ライフサイエンスコーパス: LPC

1 LPC activates protein kinase C (PKC) and increases cAMP

2 LPC and/or biliary cells generated 0.78% and 2.45% of he

3 LPC C26:0 added to ABCD1-deficient microglia in culture

4 LPC markers correlate positively with severity of liver

5 LPC markers were assessed by real-time polymerase chain

6 LPC or biliary cells terminally differentiate into funct

7 LPC transport via Mfsd2a has been shown to be necessary

8 LPC treatment of PMH or Huh7 cells induced release of EV

9 LPC-activated inflammasome also requires ASC (apoptotic

10 LPCs have intrinsic, cell proliferation-independent char

11 he proportional amounts of 18:2-LPC and 18:1-LPC in the unsaturated LPC fraction was derived from the

12 Percentage of 18:1-LPC or 18:2-LPC plasma levels compared with total satura

13 nce between the proportional amounts of 18:2-LPC and 18:1-LPC in the unsaturated LPC fraction was der

14 Percentage of 18:1-LPC or 18:2-LPC plasma levels compared with total saturated LPC leve

15 dylcholines (LPCs), including 18:1- and 18:2-LPC, were significantly decreased in CRC patients compar

16 tarch suspensions (9% w/w) containing 0.5-5% LPC were subjected to hydrolysis by porcine pancreatic a

17 ses in the initial rate of radiolabeled 2-AA-LPC and arachidonic acid (AA) production, respectively,

18 2-arachidonoyl-lysophosphatidylcholine (2-AA-LPC) from 1-palmitoyl-2-[(14)C]arachidonoyl-sn-glycero-3

19 a revealed the robust production of AA, 2-AA-LPC, and 2-docosahexaenoyl-LPC that was over 10-fold gre

20 ssels, showed that luminal but not abluminal LPC potently induced permeability, and that this require

21 n kinase C zeta (PKCzeta) activity abrogated LPC-dependent dendricity.

22 2-Acyl LPC from water fraction after precipitation in cold (-20

23 2-Acyl-lysophosphatidylcholine (2-acyl LPC), fatty acids ethyl esters (FAEEs) and free fatty ac

24 Fetal and adult LPCs had significantly greater reprogramming efficiency

25 protein (RFP)-tagged EV marker, CD63, after LPC treatment of cotransfected Huh-7 cells.

26 ted in demyelinated lesions within 5 d after LPC injection, together with enhanced astrocyte prolifer

27 astrocyte-specific Fpn knock-out mice after LPC-induced demyelination.

28 Our results indicate that although LPC can promote the initial adsorption of vesicles conta

29 on a molecular level, influenced by amylose-LPC complexation; however the effect depends on the dige

30 lly form distinct inflammasomes, activate an LPC-induced inflammasome and are important in astroglios

31 m to support mouse biliary-derived DRs as an LPC pool to replenish hepatocytes in a quantitatively re

32 ed myelin repair therapy was conducted in an LPC rat model using a mesenchymal stem cell-based hepato

33 This article reports a case of an LPC treated with guided tissue regeneration (GTR) and bo

34 y of the defect left after the removal of an LPC, GTR, along with bone grafting, can be a very useful

35 iopsy revealed the histologic features of an LPC.

36 the distinctive network dynamics in ACC and LPC during normal and pathological brain states.SIGNIFIC

37 r' responses) and by (2) examining FN400 and LPC modulation associated with false alarms.

38 We found that the FN400 and LPC were absent or attenuated in the older group relativ

39 rs together with the induction of injury and LPC proliferation.

40 rall, switched words elicited larger LAN and LPC amplitude than non-switched words.

41 However, the LPA and LPC species that increase in BAL of bleomycin-injured mi

42 ntributors to the difference in both LPE and LPC levels.

43 can contribute to the differences in LPE and LPC.

44 levels of phosphatidylethanolamine, LPE, and LPC in the cells.

45 cyltransferase function but exhibits MAG and LPC hydrolase activities.

