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1                                              MAC adopts a 'split-washer' configuration, in contrast t
2                                              MAC also influences the outcomes of cardiac surgery and
3                                              MAC and AEC were exposed to different amounts of Mn3O4 (
4                                              MAC directly lyses pathogens by a 'multi-hit' mechanism;
5                                              MAC was present in 1,149 participants (20.4%).
6                                              MACs and OCs undergo highly similar 5hmC and 5mC changes
7                                              MACs from patients were cultured with and without 10 nM
8 remaining patients were categorized into (1) MAC and (2) elective intubation for the procedure (elect
9 en of the lymphatic endothelium in an ICAM-1/MAC-1-dependent manner.
10         Sensitivity of the rapid tests and 3 MAC-ELISAs was <50%, and thus these assays are not recom
11 ters against lipid antigens extracted from a MAC strain.
12 rect analysis of the relevant forces, that a MAC balance can be achieved when the viscosity is reduce
13 s of microbe-microbe proximity reveal that a MAC-deficient diet alters monophyletic spatial clusterin
14  closed head injury in mice induced abundant MAC deposition in the brain.
15 after RIC and 25% (95% CI, 15% to 36%) after MAC ( P = .29).
16 II to IV was 32.3% after RIC and 37.5% after MAC ( P = .35).
17  disease was 61.6% after RIC and 64.7% after MAC ( P = .76).
18  a retrospective cohort study evaluating all MAC isolates obtained from pulmonary specimens at our in
19                                     Although MAC was initially thought to be an age-related degenerat
20  with increased iC3b/C3b binding avidity and MAC inhibitory activity.
21  South African Medical Research Council, and MAC AIDS Fund.
22 in the differential diagnosis of lip MEL and MAC.
23 obabilities of overall survival with RIC and MAC regimens were 45% and 48%, respectively (P = .99).
24  tumour necrosis factor alpha (TNFalpha) and MAC-conditioned media.
25 The source code, binaries for MS Windows and MAC OS X as well as test LC-MS data are available for do
26              Embedded converter machines and MACs with improved geometry were designed and fabricated
27 an MAC-PP, the highest AFS grade and no anti-MAC treatment were correlated with radiographic progress
28 ade; if these risk factors are present, anti-MAC treatment should be seriously considered.
29 logical persistence of MAC-LD was defined as MAC-PP exceeding 1 year, in contrast with the negative-c
30 y capture enzyme-linked immunosorbent assay (MAC-ELISA) followed by supplemental testing of specimens
31 y capture enzyme-linked immunosorbent assay (MAC-ELISA) for screening, followed by a confirmatory pla
32 y-capture enzyme-linked immunosorbent assay (MAC-ELISA).
33 y capture enzyme linked immunosorbent assay (MAC-ELISA).
34  capture enzyme-linked immunosorbent assays (MAC-ELISAs), and indirect immunofluorescence tests.
35 -capture enzyme-linked immunosorbent assays (MAC/GAC-ELISAs) targeted at envelope protein (E) of deng
36 tone modification and expression patterns at MAC-/OC-specific genes.
37 rential histone modification changes between MACs and OCs.
38 acquisition of differential features between MACs and OCs also depends on TET2/TDG.
39 e SETD1A is differentially recruited between MACs and OCs in a TET2-dependent manner.
40 d a C6 antisense oligonucleotide that blocks MAC formation by inhibiting C6, and we compared its ther
41 lar to that seen with CD4(+) T cells in both MAC and AGM.
42 ent-naive noncavitary nodular bronchiectatic MAC lung disease.
43 ents with noncavitary nodular bronchiectatic MAC lung disease.
44 nregulation of IP-10 and changes in ED:EI by MAC regulation of eNOS in endothelial cells.
45       Calcitriol reduced IP-10 expression by MACs (p<0.0006), and blockade of IP-10 restored the angi
46  endosome-based signaling complex induced by MACs to stabilize NIK.
47 ne-mediated activation, NIK stabilization by MACs did not involve cIAP2 or TRAF3.
48 orporating K[nido-7-CH3(CH2)15-7,8-C2B9H11] (MAC) in the bilayer membrane while encapsulating the hyd
49                Mitral annulus calcification (MAC) is a chronic, degenerative process in the fibrous b
50 stin-specific medial arterial calcification (MAC) is an arterial disease commonly referred as Monckeb
51                      Mitral annular calcium (MAC), commonly identified by cardiac imaging, is associa
52 orce, rotational force, and buoyancy (called MAC balance for Magnetic, Archimedean, Coriolis) with on
53 h a sensitive microfluidic antibody capture (MAC) chip.
