ライフサイエンスコーパス: Langerhans' cell

1 of skin langerin-expressing cells (including Langerhans cells).

2 facilitate the interaction of M. leprae with Langerhans cells.

3 The latter also included CD1a(bright) Langerhans cells.

4 ts internalization in specific organelles of Langerhans cells.

5 of VIP on TLR regulation in macrophages and Langerhans cells.

6 elocalization toward the basal epidermis and Langerhans cells.

7 s the only other cytokine expressed by human Langerhans cells.

8 se T cells recognize CD1a-lipid complexes on Langerhans cells.

9 nterleukin 22 (IL-22) in response to CD1a on Langerhans cells.

10 1c(+)CD1a(+) dermal DCs but not to epidermal Langerhans cells.

11 mice induced in situ maturation of epidermal Langerhans cells.

12 itive dendritic cells, or by radio-resistant Langerhans cells.

13 as they migrate and differentiate into oral Langerhans cells.

14 DC maturation and no binding was detected to Langerhans cells.

15 al epithelium-resident cells that are likely Langerhans cells.

16 al melanocyte hyperplasia and intraepidermal Langerhans cells.

17 ve intraepithelial macrophages and dendritic/Langerhans cells.

18 evealed the presence of GPR109A on epidermal Langerhans cells.

19 conventional DC subsets and not by epidermal Langerhans cells.

20 DCs, including a population of inflammatory Langerhans cells.

21 ing molecule that is abundantly expressed on Langerhans cells.

22 diated expression of interleukin-6 (IL-6) by Langerhans cells.

23 s (LCH) represents a clonal proliferation of Langerhans cells.

24 lation by UVB radiation, because ablation of Langerhans cells abolished the UVB-induced phenotype.

25 Mice that are deficient in epidermal Langerhans cells allow the functions of these cells in v

26 dent manner, suggesting an essential role of Langerhans cells and dendritic cells in CHQ-provoked pso

27 dermally delivered TLR agonists to stimulate Langerhans cells and dermal DCs in their natural complex

28 he MCs was efficiently taken up by epidermal Langerhans cells and dermal dendritic cells in the vicin

29 This infection of Langerhans cells and interstitial dermal dendritic cells

30 es tested whether endotoxin (LPS) stimulated Langerhans cells and macrophages (Mphi; from B6 mice) pr

31 oinflammatory cytokines, and accumulation of Langerhans cells and macrophages within 3 days of tamoxi

32 onuclear cells that are direct precursors to Langerhans cells and that EBV both latently and producti

33 In contrast, epidermal Langerhans cells and tissue macrophages were largely pre

34 s highly expressed by DC purified from skin (Langerhans cells) and bone marrow, and has been shown to

35 upffer cells), brain (microglia), epidermis (Langerhans cells) and lung (alveolar macrophages) origin

36 cluding ex vivo-generated DCs, skin-isolated Langerhans cells, and blood myeloid DCs and plasmacytoid

37 utrophils, macrophages, dendritic cells, and Langerhans cells, and colocalization of vaccine Ag withi

38 of plasmacytoid DCs (pDCs), CD8alpha(+) DCs, Langerhans cells, and conventional myeloid DCs and discu

39 ll populations including endogenous T cells, Langerhans cells, and gammadelta T cells were not requir

40 tic cells: monocyte-derived dendritic cells, Langerhans cells, and interstitial dermal dendritic cell

41 in regulates the maturation and migration of Langerhans cells, and its ligation prevents the inductio

42 receptor, expressed primarily on adipocytes, Langerhans cells, and macrophage.

43 cytes, fibroblasts, melanocytes, mast cells, Langerhans cells, and Meissner's corpuscles, as well as

44 sues, such as liver Kupffer cells, epidermal Langerhans cells, and microglia--cell populations that a

45 irect fusion, without requiring passage from Langerhans cells, and overt productive infection ensued.

46 r with myeloid cell or with dedifferentiated Langerhans cell antigens, carry the BRAFV600E mutation.