46 presence of two separate substrate (MAG and LPC)-binding sites in a single polypeptide.

47 te populations of neurons within the VTC and LPC during the visual processing of numerals and the per

48 ce of math-selective hubs within the VTC and LPC, we report here a remarkable heterogeneity of neural

49 information (face vs scene) in both VTC and LPC.

50 stablish a link between transport of DHA and LPCs by MFSD2A and human brain growth and function, pres

51 mokine expression localized to CK7(+) DR and LPCs in CFLD liver biopsies.

52 Primary cultures of hepatocytes and LPCs from AlfpCre(+)Trp53(Delta2-10/Delta2-10) mice, but

53 nd key events in DR evolution were assessed: LPC proliferation, LPC biliary differentiation, and hepa

54 ear conditions in a flow chamber, LPA or ATX/LPC strongly enhances TEM of integrin-arrested T cells a

55 6% of the total, whereas the predominant BAL LPC species contained shorter chain, saturated acyl grou

56 Because LPC produces greater disorder but positive curvature, th

57 liver parenchymal cells (LPCs; IKKalpha/beta(LPC-KO) ) were intercrossed with RIPK1(LPC-KO) or RIPK3(

58 ith a substrate-product relationship between LPC and LPA in pulmonary fibrosis.

59 t this type II NKT TCR binds with CD1d-bound LPC with micromolar affinities similar to that for sulfa

60 on of disease associated with aberrant brain LPC transport in humans.

61 Repopulation efficiency by LPC and/or biliary cells increased when extracellular ma

62 n and was prevented by LPC or OA, but not by LPC+OA.

63 cts of sPLA2-X are mediated predominantly by LPC, not AA, and we have demonstrated expression of the

64 usion channel formation and was prevented by LPC or OA, but not by LPC+OA.

65 Cytokine secretion by LPC-specific T cells is skewed toward IL-13 secretion, a

66 e in patients with laryngopharyngeal cancer (LPC) undergoing chemoradiotherapy (CRT) treatment.

67 We propose that increased cPLA(2)-catalyzed LPC production in the brain is at least one of the mecha

68 The iNKT TCR CDR loop footprint on CD1d-LPC is anchored by the conserved positioning of the CDR3

69 of the LPC headgroup but stabilizes the CD1d-LPC complex in a closed conformation.

70 iferation of STAT3-deficient EpCAM(+)CD45(-) LPCs.

71 epithelial cell adhesion molecule(+)CD45(-) LPCs isolated from DDC-fed wild-type mice.

72 easy and reproducible liver progenitor cell (LPC) isolation strategy based on aldehyde dehydrogenase

73 garding the extent of liver progenitor cell (LPC) proliferation in AH.

74 Liver progenitor cell (LPC)-enriched cell fractions were isolated from adult (6

75 ice lacking NEMO in liver parenchymal cells (LPC) spontaneously develop steatohepatitis and hepatocel

76 have indicated that liver progenitor cells (LPC) could give rise to hepatic epithelial cells during

77 ized by expansion of liver progenitor cells (LPC), which correlates with disease severity.

78 ) infiltration with lymphoplasmacytic cells (LPCs).

79 er RIPK1 or RelA in liver parenchymal cells (LPCs) did not cause spontaneous liver pathology, mice wi

80 tic IKK activity in liver parenchymal cells (LPCs; IKKalpha/beta(LPC-KO) ) were intercrossed with RIP

81 gulation of VDUP1 in lamina precursor cells (LPCs) coincided with the arrival of retinal axons into t

82 cells (LSCs) and leukemia progenitor cells (LPCs) accumulate high levels of reactive oxygen species

83 onogenic capacity of liver progenitor cells (LPCs) and hepatocytes.

