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1 f the varying manifestations associated with Lyme borreliosis.
2 ochetes' evasion of macrophages during early Lyme borreliosis.
3  the early stages of tick-transmitted murine Lyme borreliosis.
4 seroprevalence of HGE was similar to that of Lyme borreliosis.
5  for monitoring the response to treatment of Lyme borreliosis.
6 ri can prevent infection in animal models of Lyme borreliosis.
7 lian host as part of the disease process for Lyme borreliosis.
8 and 81 control subjects without a history of Lyme borreliosis.
9 variations in the clinical manifestations of Lyme borreliosis.
10 tant in the murine model of tick-transmitted Lyme borreliosis.
11  prospective studies using a murine model of Lyme borreliosis.
12 and joint histopathology during experimental Lyme borreliosis.
13  for patients with localized or disseminated Lyme borreliosis.
14 re essential for persistence associated with Lyme borreliosis.
15  burgdorferi infection in the mouse model of Lyme borreliosis.
16 le vaccine candidate to help protect against Lyme borreliosis.
17 protect mice from experimental syringe-borne Lyme borreliosis.
18 t hypervariability among isolates that cause Lyme borreliosis.
19 nvolved in pathogenic processes operative in Lyme borreliosis.
20 may be useful for the serodiagnosis of early Lyme borreliosis.
21  role of complement in the global ecology of Lyme borreliosis.
22 borne diseases should prompt a fresh look at Lyme borreliosis.
23 h2 cells in the genetic resistance to murine Lyme borreliosis.
24                          Of 96 patients with Lyme borreliosis, 9 (9.4%) demonstrated immunoserologic
25 on between the chronic skin manifestation of Lyme borreliosis, acrodermatitis chronica atrophicans, a
26 feri (Bb) sensu lato, the etiologic agent of Lyme borreliosis, adapts to distinct environments in the
27 ecover a viable RecA-deficient mutant of the Lyme borreliosis agent Borrelia burgdorferi, we characte
28 h a range of early or late manifestations of Lyme borreliosis and found that only American patients w
29 ignancy more often had signs of disseminated Lyme borreliosis and more frequently needed antibiotic r
30 ovide insight into mechanisms of immunity to Lyme borreliosis and other vector-borne diseases.
31 nes are induced by spirochetes during murine Lyme borreliosis and play roles in protective immunity.
32 ucial to the transmission and persistence of Lyme borreliosis and tick-borne relapsing fever.
33 s potential relevance to the pathogenesis of Lyme borreliosis and to the physiology of the spirochete
34 e the virulence of Bb in the murine model of Lyme borreliosis and was essential for the persistence o
35 respectively by B. burgdorferi, the agent of Lyme borreliosis, and B. hermsii, the agent of tick-born
36 f autochthonous transmission of the agent of Lyme borreliosis, and continued monitoring of exposure r
37 sensu lato known to be associated with human Lyme borreliosis (B. burgdorferi sensu stricto, B. garin
38 Borrelia are known to be causative agents of Lyme borreliosis: B. burgdorferi sensu stricto, B. garin
39 timulatory molecules on the course of murine Lyme borreliosis because experimental Lyme arthritis is
40 sed lipoproteins of relapsing fever (RF) and Lyme borreliosis Borrelia spirochetes mediate certain in
41 roteins are essential to the pathogenesis of Lyme borreliosis, but the mechanisms responsible for the
42 ary quality has clouded our understanding of Lyme borreliosis by giving Borrelia burgdorferi infectio
43  of IL-17 in the development of experimental Lyme borreliosis by infecting C3H mice devoid of the com
44 eri, the tick-transmitted etiologic agent of Lyme borreliosis, can colonize and persist in multiple t
45 ectious diseases are reviewed: tuberculosis, Lyme borreliosis, cat scratch disease, toxocariasis, con
46                                       Murine Lyme borreliosis, caused by infection with the spirochet
47                                              Lyme borreliosis, caused by the tick-borne bacterium Bor
48 e the role of B. burgdorferi-specific Abs in Lyme borreliosis, CD40 ligand-deficient (CD40L-deficient
49 relia burgdorferi, a spirochaete that causes Lyme borreliosis, contains 21 linear and circular plasmi
50  that myositis can be a prominent feature of Lyme borreliosis depending on the infecting organism and
51 f 5, 100%) and patients (31 of 43, 72%) with Lyme borreliosis developed P35 or P37 antibodies.
52                                              Lyme borreliosis differs in dogs and humans in terms of
53 e causative agents of the tick-borne disease Lyme borreliosis, disseminate hematogenously from the ti
54                The clinical presentations of Lyme borreliosis do differ in North America and Eurasia,
55 e infections in the United States other than Lyme borreliosis, emphasizing a clinical and geographic
56 are addressed using examples from tick-borne Lyme borreliosis; flea-borne plague; and mosquito-borne
57 ythema migrans skin lesions of patients with Lyme borreliosis from the United States or Slovenia.
58 naming this new South American member of the Lyme borreliosis group B. chilensis VA1 in honor of its
59 s spirochete as a new genospecies within the Lyme borreliosis group.
60 amples from patients with case-defined early Lyme borreliosis had anti-B. burgdorferi sensu stricto 5
61        The serious morbidity associated with Lyme borreliosis has focused considerable effort on the
62 viduals with confirmed clinical diagnoses of Lyme borreliosis, immunoblotting is still advised as an
63 fect of anti-interleukin (IL-12 treatment on Lyme borreliosis in C3H/HeN (C3H) mice was assessed beca
64 he specificity of the serologic diagnosis of Lyme borreliosis in cases of acute facial palsy.
