ライフサイエンスコーパス: LH

1 LH GABAergic neurons project extensively to the paravent

2 LH infusions were made in close proximity to orexin neur

3 LH is a PhD clinical training fellow and funded through

4 LH melanin-concentrating-hormone (MCH) and orexin/hypocr

5 LH rapidly reduces the production of cGMP, but how this

6 LH-21 did not affect food intake nor body weight but it

7 LH-21 is a triazol derivative that has been described as

8 LH-butter showed a significantly (P<0.05) higher fat con

9 LHs bind bacteriochlorophyll a molecules, which confer o

10 3 [2.19] vs 2.64 [1.49] microg/mL; P = .02), LH (7.78 [3.19] vs 4.56 [2.01] mIU/mL; P < .001), and pr

11 TBPB and 77-LH-28-1 are selective agonists of the M1 muscarinic acet

12 propyl]-1,2,3,4-tetrahydroquinolin-2-one (77-LH-28-1, 3) connected by an aliphatic linker of variable

13 Moreover, functional imaging disclosed a LH region tuned to repulsive odors comprised exclusively

14 both global AcbSh neuron and selective AcbSh-LH terminal stimulation facilitated depression-like beha

15 l link between altered activity in the AcbSh-LH pathway and changes in the motivation for cocaine.

16 We found that stimulation of the AcbSh-LH pathway enhanced the motivation to self-administer co

17 he anaerobic reaction between linoleic acid (LH) and its 13-hydroperoxide (LOOH) catalysed by lipoxyg

18 ify a molecularly distinct, orexin-activated LH submodule that governs physical activity in mice.

19 ults suggest that restraint stress activates LH orexin neurons, releasing orexins into the VTA to act

20 d that disruption of GABA release from adult LH GABAergic neurons reduced feeding.

21 nd DNA integrity in vitro but did not affect LH stimulated testosterone secretion from adult testis e

22 ated PRL-, inhibited ACTH- and did not alter LH/FSH/TSH-release; and 3) resistin increased ACTH-relea

23 ous burst firing was ineffective at altering LH secretion.

24 When an LH exhibits more than two genotypes, we call it a local

25 decreases in cocaine self-administration and LH self-stimulation that are reversed by dynorphin antag

26 Lower estradiol and LH levels were found in urine and saliva samples of soy

27 The mean FAI and LH/FSH ratio were was also increased in the AGA group.

28 miR-7 genomic circuits that regulate FSH and LH synthesis and secretion through their effects on pitu

29 GHD and LH/FSHD were not treated in 99.7% and 78.5% of affected

30 d development over time is noted for GHD and LH/FSHD with possible associations between nontreatment

31 tant for generation of the surge of GnRH and LH that induces ovulation.

32 parental populations (referred to as LL and LH for maternally unstressed offspring, assigned in low

33 parental populations (referred to as LL and LH for maternally unstressed offspring, assigned in low

34 Populations in LL and LH groups changed significantly in responding to initial

35 ited single-unit responses aNAcSh, mPFC, and LH, in a manner that maintains firing rate homeostasis.

36 hat, under LTM-guided attention, both RH and LH IPS0-2 exhibit bilateral responses and hemispheric sy

37 iates the clearance of glycoproteins such as LH that bear N-linked glycans terminating with beta1,4-l

38 nanocrystal surfaces and closely associated LH(+) counter-cations (protonated n-octylamine or tri-n-

39 Before LH exposure, the cGMP concentration throughout the folli

40 Before LH stimulation, the arrest is maintained by diffusion of

41 length and rescued the progesterone blockade LH surge, while RU486 into the ARC shortened LH pulse in

42 Results showed that both LH and LS had higher relative abundance of S and Fe oxid

43 uxtacapsular BST, a region that contains BST-LH projecting neurons.

44 t low salt concentration, CTD condenses, but LH only interacts with the nucleosome and one linker DNA

45 ake direct protein-protein interactions, but LH residues at a distant sequence position and L1 residu

46 Fermented butter (LH-butter) was produced by churning the cream that was f

47 The ManR clears LH thus regulating testosterone production, whereas the

48 In contrast, LH neurons downstream of VTA encode reward-predictive cu

49 n LH secretion were compared with endogenous LH pulse parameters measured from ovariectomized mice.