84 Liver progenitor cells (LPCs) are necessary for repair in chronic liver disease

85 cells (LSCs) and leukemia progenitor cells (LPCs) displaying innate and acquired resistance, respect

86 contain bipotential liver progenitor cells (LPCs) that serve as an emergency cell pool to regenerate

87 ved in DR induction, liver progenitor cells (LPCs) were treated with taurocholate, and key events in

88 isolation of primary liver progenitor cells (LPCs), directed hepatocyte differentiation of primary LP

89 roliferation of liver stem/progenitor cells (LPCs), which can differentiate into hepatocytes or bilia

90 ial cells (BECs), or liver progenitor cells (LPCs).

91 dult hepatocytes and liver progenitor cells (LPCs).

92 patient-derived leukemia-propagating cells (LPCs) in murine xenografts.

93 um yoelii synthetic linear peptide chimeras (LPCs) based on the circumsporozoite protein protects aga

94 ough hydrolysis of lysophosphatidyl choline (LPC).

95 OX9 proteins, the number of primary cilia(+) LPCs, and increased active gamma-glutamyltranspeptidase

96 d bilayer coating and are named Lipid-Pt-Cl (LPC) NPs, which showed significant antitumor activity bo

97 at complete NF-kappaB inhibition by combined LPC-specific ablation of RelA, c-Rel, and RelB did not p

98 production of observable long-period comets (LPCs) from the Oort Cloud, a vast reservoir of icy bodie

99 ated with an enhanced late positive complex (LPC, 500-800ms) largest on frontal sites.

100 vity (FRN), and the late positive component (LPC), indicate that social comparison manifests in three

101 LAN - followed by a Late Positive Component (LPC).

102 hyl-allo-threonyl hydroxamate-based compound LPC-058 is a potent inhibitor of UDP-3-O-(R-3-hydroxymyr

103 ntial patients with histologically confirmed LPC being treated with CRT.

104 By contrast, LPC did not contribute to hepatocyte regeneration during

105 Contrastingly, LPC promotes RPT aggregation at both submicellar and mic

106 og (Hh) is a signal well known to coordinate LPC proliferation and differentiation in response to ret

107 in areas within the lateral parietal cortex (LPC) and ventral temporal cortex (VTC) have been shown t

108 creased activity in lateral parietal cortex (LPC)--"retrieval success effects" that are thought to ge

109 The lateral periodontal cyst (LPC) is an unusual cyst of odontogenic origin, most freq

110 ember' responses at both delays, a decreased LPC amplitude was observed with false alarms relative to

111 del to study the behavior of patient-derived LPC clones, which provides insights relevant for experim

112 monstrate that pluripotent stem cell-derived LPCs choose hepatic fate when cultured next to healthy h

113 These studies show that dietary LPC-DHA efficiently increases brain DHA content and impr

114 ction of AA, 2-AA-LPC, and 2-docosahexaenoyl-LPC that was over 10-fold greater than wild-type mitocho

115 Moreover, an early LPC effect was observed only for switched nouns, but not

116 is associated with the late parietal effect (LPC).

117 iency-corrected reprogramming rates of fetal LPCs were 275-fold higher, compared with unsorted fetal

118 d level of performance on both early (FN400, LPC) and later [late frontal effect (LFE)] ERP indices o

119 clude that the primary signaling pathway for LPC-dependent dendrite formation in human melanocytes in

120 ptor and two G-protein-coupled receptors for LPC (G2A and GPR119) in human melanocytes.

121 The most common treatment for LPC is surgical enucleation.

122 ke, confirming the specificity of MFSD2A for LPCs having mono- and polyunsaturated fatty acyl chains.

123 hese findings indicate an essential role for LPCs in human brain development and function and provide

124 h was markedly enriched in EVs isolated from LPC-treated hepatocytes versus untreated cells.

