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1 c neuromuscular junction disorder resembling Lambert-Eaton myasthenic syndrome.
2 ome, dermatomyositis, myasthenia gravis, and Lambert-Eaton myasthenic syndrome.
3 ved in an autoimmune neuromuscular disorder, Lambert-Eaton myasthenic syndrome.
4 ion sometimes occurs in association with the Lambert-Eaton myasthenic syndrome.
5 s developed electrophysiologically confirmed Lambert-Eaton myasthenic syndrome.
6 ved in the autoimmune neuromuscular disorder Lambert-Eaton myasthenic syndrome.
7 diseases, such as diabetes mellitus and the Lambert-Eaton myasthenic syndrome.
8 ption of the opsoclonus-myoclonus ataxia and Lambert-Eaton myasthenic syndromes.
9 limbic encephalitis, 3 cerebellar ataxia, 2 Lambert-Eaton myasthenic syndrome, 1 autonomic neuropath
10 and demonstrated an autoimmune basis for the Lambert Eaton myasthenic syndrome and 'seronegative' mya
12 d GV-58 in a passive transfer mouse model of Lambert-Eaton myasthenic syndrome and have shown that we
13 ced improvement in several patients with the Lambert-Eaton myasthenic syndrome and myasthenia gravis
15 ataxia type 2, spinocerebellar ataxia 6, and Lambert-Eaton myasthenic syndrome), and the skeletal mus
16 ciated with Waldenstrom's macroglobulinemia, Lambert-Eaton myasthenic syndrome, and multifocal motor
18 ation patients without clinically identified Lambert-Eaton myasthenic syndrome had P/Q-type voltage-g
19 ubtype that is targeted by antibodies in the Lambert-Eaton myasthenic syndrome has been identified, a
22 iated with small-cell lung cancer, including Lambert-Eaton myasthenic syndrome (LEMS) and paraneoplas
26 d/or signs were present in 18 of 23 (78%) of Lambert-Eaton myasthenic syndrome (LEMS) patients evalua
30 thenic syndrome and have shown that weakened Lambert-Eaton myasthenic syndrome-model neuromuscular sy
31 t, some PNSs such as sensory neuronopathy or Lambert-Eaton myasthenic syndrome rarely occur in lympho
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