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1 f the varying manifestations associated with Lyme borreliosis.
2 ochetes' evasion of macrophages during early Lyme borreliosis.
3 the early stages of tick-transmitted murine Lyme borreliosis.
4 seroprevalence of HGE was similar to that of Lyme borreliosis.
5 for monitoring the response to treatment of Lyme borreliosis.
6 ri can prevent infection in animal models of Lyme borreliosis.
7 lian host as part of the disease process for Lyme borreliosis.
8 and 81 control subjects without a history of Lyme borreliosis.
9 variations in the clinical manifestations of Lyme borreliosis.
10 tant in the murine model of tick-transmitted Lyme borreliosis.
11 prospective studies using a murine model of Lyme borreliosis.
12 and joint histopathology during experimental Lyme borreliosis.
13 for patients with localized or disseminated Lyme borreliosis.
14 re essential for persistence associated with Lyme borreliosis.
15 burgdorferi infection in the mouse model of Lyme borreliosis.
16 le vaccine candidate to help protect against Lyme borreliosis.
17 protect mice from experimental syringe-borne Lyme borreliosis.
18 t hypervariability among isolates that cause Lyme borreliosis.
19 nvolved in pathogenic processes operative in Lyme borreliosis.
20 may be useful for the serodiagnosis of early Lyme borreliosis.
21 role of complement in the global ecology of Lyme borreliosis.
22 borne diseases should prompt a fresh look at Lyme borreliosis.
23 h2 cells in the genetic resistance to murine Lyme borreliosis.
25 on between the chronic skin manifestation of Lyme borreliosis, acrodermatitis chronica atrophicans, a
26 feri (Bb) sensu lato, the etiologic agent of Lyme borreliosis, adapts to distinct environments in the
27 ecover a viable RecA-deficient mutant of the Lyme borreliosis agent Borrelia burgdorferi, we characte
28 h a range of early or late manifestations of Lyme borreliosis and found that only American patients w
29 ignancy more often had signs of disseminated Lyme borreliosis and more frequently needed antibiotic r
31 nes are induced by spirochetes during murine Lyme borreliosis and play roles in protective immunity.
33 s potential relevance to the pathogenesis of Lyme borreliosis and to the physiology of the spirochete
34 e the virulence of Bb in the murine model of Lyme borreliosis and was essential for the persistence o
35 respectively by B. burgdorferi, the agent of Lyme borreliosis, and B. hermsii, the agent of tick-born
36 f autochthonous transmission of the agent of Lyme borreliosis, and continued monitoring of exposure r
37 sensu lato known to be associated with human Lyme borreliosis (B. burgdorferi sensu stricto, B. garin
38 Borrelia are known to be causative agents of Lyme borreliosis: B. burgdorferi sensu stricto, B. garin
39 timulatory molecules on the course of murine Lyme borreliosis because experimental Lyme arthritis is
40 sed lipoproteins of relapsing fever (RF) and Lyme borreliosis Borrelia spirochetes mediate certain in
41 roteins are essential to the pathogenesis of Lyme borreliosis, but the mechanisms responsible for the
42 ary quality has clouded our understanding of Lyme borreliosis by giving Borrelia burgdorferi infectio
43 of IL-17 in the development of experimental Lyme borreliosis by infecting C3H mice devoid of the com
44 eri, the tick-transmitted etiologic agent of Lyme borreliosis, can colonize and persist in multiple t
45 ectious diseases are reviewed: tuberculosis, Lyme borreliosis, cat scratch disease, toxocariasis, con
48 e the role of B. burgdorferi-specific Abs in Lyme borreliosis, CD40 ligand-deficient (CD40L-deficient
49 relia burgdorferi, a spirochaete that causes Lyme borreliosis, contains 21 linear and circular plasmi
50 that myositis can be a prominent feature of Lyme borreliosis depending on the infecting organism and
53 e causative agents of the tick-borne disease Lyme borreliosis, disseminate hematogenously from the ti
55 e infections in the United States other than Lyme borreliosis, emphasizing a clinical and geographic
56 are addressed using examples from tick-borne Lyme borreliosis; flea-borne plague; and mosquito-borne
57 ythema migrans skin lesions of patients with Lyme borreliosis from the United States or Slovenia.
58 naming this new South American member of the Lyme borreliosis group B. chilensis VA1 in honor of its
60 amples from patients with case-defined early Lyme borreliosis had anti-B. burgdorferi sensu stricto 5
62 viduals with confirmed clinical diagnoses of Lyme borreliosis, immunoblotting is still advised as an
63 fect of anti-interleukin (IL-12 treatment on Lyme borreliosis in C3H/HeN (C3H) mice was assessed beca
65 o is the only established etiologic agent of Lyme borreliosis in dogs and in humans in North America.
66 defense against B. burgdorferi, we examined Lyme borreliosis in mice deficient in either caspase 1 o
69 Borrelia burgdorferi, the causative agent of Lyme borreliosis, in the central nervous system, only mi
74 i sensu lato (s.l.), the tick-borne agent of Lyme borreliosis, is a bacterial species complex compris
76 orferi sensu lato, the causative organism of Lyme borreliosis, is a heterogeneous group of spirochete
78 Borrelia burgdorferi, a causative agent of Lyme borreliosis, is a zoonotic pathogen that survives i
79 he thyX gene for thymidylate synthase of the Lyme borreliosis (LB) agent Borrelia burgdorferi is loca
80 Human granulocytic ehrlichiosis (HGE) and Lyme borreliosis (LB) are acquired after Ixodes ricinus-
85 mes need to evaluate a patient for suspected Lyme borreliosis (LB) who may have acquired the infectio
88 ated with sequences from 35 genomes of eight Lyme-borreliosis (LB) group Borrelia species and 7 Relap
90 has not been studied using the experimental Lyme borreliosis model of infection of C3H mice with Bor
91 ogic testing also revealed possible cases of Lyme borreliosis (n = 142), babesiosis (n = 41), and HME
92 rom persons who had been diagnosed as having Lyme borreliosis on the basis of the presence of erythem
99 ibodies, especially IgM antibodies, in early Lyme borreliosis sera provides additional evidence that
101 In vivo studies using a murine model of Lyme borreliosis showed that while OspC F(ab)(2) fragmen
103 In contrast to RF Borrelia species, the Lyme borreliosis spirochete Borrelia burgdorferi is amen
104 , suggesting that BptA may be widely used by Lyme borreliosis spirochetes for persistence in nature.
106 Borrelia burgdorferi spirochetes that cause Lyme borreliosis survive for a long time in human serum
108 ocytoma is a rare cutaneous manifestation of Lyme borreliosis that has been reported almost exclusive
111 present studies, we use the murine model of Lyme borreliosis to define an OspA T cell epitope locate
112 es of sera from patients with early or later Lyme borreliosis to the antibody reactivities of sera fr
114 Twelve of 15 patients (80%) with suspected Lyme borreliosis, versus 0 of 10 controls, were positive
115 equence of Borrelia burgdorferi, an agent of Lyme borreliosis, was used to study the genetic content
116 intravital microscopy and the mouse model of Lyme borreliosis, we observed that Borrelia burgdorferi
118 protective immunogen in the murine model of Lyme borreliosis when mice were challenged (needle inocu
120 i) in the upper midwestern USA, which causes Lyme borreliosis with unusually high spirochaetaemia.
121 Borrelia burgdorferi, the causative agent of Lyme borreliosis, with phagocytic cells induces the acti
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