125 cholate is involved in initiating functional LPC biliary differentiation and the development of the D

126 Furthermore, LPC-mediated activation of type II NKT cells leads to an

127 eak concentration, in the presence of a high LPC concentration, which is related to less degradation

128 However, LPC numbers also correlate with disease severity and hep

129 Human LPC-CD1d dimer binding cells are T-cell receptoralphabet

130 These findings identify LPC transport via Mfsd2a as an important pathway for DHA

131 actively represents retrieved content or if LPC activity only scales with content reactivation elsew

132 B2) as predictors of locoregional failure in LPC patients treated with CRT.

133 lso report that an age-dependent increase in LPC levels in the PPT1-KO mouse brain positively correla

134 cholate induced a time-dependent increase in LPC proliferation and expression of genes associated wit

135 As encoding enzymes and proteins involved in LPC degradation (lysophosphatidylcholine acyltransferase

136 oreover, recall-related activity patterns in LPC, but not VTC, differentiated between individual even

137 Longitudinal PET studies in LPC and EAE rat models demonstrate that [11C]MeDAS uptak

138 he IL-22 signaling pathway, was activated in LPCs isolated from DDC-fed IL-22TG mice.

139 nds where VDUP1 expression was maintained in LPCs, inhibiting both cell proliferation and lamina neur

140 n LSCs, including quiescent LSCs, but not in LPCs.

141 lpha/beta-dependent RIPK1 phosphorylation in LPCs inhibits compensatory proliferation of hepatocytes

142 ith combined deficiency of RIPK1 and RelA in LPCs showed increased hepatocyte apoptosis and developed

143 reover, mice lacking both RIPK1 and TNFR1 in LPCs displayed normal tumor formation in response to DEN

144 Although an increased LPC amplitude was found associated with 'Remember' respo

145 is the key enzyme mediating the p25-induced LPC production and cPLA2 upregulation is critical in tri

146 Taurocholate induced LPCs to release MCP-1, MIP1alpha, and RANTES into condit

147 tes increased the induction of liver injury, LPC proliferation, and tumor necrosis factor-alpha produ

148 The aim of this study was to investigate LPC markers in AH and its correlation with disease sever

149 ortant, if not the dominant, source of known LPCs.

150 e and the other two players' evoked a larger LPC than the medium difference and the even condition.

151 almitoyl-, stearoyl-, oleoyl-, and linoleoyl-LPC levels after LCA exposure.

152 iosis in corpus callosum after lysolecithin (LPC)-induced focal demyelination and in cultured astrocy

153 at regulates cell 2-lysophosphatidylcholine (LPC) levels and arachidonate incorporation into phosphat

154 containing L-alpha-lysophosphatidylcholine (LPC), as observed using atomic force microscopy.

155 ncreases in LPE and lysophosphatidylcholine (LPC) contents in leaves.

156 lglycerol (MAG) and lysophosphatidylcholine (LPC) hydrolytic activities, indicating the bifunctional

157 LPA and lysophosphatidylcholine (LPC) levels in the tumor microenvironment were reduced t

158 lysolipids LGL1 and lysophosphatidylcholine (LPC).

159 ormal adult mice as lysophosphatidylcholine (LPC) (40 mg DHA/kg) for 30 days increased DHA content of

160 ospholipids such as lysophosphatidylcholine (LPC) can stimulate the sulfatide-reactive type II NKT hy

161 levated atherogenic lysophosphatidylcholine (LPC 18:1) and lysophosphatidic acids (LPAs) in the intes

162 ation in the brain, lysophosphatidylcholine (LPC)-induced focal demyelination in the spinal cord, and

163 ed sodium-dependent lysophosphatidylcholine (LPC) symporter expressed at the blood-brain barrier that

164 a sodium-dependent lysophosphatidylcholine (LPC) transporter (MFSD2A), expressed in the endothelium

165 addition of either lysophosphatidylcholine (LPC) or lysophosphatidic acid (LPA) to cells restored th

166 Finally, lysophosphatidylcholine (LPC) levels in the PFC were found to be correlated with

167 n produces LPA from lysophosphatidylcholine (LPC) via lysophospholipase D activity, but alternative e

168 dic acid (LPA) from lysophosphatidylcholine (LPC).

