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1 MAC adopts a 'split-washer' configuration, in contrast t
2 MAC also influences the outcomes of cardiac surgery and
3 MAC and AEC were exposed to different amounts of Mn3O4 (
4 MAC directly lyses pathogens by a 'multi-hit' mechanism;
5 MAC was present in 1,149 participants (20.4%).
6 MACs and OCs undergo highly similar 5hmC and 5mC changes
7 MACs from patients were cultured with and without 10 nM
8 remaining patients were categorized into (1) MAC and (2) elective intubation for the procedure (elect
12 rect analysis of the relevant forces, that a MAC balance can be achieved when the viscosity is reduce
13 s of microbe-microbe proximity reveal that a MAC-deficient diet alters monophyletic spatial clusterin
18 a retrospective cohort study evaluating all MAC isolates obtained from pulmonary specimens at our in
23 obabilities of overall survival with RIC and MAC regimens were 45% and 48%, respectively (P = .99).
25 The source code, binaries for MS Windows and MAC OS X as well as test LC-MS data are available for do
27 an MAC-PP, the highest AFS grade and no anti-MAC treatment were correlated with radiographic progress
29 logical persistence of MAC-LD was defined as MAC-PP exceeding 1 year, in contrast with the negative-c
30 y capture enzyme-linked immunosorbent assay (MAC-ELISA) followed by supplemental testing of specimens
31 y capture enzyme-linked immunosorbent assay (MAC-ELISA) for screening, followed by a confirmatory pla
35 -capture enzyme-linked immunosorbent assays (MAC/GAC-ELISAs) targeted at envelope protein (E) of deng
40 d a C6 antisense oligonucleotide that blocks MAC formation by inhibiting C6, and we compared its ther
48 orporating K[nido-7-CH3(CH2)15-7,8-C2B9H11] (MAC) in the bilayer membrane while encapsulating the hyd
50 stin-specific medial arterial calcification (MAC) is an arterial disease commonly referred as Monckeb
52 orce, rotational force, and buoyancy (called MAC balance for Magnetic, Archimedean, Coriolis) with on
56 tion of microbiota-accessible carbohydrates (MACs) from the diet results in thinner mucus in the dist
57 is of Chemotherapy in Nasopharynx Carcinoma (MAC-NPC) collaborative group to assess the addition of c
58 outcomes between monitored anesthesia care (MAC) and general anesthesia (GA) in patients presenting
59 ND & AIMS: Use of monitored anesthesia care (MAC) for gastrointestinal endoscopy has increased in the
62 (TRB) and Multiple Atmospheric Circulation (MAC) to explore the mechanism of MAC driving the drying
63 varying degrees of virulence and classifying MAC isolates into distinct species aids in identifying w
64 microphthalmia, anophthalmia, and coloboma (MAC) and inherited retinal dystrophies, collectively rep
65 driving the drying in TRB, through comparing MAC between abundant and scarce precipitation years.
66 ormed a phase III randomized trial comparing MAC with RIC in patients with acute myeloid leukemia or
67 he abundance of the membrane attack complex (MAC) and performed immunofluorescence studies on eyes fr
68 ent activation, the membrane attack complex (MAC) assembles from fluid-phase proteins to form pores i
70 interacted with the membrane attack complex (MAC) components C5, C7, and C9, thereby blocking the ass
71 Terminal complement membrane attack complex (MAC) formation is induced initially by C5b, followed by
74 C7 protein, soluble membrane attack complex (MAC), and IL-1beta expression compared with the donor C7
75 sential role in the membrane attack complex (MAC), which forms a lethal pore on the cellular surface
78 stent growth of Mycobacterium avium complex (MAC) in the lungs indicates continuous infection in MAC
82 rface assembly of membrane attack complexes (MACs), which promote inflammation by causing aberrant si
83 not exceed maximum acceptable concentration (MAC) of 0.003mgkg(-1)bw(-1)day(-1) through daily consump
84 regulatory maximum allowable concentrations (MACs), with the exception of the higher molecular weight
86 atients received myeloablative conditioning (MAC), and 21 patients received reduced-intensity conditi
87 es compared with myeloablative conditioning (MAC), making RIC-HSCT a viable option for older patients
89 (RIC) instead of myeloablative conditioning (MAC); however, the biology underlying this treatment rem
91 cently reconstituted minimal actin cortices (MACs) and here advanced our assay to investigate effects
92 whereby we specify a maximum allowable cost (MAC) per ton of CO2 stored, a priori, and determine the
93 The reaction utilizes a masked acyl cyanide (MAC) reagent, which enables the one-pot preparation of a
97 (CDC) Zika MAC-ELISA, the InBios ZIKV Detect MAC-ELISA, and the Euroimmun anti-Zika Virus IgM ELISA.
