1 e effect against the incidence of individual MS components.

2 t mediate the association between ox-LDL and MS components.

3 ciated with four of five metabolic syndrome (MS) components.

4 ly" (170 ms) and a "late" (approximately 300 ms) component].

5 tionships of endothelial function with the 5 MS components (analyzed as continuous variables) reveale

6                     The relationships of the MS components (as defined according the National Cholest

7                                       The GC-MS component co-elution was overcome by GCxGC-qMS.

8                                      The RLF-M component consists of complexes of all six Xenopus min

9 DL may reflect core mechanisms through which MS components develop and progress in parallel with insu

10 ts showing the same trend were found for all MS components except glucose concentration.

11 mportant predictors of survival were size of M-component in 24-hour urine, number of involved organs,

12 hermo-sensitive pathways, we reveal that the M component is predominantly dependent on visual input,

13                                       The 75 ms component is lost when the Fe-S protein is removed fr

14 e evolution of the ape-human group, (ii) the M component of layer 4A was subsequently modified (and p

15 est that EBNA3C can directly regulate the G2/M component of the host cell cycle machinery, allowing f

16 r channels associated with both Ileak and IK(M) components of the sustained macroscopic currents.

17 rature as evidenced by the out-of-phase (chi(M)'') component of the alternating current (ac) magnetic

18 pacitance measurements revealed a rapid (<50 ms) component of exocytosis.

19 rtex of awake animals to identify early (<32 ms) components of the population response to a 70 dB cli

20  shifts on green or orange fields, the L and M components were detected about equally well by the LUM

21 tely 10 micro s) and slow ( approximately 50 ms) components were found.