169 e D that hydrolyzes lysophosphatidylcholine (LPC) into lysophosphatidic acid (LPA), initiating signal

170 disease, including lysophosphatidylcholine (LPC).

171 emifusion inhibitor lysophosphatidylcholine (LPC), but not if a complementary-shaped lipid, oleic aci

172 he lysophospholipid lysophosphatidylcholine (LPC), from membrane preparations.

173 uble lipid mediator lysophosphatidylcholine (LPC) is released by p25 overexpressing neurons to initia

174 r concentrations of lysophosphatidylcholine (LPC) led to an increase of the MscL/MscS threshold ratio

175 sured the effect of lysophosphatidylcholine (LPC) on adsorption.

176 ribe our studies of lysophosphatidylcholine (LPC) presentation by human CD1d and its recognition by a

177 s the conversion of lysophosphatidylcholine (LPC) to lysophosphatidic acid (LPA), a bioactive lipid a

178 r the hydrolysis of lysophosphatidylcholine (LPC) to the bioactive lysophosphatidic acid (LPA) and ch

179 ined the effects of lysophosphatidylcholine (LPC) upon microglia.

180 eased production of lysophosphatidylcholine (LPC), catalyzed by the activation of cytosolic phospholi

181 cids in the form of lysophosphatidylcholine (LPC).

182 y microinjection of lysophosphatidylcholine (LPC).

183 earoyl-, and oleoyl-lysophosphatidylcholine (LPC) and marked increases in tauro-beta-muricholate, tau

184 linating effects on lysophosphatidylcholine (LPC) induced demyelination in a three-dimensional brain

185 d with palmitate or lysophosphatidylcholine (LPC).

186 enzymatic product, lysophosphatidylcholine (LPC), are involved in blood-retinal barrier (BRB) damage

187 of proinflammatory lysophosphatidylcholine (LPC), which was detectable in both HFD and MCD mice, was

188 h more radiolabeled lysophosphatidylcholine (LPC)-DHA enters the brain than NEFA-DHA, this is due to

189 in decreased serum lysophosphatidylcholine (LPC) and sphingomyelin levels due to elevated lysophosph

190 ds revealed several lysophosphatidylcholine (LPC) species.

191 ating single-tailed lysophosphatidylcholine (LPC) into a membrane bilayer using coarse-grained molecu

192 udy, we reveal that lysophosphatidylcholine (LPC), a molecule associated with neurodegeneration and d

193 we have shown that lysophosphatidylcholine (LPC), the main lysophospholipid released in response to

194 hile treatment with lysophosphatidylcholine (LPC) enhanced the expression of iNOS, demonstrating a no

195 sion complexes with lysophosphatidylcholine (LPC), that decrease the susceptibility of amylose to amy

196 PG) or zwitterionic lysophosphatidylcholine (LPC) stimulate aggregation, LPG exerted a greater effect

197 of chemoprotective lysophosphatidylcholines (LPCs).

198 levels of several lysophosphatidylcholines (LPCs), including 18:1- and 18:2-LPC, were significantly

199 Moreover, LPC-DHA treatment markedly improved the spatial learning

200 roliferation of cultured BMOL cells (a mouse LPC line).

201 human CD1d and its recognition by a native, LPC-specific iNKT TCR.

202 epatocyte apoptosis and liver tumors in NEMO(LPC-KO) mice, revealing distinct kinase-dependent and sc

203 vented hepatocellular damage and HCC in NEMO(LPC-KO) mice.

204 amming efficiency of LPCs, compared with non-LPCs.

205 eta-catenin, and surface markers with normal LPCs.

206 LPA, but not LPC, also enhanced in vivo ascites formation (by approxi

207 LPA, but not LPC, stimulated ID8 cell migration and invasion with cel

208 ependent on the digestion time and amount of LPC.