50 N kisspeptin neurons were all found to evoke LH pulses.

51 hat the immunophilin FKBP65 does not exhibit LH activity, it is likely that LH2 activity is somehow d

52 ur mesoscale chromatin model with a flexible LH CTD captures a dynamic, salt-dependent condensation m

53 0), and 30-day mortality (1 patient [2%] for LH vs 2 [4%] for OH; P > .99) were not significantly dif

54 n positive margin status (1 patient [2%] for LH vs 2 patients [4%] for OH; P > .99).

55 rative blood transfusion (1 patient [2%] for LH vs 5 [10%] for OH; P = .20).

56 rall 30-day morbidity (22 patients [42%] for LH vs 19 [37%] for OH; P = .70), and 30-day mortality (1

57 oint prevalence was 46.5% for GHD, 10.8% for LH/FSHD, 7.5% for TSHD, and 4% for ACTHD, and the cumula

58 of major complications (4 patients [8%] for LH vs 4 patients [8%] for OH; P = 10), overall 30-day mo

59 ), and readmission rate (4 patients [8%] for LH vs 5 [10%] for OH; P = .70) were similar in both grou

60 = .16), hospital length of stay (5 days for LH vs 6 days for OH; P = .13), and readmission rate (4 p

61 Operative time (219 minutes for LH vs 198 minutes for OH; P = .16), hospital length of s

62 red significantly between groups (237 mL for LH vs 387 mL for OH; P = .049), we found no difference i

63 relations to analyze the trends observed for LH inhibition in castrated rats and monkeys that served

64 studies that are consistent with a role for LH in learning.

65 gma is that these effects reflect a role for LH neurons in the control of the core motivation to feed

66 , these results demonstrate a novel role for LH/cAMP in SCAP/SREBP activation and subsequent regulati

67 to define which patients derive benefit from LH and to determine oncologic equivalence to OH.

68 h were highest by microbial communities from LH, lower by those from LS and lowest form Sediment.

69 crine changes were not observed; rather FSH, LH, inhibin B, and anti-Mullerian hormone were temporall

70 Our study clarifies how GABAergic LH inputs to the VTA can contribute to generalized behav

71 H), and optogenetic stimulation of GABAergic LH --> PVH fibers induced monosynaptic IPSCs in PVH neur

72 dence that photostimulation of the GABAergic LH-VTA component, but not the glutamatergic component, i

73 bility of ARN kisspeptin neurons to generate LH pulses.

74 -18 can increase the firing of glutamatergic LH neurons through a disinhibitory mechanism.

75 viding homeostatic feedback on episodic GnRH/LH release as well as positive feedback to control ovula

76 terone can block the oestradiol-induced GnRH/LH surge and inhibit LH pulse frequency.

77 effects of HCD on the estradiol-induced GnRH/LH surge were overcome by neuron-specific SOCS3 knock-ou

78 As the most potent stimulator of GnRH/LH release, kisspeptin is believed to mediate the positi

79 progesterone's inhibitory effect on the GnRH/LH surge and pulsatile secretion is mediated by its rece

80 efer to scaffolded SNVs as local haplotypes (LH).

81 ral photosynthesis, namely light harvesting (LH), energy transfer (EnT), reductive/oxidative electron

82 ally fluorescing proteins, light-harvesting (LH) proteins from purple bacteria constitute an ideal ob

83 opper ore bioleaching system: leaching heap (LH), leaching solution (LS) and sediment under LS.

84 at the N terminus and a short nascent helix, LH, flanked by the two Dyn2 recognition motifs.

85 eat (SD) stress model, learned helplessness (LH), and a chronic corticosterone (CORT) model in mice,

86 iant on the ability to mobilize labile heme (LH).

87 human speech perception the left hemisphere (LH) of the brain is specialized for processing intelligi

88 ss the visual field; in the left hemisphere (LH), IPS0-2 codes primarily contralateral targets.

89 Higher LH level was found in urine samples of soy formula-fed g

90 Risk of lower testosterone and higher LH and FSH levels was significantly increased for TCSs a

91 significantly associated with risk of higher LH levels at both surveys and higher FSH at SI.

92 The risk of higher (LH) or lower (testosterone) levels was assessed with chi

93 terminal domain (CTD) of the linker histone (LH) is essential for chromatin compaction.

94 ectly controlled by the luteinizing hormone (LH) acting through the LH/choriogonadotropin receptor (L

95 oocytes is paused until luteinizing hormone (LH) activates receptors in the mural granulosa cells of

96 nished levels of plasma luteinizing hormone (LH) and correspondingly attenuated levels of circulating

97 crine cells [expressing luteinizing hormone (LH) and follicle-stimulating hormone (FSH)], with adreno

98 mulating hormone (FSH), luteinizing hormone (LH) and growth hormone (GH) cells.

