ライフサイエンスコーパス: LVEDP

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1 LVEDP was elevated after L-NMMA and hemoglobin but reduc

2 relationship between increases in [ADP] and LVEDP was found (r2=.66, P=.001).

3 erfusion caused a marked increase in CPP and LVEDP and a decrease in dLVP, indicating severe cardiac

4 ) prevented the reduction in +dP/dt, Ecs and LVEDP.

5 ing (201)Tl LHR was calculated manually, and LVEDP was obtained at catheterization.

6 iac output (340+/-20/880+/-40.3 mL/min), and LVEDP (11.3+/-2.2/0.3+/-2.9 mm Hg).

7 P) to systolic blood pressure (SBP): [(DBP - LVEDP)/SBP] x 100.

8 alysis of PFR, TPFR, and 1/3FR for detecting LVEDPs of >or=18 mm Hg showed areas under the curve of 0

9 cificity, and predictive power for detecting LVEDPs of >or=18 mm Hg.

10 lidocaine, blood pressure, HR, LVSP, dp/dt, LVEDP and ESPVR decreased in CHF rats whereas lidocaine

11 c volumes and further increased the elevated LVEDP, neither of which was seen in sham rats.

12 The area under the curve for LVEDP during reperfusion was smaller in CP plus Ran vers

13 Area under the curve for LVEDP during the entire ischemic period was also smaller

14 maintained <40 micromol/L, and no change in LVEDP was observed.

15 The increase in LVEDP was closely related to the increase in free [MgADP

16 ring demand ischemia when DCS had increased (LVEDP pretachycardia versus posttachycardia, 15+/-1 vers

17 veloped left ventricular pressure (dLVP=LVSP-LVEDP), ischemia-reperfusion caused a marked increase in

18 The percent increase (from baseline) of LVEDP measured at the end of 30-minute reperfusion was s

19 in left ventricular end diastolic pressure (LVEDP) and 38% increase in the time constant of pressure

20 sed left ventricular end-diastolic pressure (LVEDP) and left ventricular end-diastolic volume (preloa

21 of left ventricular end-diastolic pressure (LVEDP) by > or =20 mm Hg (ie, cardiac contracture).

22 of left ventricular end-diastolic pressure (LVEDP) in LVH during 2DG perfusion, and this increase wa

23 little effect on LV end-diastolic pressure (LVEDP) or the end-diastolic P-V relationship (EDPVR) in

24 and left ventricular end-diastolic pressure (LVEDP) to systolic blood pressure (SBP): [(DBP - LVEDP)/

25 P), left ventricular end-diastolic pressure (LVEDP), and developed left ventricular pressure (dLVP=LV

26 d cardiac output, LV end-diastolic pressure (LVEDP), and peak LV pressure (LVPmax).

27 ced left ventricular end-diastolic pressure (LVEDP), but not left ventricular end-diastolic volume, c

28 g cardiac output, LV end-diastolic pressure (LVEDP), rate of pressure rise at LV pressure of 40 mm Hg

29 ted left ventricular end-diastolic pressure (LVEDP), which adds prognostic value in CAD.

30 HF (left ventricular end diastolic pressure (LVEDP): 6 +/- 1 versus 14 +/- 1 mmHg, respectively, P <

31 mic left ventricular end-diastolic pressure [LVEDP] increased 10 mm Hg, P<0.001, n=38).

32 ilat did not inhibit contractility or reduce LVEDP during dobutamine infusion.

33 Ran added to CP reduced LVEDP at the end of ischemia from 41+/-5 mm Hg in CP alo

34 , n=6) reduced DCS to pretachycardia values (LVEDP post-QSR, 15+/-1 mm Hg, P<0.001), ie, elicited a r

35 ftware showed a significant correlation with LVEDP.

36 PFR, and 1/3FR correlated significantly with LVEDP (r= -0.53, 0.45, and -0.45, respectively; P=0.0000

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