ライフサイエンスコーパス: Listeria

1 e mCTL recall upon secondary infections with Listeria.

2 mary and memory CD8 T cell responses against Listeria.

3 es defects in myelopoiesis and resistance to Listeria.

4 e paradoxically also being more resistant to Listeria.

5 piked whole milk and ground meat spiked with listeria.

6 uxiliary factor that promoted the ability of Listeria ActA protein to activate the Arp2/3 complex to

7 s a factor that facilitates Arp2/3-dependent Listeria actin cloud formation in the presence of Arp2/3

8 demonstrate an important role for CRMP-1 in Listeria actin comet tail formation and open the possibi

9 It also scored as an important factor for Listeria actin comet tail formation in brain cytosol.

10 2/3) complex is necessary and sufficient for Listeria actin tail assembly, previous studies suggest t

11 explored the three-dimensional structure of Listeria actin tails in Xenopus laevis egg extracts usin

12 We found that the architecture of Listeria actin tails is shared between those formed in c

13 eclared to the National Reference Center for Listeria (all microbiologically proven) between Nov 3, 2

14 acterium, Salmonella, Escherichia, Shigella, Listeria and Bartonella, using published literature.

15 and displayed much greater effect to inhibit Listeria and Salmonella than non-emulsion, aqueous formu

16 n, and conferred improved protection against Listeria and vaccinia virus challenges compared with the

17 Pathogenic bacteria such as Listeria and Yersinia gain initial entry by binding to h

18 uent autophagic capturing and degradation of Listeria antigens.

19 liferation after infection with influenza or Listeria, as compared with naive Ly49H(+) or Ly49H(-) NK

20 The protease from the listeria bacteria was detected using D-amino acid substr

21 e sensor was tested with serial dilutions of Listeria bacteria.

22 magnetic nanoparticles for the detection of listeria bacteria.

23 with recombinant SFB Ag-expressing virulent Listeria (but not wild-type virulent Listeria), suggesti

24 ontribute to the adaptive immune response to Listeria, but only CD8(+) NK1.1(+) cells were equipped w

25 at P. yoelii infections suppress immunity to Listeria by causing increased apoptosis in Listeria-spec

26 these findings provide detailed insight into Listeria cell wall-associated carbohydrates, and will gu

27 we found that in Perforin-2(-/-)macrophages,Listeria-containing vacuoles quickly (</= 15 min) acidif

28 rive from it regulating the acidification of Listeria-containing vacuoles, thereby depriving the path

29 CD1d tetramers did not stain dNKT cells, the Listeria-derived phosphatidylglycerol-loaded tetramers d

30 important role in the mechanisms underlying Listeria disease.

31 Thus, Listeria DNA is a major trigger of IFNbeta expression in

32 Here we report that in human macrophages, Listeria DNA rather than cyclic-di-AMP is stimulating th

33 CD8alpha(+) DCs are also necessary for Listeria expansion and dissemination within the host.

34 nisms that regulate CD8alpha(+) DCs to allow Listeria expansion are unclear.

35 ling limits Fas/FasL-mediated suppression of Listeria expansion within CD8alpha(+) DCs to more effect

36 nto Btla(-/-) CD8alpha(+) DCs is unaffected, Listeria fails to expand within these cells.

37 bacteria, including Burkholderia, Shigella, Listeria, Francisella, and Mycobacterium species.

38 ed against five different species within the Listeria genus.

39 DHFR originated from Chlamydia muridarum and Listeria grayi We found that the acquisition of TMP resi

40 te that HD6 may provide host defense against Listeria in the gut.

41 ability of CD8(+) T cells to protect against Listeria infection and attenuate tumor growth in vivo.

42 ) T cells were more effective at controlling Listeria infection and B16 melanoma growth in vivo, and

43 C/SLEC differentiation choice following both Listeria infection and dendritic cell immunization of mi

44 effector CD8(+) T cell differentiation upon listeria infection but did result in a severe defect in

45 we observed that ISG15 expression restricts Listeria infection in vitro and in vivo.

46 T cells in vitro was not recapitulated after Listeria infection in vivo.

47 ver, mice lacking TNFR1 in MDCs succumbed to listeria infection, although they displayed similar sens

48 characterized ISG15-dependent restriction of Listeria infection, reinforcing the view that ISG15 is a

49 susceptible than wild-type mice to systemic Listeria infection, which correlates with increased numb

50 ficient T cells are defective in controlling Listeria infection.

51 duced CD8(+) T cell response to a subsequent Listeria infection.

