ライフサイエンスコーパス: MAG

1 (JAM-C) and myelin-associated glycoprotein (MAG).

2 lin, such as myelin-associated glycoprotein (MAG).

3 n induced by myelin-associated glycoprotein (MAG).

4 3 (NT3) and myelin-associated glycoprotein (MAG).

5 tion such as myelin-associated glycoprotein (MAG).

6 netrin-1 and myelin-associated glycoprotein (MAG).

7 tic activity for acylating monoacylglycerol (MAG).

8 division of the medial preoptic nucleus (MPN mag).

9 nal levels are commonly regulated by NT3 and MAG.

10 NgR1 supports binding of Nogo-66, OMgp, and MAG.

11 lin-associated genes as Mpz, Mbp, Pmp22, and Mag.

12 asion showed the expression of both MUC1 and MAG.

13 tgrowth almost as effectively as full-length MAG.

14 ors (NgRs) as exclusive axonal receptors for MAG.

15 f endogenous regeneration inhibitors such as MAG.

16 enhance neurite outgrowth in the presence of MAG.

17 trescine's ability to overcome inhibition by MAG.

18 verexpressing p35 can overcome inhibition by MAG.

19 ilar effects on the growth and maturation of MAG.

20 vation of PKC and Rho in response to soluble MAG.

21 nsistent with C1q-mediated neutralization of MAG.

22 romotes neurite outgrowth in the presence of MAG.

23 ellar incorporation of carotenoids, FFAs and MAGs.

24 with 91% purity and 94% overall recovery of MAGs.

25 MAG (0-2.5wt%) had no remarkable impact on the chemical

26 The addition of MAG (0.5wt%) suppressed the effectiveness of alpha-tocop

27 harge of up to 120 mAhg(-1) at a rate of 100 mAg(-1).

28 after 100 cycles at a current density of 400 mAg(-1).

29 TAG; however, increased concentrations of 2-MAG (50-200 microm) resulted in decreased TAG formation.

30 Of 5580 participants who underwent MAG, 586 (11%) were women and the mean (SD) age was 60 (

31 nstrate that myelin-associated glycoprotein (MAG), a well known inhibitor of neurite outgrowth, inhib

32 and Gly(18) has been replaced with alanine (Mag-A).

33 s report establishes for the first time that MAG also promotes resistance to axonal injury and preven

34 the binding pocket in the 2 photoisomers of MAG and (ii) the degree of clamshell closure that is pos

35 itors and promotes neurite outgrowth on both MAG and CNS myelin substrates.

36 ition of neurite outgrowth by both wild-type MAG and CNS myelin.

37 the concentration of triacylglycerols, DAG, MAG and free fatty acids (FFA) and the concentration of

38 gated whether MUC1 is a counter-receptor for MAG and if their interaction contributed to pancreatic p

39 nd flow cytometry, and this occurred in NAG, MAG and IM.

40 .1 (4.5-11.7) years for the groups receiving MAG and LITA+SVG, respectively.

41 in the acyltransferase function but exhibits MAG and LPC hydrolase activities.

42 sted the presence of two separate substrate (MAG and LPC)-binding sites in a single polypeptide.

43 viously shown to bind an intronic element of MAG and modulate alternative exon inclusion from a MAG m

44 dbcAMP), can block the inhibitory effects of MAG and myelin.

45 Nogo, MAG and OMgp are three prototypical myelin inhibitors th

46 that from wild-type mice, but myelin lacking MAG and OMgp is indistinguishable from control.

47 In contrast, deletion of MAG and OMgp stimulates neither axonal growth nor enhanc

48 role for Nogo-A and synergistic actions for MAG and OMgp, presumably through shared receptors.

49 Both mAg and sAg induce F-actin accumulation and actin polyme

50 phosphorylation in BCR microclusters without mAg and sAg, but with much slower kinetics than those in

51 tion of BCR signalosomes in response to both mAg and sAg.

52 ulture and in vivo to overcome inhibition by MAG and that spermidine can promote optic nerve regenera

53 iple labeling immunohistochemistry, confocal maging and analysis to investigate the presence and colo

54 aroyl lactylate (SSL) and monoacylglycerols (MAG) and Bacillus stearothermophilus maltogenic alpha-am

55 yzes the acylation of both monoacylglycerol (MAG) and diacylglycerol (DAG) to generate DAG and TAG, r

56 A, OMgp, and myelin-associated glycoprotein (MAG) and has been proposed to function as the ligand-bin

57 ic lesions of multifocal atrophic gastritis (MAG) and intestinal metaplasia (IM) have occurred.

58 s revealed the presence of monoacylglycerol (MAG) and lysophosphatidylcholine (LPC) hydrolytic activi

59 proteins present in myelin, including Nogo, MAG, and oligodendrocyte-myelin glycoprotein (OMgp), hav

60 Our data indicate that while Nogo, MAG, and OMgp may modulate axon sprouting, they do not p

61 Three proteins in mature myelin (Nogo, MAG, and OMgp) have been purported to be critical in cau

62 Three molecules, Nogo-A, MAG, and OMgp, are produced by oligodendrocytes and shar

63 of the three major myelin inhibitors, Nogo, MAG, and OMgp, in injury-induced axonal growth, includin

64 Three proteins found in myelin--Nogo, MAG, and OMgp--inhibit axon regeneration in vitro and bi

65 f myelin-associated inhibitors such as Nogo, MAG, and OMgp.