47 Activation via RANKL prevents Langerhans cell apoptosis, thus leading to enhanced anti

48 Langerhans cells appeared in the dermis of skin borderin

49 Dermal dendritic cells and epidermal Langerhans cells are APCs that migrate from skin to drai

50 However, Flt3L-dependent DCs and resident Langerhans cells are dispensable for the inflammation.

51 Kupffer cells, microglia and Langerhans cells are only marginally replaced in one-yea

52 migratory dendritic cells and in particular Langerhans cells at governing T follicular helper and ge

53 Langerin, an endocytic receptor of Langerhans cells, binds pathogens such as human immunode

54 cific pattern recognition receptor TLR7, the Langerhans cell chemoattractant CCL20, and proinflammato

55 logical heterogeneity and differentiation of Langerhans cells, delineate the signaling pathways respo

56 No differences were identified in corneal Langerhans cell density (19.84 cells/mm2 for the control

57 corneal nerve fiber tortuosity, and corneal Langerhans cell density between healthy controls and pat

58 ree main cutaneous DC populations: epidermal Langerhans cells, dermal myeloid DCs, and dermal plasmac

59 efects, multiorgan inflammation, and lack of Langerhans cells) despite production of normal levels of

60 eraction with vaginal epithelial T cells and Langerhans cells early after infection.

61 g cutaneous inflammation when it facilitates Langerhans cell egress from skin and enables the accumul

62 ) mice contained normal numbers of epidermal Langerhans cells (eLC) and increased numbers of CD11b(+)

63 Epidermal Langerhans cells (eLCs) uniquely express the C-type lect

64 inophils, macrophages, osteoclasts, DCs, and Langerhans cells from human embryonic stem cells (hESCs)

65 ify possible features that can differentiate Langerhans cells from malignant melanocytes to prevent t

66 eric C-type lectin specifically expressed in Langerhans cells, has been reported to be a pathogen rec

67 Precursors of pathological Langerhans cells have yet to be defined.

68 ations have been observed in 57% of cases of Langerhans cell histiocytosis (LCH) and 54% of cases of

69 novel somatic ARAF mutation in a child with Langerhans cell histiocytosis (LCH) and demonstrate that

70 enesis of 2 of the most common histiocytoses-Langerhans cell histiocytosis (LCH) and Erdheim-Chester

71 Langerhans cell histiocytosis (LCH) and Erdheim-Chester

72 have been observed in half of patients with Langerhans cell histiocytosis (LCH) and in 50% to 100% o

73 heterogeneous diseases that mostly comprise Langerhans cell histiocytosis (LCH) and non-LCH.

74 Langerhans cell histiocytosis (LCH) and the non-LCH neop

75 Langerhans cell histiocytosis (LCH) combines in one noso

76 Langerhans cell histiocytosis (LCH) has a broad spectrum

77 nase (ERK) signaling pathway is activated in Langerhans cell histiocytosis (LCH) histiocytes, but onl

78 atients with refractory, risk-organ-positive Langerhans cell histiocytosis (LCH) in 2005.

79 There is little data on treatment of Langerhans cell histiocytosis (LCH) in adults.

80 Langerhans cell histiocytosis (LCH) is a clinically and

81 Langerhans cell histiocytosis (LCH) is a clonal disorder

82 Langerhans cell histiocytosis (LCH) is a myeloproliferat

83 Langerhans cell histiocytosis (LCH) is a rare disease af

84 Langerhans cell histiocytosis (LCH) is a rare disease ch

85 Langerhans cell histiocytosis (LCH) is a rare disease wi

86 Langerhans cell histiocytosis (LCH) is a rare histiocyti

87 Langerhans cell histiocytosis (LCH) is an enigmatic dise

88 Langerhans cell histiocytosis (LCH) is an inflammatory m

89 Langerhans cell histiocytosis (LCH) is an inflammatory m

90 Langerhans cell histiocytosis (LCH) is characterized by

91 Langerhans cell histiocytosis (LCH) represents a clonal

92 ing data to date on a group of patients with Langerhans cell histiocytosis (LCH), which historically

93 Langerhans cell histiocytosis (LCH)-III tested risk-adju

94 BRAF mutations have been observed in Langerhans cell histiocytosis (LCH).

95 9 patients with ECD and ECD overlapping with Langerhans cell histiocytosis (so-called mixed histiocyt

96 Included among them are Langerhans cell histiocytosis and hemophagocytic lymphoh

97 hough lymphangioleiomyomatosis and pulmonary Langerhans cell histiocytosis are perhaps more frequentl

98 Erdheim-Chester disease (ECD) is a rare non-Langerhans cell histiocytosis that most commonly affects

99 d eruptive histiocytosis (GEH) is a rare non-Langerhans cell histiocytosis with a benign, self-healin

100 "Langerhans cell histiocytosis" (LCH) describes a spectru

101 most common histiocytic disorders, including Langerhans cell histiocytosis, Erdheim-Chester disease,

102 Erdheim-Chester disease (ECD) is a rare non-Langerhans cell histiocytosis, to whose pathogenesis neo

103 those of histiocytic human diseases, such as Langerhans cell histiocytosis.

