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1 wal [4, 5] and feeding [6] in the pond snail Lymnaea.
2 o 6.2 mM for Aplysia and 0.12 to 0.22 mM for Lymnaea.
5 that has the highest known affinity for the Lymnaea AChBP and also potently blocks the alpha7 nAChR
6 on are possible for approximately 56% of the Lymnaea AChBP sequence, covering primarily the outer sur
7 r of 4,6-disubstituted 2-aminopyrimidines to Lymnaea AChBP, with different molecular variants exhibit
9 f nitric oxide (NO) production in the CNS of Lymnaea, an established model for molecular analysis of
11 ing substituted anabaseines with AChBPs from Lymnaea, Aplysia, and Bulinus species and correlated the
12 Secretions of the exocrine albumen gland of Lymnaea are packaged in the eggs of an egg mass before t
18 atively damped swelling-tension responses of Lymnaea neurons (no BAPTA) were consistent with feedback
19 hods to measure Ca2+ channel inactivation in Lymnaea neurons-one method, based upon the conventional
23 al approach, we demonstrate that the mollusc Lymnaea performs a sophisticated form of decision-making
24 g masses of the freshwater gastropod mollusc Lymnaea provide a microenvironment for developing embryo
28 ith distinct pharmacological profiles [i.e., Lymnaea stagnalis (Ls) AChBP of low neonicotinoid and hi
29 2 and the acetylcholine-binding protein from Lymnaea stagnalis (Ls-AChBP) confirms Ls-AChBP as struct
30 , we chronically exposed freshwater snails ( Lymnaea stagnalis ) to synthetic water spiked with Cu th
31 alifornica, Pleurobranchaea californica, and Lymnaea stagnalis -three well-recognized models in cellu
32 erred to as model I, was built from both the Lymnaea stagnalis acetylcholine binding protein (AChBP)
33 constructed from the human alpha(7) AChR and Lymnaea stagnalis acetylcholine binding protein (AChBP),
34 tinic acetylcholine receptor (nAChR) and the Lymnaea stagnalis acetylcholine binding protein (Ls-AChB
35 L112Q, and M114T, into the binding pocket of Lymnaea stagnalis acetylcholine-binding protein (Ls-AChB
37 r and reward, and research on the pond snail Lymnaea stagnalis elucidated the role of a behavior-init
38 e of Cu were evaluated in the benthic grazer Lymnaea stagnalis following 4-5 h exposures to Cu adsorb
39 entity over a stretch of 294 residues with a Lymnaea stagnalis G-protein-linked receptor (LSGLR).
41 ly, we trained intact specimens of the snail Lymnaea stagnalis in a single conditioning trial using a
44 ic behaviour we show that the feeding CPG of Lymnaea stagnalis is itself associated with another, and
45 ted during the maturation of the pond snail, Lymnaea stagnalis L., applying light and electron micros
47 tral nervous system of the gastropod mollusk Lymnaea stagnalis produces a soluble protein that specif
48 re of the acetylcholine binding protein from Lymnaea stagnalis to model the chicken alpha7 agonist-bi
49 ntified neurons in the CNS of the pond snail Lymnaea stagnalis to study the role of endogenous NO sig
52 re of the acetylcholine-binding protein from Lymnaea stagnalis with NS9283, a stoichiometry selective
53 these transposon families in the pond snail Lymnaea stagnalis, a cosmopolitan vector of trematodes i
55 protein (AChBP) from the fresh water snail, Lymnaea stagnalis, shows it to be a structural homolog o
56 using central neurons from the invertebrate Lymnaea stagnalis, we demonstrate that menin coordinates
57 te circuit generating feeding in the mollusk Lymnaea stagnalis, we identified three independent pathw
67 etopleura, the limpet Tectura, and the snail Lymnaea, the MAPK pathway is activated in the 3D cell bu
68 rs to produce significant amounts of a novel Lymnaea trypsin inhibitor (LTI), a second peptide that w
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