209 ylate either sn position of ether analogs of LPC The data show that the activities of LPEAT1 and LPEA

210 ffects were particularly evident in areas of LPC (namely, angular gyrus) in which activity scaled wit

211 We investigated the capacity of LPC to differentiate into hepatocytes in vivo and contri

212 CDE models indicated that no contribution of LPC to hepatocytes was associated with LPC expression of

213 protein reporter mice, to follow the fate of LPC and biliary cells.

214 The generation of LPC by PMVECs required Prdx6-PLA2 We propose that Prdx6-

215 ulates NOX2 activation through generation of LPC for conversion to LPA; binding of LPA to LPAR1 signa

216 2 modulates NOX2 activation by generation of LPC that is converted to LPA by the lysophospholipase D

217 olyclonal, consisting of tens to hundreds of LPC clones.

218 Specifically, the inclusion of LPC and eggPE significantly altered the lipid diffusion,

219 ng, we show that asymmetric incorporation of LPC can lead to significant curvature in a bilayer.

220 tive channels by asymmetric incorporation of LPC into membrane patches in patch-clamp experiments.

221 cyte-derived macrophages in the induction of LPC proliferation using clodronate liposome deletion of

222 rogenic genes, likely adaptations to loss of LPC transport.

223 Downstream mediators of LPC-dependent dendricity include Rac and Rho.

224 beta(+) biliary duct cells are the origin of LPC.

225 roscopy demonstrated that in the presence of LPC as opposed to cholesterol, N-ethylmaleimide-sensitiv

226 Direct comparison of the uptake rate of LPC-DHA and NEFA-DHA demonstrates that uptake of NEFA-DH

227 croarray analysis showed an up-regulation of LPC markers in patients with AH.

228 However, the origin and role of LPC in liver physiology and in hepatic injury remains a

229 However, the functional significance of LPC activation during memory retrieval remains a subject

230 ing to spread to cells distal to the site of LPC substrate binding by ATX.

231 haly syndrome linked to inadequate uptake of LPC lipids.

232 ferentiation toward hepatocyte-like cells of LPCs with high ALDH activity is also successfully applic

233 ificantly increased plasma concentrations of LPCs containing mono- and polyunsaturated fatty acyl cha

234 The increased reprogramming efficiency of LPCs, compared with differentiated liver cells, occurred

235 ed the increased reprogramming efficiency of LPCs, compared with non-LPCs.

236 High ALDH activity is a feature of LPCs that can be taken advantage of to isolate these cel

237 The immunogenicity of LPCs is significantly enhanced by spontaneous polymeriza

238 Proliferation of LPCs in mice was induced by feeding a diet that containe

239 factor for hepatocytes, on proliferation of LPCs in patients with chronic hepatitis B virus (HBV) in

240 ings link inflammation with proliferation of LPCs in patients with HBV infection.

241 oduce IL-22, which promotes proliferation of LPCs via STAT3.

242 e grade of inflammation and proliferation of LPCs.

243 g adenovirus, had increased proliferation of LPCs.

244 inflammation might promote proliferation of LPCs.

245 ers of IL-22TG mice reduced proliferation of LPCs.

246 r-alpha, and the subsequent proliferation of LPCs.

247 Significantly, biliary fate selection of LPCs was not observed in the absence of hepatocytes nor

248 CMCs was not correlated with bevacizumab or LPC treatment.

249 Treatment of melanocytes with sPLA2-X or LPC induced phosphorylation of the zeta isoform of PKC,

250 f experimental evidence establishing BECs or LPCs as the origin of ICCs, other liver cell types have

251 higher amounts of lyso phosphatidylcholine (LPC) compared to SP2/0 and CHO cell lines.

252 of ethanolamine), lyso-phosphatidylcholine (LPC), and lyso-phosphatidylethanolamine (LPE).

253 treal bevacizumab or laser photocoagulation (LPC) and untreated eyes.

254 and Firmicutes and Cyanobacteria with plasma LPC 18:1.

255 Ex vivo, ATX plus LPC or LPA itself induces the polarization of mouse naiv

256 ndent fatty acid release and polyunsaturated LPC production.

