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1 ough a c12-gp130 complex; it did not use the LIF receptor.
2 SM receptor; human OSM instead activates the LIF receptor.
3 s PBEF via its OSM receptor beta and not its LIF receptor.
4 tenuated by immuneutralization of either the LIF receptor (35%, P<0.05) or the gp130 affinity convert
5                       We conclude that gp130/LIF receptor and ET(A) receptor activation are essential
6 estigated the interaction between CT-1/gp130/LIF receptor and ET-1/endothelin type A (ET(A)) receptor
7 ctor (LIF) consists of two polypeptides, the LIF receptor and gp130.
8        Both murine LIF (cannot bind to human LIF receptor) and human LIF caused expansion of human CD
9  tripartite complex comprised of CNTFRalpha, LIF receptor, and gp130.
10         Leukemia inhibitory factor (LIF) and LIF receptors are expressed in adenohypophyseal cells an
11                            During this time, LIF receptors are expressed predominantly in the luminal
12 endogenous and exogenous CT-1 requires gp130/LIF receptor as well as ET(A) receptor activation.
13 TF, and CT-1 act through the known receptors LIF receptor beta (LIFRbeta) and gp130 and well defined
14 3T3 cells (which endogenously express gp130, LIF receptor beta [LIFRbeta], and the protein product, c
15 n M and ciliary neurotrophic factor, and the LIF receptor beta and glycoprotein gp130 in normal human
16                                          The LIF receptor beta is expressed during the proliferative
17 nd FoxD3 as well as in the expression of the LIF receptor complex LIFR/IL6ST (gp130).
18                 We recently reported LIF and LIF receptor expression in human fetal pituitary cortico
19  uterus increased HLA-G, EGF-Receptor-2, and LIF-Receptor expression on EVT, presumably representing
20 f mice carrying a targeted disruption of the LIF receptor gene, a common component of the receptor co
21 sociated signal-transducing component of the LIF receptor, gp130, is also expressed in both the lumin
22         Leukemia inhibitory factor (LIF) and LIF receptors have been implicated previously in astrocy
23                         Blockade of gp130 or LIF receptor inhibited basal growth as well as CT-1- or
24 keletal muscles, we examined whether the LIF-LIF receptor (LIF-R) axis affects the biology of rhabdom
25 -6 promoter after fibroblast activation, and LIF receptor (LIFR) and STAT4 formed a molecular complex
26  (LIF, OSM, CNTF, and CT-1) also utilize the LIF receptor (LIFR) as a component of their receptor com
27 tes giant cell differentiation in vitro, and LIF receptor (LIFR) deficiency results in loss of giant
28 he hypothesis that the reduced expression of LIF receptor (LIFR) observed in hepatoma cells is mediat
29 LIF/OSM receptor (a heterodimer of gp130 and LIF receptor (LIFR)) and the OSM-specific receptor (a he
30 d murine LIF (mLIF) act in mouse cells via a LIF receptor (LIFR)-glycoprotein 130 (gp130) heterodimer
31  receptor complex composed of a low affinity LIF receptor (LIFRbeta) and gp130.
32 weeks with a monoclonal antibody against the LIF receptor motif, gp130 (0.833 mg ml-1) (P < 0.05, Man
33 cells, we hypothesized that LIF binds to the LIF receptor on AC6.21 stromal cells, leading to up-regu
34 n130 (gp130) and leukemia inhibitory factor (LIF) receptor, on cardiac fibroblast growth in cultured
35 ation of SHP-2 with the gp130 subunit of the LIF receptor signaling complex.
36                Treatment with either soluble LIF receptor (sLIFR), a LIF antagonist, or the mTOR inhi
37 y despite the presence of constant levels of LIF receptor throughout the preimplantation period.
38                               CT-1 and gp130/LIF receptor were widely present in the cytoplasm of the

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