitudinal LDH levels, and outcomes after pump thrombosis.

192 Significantly lower LDH levels (p < 0.05) were observed as were lower DCF fl

193 decrease in lactate dehydrogenase isoform M (LDH-M) activity and an increase in cellular protection a

194 ormation by a direct effect on mitochondria, LDH and arachidonic acid release were blocked by CsA and

195 grees C, the distribution of single-molecule LDH activities from solutions of individual crystals bro

196 localization of ssDNA-FITC suggest that nano-LDHs have potential application as a novel gene carrier

197 layered double hydroxide lactate nanosheets (LDH-lactate-NS) with a 0.52 nm thickness and 3060 nm dia

198 r I(FITC) and DNA molecules, forming neutral LDH-nanosheet conjugates.

199 ckel-aluminum double layered hydroxide (NiAl-LDH) nanoplates on carbon nanotubes (CNTs) network.

200 t results in fully integrated amorphous NiFe-LDH/C nanohybrid, allowing the harness of the high intri

201 The confined growth of both NiFe-LDH and carbon in one single sheet results in fully inte

202 The crystalline NiFe-LDH phase in nanoplate form is found to be highly active

203 For NiFe-LDH grown on a network of CNTs, the resulting NiFe-LDH/C

204 morphous NiFe-layered double hydroxide (NiFe-LDH) (<5 nm) and nanocarbon using the molecular precurso

205 n nickel-iron layered double hydroxide (NiFe-LDH) nanoplates on mildly oxidized multiwalled carbon na

206 rness of the high intrinsic activity of NiFe-LDH due to (i) amorphous and distorted LDH structure, (i

207 During the solvothermal synthesis of NiFe-LDH, the organic ligand decomposes and transforms to amo

208 As such, the resultant NiFe-LDH/C exhibits superior activity and stability.

209 own on a network of CNTs, the resulting NiFe-LDH/CNT complex exhibits higher electrocatalytic activit

210 and/or t(4;14) and/or t(14;16)], and normal LDH level (less than the upper limit of normal range); R

211 ine the categorization of age and normalized LDH.

212 We also found that the absence of LDH renders the pneumococci avirulent after intravenous

213 observed, indicating that in the absence of LDH the redox balance is maintained through alcohol dehy

214 rom GBM patients contain elevated amounts of LDH, which correlate with expression of NKG2D ligands on

215 ets, as well as the emerging applications of LDH nanosheets in catalyzing oxygen evolution reactions

216 We provide evidence for the degradation of LDH, dsRNA uptake in plant cells and silencing of homolo

217 Myeloid-specific deletion of LDH-A promoted accumulation of macrophages with a CD86(h

218 r, the fluorescence signal upon detection of LDH was amplified by 10 and 5-fold compared to that of a

219 d expression of NaV1.7 and NaV1.8 in DRGs of LDH rats.

220 tantly, PGC-1alpha reduces the expression of LDH A and one of its regulators, the transcription facto

221 Our results suggest that expressions of LDH-A and lactate by macrophage in the tumor microenviro

222 c self-assembly process for the formation of LDH/C composite, this method offers one new opportunity

223 Herein we identified unspliced forms of LDH and ENO transcripts produced during transition betwe

224 Inhibition of LDH activity by small hairpin ribonucleic acid or expres

225 ir return to quiescence, while inhibition of LDH activity rescued AMPKalpha1(-/-) MuSC self-renewal.

226 otent enzymatic and cell-based inhibition of LDH enzymatic activity.

227 we used FX11, a small-molecule inhibitor of LDH-A, to investigate this possible vulnerability in a p

228 tudy is to explore chelator-free labeling of LDH nanoparticles with radioisotopes for in vivo PET ima

229 icity as demonstrated by increased levels of LDH, in parallel to the presence of numerous vacuoles in

230 Loss of LDH led to a dramatic reduction of the growth rate, pinp

231 effects to resolve the chemical mechanism of LDH and establish the coupling of fs-ps protein dynamics

232 G) neurons were sensitized in a rat model of LDH.

233 VGSCs in a previously validated rat model of LDH.

234 cribe the identification and optimization of LDH-A inhibitors by fragment-based drug discovery.

235 .5 U/ml, covering the concentration range of LDH in normal as well as damaged tissues.

236 Complete recovery of LDH, CRP, and PSA levels was achieved post-rehydration w

237 al crystals broadened and approached that of LDH obtained from the original solution.

238 ions were utilized to construct the base of LDHs in an alkaline solution.

239 nly exhibits the alternative exploitation of LDHs, but also provides new insights into the removal of

240 ion of AY25 molecules into the interlayer of LDHs during their structural arrangement, where Mg(2+) a

241 However, the stacking structure of LDHs limits the exposure of the active sites.

242 assay, suggesting that off-target effects on LDH production may be responsible.

243 ificantly, a single spray of dsRNA loaded on LDH (BioClay) afforded virus protection for at least 20

244 Once loaded on LDH, the dsRNA does not wash off, shows sustained releas

245 viability was determined by MTT reduction or LDH release assays.