99 involve strategies that incorporate dietary MACs as well as taxa not currently present in the Wester
101 at taxa driven to low abundance when dietary MACs are scarce are inefficiently transferred to the nex
102 es continuous infection in MAC lung disease (MAC-LD), but its clinical significance has not been inve
105 D receptor was transiently expressed during MAC differentiation and that in vitro, calcitriol increa
108 imize an IgM and IgG antibody-capture ELISA (MAC/GAC-ELISA) to detect anti-NS1 antibodies and compare
109 ence of bacteria-induced metamorphic events: MACs induce larval settlement; then, particular properti
110 tern, and positive AFS had an OR of 17.7 for MAC-PP, and those with >/=2 of the factors had a 4.5-fol
113 dictors of persistent culture-positivity for MAC (MAC-PP) and its impact on radiographic deterioratio
115 re frequent and significantly different from MAC: epidermal disarray; pagetoid infiltration of dendri
126 the lungs indicates continuous infection in MAC lung disease (MAC-LD), but its clinical significance
130 V infection, we show that the CDC and InBios MAC-ELISAs perform comparably to each other, with positi
131 immun ZIKV ELISA to either the CDC or InBios MAC-ELISAs resulted in positive agreement, negative agre
133 have described the structures of individual MAC components and subcomplexes; however, the molecular
139 increased differentiation of monocytes into MACs in both SLE and in a model using the prototypic SLE
140 hich C7 defends against bacteria may involve MAC formation, leading to NLRP3 inflammasome activation
142 investigated mechanisms crucial for limiting MAC assembly and preserving cellular integrity in the RP
143 es in the microbiota of mice consuming a low-MAC diet and harbouring a human microbiota are largely r
144 However, over several generations, a low-MAC diet results in a progressive loss of diversity, whi
145 rs of persistent culture-positivity for MAC (MAC-PP) and its impact on radiographic deterioration in
148 anscriptionally active somatic macronucleus (MAC) and the transcriptionally silent germ-line micronuc
153 transcriptionally active genes in the mature MAC genome, making these ciliates model organisms to stu
155 ar, Baldcypress, and Blue spruce) had median MAC values ranging from 1.4 x 10(-2) m(2) g(-1) to 7.9 x
156 Methyl vinyl ketone (MVK) and methacrolein (MAC) are key oxidation products (iox) of isoprene, the m
157 ambient iox (from 0 to 6 ppbv of a 1:1 = MVK/MAC mixture) in all the investigated species, indicating
161 fg (500 nm </= mobility diameter </=950 nm), MAC values in parentheses are the 16(th) and 84(th) perc
162 mbining the use of multiantigen VLP- and NS1-MAC-ELISAs was developed and can be practically applied
164 ural protein 1 (NS1)-specific MAC-ELISA (NS1-MAC-ELISA) on archived acute-phase serum specimens from
165 ed positive-to-negative (P/N) ratios for NS1-MAC/GAC-ELISAs after the depletion of anti-prM/E antibod
167 along with naive sera, were subjected to NS1-MAC/GAC-ELISAs before or after depletion of anti-prM/E a
168 Consistent with these in vitro observations, MAC internalization occurred in human coronary ECs in vi
169 entives to promote appropriate allocation of MAC based on clinically meaningful patient factors.
