ライフサイエンスコーパス: LGV

1 mbinant strains D/B120, G/UW-57, E/Bour, and LGV-98 identified in this study.

2 correctly identified the DNA from D to K and LGV reference strains and did not cross-react with the D

3 manifested itself in ocular, urogenital and LGV C. trachomatis strains, including the epidemic LGV s

4 the 15 serovars into ocular, urogenital, and LGV groups, and both proteins have been localized to the

5 ced two isolates from the remaining biotype, LGV, a long-term laboratory passaged strain and the rece

6 mydia trachomatis strains (L2/434/Bu [biovar LGV] and E/DK20/ON [biovar trachoma]) to induce putative

7 echnologies to show that the recent clinical LGV isolate causing proctitis is unlikely to be a newly

8 Newer molecular tests to confirm LGV infection are sensitive and specific, but are genera

9 irically with a recommended regimen to cover LGV infection.

10 trachomatis strains, including the epidemic LGV serotype L2b.

11 ry passaged strain and the recent "epidemic" LGV isolate-causing proctitis.

12 21 days) remains the treatment of choice for LGV.

13 r symptoms of proctitis should be tested for LGV, or if confirmatory testing is not available, should

14 Tarps from LGV strains contained the highest number of tyrosine-ric

15 hough with lower sensitivity than they lysed LGV-infected target cells.

16 rent pathologies associated with LGV and non-LGV strains.

17 e most divergent, compared to ocular and non-LGV urogenital disease groups.

18 No such associations were found for non-LGV-inducing strains.

19 ranuloma venereum (LGV); 1 plausible case of LGV was found by MIF.

20 With the exception of LGV-inducing strains, no evidence was found that differe

21 commonly reported clinical manifestations of LGV, with symptoms resembling those of inflammatory bowe

22 A simplex QTL allele was found on LGV of Stirling close to marker STM3179, which was assoc

23 there was no significant effect of a QTL on LGV, but a duplex QTL allele for resistance was found on

24 indings of an extensive literature review on LGV clinical presentation, diagnosis, and treatment that

25 confirmed these results using group-specific LGV real-time PCR and restriction fragment length polymo

26 metabolism was essential to induction by the LGV strain.

27 The assay also identified the LGV-II genotype in 24 of 48 rectal swab specimens contai

28 Although the genome of the LGV strain shows no additional genes that could account

29 spite minimal chromosomal polymorphisms, the LGV strain L2(25667R) was indistinguishable from plasmid

30 d serotypes D-K are noninvasive, whereas the LGV strains are invasive, causing a disseminating infect

31 B4855 are associated with factors mapping to LGV and LGX.

32 itative proteomic analysis of C. trachomatis LGV-L2 EB and RB forms.

33 the predicted proteins in the C. trachomatis LGV-L2 proteome.

34 f elementary bodies of Chlamydia trachomatis LGV serovar L2 and mass spectrometry to identify the lab

35 dia), and invasive lymphogranuloma venereum (LGV) and the murine strain Chlamydia muridarum share 99%

36 both trachoma and lymphogranuloma venereum (LGV) biovars from temporally and geographically diverse

37 ular, genital, and lymphogranuloma venereum (LGV) clinical isolates demonstrated an evolutionary rela

38 Lymphogranuloma venereum (LGV) has emerged as an important cause of proctitis and

39 differentiates the lymphogranuloma venereum (LGV) serovar from other serovars and may contribute the

40 Lymphogranuloma venereum (LGV) serovars were the most closely related for the pmp

41 Men with lymphogranuloma venereum (LGV)-inducing strains were more likely to be infected wi

42 serotypes D-K and lymphogranuloma venereum (LGV).

43 three genovars of lymphogranuloma venereum (LGV).

44 nital disease, and lymphogranuloma venereum (LGV).

45 lly diagnosed with lymphogranuloma venereum (LGV); 1 plausible case of LGV was found by MIF.

46 C. trachomatis (lymphogranuloma venereum [LGV])-elicited immune murine spleen cells lysed target c

47 te the different pathologies associated with LGV and non-LGV strains.

48 ry relationship with disease phenotype, with LGV and ocular isolates branched into clades that were s