99 sing hormone (GnRH) and luteinizing hormone (LH) are pivotal events in female reproductive function.

100 FSH and luteinizing hormone (LH) are secreted constitutively or in pulses, respective

101 The half-life for luteinizing hormone (LH) clearance increases in Mrc1(-/-) and Mrc1(-/-)Asgr2(

102 ating hormone (FSH) and luteinizing hormone (LH) gene expression in the pituitary.

103 he pulsatile release of luteinizing hormone (LH) is critical for mammalian fertility.

104 pression of circulating luteinizing hormone (LH) levels and no change in the lipid and triglyceride l

105 ted with an increase in luteinizing hormone (LH) pulse frequency, implicating abnormal steroid hormon

106 ious reports of in vivo luteinizing hormone (LH) pulse frequency.

107 including expression of luteinizing hormone (LH) receptor by the granulosa cells.

108 neurons and subsequent luteinizing hormone (LH) release.

109 ould generate pulsatile luteinizing hormone (LH) secretion in vivo.

110 red to evoke a pulse of luteinizing hormone (LH) secretion.

111 ating hormone (FSH) and luteinizing hormone (LH) secretion.

112 Luteinizing hormone (LH) stimulates steroidogenesis largely through a surge i

113 e preovulatory surge of luteinizing hormone (LH) that acts on the ovary and triggers the rupture of t

114 r glycoproteins such as luteinizing hormone (LH) that undergo regulated release into the circulation

115 ating hormone (FSH) and luteinizing hormone (LH), both of which are heterodimers specified by unique

116 estrone, testosterone, luteinizing hormone (LH), follicle-stimulating hormone (FSH) and sex hormone-

117 terone sulfate (DHEAS), luteinizing hormone (LH), follicle-stimulating hormone (FSH), prolactin, fast

118 drimer 3 showed similar luteinizing hormone (LH)-release activity to triptorelin in mice.

119 meiosis in response to luteinizing hormone (LH).

120 the resulting surge of luteinizing hormone (LH); however, the neural circuits that control the lepti

121 functionally integrated in the lateral horn (LH) of the vinegar fly, Drosophila melanogaster.

122 -) mice but not in Asgr2(-/-) mice; however, LH and testosterone are elevated in all three knockouts.

123 s in the genes coding for lysyl hydroxylase (LH) 2 or peptidyl-prolyl cis-trans isomerase (PPIase) FK

124 ress in mice activates lateral hypothalamic (LH) orexin neurons, increases levels of orexin A and 2-a

125 The lateral hypothalamic (LH) projection to the ventral tegmental area (VTA) has b

126 e rewarding effects of lateral hypothalamic (LH) stimulation, eliminates cocaine-induced impulsivity,

127 tudied the role of the lateral hypothalamus (LH) and its forebrain projections in this effect.

128 c projections into the lateral hypothalamus (LH) and receives extensive glutamatergic inputs from, am

129 neurons located in the lateral hypothalamus (LH) and zona incerta (ZI), but MCHR1 mRNA is widely expr

130 Lesions of the lateral hypothalamus (LH) cause hypophagia.

131 Damage to the lateral hypothalamus (LH) causes profound physical inactivity in mammals.

132 The lateral hypothalamus (LH) controls energy balance.

133 ivation of a subset of lateral hypothalamus (LH) GABAergic neurons induced both appetitive (food-seek

134 cal stimulation of the lateral hypothalamus (LH) has two motivational effects: long trains of stimula

135 The lateral hypothalamus (LH) is a key regulator of multiple vital behaviors.

136 The lateral hypothalamus (LH) is a neuroanatomical region essential for appetitive

137 However, the lateral hypothalamus (LH) is also a key reward-control locus in the brain.

138 reased activity in the lateral hypothalamus (LH) promotes feeding.

139 septal nucleus to the lateral hypothalamus (LH) that contribute to neural regulation of food intake

140 Projections from the lateral hypothalamus (LH) to the ventral tegmental area (VTA), containing both

141 including those to the lateral hypothalamus (LH), a target region known to influence addictive behavi

142 al habenula (LHb), the lateral hypothalamus (LH), and the midbrain are not only reciprocally connecte

143 ing neurons within the lateral hypothalamus (LH), as well as their function in the arousal network, r

144 used directly into the lateral hypothalamus (LH), but not the ventral tegmental area (VTA).