54 and involves the methylene alkoxy carbamate (MAC) self-immolative unit.
55         Microbiota-accessible carbohydrates (MACs) found in dietary fibre have a crucial involvement
56 tion of microbiota-accessible carbohydrates (MACs) from the diet results in thinner mucus in the dist
57 is of Chemotherapy in Nasopharynx Carcinoma (MAC-NPC) collaborative group to assess the addition of c
58  outcomes between monitored anesthesia care (MAC) and general anesthesia (GA) in patients presenting
59 ND & AIMS: Use of monitored anesthesia care (MAC) for gastrointestinal endoscopy has increased in the
60                    Myeloid angiogenic cells (MACs) promote angiogenesis to restore damaged vessels.
61                    Myeloid angiogenic cells (MACs), derived from circulating monocytes, augment vascu
62  (TRB) and Multiple Atmospheric Circulation (MAC) to explore the mechanism of MAC driving the drying
63 varying degrees of virulence and classifying MAC isolates into distinct species aids in identifying w
64  microphthalmia, anophthalmia, and coloboma (MAC) and inherited retinal dystrophies, collectively rep
65 driving the drying in TRB, through comparing MAC between abundant and scarce precipitation years.
66 ormed a phase III randomized trial comparing MAC with RIC in patients with acute myeloid leukemia or
67 he abundance of the membrane attack complex (MAC) and performed immunofluorescence studies on eyes fr
68 ent activation, the membrane attack complex (MAC) assembles from fluid-phase proteins to form pores i
69 designed to inhibit membrane attack complex (MAC) assembly at sites of C3b/iC3b deposition.
70 interacted with the membrane attack complex (MAC) components C5, C7, and C9, thereby blocking the ass
71 Terminal complement membrane attack complex (MAC) formation is induced initially by C5b, followed by
72 ed by inhibition of membrane attack complex (MAC) formation.
73                 The membrane attack complex (MAC) of the complement system is detected in the traumat
74 C7 protein, soluble membrane attack complex (MAC), and IL-1beta expression compared with the donor C7
75 sential role in the membrane attack complex (MAC), which forms a lethal pore on the cellular surface
76 paid to that of the membrane attack complex (MAC).
77                 The membrane attack complex (MAC)/perforin-like protein complement component 9 (C9) i
78 stent growth of Mycobacterium avium complex (MAC) in the lungs indicates continuous infection in MAC
79  bronchiectatic Mycobacterium avium complex (MAC) lung disease, supporting data are limited.
80  mycobacterial (Mycobacterium avium complex [MAC] or Mycobacterium abscessus) disease.
81        Complement membrane attack complexes (MACs) promote inflammatory functions in endothelial cell
82 rface assembly of membrane attack complexes (MACs), which promote inflammation by causing aberrant si
83 not exceed maximum acceptable concentration (MAC) of 0.003mgkg(-1)bw(-1)day(-1) through daily consump
84 regulatory maximum allowable concentrations (MACs), with the exception of the higher molecular weight
85              Within mobile air-conditioning (MAC), the largest RAC sector and most significant UK sou
86 atients received myeloablative conditioning (MAC), and 21 patients received reduced-intensity conditi
87 es compared with myeloablative conditioning (MAC), making RIC-HSCT a viable option for older patients
88 S) compared with myeloablative conditioning (MAC).
89 (RIC) instead of myeloablative conditioning (MAC); however, the biology underlying this treatment rem
90  those receiving myeloablative-conditioning (MAC) regimens.
91 cently reconstituted minimal actin cortices (MACs) and here advanced our assay to investigate effects
92 whereby we specify a maximum allowable cost (MAC) per ton of CO2 stored, a priori, and determine the
93 The reaction utilizes a masked acyl cyanide (MAC) reagent, which enables the one-pot preparation of a
94                         Masked acyl cyanide (MAC) reagents are shown to be effective umpolung synthon
95                             With dermoscopy, MAC were correctly classified as benign in 13 of 16 case
96 s, the draped pattern-a previously described MAC RCM feature-was identified in all cases.
97 (CDC) Zika MAC-ELISA, the InBios ZIKV Detect MAC-ELISA, and the Euroimmun anti-Zika Virus IgM ELISA.
98  of missing taxa in combination with dietary MAC consumption.
