ライフサイエンスコーパス: LVI

1 ing microvial insert large volume injection (LVI).

2 cular spaces termed lymphovascular invasion (LVI).

3 high grade without lymphovascular invasion (LVI).

4 sis, all factors remained significant except LVI.

5 breast cancer who have grade 3 tumors and/or LVI.

6 et)) levels of spontaneous metastasis but no LVI.

7 re widely negative at > 5 mm and there is no LVI.

8 in the presence of lymphovascular invasion (LVI; 0.25 +/- 0.02 v 0.17 +/- 0.02; P < .0001) or lymph

9 75 mm, MR >/= 1, presence of ulceration, and LVI (all P = .001) were significantly associated with se

10 Our findings suggest including LVI and regression as new prognostic factors in the mela

11 carcinoma (IC), or lymphovascular invasion (LVI), and 8% lobular neoplasia (lobular carcinoma in sit

12 Lung weight index (LWI), lung volume index (LVI), and alveolar cell proliferation index (CPI) were m

13 ration, blood and lymphatic vessel invasion (LVI), and lymph node and lung metastasis.

14 e (MR), ulceration, lymphovascular invasion (LVI), and regression; incidence was lower and subgroup d

15 BE]), nodal status, lymphovascular invasion (LVI), and the presence of multifocal neoplasia (MFN) (hi

16 in further significant enhancements of LWI, LVI, and CPI in group PK.

17 As expected, LWI, LVI, and CPI were significantly increased after pneumone

18 in adult rats as indicated by increased LWI, LVI, and CPI.

19 The combined high incidence of MFN, LVI, and occult nodal metastases does not support the us

20 R >/= 1, presence of ulceration, presence of LVI, and regression >/= 50%.

21 breast tumors is essential for BVI, but not LVI, and that MT1-MMP should be further explored as a pr

22 th postcolumn infusions) and compared across LVI- and SPE-based methods at constant (high and low) an

23 nts with associated lymphovascular invasion (LVI) are at increased risk of occult metastases.

24 esults from this study demonstrated that the LVI-based method produced analytical signals of quality

25 ted responses of interneurons in LII-III and LVI completely desensitized, while cholinergic responses

26 All 9 patients with LVI had T1b tumors.

27 atrixes using direct large-volume injection (LVI) high-performance liquid chromatography (HPLC) tande

28 rs are younger age, lymphovascular invasion (LVI), high Ki-67, and larger tumors within the T1a,b sub

29 versely, there was a significant decrease in LVI in re-stented segments B and C (P<0.05).

30 Prevalence of LVI increased with higher pathologic stage (9.0%, 23%, 6

31 LVI is an independent predictor of recurrence and decrea

32 crete sequence of three amino acid residues (LVI), located at the carboxyl-terminal end of the impair

33 LVI maintained its independent predictor status in compe

34 Patients with high-grade tumors and/or LVI may have 10-year RFS rates of less than 75% in the a

35 status, presence of lymphovascular invasion (LVI), number of SLN(s) identified, number of positive SL

36 to sham-operated animals did not alter LWI, LVI, or CPI.

37 P expression in lymphatic endothelial cells, LVI, or lymph node metastasis.

38 ith T1a and 25 of 45 (58%) with T1b had MFN, LVI, or nodal metastases.

39 de of both DCIS (P = .002) and IC (P = .03), LVI (P = .03), and lymph node involvement (P = .02) by u

40 tension (P < 0.001), tumor size (P = 0.001), LVI (P = 0.019), and histology (P = 0.034).

41 Furthermore, LVI presented a clear advantage over SPE because it was

42 cholinergic responses in LV interneurons and LVI pyramidal cells showed less desensitization.

43 ized by exaggerated lymphovascular invasion (LVI), recapitulated in our human xenograft, MARY-X.

44 Age, MR, ulceration, LVI, regression, and sentinel node status were independe

45 with transitional-cell carcinoma (n = 776), LVI status was available for 750.

46 combining the 4,871 and 8,596 Da peaks with LVI, the area under the receiver operating characteristi

47 e, and number of pelvic lymph nodes removed, LVI was an independent predictor of local (HR = 2.03, P

48 LVI was an independent predictor of overall (HR = 1.84,

49 LVI was defined as the presence of tumor cells within an

50 In patients treated with (192)Ir radiation, LVI was maintained from baseline to follow-up only in no

51 LVI was not a predictor of recurrence or survival in nod

52 LVI was performed by the direct injection of 900 muL of

53 LVI was present in 36.4% (273 of 750) overall, involving

54 ed segment A (P<0.05).At follow-up, however, LVI was similar in all 4 segments secondary to the incre

55 onmental analysis by large-volume injection (LVI) was compared to solid-phase extraction (SPE) based

56 tients, postintervention lumen volume index (LVI) was significantly greater in re-stented segments B

57 Lymphovascular invasion (LVI) was the only clinicopathologic factor predictive of

58 Serum DNA integrity and LVI were significant for predicting LN metastasis in a m