66 icity, is a high-affinity receptor for Nogo, MAG, and OMgp.

67 ent in two major myelin inhibitors, Nogo and MAG, and their common receptor NgR1 (or NgR).

68 of triacylglycerides, formation of FFAs and MAGs, and micellar incorporation of carotenoids, FFAs an

69 solin, are recruited to BCR clusters in both mAg- and sAg-stimulated B cells but with different kinet

70 and its role in BCR signalosome formation in mAg- and sAg-stimulated B cells.

71 selectively neutralizing the pathogenic anti-MAG antibodies with carbohydrate-based ligands mimicking

72 of blood IgM memory B cells that recognized MAG antigen.

73 is mediated by IgM autoantibodies binding to MAG antigen.

74 t the adhesive interactions between MUC1 and MAG are of biological significance in pancreatic cancer

75 Domains Ig3-Ig5 of MAG are sufficient to inhibit neurite outgrowth but fail

76 Therefore, MAGEs are implicated in a broad range of diseases includ

77 glycerol was proven to be an alternative to MAG as acyl-group acceptor.

78 DGAT1 (IC(50) of human DGAT1: 16.6+/-4.0 nM (MAG as substrate) and 1499+/-318 nM (DAG as substrate);

79 The possible roles of 2-MAGs as intermediates in cutin synthesis are discussed.

80 or scalable production of n-3 PUFAs enriched MAGs as potential food emulsifier and ingredient.

81 and show the utility of the SGM approach and MAGS as resources for defining novel glycan recognition

82 emical and biophysical studies indicate that MAGEs assemble with E3 RING ubiquitin ligases to form MA

83 enicity and demyelinating properties of anti-MAG autoantibodies are well established, current treatme

84 reological methods to determine when the MPN mag becomes sexually differentiated.

85 show that the first three Ig-like domains of MAG bind with high affinity and in a sialic acid-depende

86 on of NgR2, but not NgR1, are sufficient for MAG binding, and when expressed in neurons, exhibit cons

87 -Cys(336) is deleted and followed by a 13 aa MAG-binding motif of the NgR2 stalk, shows superior bind

88 e inhibitor, myelin-associated glycoprotein (MAG), binds to sialoglycans and other receptors on axons

89 contain the myelin-associated glycoprotein (MAG) but not P(0) or proteolipid protein (PLP), the stru

90 arginine 118 in the extracellular portion of MAG, but it is independent of Nogo signaling in the axon

91 r that selectively inhibits the acylation of MAG by DGAT1 (IC(50) of human DGAT1: 16.6+/-4.0 nM (MAG

92 nd that triacylglycerol (TAG) synthesis from MAG by DGAT1 does not behave according to classic Michae

93 g through IIS pathway regulate maturation of MAG by promoting the growth of MAG cells.

94 MAG can display clarithromycin resistance through the in

95 embers of the Mycobacterium abscessus group (MAG) cause lung, soft tissue, and disseminated infection

96 maturation of MAG by promoting the growth of MAG cells.

97 ce extend significantly shorter processes on MAG compared with wild-type DRG neurons, and regeneratio

98 shows superior binding of OMgp, Nogo-66, and MAG compared with wild-type NgR1 or NgR2.

99 ell envelope is the mycolyl-arabinogalactan (mAG) complex.

100 Elucidating microbe-specific differences in mAG composition could advance biotechnological applicati

101 the brains of double mutants (Phr1(Delta8,9/Mag)), confirming that Pam displays dual regulation of t

102 We here showed that SSL but not MAG delays wheat starch hydrolysis by BStA.

103 led study of rituximab in patients with anti-MAG demyelinating polyneuropathy (A-MAG-DP).

104 This NT3- and MAG-dependent axonal mRNA transport requires activation

105 ssential source of cAMP for BDNF to overcome MAG-dependent inhibition of neurite outgrowth.

106 The data show that (1) neurons in the MPN mag display two distinct phenotypes, those with a single

107 A soluble proteolytic fragment of native MAG, dMAG, also inhibited neurite outgrowth.

108 irst drug that improves some patients with A-MAG-DP in a controlled study.

109 Thirteen A-MAG-DP patients were randomized to rituximab and 13 to p

110 ith anti-MAG demyelinating polyneuropathy (A-MAG-DP).

111 e fatty acids, FFAs, and monoacylglycerides, MAGs) during in vitro digestion of oil-in-water emulsion

112 urther work is required to determine whether MAG dysregulation is a cause or consequence of audiogeni

113 stinct and cell type-specific mechanisms for MAG-elicited growth inhibition.

114 evealed that increased expression of MUC1 or MAG enhanced adhesion.

115 -mRNA and modulates alternative inclusion of MAG exons.

116 covered that myelin-associated glycoprotein (MAG) expression is dramatically decreased in Frings mice

117 d substrata adsorbed with full-length native MAG extracted from purified myelin.