52 hagocytic cells is dramatically induced upon Listeria infection.

53 We found that, although Listeria infections reduced expression of CCL21 in murin

54 vity of the essential oil components against Listeria innocua and Escherichia coli compared to free c

55 Here, we show for the Listeria innocua integrase (LI Int) system that the CC d

56 ch as Escherichia coli, Salmonella enterica, Listeria innocua, Mycobacterium parafortuitum, and Sacch

57 s Escherichia coli, Salmonella enterica, and Listeria innocua, on stainless steel surfaces and on org

58 A nanoparticles were effective inhibitors of Listeria innocua, with lower (P<0.05) GPE concentrations

59 ng elongation of the Gram-positive bacterium Listeria innocua.

60 ere we showed that Shigella, Salmonella, and Listeria interfere with spliceosomal U snRNA maturation

61 our data indicate that delivery of LJM11 by Listeria is a promising vaccination strategy against cut

62 is of clinical features, characterisation of Listeria isolates, and determination of predictors of 3-

63 Listeria (L.) monocytogenes is an opportunistic pathogen

64 ic T cell responses were not impaired during Listeria + LCMV coinfection, arguing against a major rol

65 ngitis were rare in the first month, whereas Listeria meningitis was seen only in the first month of

66 Recently, AcrIIA2 and AcrIIA4, encoded by Listeria monocytogene prophages, were shown to block the

67 thogens such as Salmonella Typhimurium, (7%) Listeria monocytogenes (3%) and Escherichia coli (0%).

68 ell transfers in the well-established murine Listeria monocytogenes (L.m.) infection model.

69 Remarkably Hfq from Gram-positive Listeria monocytogenes (Lm) binds (GU)3G on its proximal

70 Listeria monocytogenes (Lm) causes severe foodborne illn

71 ed a novel approach utilizing infection with Listeria monocytogenes (LM) encoding proteolipid protein

72 Listeria monocytogenes (Lm) is a major human foodborne p

73 nonphagocytic cells, a critical property of Listeria monocytogenes (Lm) that enables it to cross hos

74 tabolic pathway from the food-borne pathogen Listeria monocytogenes (Lm).

75 hemokine production following infection with Listeria monocytogenes (Lm).

76 nvestigate novel live attenuated recombinant Listeria monocytogenes (rLm) vaccines expressing the Myc

77 Pdx-1-Cre mice with attenuated intracellular Listeria monocytogenes (which induces CD4(+) and CD8(+)

78 ular pathogens such as Shigella flexneri and Listeria monocytogenes achieve dissemination in the inte

79 as a factor that stimulates the formation of Listeria monocytogenes actin comet tails, thereby implic

80 galovirus and DNA, and the infectious agents Listeria monocytogenes and Aspergillus fumigatus.

81 resentative of CsoRs from pathogenic bacilli Listeria monocytogenes and Bacillus anthracis.

82 neity in manifestations of disease caused by Listeria monocytogenes and demonstrate that a previously

83 llin-resistant Staphylococcus aureus (MRSA), Listeria monocytogenes and Enterococcus faecalis, and ag

84 es for Salmonella spp. and other pathogens ( Listeria monocytogenes and Escherichia coli ) are compar

85 multaneous detection of Salmonella enterica, Listeria monocytogenes and Escherichia coli based on tri

86 the survival of common food borne pathogens, Listeria monocytogenes and Escherichia coli O157:H7.

87 this question with the facultative pathogen Listeria monocytogenes and its PrfA virulence regulon.

88 showed potent antibacterial activity against Listeria monocytogenes and methicillin-resistant Staphyl

89 g infection with the Th1-associated pathogen Listeria monocytogenes and observed that IS001 enhanced

90 Listeria monocytogenes and other pathogenic bacteria mod

91 ry mechanisms employed by two intracellular [Listeria monocytogenes and Salmonella enterica serovar T

92 ew recent advances in the field showing that Listeria monocytogenes and Shigella flexneri have evolve

93 ting the growth of Staphylococcus aureus and Listeria monocytogenes and showed high inhibitory capaci

94 hylogenetically related human pathogens like Listeria monocytogenes and Staphylococcus aureus possess

95 Toll-like receptor 5 ligand flagellin A from Listeria monocytogenes and the birch pollen allergen Bet

96 n in vivo T-cell priming during infection by Listeria monocytogenes and vesicular stomatitis virus.

