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   1 oiety from a culture of a virulent strain of M. arthritidis.                                         
     2  genome sequence and a transposon library of M. arthritidis.                                         
     3 oduct) predicted from the genome sequence of M. arthritidis.                                         
     4 poprotein designated MAA1 in cytadherence of M. arthritidis.                                         
     5 ed in M. pulmonis, and Tn4001T transposed in M. arthritidis.                                         
     6  by the lack of genetic systems for use with M. arthritidis.                                         
     7 nsfer of Tn916 from an enterococcal donor to M. arthritidis.                                         
     8 h the recently described MlpD lipoprotein of M. arthritidis.                                         
  
    10 are procedures for genetic transformation of M. arthritidis and conjugal transfer of Tn916 from an en
    11 from a 41-kDa known bioactive lipoprotein of M. arthritidis, avidly bound to purified apoA-1 that sep
    12  and upstream DNA sequences were cloned from M. arthritidis clonal variants differing in MAA2 express
    13     We suggest that macrophage activation by M. arthritidis could play a significant role in the infl
    14 , the process of obtaining purified MAM from M. arthritidis culture supernatants is extremely time-co
    15 ge-activating lipopeptide-2, activity of the M. arthritidis-derived 28-kDa component was dependent up
  
    17 ed that bioactive lipopeptides prepared from M. arthritidis grown in serum-free medium and also from 
    18 ritis in C3H/HeJ mice following injection of M. arthritidis in comparison to the mild disease seen in
  
  
  
    22 To study the pathogenic significance of MAM, M. arthritidis mutants that overproduced or failed to pr
  
  
  
  
    27 ing and sequencing of the maa2 gene from two M. arthritidis strains, 158p10p9 and H606, expressing tw
  
    29  serum-free medium supplemented with starch, M. arthritidis synthesized higher levels of rhamnose, wi
    30  examining the role of the superantigen MAM (M. arthritidis T-cell mitogen) in the development of aut
    31 The preparations from the virulent strain of M. arthritidis were also more potent in activating dendr
  
    33 riton X-114 extracts of a virulent strain of M. arthritidis were found to be more potent in activatin
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