104 ytic conditions, Erdheim-Chester disease and Langerhans cell histiocytosis.

105 lymphomas, neuroblastoma, some sarcomas, and Langerhans cell histiocytosis.

106 us on lymphangioleiomyomatosis and pulmonary Langerhans cell histiocytosis.

107 n the cohort with Erdheim-Chester disease or Langerhans'-cell histiocytosis, the response rate was 43

108 ng cancer and in Erdheim-Chester disease and Langerhans'-cell histiocytosis.

109 ted from human blood and differentiated into Langerhans cells (hLC-L) also showed GPR109A expression.

110 scopy of the t.c. immunization site revealed Langerhans cells in areas of the skin containing the Abe

111 led an increased number of epidermal CD83(+) Langerhans cells in FLG-null subjects.

112 Although Langerhans cells in LCH are clonal, their benign morphol

113 NTB failed to induce emigration of epidermal Langerhans cells in naive individuals.

114 cts with FLG-null mutations have more mature Langerhans cells in nonlesional skin irrespective of whe

115 y an abnormality in corneal nerve fibers and Langerhans cells in patients with and without HIV-SN.

116 IL-10 production by (regulatory) T cells and Langerhans cells in regulating CHS has been established

117 Flt3L augmented the appearance of Langerhans cells in response to both injury and infectio

118 rai and coworkers explore the involvement of Langerhans cells in skin graft rejection and describe fa

119 ection against HIV infection to skin-derived Langerhans cells in the ex vivo system, suggesting Marav

120 arbohydrate-dependent uptake of pathogens by Langerhans cells in the first step of antigen presentati

121 ated by the release of prostaglandin D2 from Langerhans cells in the skin.

122 oid and myeloid DCs, but also from epidermal Langerhans cells, indicating a distinct DC entity.

123 over NL4-3 in immature MDDCs and in ex vivo Langerhans cells, indicating that these laboratory-adapt

124 f Th1 and Th2 polarization in the setting of Langerhans cell (LC) Ag presentation to responsive T cel

125 vasoactive intestinal peptide (VIP) suppress Langerhans cell (LC) antigen presentation and modulate c

126 ssociated with systemic effects on epidermal Langerhans cell (LC) function and, specifically, the mig

127 RC1 but not mTORC2 is required for epidermal Langerhans cell (LC) homeostasis.

128 Cs in SDLNs, and augmented TRITC/DBP-induced Langerhans cell (LC) migration 72 hours post TRITC treat

129 e required for CCL19 production and adequate Langerhans cell (LC) migration both ex vivo and in vivo.

130 Langerhans cell (LC) networks play key roles in immunity

131 Human Langerhans cell (LC) precursors populate the epidermis e

132 In this study, the role of the epidermal Langerhans cell (LC) subset of skin dendritic cells in t

133 dritic cells (DC) in the skin and mucosa are Langerhans cells (LC) and interstitial dermal DC (iDDC).

134 1-negative adults and neonates (moDC) and by Langerhans cells (LC) and interstitial, dermal-intestina

135 ransit through the epidermis, which contains Langerhans cells (LC) and keratinocytes (KC), among othe

136 Langerhans cells (LC) and other antigen-presenting cells

137 These include Langerhans cells (LC) and some subtypes of circulating b

138 Langerhans cells (LC) are a subset of skin-resident dend

139 Langerhans cells (LC) are APC that reside at the barrier

140 Langerhans cells (LC) are epidermal dendritic cells capa

141 etween these cells and langerin(+) epidermal Langerhans cells (LC) are incompletely characterized.

142 Langerhans cells (LC) are the dendritic APC population o

143 Langerhans cells (LC) are the resident APCs at the site

144 Mice lacking epidermal Langerhans cells (LC) develop exaggerated contact-hypers

145 dly, huLangerin-DTA mice (DeltaLC) that lack Langerhans cells (LC) developed increased skin inflammat

146 Epidermal Langerhans cells (LC) expressing the high-affinity recep

147 e literature regarding the role of epidermal Langerhans cells (LC) in promoting skin immune responses

148 Presentation of foreign antigen by Langerhans cells (LC) in the absence of exogenous adjuva

149 isrupted, allowing easier access of HIV-1 to Langerhans cells (LC) in the epidermis and perhaps even