97 The emergence of Listeria monocytogenes as a promising immunotherapeutic

98 this study, we used an attenuated strain of Listeria monocytogenes as a vaccine expression system fo

99 the in situ detection and discrimination of Listeria monocytogenes at a concentration of single cell

100 particular Staphylococcus aureus ATCC 6538, Listeria monocytogenes ATCC 13932 and methicillin-resist

101 PGRN-deficient mice are sensitive to Listeria monocytogenes because of deficits in xenophagy,

102 Listeria monocytogenes binds to the epithelial host cell

103 ng of the MOLF strain in response to HSV and Listeria monocytogenes both in vitro and in vivo.

104 hat c-di-AMP regulates central metabolism in Listeria monocytogenes by inhibiting its pyruvate carbox

105 owth of the foodborne intracellular pathogen Listeria monocytogenes by promoting mechanisms that damp

106 iated with the functional transitions in the Listeria monocytogenes Ca(2+)-ATPase (LMCA1), an ortholo

107 The bacterial pathogen Listeria monocytogenes causes foodborne systemic disease

108 The facultative intracellular pathogen Listeria monocytogenes causes listeriosis, a rare but li

109 The bacterial pathogen Listeria monocytogenes causes spontaneous abortion, stil

110 Listeria monocytogenes cells were grown at 37 degrees C

111 iled to protect against a lethal recombinant Listeria monocytogenes challenge.

112 vaccination increasing protection against a Listeria monocytogenes challenge.

113 EPS had the largest inhibition zone against Listeria monocytogenes CMCC 54001.

114 w that a diverse microbiota markedly reduces Listeria monocytogenes colonization of the gut lumen and

115 The pathogenesis of Listeria monocytogenes depends on the ability of this ba

116 ed by the Gram-positive facultative pathogen Listeria monocytogenes during an in vivo infection.

117 es of the facultative intracellular pathogen Listeria monocytogenes encode two functional enoyl-acyl

118 Immunization of KPC mice with Listeria monocytogenes engineered to express Kras(G12D),

119 enically express a TCR specific for the same Listeria monocytogenes epitope, elicited distinct interl

120 -2S4B6-treated HSCT recipients infected with Listeria monocytogenes exhibited decreased bacterial lev

121 D8 cell adoptive transfer and challenge with Listeria monocytogenes expressing a cognate antigen, we

122 ely can be expanded by secondary exposure to Listeria monocytogenes expressing recombinant Plasmodium

123 prevents the human gastrointestinal pathogen Listeria monocytogenes from invading cultured mammalian

124 The peptide sensor also selectively detected Listeria monocytogenes from other Gram-positive strains

125 Diagnosis is made by culturing Listeria monocytogenes from sterile body fluids or from

126 Listeria monocytogenes FrvA (Lmo0641) is critical for vi

127 crobial properties of SAMN@TA were tested on Listeria monocytogenes in comparison with free TA, showi

128 We show that clearance of Listeria monocytogenes in macrophages requires IRF8-depe

129 the antibacterial activity observed against Listeria monocytogenes in vitro, in cell culture, and in

130 A fruit extract matrix was selected and Listeria monocytogenes inactivation was followed from th

131 We found that oral infection with Listeria monocytogenes induced a robust intestinal CD8 T

132 Importantly, Listeria monocytogenes induces NLRP3-dependent rapid cas

133 Listeria monocytogenes infected CD8alpha(+) DCs in the s

134 MHC class II-specific GC-Tfh cells following Listeria monocytogenes infection and a 2-fold decrease f

135 defensive role of the gut microbiota against Listeria monocytogenes infection and identify intestinal

136 cells conferred increased protection against Listeria monocytogenes infection in susceptible IFN-gamm

137 a T cells are important for the clearance of Listeria monocytogenes infection in the intestinal mucos

138 ate that fetal wastage triggered by prenatal Listeria monocytogenes infection is driven by placental

139 ainst microbial infections, we have used the Listeria monocytogenes infection model to explore the im

140 f cardiac transplantation, we show that when Listeria monocytogenes infection precipitates acute reje

141 l uptake and were more susceptible to lethal Listeria monocytogenes infection than were DT-treated CL

142 Upon acute Listeria monocytogenes infection, deleting miR-23a in T

143 cing CD4(+) and CD8(+) T cells responding to Listeria monocytogenes infection.

144 tion of memory CD8(+) T cells in response to Listeria monocytogenes infection.

145 Tim-3 affects CD8 T cell responses to acute Listeria monocytogenes infection.

146 and conferred protection against recombinant Listeria monocytogenes infection.

