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1 ic infection, and birth on site as risks for MAC infection.
2 aB to promote monocyte migration to sites of MAC infection.
3 or disseminated Mycobacterium avium complex (MAC) infection.
4 d prevalence of Mycobacterium avium complex (MAC) infections.
5 investigating the influence of the level of MAC infection and the effect of other drugs on the frequ
6 xic, IL-12 is an effective immunotherapy for MAC infection, and combination with clarithromycin reduc
9 nt of disseminated Myocardium avium complex (MAC) infection, beige mice were infected with MAC strain
11 epidemiology of Mycobacterium avium complex (MAC) infections in children with and without human immun
12 nd disseminated Mycobacterium avium-complex [MAC] infection) in persons whose CD4(+) T lymphocyte cou
13 week study to determine whether treatment of MAC infection is associated with decreases in plasma tum
14 stages of AIDS, Mycobacterium avium complex (MAC) infection is the most common disseminated bacterial
16 nts of less than 50 cells/mL and subclinical MAC infection may be associated with a severe illness, c
19 ents to further define the mechanism whereby MAC infection of macrophages initiates monocyte migratio
20 ionship between Mycobacterium avium complex (MAC) infection of blood and bone marrow was studied in h
23 suggest that with an initially high level of MAC infection, the addition of ethambutol may only delay
24 prophylaxis for Mycobacterium avium complex (MAC) infection to describe the specific signs, symptoms,
25 g therapy was 77 weeks, and the incidence of MAC infection was 1.44/100 person-years (95% confidence
26 lammatory response to previously subclinical MAC infection was associated with leucocytosis in all pa
27 rophages were treated either before or after MAC infection with different concentrations of IL-7.
28 of experimental murine Mycobacterium avium (MAC) infection with interleukin-12 (IL-12) significantly
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