150 Langerhans cells (LC) in the epidermis of patients with

151 plasmacytoid DC (pDC) and in vitro-generated Langerhans cells (LC) obtained from AD patients with HSV

152 After activation, Langerhans cells (LC), a distinct subpopulation of epide

153 ce had multiorgan inflammation, lack of skin Langerhans cells (LC), and a shortened lifespan, consist

154 MHD1 is also expressed in epidermis-isolated Langerhans cells (LC), but degradation of SAMHD1 does no

155 There are at least three subsets of skin DC- Langerhans cells (LC), Langerin(+) dermal DCs (dDCs), an

156 Tissue-resident dendritic cells, such as Langerhans cells (LC), normally carry Ags from tissues t

157 HPV does not activate Langerhans cells (LC), the APC at the site of infection,

158 During infection, HPV16 also interacts with Langerhans cells (LC), the APC of the epithelium, induci

159 Langerhans cells (LC), the dendritic cells of the epider

160 leting or altering the function of epidermal Langerhans cells (LC).

161 s--Trier public speaking test--on: epidermal Langerhans' cell (LC) frequency; and cutaneous nerve fib

162 CD207+ Langerhans cells (LCs) and CD11c+ myeloid dendritic cell

163 Langerhans cells (LCs) and CD8(+) tissue-resident memory

164 dritic cell (DC) subsets, epidermal CD207(+) Langerhans cells (LCs) and dermal CD14(+) DCs.

165 We tested the capacity of human Langerhans cells (LCs) and dermal dendritic cells (DDCs)

166 of TGF-beta1 on the differentiation of human Langerhans cells (LCs) and identified Axl as a key TGF-b

167 he two best-characterized skin-resident DCs, langerhans cells (LCs) and Langerin(+) dermal DCs (dDCs)

168 by heterogeneous lesions containing CD207(+) Langerhans cells (LCs) and lymphocytes that can arise in

169 Langerhans cells (LCs) and microglia develop from embryo

170 The development of both Langerhans cells (LCs) and microglia is highly dependent

171 for all mouse DC subsets revealed that human Langerhans cells (LCs) and the mouse XCR1(+)CD8alpha(+)C

172 Langerhans cells (LCs) are a distinct population of dend

173 Langerhans cells (LCs) are a subset of dendritic cells (

174 Langerhans cells (LCs) are able to orchestrate adaptive

175 Langerhans cells (LCs) are antigen-presenting cells that

176 Langerhans cells (LCs) are bone marrow (BM)-derived epid

177 Langerhans cells (LCs) are dendritic cells (DCs) localiz

178 Langerhans cells (LCs) are dendritic cells (DCs) residin

179 eport that dermal dendritic cells (DDCs) and Langerhans cells (LCs) are differentially mobilized duri

180 Langerhans cells (LCs) are distinct dendritic cells (DCs

181 Langerhans cells (LCs) are epidermis-resident antigen-pr

182 Langerhans cells (LCs) are epithelial APCs that sense da

183 esident member of the dendritic cell family, Langerhans cells (LCs) are generally regarded to functio

184 Human Langerhans cells (LCs) are highly efficient at priming c

185 Langerhans cells (LCs) are immediate targets of exogenou

186 Langerhans cells (LCs) are known as "sentinels" of the i

187 We recently reported that human epidermal Langerhans cells (LCs) are more efficient than dermal CD

188 Langerhans cells (LCs) are myeloid cells of the epidermi

189 Langerhans cells (LCs) are professional antigen-presenti

190 Langerhans cells (LCs) are self-renewing epidermal myelo

191 Langerhans cells (LCs) are self-renewing in the steady s

192 Langerhans cells (LCs) are skin-resident dendritic cells

193 Langerhans cells (LCs) are the dendritic cells (DCs) of

194 Langerhans cells (LCs) are the only DC subset in the hea

195 Langerhans cells (LCs) are the only dendritic cells of t

196 Langerhans cells (LCs) are the sole dendritic cell type

197 Langerhans cells (LCs) are the unique dendritic cells fo

198 Langerhans cells (LCs) can be targeted with DNA-coated g

199 Langerhans cells (LCs) comprise a dendritic network with

200 Langerhans cells (LCs) comprise a network of dendritic c

201 Langerhans cells (LCs) constitute a network of immune se

202 Following MN immunization, Langerhans cells (LCs) constituted the major skin DC sub

203 Human epidermal and mucosal Langerhans cells (LCs) express the C-type lectin recepto

204 dent DCs remain controversial, and epidermal Langerhans cells (LCs) have been referred to recently as