147 survival of CD8(+) T cells in vivo following Listeria monocytogenes infection.

148 n the innate immune response of mice against Listeria monocytogenes infection.

149 and dampened innate immune responses against Listeria monocytogenes infection.

150 ophils in the spleen than did WT mice during Listeria monocytogenes infection.

151 uced tolerance and more effective control of Listeria monocytogenes infection.

152 Listeria monocytogenes is a bacterial parasite that uses

153 The Gram-positive bacterium Listeria monocytogenes is a facultative intracellular pa

154 Listeria monocytogenes is a facultative intracellular pa

155 The Gram-positive bacterium Listeria monocytogenes is a facultative intracellular pa

156 Listeria monocytogenes is a food-borne pathogen that can

157 Listeria monocytogenes is a foodborne pathogen capable o

158 Listeria monocytogenes is a foodborne pathogen responsib

159 Listeria monocytogenes is a foodborne pathogen that caus

160 Listeria monocytogenes is a foodborne pathogen that caus

161 Listeria monocytogenes is a Gram-positive facultative in

162 Listeria monocytogenes is a gram-positive facultative in

163 Listeria monocytogenes is a Gram-positive intracellular

164 Listeria monocytogenes is a Gram-positive intracellular

165 Listeria monocytogenes is a highly adaptive bacterium th

166 Listeria monocytogenes is a major cause of mortality res

167 Listeria monocytogenes is a major intracellular human fo

168 Listeria monocytogenes is a serious cause of human foodb

169 Listeria monocytogenes is an intracellular Gram-positive

170 Listeria monocytogenes is an intracellular pathogen that

171 Infection by the human bacterial pathogen Listeria monocytogenes is mainly controlled by the posit

172 Listeria monocytogenes is responsible for gastroenteriti

173 Listeria monocytogenes is responsible for the life-threa

174 We performed WGS on Listeria monocytogenes isolates from patients and availa

175 To address this question, we used the Listeria monocytogenes model of infection and followed C

176 ages and is induced following infection with Listeria monocytogenes or stimulation with TLR ligands (

177 ibited a modest expansion defect early after Listeria monocytogenes or vesicular stomatitis virus inf

178 Listeria monocytogenes PdxR is a member of the poorly ch

179 -A CRISPR-Cas9 inhibitor proteins encoded by Listeria monocytogenes prophages.

180 Using chemical proteomics, we identified the Listeria monocytogenes protein PgpH as a molecular targe

181 facultative intracellular bacterial pathogen Listeria monocytogenes remodels its transcriptional prog

182 Pathogenic Listeria monocytogenes replicates within the host cytoso

183 e to apoptotic immune cells and live or dead Listeria monocytogenes scavenger receptor BI (SR-BI), an

184 analyzed for antimicrobial activity against Listeria monocytogenes Scott A and Escherichia coli K12.

185 Listeria monocytogenes serotype 4b was more common in pr

186 DeltagpsB mutants of the human pathogen Listeria monocytogenes show severe lysis, division and g

187 packaged leafy green salad contaminated with Listeria monocytogenes singleton sequence type 382 (ST38

188 estinal pathogens Salmonella typhimurium and Listeria monocytogenes to induce the expression of IL-8.

189 Using recombinant Listeria monocytogenes to prime stably differentiated Th

190 he sensitivity of the intracellular pathogen Listeria monocytogenes to various beta-lactams by inhibi

191 The Gram-positive bacterium Listeria monocytogenes transitions from an environmental

192 facultative intracellular bacterial pathogen Listeria monocytogenes Two days after foodborne infectio

193 tinase, as well as unrelated chitinases from Listeria monocytogenes using the fluorescently labeled s

194 Listeria monocytogenes V7 and Salmonella enterica serova

195 e tested this hypothesis using a recombinant Listeria monocytogenes vaccine platform that targets CD1

196 inhibition against Staphylococcus aureus and Listeria monocytogenes was >89% when fPEM extracts were

197 proliferation of the intracytosolic pathogen Listeria monocytogenes Within a few hours of systemic in

198 ere promoted by the TLR2 ligand (heat killed Listeria monocytogenes) and the TLR4 ligand (lipopolysac

199 ia coli O157:H7, Salmonella typhimurium, and Listeria monocytogenes) in buffer.

200 d by two other pathogens (vaccinia virus and Listeria monocytogenes).

201 anscriptional regulators to the virulence of Listeria monocytogenes, a Gram-positive facultative intr

202 e viral infection localized to the lung, and Listeria monocytogenes, a systemic bacterial infection.