99  involve strategies that incorporate dietary MACs as well as taxa not currently present in the Wester
100 overable after the reintroduction of dietary MACs.
101 at taxa driven to low abundance when dietary MACs are scarce are inefficiently transferred to the nex
102 es continuous infection in MAC lung disease (MAC-LD), but its clinical significance has not been inve
103 he clinical courses associated with distinct MAC species.
104 he modified Rankin scale score distribution (MAC: OR, 1.52; 95% CI, 0.80-2.90; P = .19).
105  D receptor was transiently expressed during MAC differentiation and that in vitro, calcitriol increa
106 ti-NS1 antibodies and compare it with anti-E MAC/GAC-ELISA.
107              We find no evidence that either MAC or PAC are associated with LTL in offspring across t
108 imize an IgM and IgG antibody-capture ELISA (MAC/GAC-ELISA) to detect anti-NS1 antibodies and compare
109 ence of bacteria-induced metamorphic events: MACs induce larval settlement; then, particular properti
110 tern, and positive AFS had an OR of 17.7 for MAC-PP, and those with >/=2 of the factors had a 4.5-fol
111 tivated Akt to MAC(+) endosomes, but not for MAC internalization or for Akt activation.
112  the factors had a 4.5-fold increased OR for MAC-PP relative to the comparison group.
113 dictors of persistent culture-positivity for MAC (MAC-PP) and its impact on radiographic deterioratio
114                  Patients who converted from MAC to GA during the procedure were included in the MAC
115 re frequent and significantly different from MAC: epidermal disarray; pagetoid infiltration of dendri
116 tures that assist in distinguishing MEL from MAC using dermoscopy and RCM.
117                     The conversion rate from MAC to GA was 13% (n = 8).
118  C9 subunits can be recruited to the growing MAC subsequent to membrane insertion.
119                     Sixty-three patients had MAC (36%) and 113 patients had GA (64%).
120  electron cryo-microscopy structure of human MAC at subnanometre resolution.
121  a gene expression signature that implicates MAC as a driver of tumor progression.
122                                           In MAC elevated ROS levels, decreased metabolic activity an
123  its impact on radiographic deterioration in MAC-LD.
124 ection levels were similar to those found in MAC pDC.
125 provement was associated with an increase in MAC number and function.
126  the lungs indicates continuous infection in MAC lung disease (MAC-LD), but its clinical significance
127                      Nucleosome occupancy in MAC and MIC are nonetheless highly correlated with each
128 terminants promote nucleosome positioning in MAC.
129          The improved angiogenic capacity in MACs might be mediated via downregulation of IP-10 and c
130 V infection, we show that the CDC and InBios MAC-ELISAs perform comparably to each other, with positi
131 immun ZIKV ELISA to either the CDC or InBios MAC-ELISAs resulted in positive agreement, negative agre
132                                      Indeed, MAC is associated with an increased incidence of cardiov
133  have described the structures of individual MAC components and subcomplexes; however, the molecular
134  (85%) and serum levels (90%), and inhibited MAC deposition in the injured brain (91-96%).
135                       CD59-2a-CRIg inhibited MAC formation and prevented complement-mediated lysis in
136                       CD59-2a-CRIg inhibited MAC-induced inflammasome activation and IL-1beta product
137        Moreover, PGK significantly inhibited MAC deposition via both the classical and alternative pa
138 ulator CD59 to the RPE cell surface inhibits MAC formation.
139  increased differentiation of monocytes into MACs in both SLE and in a model using the prototypic SLE
140 hich C7 defends against bacteria may involve MAC formation, leading to NLRP3 inflammasome activation
141                 Another possibility involves MAC waves, which arise from the interplay between magnet
142 investigated mechanisms crucial for limiting MAC assembly and preserving cellular integrity in the RP
143 es in the microbiota of mice consuming a low-MAC diet and harbouring a human microbiota are largely r
144     However, over several generations, a low-MAC diet results in a progressive loss of diversity, whi
145 rs of persistent culture-positivity for MAC (MAC-PP) and its impact on radiographic deterioration in
146 an immunodeficiency virus (SIV) in macaques (MAC) lead to chronic inflammation and AIDS.
147 through the presence of somatic macronuclei (MAC) and germline micronuclei (MIC).
148 anscriptionally active somatic macronucleus (MAC) and the transcriptionally silent germ-line micronuc
149 ty-were studied in rat alveolar macrophages (MAC) and epithelial cells (AEC).
