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1 which RGCs are specifically affected (e.g., Leber hereditary optic neuropathy).
2 may be useful for treatment of patients with Leber hereditary optic neuropathy.
3 the optic nerve degeneration associated with Leber hereditary optic neuropathy.
4 truction and demyelination, the hallmarks of Leber hereditary optic neuropathy.
5 xpression opens the door for gene therapy of Leber Hereditary Optic Neuropathy.
6 y be safe for the treatment of patients with Leber hereditary optic neuropathy.
7 To produce a mouse model of Leber hereditary optic neuropathy.
9 ly, we apply our methods to three data sets: Leber hereditary optic neuropathy, a disease that has be
11 of human neurodegenerative diseases such as Leber hereditary optic neuropathy and Parkinson's diseas
14 D6) gene has been identified as the cause of Leber hereditary optic neuropathy (LHON) and/or pediatri
15 developed a novel strategy for treatment of Leber hereditary optic neuropathy (LHON) caused by a mut
16 Pedigree analysis of British families with Leber hereditary optic neuropathy (LHON) closely fits a
17 ant human mitochondrial gene responsible for Leber hereditary optic neuropathy (LHON) into the mouse
22 Establishing the natural history of G11778A Leber hereditary optic neuropathy (LHON) is important to
23 family that is heteroplasmic for the primary Leber hereditary optic neuropathy (LHON) mutation at nuc
26 mtDNA) sequences for 63 Dutch pedigrees with Leber hereditary optic neuropathy (LHON) were determined
27 o play a pivotal role in the pathogenesis of Leber hereditary optic neuropathy (LHON), caused by muta
30 ances in molecular research that have linked Leber Hereditary Optic Neuropathy to mutations in mitoch
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