203 sed by diverse pathogens (Trypanosoma cruzi, Listeria monocytogenes, and adenovirus) to promote their

204 gainst Gram-positive (Staphylococcus aureus, Listeria monocytogenes, and Bacillus cereus) and Gram-ne

205 three unrelated bacteria: Escherichia coli, Listeria monocytogenes, and Mycobacteria tuberculosis.

206 e intracellular pathogens Toxoplasma gondii, Listeria monocytogenes, and Mycobacterium tuberculosis H

207 Escherichia coli O157:H7, non-O157 E. coli, Listeria monocytogenes, and Salmonella spp.) was modeled

208 high-affinity Fe(2+) efflux transporter from Listeria monocytogenes, as an inducible genetic tool to

209 oximately 300 nM) had activity against MRSA, Listeria monocytogenes, Bacillus anthracis, and a vancom

210 y against Gram positive foodborne pathogens (Listeria monocytogenes, Bacillus cereus and Staphylococc

211 tificially inoculated with Escherichia coli, Listeria monocytogenes, Bacillus cereus, Staphylococcus

212 om Escherichia coli, Pseudomonas aeruginosa, Listeria monocytogenes, Bacillus subtilis, and Staphyloc

213 ctivities compared to free LAE in inhibiting Listeria monocytogenes, but was less effective against E

214 Here we show that a bacterial pathogen, Listeria monocytogenes, can exploit efferocytosis to pro

215 lococcus epidermidis, Staphylococcus aureus, Listeria monocytogenes, Candida albicans, and Candida pa

216 In the human pathogen Listeria monocytogenes, cdiA is an essential molecule th

217 Indeed, following infection with Listeria monocytogenes, DNA-PKcs-deficient murine macrop

218 the highest on tyramine production (55%) by Listeria monocytogenes, following Lc. lactis subsp. lact

219 ive (Bacillus cereus, Staphylococcus aureus, Listeria monocytogenes, Geobacillus stearothermophilus)

220 Indeed, following systemic infection with Listeria monocytogenes, germ-free and oral-antibiotic-tr

221 rin alone had antimicrobial activity against Listeria monocytogenes, however, films incorporating cit

222 ce also harboured a strong TH1 cell inducer, Listeria monocytogenes, in their intestine.

223 Listeriosis, caused by Listeria monocytogenes, is an important foodborne diseas

224 atitis and in models of bacterial infection (Listeria monocytogenes, lipopolysaccharide).

225 facultative intracellular bacterial pathogen Listeria monocytogenes, most of the bacterial burden in

226 In Listeria monocytogenes, mutations that prevent addition

227 Escherichia coli K1, Haemophilus influenzae, Listeria monocytogenes, Neisseria meningitidis, Streptoc

228 ith the Gram-positive intracellular pathogen Listeria monocytogenes, neutrophils are recruited from t

229 Both compounds appeared effective against Listeria monocytogenes, one of the most important foodbo

230 itive bacteria, including the human pathogen Listeria monocytogenes, possess an additional nonessenti

231 ood pathogens, namely Staphylococcus aureus, Listeria monocytogenes, Salmonella enteritidis and Esche

232 omposition and volatile molecule profiles of Listeria monocytogenes, Salmonella enteritidis, Escheric

233 Escherichia coli, Listeria monocytogenes, Salmonella sp. and Staphylococcu

234 Since Listeria monocytogenes, Shigella flexneri, and Vaccinia

235 the spoilage food bacteria Escherichia coli, Listeria monocytogenes, Staphylococcus aureus and Salmon

236 In Listeria monocytogenes, the sole diadenylate cyclase, Da

237 ogens such as Zika virus, Toxoplasma gondii, Listeria monocytogenes, Treponema pallidium, parvovirus,

238 In Listeria monocytogenes, two enzymes are required for the

239 this function upon secondary challenges with Listeria monocytogenes, vesicular stomatitis virus, or V

240 ed by the facultative intracellular pathogen Listeria monocytogenes, was posttranslationally modified

241 -di-AMP-interacting proteins in the pathogen Listeria monocytogenes, we identified several broadly co

242 ine kinase Src upon incubation of cells with Listeria monocytogenes, we searched for novel host prote

243 opy of the bloodstream of mice infected with Listeria monocytogenes, we show that bacterial clearance

244 ed Drosophila melanogaster with the pathogen Listeria monocytogenes, we tested this framework, findin

245 ine, DMOT4039A, BMS-986148), live attenuated Listeria monocytogenes-expressing mesothelin (CRS-207, J

246 CRS-207, live-attenuated Listeria monocytogenes-expressing mesothelin, induces in

247 sensitive against Staphylococcus aureus and Listeria monocytogenes.