150 effectors of innate immunity as macrophages (MACs) and bone remodelling as osteoclasts (OCs).
151                    Benign melanotic macules (MAC) are the most frequent cause of lip pigmentation and
152                        We test for maternal (MAC) and paternal age at conception effects on offspring
153 transcriptionally active genes in the mature MAC genome, making these ciliates model organisms to stu
154                          The median measured MAC at lambda = 660 nm for smoldering oak particles was
155 ar, Baldcypress, and Blue spruce) had median MAC values ranging from 1.4 x 10(-2) m(2) g(-1) to 7.9 x
156  Methyl vinyl ketone (MVK) and methacrolein (MAC) are key oxidation products (iox) of isoprene, the m
157 ambient iox (from 0 to 6 ppbv of a 1:1 = MVK/MAC mixture) in all the investigated species, indicating
158 al myeloid differentiation processes, namely MAC and OC differentiation.
159 tion, C9 assembles directly onto the nascent MAC from solution.
160 isplacement conversion efficiency of the new MACs over the existing ones was demonstrated.
161 fg (500 nm </= mobility diameter </=950 nm), MAC values in parentheses are the 16(th) and 84(th) perc
162 mbining the use of multiantigen VLP- and NS1-MAC-ELISAs was developed and can be practically applied
163 erall assay performances of the VLP- and NS1-MAC-ELISAs.
164 ural protein 1 (NS1)-specific MAC-ELISA (NS1-MAC-ELISA) on archived acute-phase serum specimens from
165 ed positive-to-negative (P/N) ratios for NS1-MAC/GAC-ELISAs after the depletion of anti-prM/E antibod
166                                 When the NS1-MAC-ELISA was used to confirm positive results in the VL
167 along with naive sera, were subjected to NS1-MAC/GAC-ELISAs before or after depletion of anti-prM/E a
168 Consistent with these in vitro observations, MAC internalization occurred in human coronary ECs in vi
169 entives to promote appropriate allocation of MAC based on clinically meaningful patient factors.
170                    Using network analysis of MAC-responsive expression changes, we demonstrate a clus
171 toxin C5a and C5b, an essential component of MAC.
172 nsistent, clinically relevant description of MAC on the basis of contemporary imaging.
173 olism, also contribute to the development of MAC.
174  mechanisms contributing to the formation of MAC and the clinical implications of this disease entity
175            The biological functionalities of MAC has been previously investigated by using either the
176 idence, to our knowledge, that inhibition of MAC formation in otherwise complement-sufficient animals
177 nt opsonization, site-targeted inhibition of MAC should be considered to promote recovery postneurotr
178 cking the assembly and membrane insertion of MAC resulting in significant inhibition of the hemolytic
179          However, the molecular mechanism of MAC assembly remains to be understood.
180 irculation (MAC) to explore the mechanism of MAC driving the drying in TRB, through comparing MAC bet
181               Microbiological persistence of MAC-LD was defined as MAC-PP exceeding 1 year, in contra
182                            The predictors of MAC-PP are low BMI, NB pattern, and high AFS grade; if t
183                    Independent predictors of MAC-PP were low body mass index (BMI), radiographic nodu
184 ignificantly associated with the presence of MAC (odds ratio [OR] per triglyceride GRS unit: 1.73; 95
185 e levels was associated with the presence of MAC, a risk factor for clinically significant mitral val
186 ma lipids is associated with the presence of MAC.
187                         The adjusted rate of MAC use in the VHA increased 17% per year (odds ratio fo
188  (IES) are excised, followed by rejoining of MAC-destined sequences, while fragmentation occurs at co
189 ite its frequency, the clinical relevance of MAC is grossly underappreciated.
190  should focus on better defining the role of MAC and facility and organizational factors that affect
191                                  A series of MAC beta-glucuronide model constructs were prepared to e
192 /-), which is associated with suppression of MAC-mediated inflammasome activation in mC9(-/-).
193 and promote CDC with a very low threshold of MAC binding, thus providing additional insight into thei
194 ects had the greatest effect on the trend of MAC use.
195                These data support the use of MAC as the standard of care for fit patients with acute
196  investigated factors associated with use of MAC in an integrated health care delivery system with a
197                                   VHA use of MAC was associated with patient-level factors that inclu
198 ity factors were also associated with use of MAC, although again the magnitude of these associations
199 ominent factor influencing increasing use of MAC.
