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1 Lambert Eaton myasthenic syndrome and acquired neuromyot
2 Lambert et al. make clever use of psoriasis patient gene
3 Lambert et al. reported that silicate ions catalyze the
4 Lambert-Eaton myasthenic syndrome (LEMS) is a paraneopla
5 Lambert-Eaton myasthenic syndrome (LEMS) is an autoimmun
6 Lambert-Eaton myasthenic syndrome (LEMS) is an autoimmun
7 Lambert-Eaton myasthenic syndrome IgG did not selectivel
8 mplitudes were correlated with decibel and 1/Lambert DLS for the central 18 degrees of the visual fie
9 ensitivities (both in logarithmic [dB] and 1/Lambert scales [1/L]), were investigated with components
10 There was a linear correlation between 1/Lambert DLS and PERG amplitude (transient PERG: R(2) = 0
11 im area, and a linear relationship between 1/Lambert DLS and PERG amplitude and neuroretinal rim area
12 There was a linear correlation between 1/Lambert DLS and temporal neuroretinal rim area (R(2) = 0
13 .75%, and -0.78% per year for decibel DLS, 1/Lambert DLS, transient PERG, and SS PERG, respectively.
16 limbic encephalitis, 3 cerebellar ataxia, 2 Lambert-Eaton myasthenic syndrome, 1 autonomic neuropath
17 ataxia type 2, spinocerebellar ataxia 6, and Lambert-Eaton myasthenic syndrome), and the skeletal mus
23 microscopic optical path lengths, both Beer-Lambert's law and the law of superposition were found to
24 e obtained, quantitatively obeying both Beer-Lambert's law and the law of superposition, despite the
26 on in the ITO, causing a deviation from Beer-Lambert absorption that results in an optimum ITO thickn
28 on plots are assessed in the context of Beer-Lambert's law and provide combined with time-dependent d
30 in coincide with those predicted by the Beer-Lambert law for a range of ferricyanide concentrations f
41 o coimmunogenic tumor antigens, for example, Lambert-Eaton syndrome (P/Q-type Ca(2+) channel antibody
42 ation patients without clinically identified Lambert-Eaton myasthenic syndrome had P/Q-type voltage-g
45 iated with small-cell lung cancer, including Lambert-Eaton myasthenic syndrome (LEMS) and paraneoplas
47 ciated with Waldenstrom's macroglobulinemia, Lambert-Eaton myasthenic syndrome, and multifocal motor
48 d/or signs were present in 18 of 23 (78%) of Lambert-Eaton myasthenic syndrome (LEMS) patients evalua
49 d GV-58 in a passive transfer mouse model of Lambert-Eaton myasthenic syndrome and have shown that we
51 t, some PNSs such as sensory neuronopathy or Lambert-Eaton myasthenic syndrome rarely occur in lympho
53 syn I, DeMarini DM, Caldwell JC, Kavlock RJ, Lambert P, Hecht SS, Bucher JR, Stewart BW, Baan R, Cogl
58 and demonstrated an autoimmune basis for the Lambert Eaton myasthenic syndrome and 'seronegative' mya
59 ubtype that is targeted by antibodies in the Lambert-Eaton myasthenic syndrome has been identified, a
61 ced improvement in several patients with the Lambert-Eaton myasthenic syndrome and myasthenia gravis
64 ls of gabapentin funded by Pfizer and Warner-Lambert's subsidiary, Parke-Davis (hereafter referred to
65 thenic syndrome and have shown that weakened Lambert-Eaton myasthenic syndrome-model neuromuscular sy
66 ample, P/Q-type Ca(2+)-channel antibody with Lambert-Eaton syndrome (n = 5), anti-neuronal nuclear an
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