145 eep, we focused on the lateral hypothalamus (LH).

146 its projections to the lateral hypothalamus (LH).

147 The Fe(II) complexing constants were KFe(II)-LH=3x10(10) and KFe(II)-L=10(7), and the corresponding c

148 We confirmed these effects in LH and the CORT model.

149 s (a neuronal activity marker) expression in LH.

150 associated with increased Fos expression in LH.

151 nformation about the cues leading to food in LH itself.

152 how that the carboxyterminal heptapeptide in LH is a gonadotropin-sorting determinant in vivo that di

153 Increases in LH secretion were compared with endogenous LH pulse para

154 ked high amplitude, pulse-like increments in LH secretion in anesthetized male mice.

155 tivation generated significant increments in LH secretion.

156 er cholera toxin subunit B (CTb; injected in LH) with Fos.

157 ever, activation of glutamatergic neurons in LH inhibits feeding.

158 te nucleus kisspeptin (ARN(KISS)) neurons in LH pulse generation.

159 In contrast, LPXRFa-R are expressed only in LH, ACTH, and alpha-MSH cells.

160 r functional potential for S(0) oxidation in LH microbial communities.

161 the lateral hypothalamic area/zona incerta (LH) and the arcuate (Arc) nucleus.

162 ampling confirmed that PNA elicits increased LH pulse frequency and impaired progesterone negative fe

163 ORs for increased LH and FSH were 4.4 (95% CI, 3.1 to 6.5) and 18.9 (95% C

164 KERKO mice had increased LH pulse frequency, indicating loss of negative feedback

165 an unidentified glycoprotein that increases LH levels.

166 hundreds of cells, we identified individual LH GABAergic neurons that preferentially encode aspects

167 In experiment 1, E2 benzoate-induced LH surges in ovariectomized female monkeys were severely

168 oestradiol-induced GnRH/LH surge and inhibit LH pulse frequency.

169 ed the hypothesis that progesterone inhibits LH surge and pulsatile secretion via its receptor in the

170 ocked by muscimol + baclofen injections into LH.

171 functional microcircuit evidence that intra-LH feedforward loops may facilitate appropriate switchin

172 he key processes in photosynthesis, that is, LH, EnT, ET, and CAT, define the structure of this revie

173 KNM-LH 1 is securely dated to the Late Pleistocene, and samp

174 National Museums Lukenya Hill Hominid 1 (KNM-LH 1) is a Homo sapiens partial calvaria from site GvJm-

175 Features of cranial shape distinguish KNM-LH 1 and other Middle and Late Pleistocene African fossi

176 an age range of 23,576-22,887 y B.P. for KNM-LH 1, confirming prior attribution to the Last Glacial M

177 Comparative morphometric analyses of the KNM-LH 1 cranium document the temporal and spatial complexit

178 Teitelbaum SL, Pinney SM, Windham GC, Kushi LH, Biro FM, Valentin-Blasini L, Blount BC, Wolff MS, fo

179 rol HCD-mice experienced a preovulatory-like LH surge, LH surges could be induced in almost all neuro

180 g elevated in 1-week-old animals despite low LH levels.

181 f LH is heterogeneous; the cytosol maintains LH at approximately 20-40 nM, whereas the mitochondria a

182 g rat follicles, we show that within 10 min, LH signaling causes dephosphorylation and inactivation o

183 : 1) Leptin stimulated PRL/ACTH/FSH- but not LH/TSH-release; 2) adiponectin stimulated PRL-, inhibite

184 isms underlying the pro-ovulatory actions of LH in vertebrates and may prove important in clinical as

185 role mediating the pro-ovulatory actions of LH is not well established.

186 Food motivation was reduced by activation of LH GLP-1R.

187 lective and acute chemogenetic activation of LH VGAT(+) neurons was profoundly wake promoting, wherea

188 c arrest, but it is not clear how binding of LH to receptors that are located up to 10 cell layers aw

189 onversely, acute pharmacological blockade of LH GLP-1R increased food motivation but only in male rat

190 gation of the oestrous cycle and blockade of LH surge.

191 bout the type, function, and connectivity of LH neurons and identifies a neural circuit that selectiv

192 We find that the subcellular distribution of LH is heterogeneous; the cytosol maintains LH at approxi

193 ein, we elucidate the nature and dynamics of LH using genetically encoded ratiometric fluorescent hem

194 We next determined the effect of LH reversible inactivation by GABAA + GABAB receptor ago

195 quired to generate a pulse-like increment of LH.