257 Human CD1d presenting LPC adopts an altered conformation from that of CD1d pre

258 rected hepatocyte differentiation of primary LPCs and pluripotent stem cells, findings of transdiffer

259 ed genomic instability in LSCs and primitive LPCs, which could be targeted to prevent the relapse and

260 n species (ROS) in CML-CP LSCs and primitive LPCs.

261 evolution were assessed: LPC proliferation, LPC biliary differentiation, and hepatic stellate cell (

262 xagonal-II (H(II)) phase and adsorb rapidly, LPC instead had little effect on initial adsorption but

263 DHA-deficient and had significantly reduced LPC/DHA transport in vivo Fluorescein angiography indica

264 /beta(LPC-KO) ) were intercrossed with RIPK1(LPC-KO) or RIPK3(-/-) mice to examine whether RIPK1 or R

265 A model based on total saturated LPC and the difference between the proportional amounts

266 plasma levels compared with total saturated LPC levels, either individually or in combination, may r

267 an association between the changes in serum LPC and bile acids and proinflammatory cytokines.

268 is through TGF-beta signaling and that serum LPC is a biomarker for biliary injury.

269 A-DHA into the brain is 10-fold greater than LPC-DHA.

270 t diabetes-mediated BRB dysfunction and that LPC impacts on the retinal vascular endothelium to induc

271 hese findings provide striking evidence that LPC not only signals that memories have been successfull

272 This study suggests that LPC proliferation may be an important feature of AH path

273 The LPC has been shown to be generally sensitive to correct

274 the HPC intervention compared with after the LPC intervention.

275 iNKT TCR requires a 7-A displacement of the LPC headgroup but stabilizes the CD1d-LPC complex in a c

276 n accompanied by decreased expression of the LPC receptor G2A, whereas MS patient samples show increa

277 Differences in the amplitude of the LPC were observed between correct and incorrect source d

278 so revealed differences, suggesting that the LPC is also sensitive to variations in the strength of s

279 Furthermore, alteration of the LPCs and Lpcat1/2/4 and Smpd3 expression was attenuated

280 We use this LPC production to place observationally motivated constr

281 ct cells and to assess their contribution to LPC expansion and hepatocyte generation.

282 , and provide growth and survival signals to LPCs through several TNF family ligands (CD40L, a prolif

283 d at the blood-brain barrier that transports LPCs containing DHA and other long-chain fatty acids.

284 of 18:2-LPC and 18:1-LPC in the unsaturated LPC fraction was derived from the TS.

285 tion to acyl-CoA, GPCAT efficiently utilizes LPC and lysophosphatidylethanolamine as acyl donors in t

286 ison showed distinct behaviors of LPG versus LPC monomers and micelles plausibly originating from the

287 nal thickening at the foveal center, whereas LPC is associated with earlier extrusion of the inner re

288 theories are divided with respect to whether LPC actively represents retrieved content or if LPC acti

289 on of LPC to hepatocytes was associated with LPC expression of genes related to telomere maintenance,

290 Slow catalysis with LPC reveals the potential for LPA signaling to spread to

291 Radiographs revealed a cystic lesion with LPC characteristics.

292 d hydrolysis are slow and rate-limiting with LPC.

293 Treatment of melanocytes with LPC induced rapid activation of Rac that peaked at 30 mi

294 In contrast, mice with LPC-specific knockout of Ripk1 showed reduced diethylnit

295 cells following two liver injury models with LPC expansion, a diethoxycarbonyl-1,4-dihydro-collidin (

296 c factor increased in all brain regions with LPC-DHA, but not with free DHA.

297 Eyes treated with LPC had a lower probability of having all inner retinal

298 for and compare content reactivation within LPC and VTC.

299 rt of these studies, we demonstrated that WM LPCs secrete soluble CD27 (sCD27), which is elevated in

300 ethyl-N-(7-nitrobenz-2-oxa-1,3-diazol-4-yl))-LPC.