205 Although Langerhans cells (LCs) have been reported to express IL-

206 Langerhans cells (LCs) have been shown to be sufficient

207 antagonist, to block HIV infection of human Langerhans cells (LCs) in an epithelial environment that

208 However, the roles of skin-resident Langerhans cells (LCs) in eliciting immune responses hav

209 The precise role of human epidermal Langerhans cells (LCs) in immune response is highly cont

210 Since their discovery in 1868, the role of Langerhans cells (LCs) in skin immunity has been researc

211 ency virus type 1 (HIV-1) is internalized by Langerhans cells (LCs) in stratified epithelia and trans

212 Although implied by other models, proof that Langerhans cells (LCs) in the human vagina participate i

213 Understanding the function of Langerhans cells (LCs) in vivo has been complicated by c

214 Langerhans cells (LCs) induce type 2 antibodies reactive

215 Langerhans cells (LCs) may function as inducers of immun

216 assumptions on the identity and functions of Langerhans cells (LCs) of the epidermis have undergone c

217 Langerhans cells (LCs) of the epidermis, although of mye

218 Epidermal Langerhans cells (LCs) of the skin represent the prototy

219 The ontogeny of human Langerhans cells (LCs) remains poorly characterized, in

220 nfection, we showed that in vivo ablation of Langerhans cells (LCs) resulted in enhanced bone loss.

221 Langerhans cells (LCs) serve as epidermal sentinels of t

222 We tested the contribution of mucosal Langerhans cells (LCs) to alveolar bone homeostasis in m

223 with monocyte-derived DCs (MDDCs) and MUTZ3 Langerhans cells (LCs) to investigate their relevance as

224 In vivo studies questioned the ability of Langerhans cells (LCs) to mediate CD8(+) T cell priming.

225 regulates the outcome of Ag presentation by Langerhans cells (LCs) to T cells through actions on mic

226 To determine the relative contribution of Langerhans cells (LCs) to the ensuing GVHD-like reaction

227 tion of the epidermal mononuclear phagocytes Langerhans cells (LCs) to this phenomenon because of the

228 At steady state, Langerhans cells (LCs) were lineage traced but also expr

229 ited in recent years, and the involvement of Langerhans cells (LCs), a population of epidermal dendri

230 Mice lacking Langerhans cells (LCs), a signatory epidermal dendritic

231 interplay of the C-type lectins, Langerin on Langerhans cells (LCs), and dendritic cell-specific inte

232 e fate of cutaneous DCs (cDCs), specifically Langerhans cells (LCs), and observed that they undergo a

233 A specialized subset of DCs, the Langerhans cells (LCs), are located in the stratified sq

234 s used to identify human and mouse epidermal Langerhans cells (LCs), as well as migratory LCs in the

235 Based on our ability to discriminate Langerhans cells (LCs), conventional DCs, monocytes, mon

236 ered intradermally are taken up by epidermal Langerhans cells (LCs), dermal Langerin(neg) DCs, and de

237 ts of skin-resident DC have been identified: Langerhans cells (LCs), dermal Langerin+ DC (Lang+ dDC),

238 Skin-resident DCs, namely, Langerhans cells (LCs), have been implicated in regulati

239 n two distinct microanatomical compartments: Langerhans cells (LCs), mainly in the epidermis, and der

240 In addition to Langerhans cells (LCs), other dendritic cells (CD11c(+))

241 ance in mice critically depends on epidermal Langerhans cells (LCs), which capture DNTB and migrate t

242 ers the morphology and function of epidermal Langerhans cells (LCs), which play a role in UV-induced

243 for maintaining the homeostasis of epidermal Langerhans cells (LCs).

244 ltrastructural features similar to cutaneous Langerhans cells (LCs).

245 intraepidermal dendritic cells (DCs) called Langerhans cells (LCs).

246 ation of graft antigen by resident epidermal Langerhans cells (LCs).

247 henotypic characteristics of human epidermal Langerhans cells (LCs).

248 s; CD1c), plasmacytoid DCs (pDCs; CD303) and Langerhans cells (LCs; CD1a, CD207) in uncinate tissue (

249 Using FACS, we isolated Langerhans cells (LCs; HLA-DR(+)CD207(+) cells) and derm

250 Langerhans' cells (LCs) are a subset of periphery reside

251 host-derived DCs in CLNs was consistent with Langerhans' cells (LCs).

252 matic disease defined by the accumulation of Langerhans cell-like dendritic cells (DCs).

253 nd T-helper type 1 skewing function in human Langerhans cell-like dendritic cells (LC-DCs).