65 o is the only established etiologic agent of Lyme borreliosis in dogs and in humans in North America.
66  defense against B. burgdorferi, we examined Lyme borreliosis in mice deficient in either caspase 1 o
67 to describe a novel Borrelia species causing Lyme borreliosis in the USA.
68                                              Lyme borreliosis in transgenic mice tolerant to Borrelia
69 Borrelia burgdorferi, the causative agent of Lyme borreliosis, in the central nervous system, only mi
70                                              Lyme borreliosis is a multisystemic disease caused by va
71                                       Murine Lyme borreliosis is characterized by arthritis and cardi
72                                              Lyme borreliosis is prevalent throughout the Northern He
73                                              Lyme borreliosis is the most common tick-borne disease i
74 i sensu lato (s.l.), the tick-borne agent of Lyme borreliosis, is a bacterial species complex compris
75  the typical cutaneous manifestation of late Lyme borreliosis, is a chronic skin condition.
76 orferi sensu lato, the causative organism of Lyme borreliosis, is a heterogeneous group of spirochete
77 e characteristic skin manifestation of acute Lyme borreliosis, is a self-limited lesion.
78   Borrelia burgdorferi, a causative agent of Lyme borreliosis, is a zoonotic pathogen that survives i
79 he thyX gene for thymidylate synthase of the Lyme borreliosis (LB) agent Borrelia burgdorferi is loca
80    Human granulocytic ehrlichiosis (HGE) and Lyme borreliosis (LB) are acquired after Ixodes ricinus-
81                            In North America, Lyme borreliosis (LB) is a tick-borne disease caused by
82                             The diagnosis of Lyme borreliosis (LB) is commonly made by serologic test
83                                        Human Lyme borreliosis (LB) is the most prevalent arthropod-bo
84 s (72.2%); other objective manifestations of Lyme borreliosis (LB) were present in 11 (7.6%).
85 mes need to evaluate a patient for suspected Lyme borreliosis (LB) who may have acquired the infectio
86 demic and much-debated tick-borne infection, Lyme borreliosis (LB), are unknown.
87 ers, such as depression, are associated with Lyme borreliosis (LB).
88 ated with sequences from 35 genomes of eight Lyme-borreliosis (LB) group Borrelia species and 7 Relap
89                                              Lyme borreliosis (Lyme disease) is often said to be asso
90  has not been studied using the experimental Lyme borreliosis model of infection of C3H mice with Bor
91 ogic testing also revealed possible cases of Lyme borreliosis (n = 142), babesiosis (n = 41), and HME
92 rom persons who had been diagnosed as having Lyme borreliosis on the basis of the presence of erythem
93                                              Lyme borreliosis, or Lyme disease (LD), is a tick-borne
94  with the development of neuroborreliosis in Lyme borreliosis patients in Europe.
95 ned after successful antibiotic treatment of Lyme borreliosis patients.
96        Borrelia burgdorferi, the pathogen of Lyme borreliosis, persists in nature through a tick-rode
97                Humans and other animals with Lyme borreliosis produce antibodies to a number of compo
98           Although tick immunity can prevent Lyme borreliosis, protection is not afforded against gra
99 ibodies, especially IgM antibodies, in early Lyme borreliosis sera provides additional evidence that
100                                        Early Lyme borreliosis sera with significant titers of anti-ou
101      In vivo studies using a murine model of Lyme borreliosis showed that while OspC F(ab)(2) fragmen
102 osts rather than tick species are the key to Lyme borreliosis spirochaete diversity.
103      In contrast to RF Borrelia species, the Lyme borreliosis spirochete Borrelia burgdorferi is amen
104 , suggesting that BptA may be widely used by Lyme borreliosis spirochetes for persistence in nature.
105 ith the concept of selective transmission of Lyme borreliosis spirochetes.
106  Borrelia burgdorferi spirochetes that cause Lyme borreliosis survive for a long time in human serum
107 Experts disagree on the significance of post-Lyme borreliosis symptoms.
108 ocytoma is a rare cutaneous manifestation of Lyme borreliosis that has been reported almost exclusive
109                                    In murine Lyme borreliosis, the absence of gamma delta T lymphocyt
110                        In the mouse model of Lyme borreliosis, the host immune response during infect
111  present studies, we use the murine model of Lyme borreliosis to define an OspA T cell epitope locate
112 es of sera from patients with early or later Lyme borreliosis to the antibody reactivities of sera fr
113                                A recombinant Lyme borreliosis vaccine consisting of outer surface pro
114   Twelve of 15 patients (80%) with suspected Lyme borreliosis, versus 0 of 10 controls, were positive
115 equence of Borrelia burgdorferi, an agent of Lyme borreliosis, was used to study the genetic content
116 intravital microscopy and the mouse model of Lyme borreliosis, we observed that Borrelia burgdorferi
117 rain to use for the development of a generic Lyme borreliosis Western blot for Europe.
118  protective immunogen in the murine model of Lyme borreliosis when mice were challenged (needle inocu
119                       These diseases include Lyme borreliosis, which is caused by Borrelia burgdorfer
120 i) in the upper midwestern USA, which causes Lyme borreliosis with unusually high spirochaetaemia.
121 Borrelia burgdorferi, the causative agent of Lyme borreliosis, with phagocytic cells induces the acti

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