ライフサイエンスコーパス: LV fractional shortening

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1 ejection fraction, with a trend toward lower LV fractional shortening.

2 f children had similar low left ventricular (LV) fractional shortening.

3 ) and vehicle (39.2 + or - 2.07%), increased LV fractional shortening 1.25-fold over wild-type MSCs a

4 tio, 2.06; P=0.036), worse left ventricular (LV) fractional shortening (19.7+/-1.5% versus 27.2+/-1.5

5 3.2+/-1.3%, P<0.05) dimensions and augmented LV fractional shortening (24.7+/-10.5%, P<0.03).

6 The z scores for LV fractional shortening (a measure of cardiac function)

7 d a progressive decline in left ventricular (LV) fractional shortening accompanied by ventricular dil

8 d for congestive heart failure and decreased LV fractional shortening among those with familial DCM (

9 septal wall thickness z-scores and increased LV fractional shortening and contractility up to age 2 y

10 With candesartan pretreatment, LV fractional shortening and ejection fraction increased

11 >50% increase in LV dimension, a 56% fall in LV fractional shortening, and a 33% decline in myocyte v

12 stolic blood pressure, LV ejection fraction, LV fractional shortening, and systolic wall thickness we

13 s revealed LV dilation, as well as decreased LV fractional shortening (CIH, 29.7+/-9.8%; HC, 37.4+/-7

14 Echocardiographic endocardial and midwall LV fractional shortening/circumferential end-systolic st

15 LV fractional shortening decreased after RVP (17 +/- 5 v

16 V interstitial collagen, and did not improve LV fractional shortening despite decreased LVES pressure

17 2 versus 2197 +/- 170 mm Hg/s; P < .05), and LV fractional shortening determined by echocardiography

18 th increased by 15% from control values, and LV fractional shortening fell by 20%.

19 For example, the mean LV fractional shortening fell by approximately two SD in

20 in children who, at baseline, had depressed LV fractional shortening (FS) or contractility; increase

21 associated with increased left ventricular (LV) fractional shortening (FS) during tilt testing, whic

22 In the pacing group, LV fractional shortening (LVFS) fell (13.4+/-1.4% versus

23 genic mice, as assessed by left ventricular (LV) fractional shortening (LVFS) and LV ejection fractio

24 igher LV mass and wall thicknesses and lower LV fractional shortening, midwall shortening, and stress

25 there was an inverse relationship of midwall LV fractional shortening to microtubule protein.

26 Midwall LV fractional shortening versus mean LV wall stress in t

27 For neuromuscular disease (n=139), lower LV fractional shortening was associated equally with bot

28 In the GHS group, LV fractional shortening was higher (29+/-2%) and LV pea

29 In the pacing CHF and GH groups, LV fractional shortening was higher and LV wall stress l

30 In the absence of propranolol, the LV fractional shortening was higher in TG compared with

31 In ART+ infants, LV fractional shortening was higher than in ART- infants

32 With RVP plus ETA blockade, LV fractional shortening was increased (33 +/- 6%) and e

33 With pacing CHF, LV fractional shortening was reduced (19+/-1 versus 45+/

34 In the untreated pacing CHF group, LV fractional shortening was reduced (21+/-2% versus 47+

35 In the untreated pacing CHF group, LV fractional shortening was reduced and peak wall stres

36 ness were lower whereas LV contractility and LV fractional shortening were higher when compared to th

37 a lower nadir CD4 percentage had lower mean LV fractional shortening z scores, whereas the mean z sc

38 lower cardiac function (LV contractility and LV fractional shortening z scores; all P = .001) and an

39 e heart failure, and lower left ventricular (LV) fractional shortening z score were independently ass

40 positively associated with left ventricular (LV) fractional shortening (z-score for difference = 1.07

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