200 ctors are only weakly associated with use of MAC.
201 of IP-10 restored the angiogenic capacity of MACs from patients with SLE.
202 ol also augmented the angiogenic capacity of MACs via the down-regulation of CXCL-10.
203 ad this response required internalization of MACs in a clathrin-, AP2-, and dynamin-dependent manner
204 eficient patients had an increased number of MACs compared with controls (p=0.04) but impaired migrat
205 l settlement; then, particular properties of MACs encoded by a specific locus in P. luteoviolacea ini
206                            A further type of MACs with conversion ability, viz.
207 use of ruxolitinib pretransplant, optimizing MAC to decrease toxicity, and use of posttransplant JAK2
208  C9 to the C5b-8 complex forms the C5b-9 (or MAC).
209 g either the mice deficient in C5 and C6, or MAC's regulator CD59.
210                  A novel approach to perform MAC/GAC-ELISAs for NS1 antibody detection was successful
211                                           RA-MAC cases also had a better presenting and final visual
212                                           RA-MAC is a reasonable alternative to GA for the repair of
213                                           RA-MAC was selected more often for Zone 1 and Zone 2 injuri
214 ar block) with Monitored Anesthesia Care (RA-MAC) or General Anesthesia (GA) for open globe injury re
215 ries, there are equal selection ratio for RA-MAC and GA.
216        Open globe injuries repaired under RA-MAC had significantly shorter wound length (p<0.001), mo
217   Globe injury repair was performed using RA-MAC in 351/448 (78%) patients and general anesthesia in
218            Due to antibody cross-reactivity, MAC-ELISA-positive samples may be confirmed with a time-
219 ts pre-HCT were randomly assigned to receive MAC (n = 135) or RIC (n = 137) followed by HCT from HLA-
220 to sites of injury and significantly reduced MAC deposition, microglial accumulation, mitochondrial s
221 ystemic toxicity in patients with refractory MAC lung disease.
222 ) with a myeloablative conditioning regimen (MAC) before allogeneic transplantation in patients with
223 ad a mass-specific absorption cross section (MAC) of 7.89 +/- 0.25 m(2) g(-1) at lambda = 550 nm and
224  as mass-specific absorption cross sections (MAC).
225             Interestingly, PGK bound several MAC proteins simultaneously.
226 m of vitamin D (calcitriol) restored the SLE MAC phenotype towards that of healthy subjects with redu
227 ther enhanced by calcitriol treatment of SLE MACs.
228                         We observed that SLE MACs are dysfunctional and secrete pro-inflammatory cyto
229 can be isolated by FACS or magnetic sorting (MAC) which give rise to dopamine neurons expressing TH a
230 particle therapy to reverse elastin-specific MAC and circumvent side effects associated with systemic
231 nous NP injections reversed elastin-specific MAC in rats after four injections over a 2-week period.
232  anti-nonstructural protein 1 (NS1)-specific MAC-ELISA (NS1-MAC-ELISA) on archived acute-phase serum
233 rM/E containing virus-like particle-specific MAC-ELISA (VLP-MAC-ELISA).
234           Our findings suggest that specific MAC species have varying degrees of virulence and classi
235 rapy for acute posterior circulation stroke, MAC is feasible and appears to be as safe and effective
236 morphosis-associated contractile structures (MACs).
237 y a 'multi-hit' mechanism; however, sublytic MAC pores on host cells activate signalling pathways.
238 ds light on the mechanisms by which sublytic MAC causes tumor cell responses and exposes a gene expre
239                                   Other than MAC-PP, the highest AFS grade and no anti-MAC treatment
240                    Here, we demonstrate that MAC features well-positioned nucleosomes downstream of t
241                        The results show that MAC affects both the magnitude and spatial distribution
242                             Here I show that MAC waves provide a good description of time-dependent z
243                             We conclude that MACs activate noncanonical NF-kappaB by forming a novel
244 ring five developmental stages revealed that MACs induce the regulation of groups of genes important
245                            We also show that MACs were able to positively modulate eNOS expression in
246                                      All the MAC model compounds efficiently released alcohol drug su
247 rstanding of the roles of complement and the MAC in tumor development and progression, which in turn
248                                To assess the MAC technology for ADCs, the potent microtubule-disrupti
249 dies on eyes from 117 donors to evaluate the MAC in aging, early AMD, and advanced AMD.