248 ted osmotic stress in the bacterial pathogen Listeria monocytogenes.

249 to infection with the intracellular bacteria Listeria monocytogenes.

250 immunoassay for ultrasensitive detection of Listeria monocytogenes.

251 ection by the inflammasome-evasive bacterium Listeria monocytogenes.

252 -positive facultative intracellular pathogen Listeria monocytogenes.

253 ffector responses with improved clearance of Listeria monocytogenes.

254 ysaccharide (EPS) in the food-borne pathogen Listeria monocytogenes.

255 idomas with lipid extracts from the pathogen Listeria monocytogenes.

256 ginosa but not in the Gram-positive pathogen Listeria monocytogenes.

257 ce that were subsequently infected i.v. with Listeria monocytogenes.

258 n pathogens such as Enterococcus faecium and Listeria monocytogenes.

259 red for the growth of the bacterial pathogen Listeria monocytogenes.

260 ansgenic granulysin are better able to clear Listeria monocytogenes.

261 e protected from lethal infection induced by Listeria monocytogenes.

262 ation of 100S ribosomes by an HPF homolog in Listeria monocytogenes.

263 responses to immunization or infection with Listeria monocytogenes.

264 n of epithelial and macrophage-like cells by Listeria monocytogenes.

265 , including Bacillus cereus and the pathogen Listeria monocytogenes.

266 ed by the facultative intracellular pathogen Listeria monocytogenes.

267 ion properties of the intracellular pathogen Listeria monocytogenes.

268 in of internalins of the food borne pathogen Listeria monocytogenes.

269 was created by intraperitoneal injection of Listeria monocytogenes.

270 icidal activity against Escherichia coli and Listeria monocytogenes.

271 lethal infection with the bacterial pathogen Listeria monocytogenes.

272 tic pathogens, such as influenza viruses and Listeria monocytogenes.

273 s showed high antibacterial activity against Listeria monocytogenes: cIsf pool had a minimum inhibito

274 ing a panel of food-contaminating pathogens (Listeria monocytogenesis 19115 and E. coli O157:H7), cli

275 ponsible for bacterial growth restriction of Listeria monocytogenesL.

276 ection of I-A(12%) mice with graded doses of Listeria monotcytogenes or influenza virus revealed comp

277 ts favor a mechanism of force generation for Listeria movement where the stress is released into prop

278 We identified Listeria phosphatidylglycerol as a microbial Ag that was

279 The structure of Listeria phosphatidylglycerol was distinct from mammalia

280 iated strategy used by the host that permits Listeria proliferation to enable increasing T cell respo

281 vage of the specific peptide sequence by the Listeria protease.

282 The members of the second clade, "Listeria sensu lato", are believed to be solely environm

283 51 candidate genes that are conserved in the Listeria sensu stricto species.

284 solated from symptom-free animals, form the "Listeria sensu stricto" clade.

285 ulent L. monocytogenes strains but absent in Listeria species that are nonpathogenic for humans.

286 rmine the structural complexity of WTAs from Listeria species.

287 xperimentation revealed greater apoptosis of Listeria-specific effector T cells as the main mechanism

288 ative mechanism whereby P. yoelii suppresses Listeria-specific T cell responses.

289 We found that Listeria-specific T cells expanded more slowly and resul

290 o Listeria by causing increased apoptosis in Listeria-specific T cells, resulting in a slower expansi

291 ss the recruitment or proliferation rates of Listeria-specific T cells.

292 t was evaluated against different strains of Listeria spp. in milk at 37 degrees C for 24h.

293 at 10h, against four of the five strains of Listeria spp. tested.

294 d (ss) DNA aptamers with binding affinity to Listeria spp. were selected using a whole-cell SELEX (Sy

295 irulent Listeria (but not wild-type virulent Listeria), suggesting the CLP-induced polymicrobial seps

296 r accumulates, building up stress around the Listeria surface.

297 ll-mediated hepatitis and in the response to listeria, thereby identifying the opposing role of MDC T

298 In particular, Listeria uses Arp2/3-mediated actin filament nucleation

299 detection limit of the developed sensor for Listeria was found to be 2.17x10(2) colony forming unit/

300 revealed three conjugated CPPs rapidly kill Listeria within 20 minutes without disrupting the bacter