246 areas under the curve achieved with S100 or LDH markers.

247 ly 186 mg g(-1), comparable to that of other LDH-based methods.

248 nit LDHBx can also co-import LDHA, the other LDH subunit, into peroxisomes.

249 m LDH is not regulated by FBP, but the other LDHs are activated with increasing sensitivity in the fo

250 Our LDH/RGO-AuNPs/SPCE based biosensor thus performs as elec

251 t presents with chest pain, our carrying out LDH and D-Dimer tests will not exclude PTE without CTPA.

252 Peroxisomal LDH is conserved in mammals and likely contributes to re

253 Lactobacillus plantarum LDH is not regulated by FBP, but the other LDHs are acti

254 the heart allograft recipients, lower plasma LDH levels were observed.

255 d low temperature incubation did not prevent LDH-lactate-NS internalization, suggesting that LDH-lact

256 lar lavage fluid neutrophils, total protein, LDH, CXCL1/KC and CCL2/MCP-1 were also increased (P < 0.

257 < L. lactis LDH1 </= Streptococcus pyogenes LDH.

258 from the accumulation of pyruvate, revealing LDH as the most efficient enzyme in pyruvate conversion.

259 multivariate analysis, a high baseline serum LDH level was associated with decreased progression-free

260 Baseline serum LDH level was moderately accurate for predicting progres

261 months (AUC = 0.813) than did baseline serum LDH levels alone for prediction of progression-free surv

262 The combination of baseline serum LDH levels and evaluation with MASS criteria at the firs

263 A combination of baseline serum LDH levels and evaluation with MASS criteria at the firs

264 o-thirds the upper limit of normal for serum LDH) were absent (LR, 0.04; 95% CI, 0.02-0.11).

265 baseline clinical variables including serum LDH and imaging findings with progression-free and overa

266 >0.5, a ratio of pleural fluid LDH to serum LDH >0.6, or pleural fluid LDH >two-thirds the upper lim

267 d ionic strength (NaCl concentration) on six LDHs from four LABs studied at pH 6 and pH 7.

268 ysis time threefold compared to the standard LDH assay in solution.

269 supporting the concept of targeting stromal LDH-A as an effective strategy to blunt tumoral immune e

270 al responses stimulated by newly synthesized LDHs to be predicted in advance from these three paramet

271 Our finding demonstrates that LDH buffer layer represents a new concept for substrate

272 The present work demonstrates that LDH is an effective sorbent for selective extraction of

273 However, we suggest that LDH isoenzymes should be studied in further research.

274 -lactate-NS internalization, suggesting that LDH-lactate-NS penetrated the plasma membrane via non-en

275 These results support that LDH is a versatile platform that can be labeled with var

276 rted structure and compressive strain in the LDH nanosheets, which significantly enhances N2 chemisor

277 not label LDH since it does not fit into the LDH crystal structure.

278 nd APTES-GA treatments successfully link the LDH molecule to those surfaces while retaining its activ

279 erties in the allosteric binding site of the LDH enzymes.

280 ivities of the compounds on each half of the LDH redox reaction.

281 The turnover frequency (TOF) of the LDH-Au/CNTs COE catalyst was much higher than the previo

282 e orange (AO) was successfully loaded on the LDH layers, which significantly inhibited fluorescence q

283 th changes in adsorbed amount of dyes on the LDH.

284 sults in different regulatory effects on the LDHs of different LABs.

285 (LDH) suggesting that metabolic flux through LDH was required.

286 hemical detection was achieved with the TKec/LDH modified glassy carbon electrode (GCE).

287 The TKec/LDH/GCE biosensor was optimized using the best TK donor

288 dehydrogenase 1 (MDH1) is an alternative to LDH as a supplier of NAD.

289 o identify low affinity fragments binding to LDH-A.

290 cer as a biomarker to predict sensitivity to LDH-A inhibition, with regard to both real-time noninvas

291 y in glycolysis reduces the carbon supply to LDH.

292 ultivacancies in the as-exfoliated ultrathin LDHs nanosheets.

293 complexa, convergent evolution of an unusual LDH from MDH produced a difference in specificity exceed

294 after 7 weeks in adipose and muscle, whereas LDH mRNA expression increased 12-fold after 7 weeks in a

295 itron emission tomography (PET) imaging with LDH nanoparticles has not been achieved.

296 urvival (75% [70-81]), whereas patients with LDH concentration at least two times the upper limit of

297 egy for chronic pain relief in patients with LDH.

298 ES modified surfaces can directly react with LDH via physical attachment.

299 T hybrids to produce a composite with 10 wt% LDH and 2 wt% OCNT, the 50% weight loss temperature was

300 contribution, AMO [Mg3Al(OH)8](CO3)0.5.yH2O LDH-oxidized carbon nanotube (AMO-LDH-OCNT) hybrids are