250 rs (interquartile range, 60-75 years) in the MAC group vs 67 years (interquartile range, 55.5-78.5 ye
251    Fifty-four percent of the patients in the MAC group were men vs 41% in the GA group (P = .44).
252 GA during the procedure were included in the MAC group.
253  a structural model of C9, as present in the MAC, hints at their putative roles in pore formation or
254 6 patients with MAC-LD, 75 (60%) were in the MAC-PP group; these patients had a higher proportion of
255                           Conjugation of the MAC beta-glucuronide AE drug linker to the anti-CD30 ant
256       However, a significant revision of the MAC servicing rate, coupled with a reassessment of non-R
257 plication in TBI patients, inhibition of the MAC should be considered to reduce posttraumatic neurolo
258 n symmetric and asymmetric components of the MAC underpin a molecular basis for pore formation and su
259 mponent 9 (C9) is the major component of the MAC, a multi-protein complex that forms pores in the mem
260                                 Three of the MAC-ELISA kits and 1 indirect immunofluorescence kit had
261 soluble form of the poly-C9 component of the MAC.
262 ear more or less attractive depending on the MAC scenario considered.
263                   These studies validate the MAC self-immolative unit for alcohol-containing payloads
264                                In vitro, the MAC significantly triggered NLRP3 inflammasome activatio
265  design trebled the energy absorption of the MACs.
266 se-separated lipid mono- and bilayers to the MACs.
267 required for recruitment of activated Akt to MAC(+) endosomes, but not for MAC internalization or for
268  and SM pDC were found to be, in contrast to MAC pDC, predominantly negative for CCR5.
269 ation in mediating the observed responses to MAC exposure.
270 ree survival and overall survival similar to MAC in patients with MDS or secondary acute myeloid leuk
271                Media from calcitriol-treated MACs more strongly attenuated TNFalpha-mediated downregu
272                Media from calcitriol-treated MACs significantly increased angiogenesis compared with
273                                    Using two MAC mutations that block P. luteoviolacea from inducing
274  express extremely low levels of CD4, unlike MAC and human pDC.
275  aortic endothelial cells than did untreated MACs from patients with SLE (p=0.01).
276 creased angiogenesis compared with untreated MACs (p=0.01).
277 se of high relapse incidence with RIC versus MAC (48.3%; 95% CI, 39.6% to 56.4% and 13.5%; 95% CI, 8.
278 und between the newly developed NS1- and VLP-MAC/GAC-ELISAs.
279  virus-like particle-specific MAC-ELISA (VLP-MAC-ELISA).
280  used to confirm positive results in the VLP-MAC-ELISA, the specificity of serodiagnosis, especially
281 ts, the triglyceride GRS was associated with MAC (OR per triglyceride GRS unit: 1.79; 95% CI: 1.32 to
282 sterol GRS was significantly associated with MAC.
283 ower in patients receiving RIC compared with MAC and in patients having progression compared with pat
284 r TRM but higher relapse rates compared with MAC, with a statistically significant advantage in RFS w
285 gnificantly prolonged survival compared with MAC-transplanted recipients (TBI of 850 cGy plus cycloph
286 ls was about the double in MEL compared with MAC.
287 tly in patients without cystic fibrosis with MAC and was sustained 1 year after LAI.
288 ions of lipid genetic risk scores (GRS) with MAC in 3 large patient cohorts: the Framingham Health St
289                Conclusion OS was higher with MAC, but this was not statistically significant.
290 SD] age, 34.5 [6.1] years), 16 patients with MAC (mean [SD] age, 49.6 [17.9] years), and 5 patients w
291 ypes, and Group 2 consisted of patients with MAC and MEL; RCM and dermoscopy were used for lips analy
292                                Patients with MAC-LD at multiple medical centers from 2011 to 2016 wer
293 Microbiological persistence in patients with MAC-LD is not uncommon and leads to an increased risk of
294                      Among 126 patients with MAC-LD, 75 (60%) were in the MAC-PP group; these patient
295 in all patients with MEL and 5 patients with MAC.
296 p, patients who underwent the procedure with MAC had similar rates of successful reperfusion, good cl
297 y using multilevel logistic regression, with MAC use modeled as a function of procedure year, patient
298 tistically significant advantage in RFS with MAC.
299 rial that compared a busulfan-based RIC with MAC in patients with MDS or secondary acute myeloid leuk
300 or Disease Control and Prevention (CDC) Zika MAC-ELISA, the InBios ZIKV Detect MAC-ELISA, and the Eur

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