254 o neutrophils, eosinophils, dendritic cells, Langerhans cells, macrophages and osteoclasts.

255 -derived IL-1beta alone reduced infection of Langerhans cells, macrophages, and dermal dendritic cell

256 culation led to recruitment and infection of Langerhans cells, macrophages, and dermal dendritic cell

257 Langerhans cells, macrophages, and T lymphocytes were st

258 NV infected a wide range of cells, including Langerhans cells, macrophages, dermal dendritic cells, m

259 A-DR, CD83, costimulatory molecules, and the Langerhans cell marker CD1a, whereas pDC expressed low l

260 Unconventional immune regulatory roles for Langerhans cells, mast cells, and natural killer T (NKT)

261 The same receptor in skin Langerhans cells mediates the common flushing side effec

262 Langerin, a C-type lectin on Langerhans cells, mediates carbohydrate-dependent uptake

263 response to oxazolone and oxazolone-induced Langerhans cell migration from epidermis were both preve

264 The presence of hyperreflective Langerhans cells mimicking malignant melanocytes was the

265 Strikingly, using Langerhans cells model systems (mutz-3-derived LC, monoc

266 Together, these data suggest that Langerhans cell/Mphi recognition of microbial LPS regula

267 langerin (Lang; CD207)(neg) DCs, but neither Langerhans cells nor Lang(+) DCs were required for CD8(+

268 is functional, resulting in slightly reduced Langerhans cell numbers.

269 uman immunodeficiency virus-1) transmission, Langerhans cells of genital mucosa play a protective rol

270 terstitial dendritic cells of the dermis and Langerhans cells of the epidermis, in a dose- and time-d

271 Langerhans cells participate in the immune response in l

272 st cells, basophils, eosinophils, monocytes, Langerhans cells, platelets, and neutrophils.

273 ood to oral epithelium in which EBV-infected Langerhans cell precursors serve to transport EBV to the

274 nces in the numbers of T or B lymphocytes or Langerhans cells present in StrataGraft skin substitute

275 By contrast, HIV-1 entered CD1a(+) Langerhans cells primarily by endocytosis, by means of m

276 utrophils, macrophages, dendritic cells, and Langerhans cells remained in the tissue at least 1 wk.

277 y factors (IRFs) as controllers of the human Langerhans cell response to epidermal cytokines was reve

278 e effect by interacting with GPR109A on skin Langerhans cells, resulting in release of PGD(2).

279 Antigen-presenting Langerhans cells show a differential migration phenotype

280 okine for mature DCs) in vitro, and in vivo, Langerhans cells show reduced emigration into draining l

281 Skin-migratory Langerhans cells (smiLCs) were the main cutaneous DC sub

282 Langerhans cells stained for phospho-mitogen-activated p

283 1 TCRgammadelta+ intraepithelial T cells and Langerhans cells, swiftly followed by epithelial infiltr

284 Langerhans cells take up antigen applied to skin from wh

285 Langerin is a C-type lectin present on Langerhans cells that mediates capture of pathogens in a

286 ology of the skin, including the function of Langerhans cells, the migration of immune cells in skin,

287 The working theory is that niacin provokes Langerhans cells to produce prostaglandin D2 (PGD2), sti

288 )5V(delta)1+ T cells limited carcinogenesis, Langerhans cells unexpectedly promoted it.

289 s, mast cells, eosinophils, macrophages, and Langerhans cells; upregulation of chemokine and cytokine

290 epithelial, monocyte-derived dendritic, and Langerhans cells via direct cell-cell transmission.

291 of Flt3L at the burn wound, localization of Langerhans cells was examined.

292 Increased numbers of CD1a-expressing Langerhans cells were detected both in the epithelium an

293 Langerhans cells were required for eliciting immune resp

294 DCs were able to cross-prime CD8(+) T cells, Langerhans cells were unexpectedly found to potently cro

295 ted production of prostaglandin D2 and E2 in Langerhans' cells which act on DP1 and EP2/4 receptors i

296 These two markers are mainly expressed by Langerhans cells, which are one of several functionally

297 angerin and the ephrin A2 receptor to infect Langerhans cells, which support full HHV-8 lytic replica

298 S-100-positive cells included CD1a-positive Langerhans cells, while CK20 did not identify any Merkel

299 althy skin biopsies, human keratinocytes and Langerhans cells with IL-4.

300 angerin, the distinguishing C-type lectin of Langerhans cells, would recognize the highly mannosylate