196 e rat to show that optogenetic inhibition of LH gamma-aminobutyric acid (GABA) neurons restricted to

197 delivery, the co-translational insertion of LH polypeptides and their folding and assembly to form p

198 Chronic knockdown of LH GLP-1R induced by intraparenchymal delivery of an ade

199 Then, within 1 min of LH application, cGMP begins to decrease in the periphera

200 two mole of TEMPOL consumed for one mole of LH and one mole of LOOH.

201 ings from molecularly defined populations of LH neurons in awake freely moving mice, we show that MCH

202 xual maturation will increase the potency of LH in vivo, whereas increased expression during pregnanc

203 However, the properties of LH pools, including concentration, oxidation state, dist

204 roximate analysis and rheology properties of LH-butter were compared with butter produced using unfer

205 (KISS) neurons for 1 min generated pulses of LH in freely behaving mice, whereas inhibition with arch

206 ptin neuronal population generates pulses of LH.

207 The results demonstrate an important role of LH in renewal of alcohol seeking after punishment-impose

208 These data reveal a role of LH in vertebrate germ cells, whereby a Lhcgrba-activated

209 However, a more direct, nonsteroidal role of LH mediating the spermiogenic pathway remains unclear.

210 imulation of a molecularly defined subset of LH GABAergic (Vgat-expressing) neurons enhances both app

211 Several molecularly distinct types of LH neurons have been identified, including orexin cells

212 he stimulatory action of exogenous leptin on LH secretion and prevented the restoration of fertility

213 th halorhodopsin were found to reset ongoing LH pulsatility.

214 rding from neurons in the aNAcSh, or mPFC or LH.

215 o underwent partial hepatic resection (OH or LH) for benign or malignant disease were matched first b

216 Dyn2 binding also orders LH residues of Pac11.

217 lts suggest a potential importance for other LH neurons in stimulating feeding.

218 terion screening in vitro and phosphorylated LH in vivo.

219 bility of exogenous GnRH to induce pituitary LH secretion.

220 ported that progesterone prevented premature LH surges during ovarian hyperstimulation in women.

221 increased ACTH-release and did not alter PRL/LH/FSH/TSH-secretion.

222 asure the clinico-endocrinological profiles (LH, FSH, SHBG, DHEAS, and testosterone levels) of men wi

223 The firing of brain-wide-projecting LH neurons releases neuropeptides promoting wakefulness

224 ght into how GnRH neurons generate pulsatile LH secretion in vivo.

225 be near-perfectly correlated with pulsatile LH secretion.

226 structure of the monoprotonated species [Re(LH)L2](+), an intermediate in both catalytic H2 evolutio

227 ased expression during pregnancy will reduce LH potency until progesterone and receptor levels fall p

228 2 min was sufficient to generate repetitive LH pulses.

229 en nucleosome configuration; at higher salt, LH interacts with the nucleosome and two linker DNAs, pr

230 week later were subjected to 2 weeks of SD, LH training, or 3 weeks of CORT.

231 epared from pecans and separated by Sephadex LH-20 column chromatography into low- and high-molecular

232 , and fractions (fractionationed by Sephadex LH-20 column) from these two legumes.

233 CCC runs, and final purification on Sephadex LH-20.

234 size exclusion chromatography with Sephadex LH-20 without the need for any previous SPE of phenolic

235 LH surge, while RU486 into the ARC shortened LH pulse interval in the progesterone treated rats.

236 evidence that a range of mammal species show LH specialization when processing conspecific vocalizati

237 We found that dogs showed a significant LH bias when presented with a familiar spoken command in

238 intrinsic cofactors, at a level of a single LH protein, which opens a new, to our knowledge, route f

239 These data reveal cell-type-specific LH dynamics during sensory integration, and identify dir

240 ssion of its receptor permits the subsequent LH surge to trigger the release of ligands that activate

241 n with archaerhodopsin for 30 min suppressed LH pulsatility.

242 ce experienced a preovulatory-like LH surge, LH surges could be induced in almost all neuron-specific

243 Furthermore, this targeted LH subpopulation is distinct from cells containing the f

244 These data indicate that LH GABAergic neurons modulate consummatory behaviors reg

245 We show that LH-VTA neurons encode the learned action of seeking a re

246 These results suggest that LH-21 can be a new candidate to fight against diabetes o

247 bors, including MCH neurons, suggesting that LH may sometimes coengage its antagonistic outputs.