174 mechanisms contributing to the formation of MAC and the clinical implications of this disease entity
176 idence, to our knowledge, that inhibition of MAC formation in otherwise complement-sufficient animals
177 nt opsonization, site-targeted inhibition of MAC should be considered to promote recovery postneurotr
178 cking the assembly and membrane insertion of MAC resulting in significant inhibition of the hemolytic
180 irculation (MAC) to explore the mechanism of MAC driving the drying in TRB, through comparing MAC bet
184 ignificantly associated with the presence of MAC (odds ratio [OR] per triglyceride GRS unit: 1.73; 95
185 e levels was associated with the presence of MAC, a risk factor for clinically significant mitral val
188 (IES) are excised, followed by rejoining of MAC-destined sequences, while fragmentation occurs at co
190 should focus on better defining the role of MAC and facility and organizational factors that affect
193 and promote CDC with a very low threshold of MAC binding, thus providing additional insight into thei
196 investigated factors associated with use of MAC in an integrated health care delivery system with a
198 ity factors were also associated with use of MAC, although again the magnitude of these associations
203 ad this response required internalization of MACs in a clathrin-, AP2-, and dynamin-dependent manner
204 eficient patients had an increased number of MACs compared with controls (p=0.04) but impaired migrat
205 l settlement; then, particular properties of MACs encoded by a specific locus in P. luteoviolacea ini
207 use of ruxolitinib pretransplant, optimizing MAC to decrease toxicity, and use of posttransplant JAK2
214 ar block) with Monitored Anesthesia Care (RA-MAC) or General Anesthesia (GA) for open globe injury re
217 Globe injury repair was performed using RA-MAC in 351/448 (78%) patients and general anesthesia in
219 ts pre-HCT were randomly assigned to receive MAC (n = 135) or RIC (n = 137) followed by HCT from HLA-
220 to sites of injury and significantly reduced MAC deposition, microglial accumulation, mitochondrial s
222 ) with a myeloablative conditioning regimen (MAC) before allogeneic transplantation in patients with
223 ad a mass-specific absorption cross section (MAC) of 7.89 +/- 0.25 m(2) g(-1) at lambda = 550 nm and
226 m of vitamin D (calcitriol) restored the SLE MAC phenotype towards that of healthy subjects with redu
229 can be isolated by FACS or magnetic sorting (MAC) which give rise to dopamine neurons expressing TH a
230 particle therapy to reverse elastin-specific MAC and circumvent side effects associated with systemic
231 nous NP injections reversed elastin-specific MAC in rats after four injections over a 2-week period.
232 anti-nonstructural protein 1 (NS1)-specific MAC-ELISA (NS1-MAC-ELISA) on archived acute-phase serum
235 rapy for acute posterior circulation stroke, MAC is feasible and appears to be as safe and effective
237 y a 'multi-hit' mechanism; however, sublytic MAC pores on host cells activate signalling pathways.
238 ds light on the mechanisms by which sublytic MAC causes tumor cell responses and exposes a gene expre
244 ring five developmental stages revealed that MACs induce the regulation of groups of genes important
247 rstanding of the roles of complement and the MAC in tumor development and progression, which in turn
250 rs (interquartile range, 60-75 years) in the MAC group vs 67 years (interquartile range, 55.5-78.5 ye
253 a structural model of C9, as present in the MAC, hints at their putative roles in pore formation or
254 6 patients with MAC-LD, 75 (60%) were in the MAC-PP group; these patients had a higher proportion of
257 plication in TBI patients, inhibition of the MAC should be considered to reduce posttraumatic neurolo
258 n symmetric and asymmetric components of the MAC underpin a molecular basis for pore formation and su
259 mponent 9 (C9) is the major component of the MAC, a multi-protein complex that forms pores in the mem
267 required for recruitment of activated Akt to MAC(+) endosomes, but not for MAC internalization or for
270 ree survival and overall survival similar to MAC in patients with MDS or secondary acute myeloid leuk
277 se of high relapse incidence with RIC versus MAC (48.3%; 95% CI, 39.6% to 56.4% and 13.5%; 95% CI, 8.
280 used to confirm positive results in the VLP-MAC-ELISA, the specificity of serodiagnosis, especially
281 ts, the triglyceride GRS was associated with MAC (OR per triglyceride GRS unit: 1.79; 95% CI: 1.32 to
283 ower in patients receiving RIC compared with MAC and in patients having progression compared with pat
284 r TRM but higher relapse rates compared with MAC, with a statistically significant advantage in RFS w
285 gnificantly prolonged survival compared with MAC-transplanted recipients (TBI of 850 cGy plus cycloph
288 ions of lipid genetic risk scores (GRS) with MAC in 3 large patient cohorts: the Framingham Health St
290 SD] age, 34.5 [6.1] years), 16 patients with MAC (mean [SD] age, 49.6 [17.9] years), and 5 patients w
291 ypes, and Group 2 consisted of patients with MAC and MEL; RCM and dermoscopy were used for lips analy
293 Microbiological persistence in patients with MAC-LD is not uncommon and leads to an increased risk of
296 p, patients who underwent the procedure with MAC had similar rates of successful reperfusion, good cl
297 y using multilevel logistic regression, with MAC use modeled as a function of procedure year, patient
299 rial that compared a busulfan-based RIC with MAC in patients with MDS or secondary acute myeloid leuk
300 or Disease Control and Prevention (CDC) Zika MAC-ELISA, the InBios ZIKV Detect MAC-ELISA, and the Eur
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