ライフサイエンスコーパス: M. hominis

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1 a suggest donor-to-recipient transmission of M. hominis.

2 d to 24 (29.6%) of the 81 women negative for M. hominis.

3 donor for patient 1, which subsequently grew M. hominis.

4 mutator element in the vaa gene that governs M. hominis adherence and highlight the importance of mut

5 Increasing levels of G. vaginalis and M. hominis and decreasing levels of lactobacilli were si

6 ergence of Vaa could affect the adherence of M. hominis and evasion of antibody-mediated immunity, th

7 he results of these studies demonstrate that M. hominis and M. hominis antigen are potent stimulators

8 acteria will not be reliable for recovery of M. hominis and that specialized media and incubation con

9 ase demonstrates the pathogenic potential of M. hominis and the need for rapid recognition of the org

10 hree log(10) bacterial counts (G. vaginalis, M. hominis, and lactobacilli) in our model improved the

11 hese studies demonstrate that M. hominis and M. hominis antigen are potent stimulators of type II epi

12 tative bacterial counts for lactobacilli and M. hominis are better correlates of CVL HIV RNA than are

13 m the joint synovial fluid of a patient with M. hominis-associated arthritis, which indicated that Va

14 terial counts (P=.006; inverse association), M. hominis bacterial counts (P=.0001; positive associati

15 ate analyses, we found that G. vaginalis and M. hominis bacterial counts, Candida vaginitis, and herp

16 Clinicians should be aware that M. hominis can cause surgical site infections, and may n

17 had log(10) G. vaginalis counts and log(10) M. hominis counts greater than 6.81 and 4.82, respective

18 Here we describe the recovery of M. hominis from a brain abscess associated with a postpa

19 PCR quantification of G. vaginalis and M. hominis from CVL is significantly more sensitive than

20 e to challenge with LPS, U. urealyticum, and M. hominis in a concentration-dependent fashion.

21 No BacT/ALERT bottles containing M. hominis in simulated blood cultures were flagged posi

22 e data strongly suggest that the presence of M. hominis in the lungs of premature infants may initiat

23 Surfactant suppressed LPS and M. hominis induced TNF-alpha production in a dose-depend

24 ng chronic, active arthritis associated with M. hominis infection and is highly immunogenic in the hu

25 cated that Vaa phase variation occurs during M. hominis infection in the natural host.

26 ransplant recipients presented with invasive M. hominis infections at multiple sites characterized by

27 In a single center, a cluster of M. hominis infections were identified in lung transplant

28 eloped surgical site infections, including 2 M. hominis infections.

29 ICU were found to have genetically unrelated M. hominis isolates, excluding patient-to-patient transm

30 The M. hominis-like mycoplasma neither inhibits nor enhances

31 Only 7 women were positive for M. hominis; none were allele 2 homozygotes as opposed to

32 We investigated a cluster of M. hominis surgical site infections in patients who unde

33 ich correlated precisely with the ability of M. hominis to adhere to cultured human cells.

34 n requirements and relatively slow growth of M. hominis warrant that dependence on automated systems

35 s, Ureaplasma spp. was detected in 23 (82%), M. hominis was detected in 3 (11%), and both were detect

36 Viability of clinical isolates of M. hominis was maintained for 7 days in BacT/ALERT media

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