248 The LH/FSH ratio was not significantly different among the P

249 driven by charge neutralization between the LH and linker DNA.

250 the uneven interaction observed between the LH and the linker DNAs.

251 of bound pigments, which is governed by the LH protein dynamics.

252 Conversely, the LH of ppDIO-exposed mice had reduced contents of seroton

253 FSH released from the LH secretory pathway rescued ovarian defects in Fshb-nul

254 hway in which GABAergic projections from the LH to the PVH promote feeding.

255 Hb requires glutamatergic signaling from the LH, but not from the midbrain.

256 Our data identify the LH GLP-1R as an indispensable element of normal food rei

257 ptide YY 3-36 increased 5-HT turnover in the LH and ameliorated the ppDIO-induced sleep disturbances,

258 warding stimuli as well as its effect in the LH and mPFC are not well understood.

259 r demonstrated high expression levels in the LH and ZI, were tested to determine whether they are exp

260 en excitatory and inhibitory activity in the LH can induce changes in food intake.

261 eceptor (GLP-1R) activity selectively in the LH can profoundly affect food reward behavior, ultimatel

262 GABAergic neurons in the LH have been shown to mediate appetitive and feeding-rel

263 In the LH model, latency to escape was increased following trai

264 ntaining neurons or their projections in the LH reduced food intake in mice.

265 population of VLPO-projecting neurons in the LH that express the vesicular GABA transporter (VGAT; a

266 T neurons to a subset of vGAT neurons in the LH, an area involved in homeostatic and hedonic control

267 vide evidence for a feature-based map in the LH, and elucidate its role as the center for integrating

268 stimulate AcbSh terminal projections in the LH, in rats self-administering cocaine.

269 these features into separate domains in the LH.

270 acological or genetic GLP-1R blockade in the LH.

271 We show that inhibiting the LH-VTA pathway reduces "compulsive" sucrose seeking but

272 fibers originating in the bed nucleus of the LH and projecting to the ventral tegmental area (VTA).

273 bunit, as a critical signal for entry of the LH dimer into the regulated pathway.

274 show that inhibition of the terminals of the LH GABA neurons in ventral-tegmental area (VTA) facilita

275 we show that the asymmetric structure of the LH globular head is responsible for the uneven interacti

276 ll as molecular and efferent targets, of the LH GLP-1R activation were also evaluated.

277 the selective chemogenetic silencing of the LH-to-LHb pathway impairs aversion-driven escape behavio

278 we show that the GABAergic component of the LH-VTA pathway supports positive reinforcement and place

279 ry projection neurons (iPNs) that target the LH exclusively, at morphological, functional and behavio

280 implications of these findings are that the LH differs between females and males in its ability to c

281 These results suggest that the LH GABA neurons are critical for storing and later disse

282 We reveal that the LH sends excitatory and inhibitory input onto VTA dopami

283 luteinizing hormone (LH) acting through the LH/choriogonadotropin receptor (LHCGR) to stimulate andr

284 s, but whether individual neurons within the LH differentially contribute to these interconnected pro

285 processing, but the computations within the LH-VTA loop that give rise to specific aspects of behavi

286 e used, the data reported suggest that these LH GABAergic neurons may modulate behaviors that functio

287 onal activation in brain areas projecting to LH during context-induced renewal tests by measuring dou

288 ion of accumbens shell neurons projecting to LH.

289 est a role of accumbens shell projections to LH in this form of relapse.

290 o gonadotropin-releasing hormone, similar to LH.

291 Patients who undergo LH have similar short-term outcomes when compared with t

292 ess, we included 104 patients (52 undergoing LH and 52 undergoing OH) in the study.

293 Unexpectedly, LH-21 induced anxiolysis and reverted obesity-induced an

294 uscle mass and exercise tolerance; untreated LH/FSHD was associated with hypertension, dyslipidemia,

295 le charge transfer processes that occur when LH pigments are concentrated to enhance and direct the f

296 l and textural properties of butter in which LH-butter contained higher health beneficial unsaturated

297 ; white race was significant associated with LH/FSHD and TSHD.

298 th GHD; doses >/= 22 Gy were associated with LH/FSHD; and doses >/= 30 Gy were associated with TSHD a

299 d obesity were significantly associated with LH/FSHD; white race was significant associated with LH/F

300 -fat diet for 15 weeks and then treated with LH-21 or vehicle for two weeks.