118 When applied acutely, however, MAG-Fc and OMgp-Fc induce a modest degree of growth cone

119 ag-fluo-4 was identical to furaptra's; thus, Mag-fluo-4 also yields reliable kinetic information abou

120 However, because the resting fluorescence of Mag-fluo-4 probably arises largely from indicator that i

121 t is bound with Mg(2+), the amplitude of the Mag-fluo-4 signal, and its calibration in Delta[Ca(2+)]

122 In contrast, the DeltaF time course of Mag-fluo-4 was identical to furaptra's; thus, Mag-fluo-4

123 ators OGB-5N, Fluo-5N, fura-5N, Rhod-5N, and Mag-fluo-4 were evaluated for their ability to accuratel

124 Mag-fluo-4's DeltaF has a larger signal/noise ratio than

125 A low-affinity Ca2+ indicator (mag-fluo-4) trapped within the ER of permeabilized cells

126 e was micro-injected with either furaptra or mag-fluo-4, two low-affinity rapidly responding Ca(2+) i

127 w-twitch fibres that had been AM-loaded with mag-fluo-4: 12.4 +/- 0.8 ms and 17.2 +/- 1.7 ms.

128 smic and ER-Ca(2+) (measured with Fura-2 and Mag-Fluo4) levels are decreased and store-operated Ca(2+

129 s and subgroups support the consideration of MAG for a broader spectrum of patients who are undergoin

130 me the inhibitory effects of both myelin and MAG for cortical, hippocampal, and DRG neurons.

131 that SPD is able to concentrate n-3 PUFAs in MAG form by distilling at proper TE e.g. 125 degrees C,

132 AGs reveals two high-coverage, low-diversity MAGs from Piccard enriched in unique genes related to th

133 The release of FFAs and MAGs from TAGs proceeded faster than their incorporation

134 reconstruct 73 metagenome-assembled genomes (MAGs) from two geochemically distinct vent fields in the

135 SR luminal Ca2+ measured using Mag-Fura-2 was not altered by Casq2 reduction.

136 Competition with chelators mag-fura-2, nitrilotriacetic acid, EDTA, and EGTA estima

137 Using the fluorescent indicator mag-fura-2, the metal released from DHHC3 was identified

138 ing two different calcium probes, Fura-2 and Mag-Fura-2, we found that inactivation of PS in primary

139 C marker S100 and MBP expressions increased; MAG, GFAP, and SCMP expressions were very low.

140 A focal MAG gradient induced polarized endocytosis and concomita

141 Here, we present a comprehensive guide to MAGEs highlighting the molecular mechanisms of MRLs and

142 ever, only the chimeric molecules containing MAG Ig-5 inhibited neurite outgrowth.

143 more, peptides corresponding to sequences in MAG Ig-5, but not Ig-4 or Sn Ig-5, are able to block inh

144 a correlation between the reduction of anti-MAG IgM levels and clinical improvement, an immunologica

145 MAG neuropathy producing high levels of anti-MAG IgM was developed.

146 e, considering the multivalent nature of the MAG-IgM interaction, polylysine polymers of different si

147 he ability of cAMP to overcome inhibition by MAG in culture involves the upregulation of the enzyme a

148 Deletion of Jam-c or Mag in mice recapitulates pathology in HNPP.

149 The physical properties of DAG and MAG in the SSO may be related to the chemical stability

150 inhibitory and repulsive turning effects of MAG in vitro.

151 umulation of myelin-associated glycoprotein (MAG) in LAMP1(+)perinuclear vesicles that fail to migrat

152 irectly with myelin associated glycoprotein (MAG) in myelin, resulting in reduced activation of growt

153 and maturation of the male accessory gland (MAG) in the red flour beetle, Tribolium castaneum.

154 soluble Ag (sAg) and membrane-associated Ag (mAg) in the secondary lymphoid tissue, yet how the physi

155 termed metadata-assisted glycan sequencing (MAGS), in which we combine information from analyses of

156 The size of MAG increased from day 1 to day 5 post-adult emergence (

157 ing of fluorescence (SEER) was used to image mag-indo-1 trapped in the tubular (t) system of mechanic

158 MAG inhibition of axon outgrowth in some neurons is reve

159 hibition of RGCs; however, it does attenuate MAG inhibition of cerebellar granule neurons.

160 A smaller percentage of MAG inhibition of DRGN outgrowth was via gangliosides, e

161 The receptors responsible for MAG inhibition of neurite outgrowth varied with nerve ce

162 t VCN treatment is not sufficient to release MAG inhibition of RGCs; however, it does attenuate MAG i

163 In contrast to DRGNs, in CGNs MAG inhibition was exclusively via gangliosides, whereas

164 In DRGNs, most of the MAG inhibition was via NgRs, evidenced by reversal of in

165 ult in a substantial release of L-MAG (large MAG) inhibition.

166 Our data indicate that MAG inhibits axon outgrowth via two independent receptor

167 A subset of MAGEs initially garnered interest as cancer biomarkers a

168 AMP and putrescine to overcome inhibition by MAG is abolished in the presence of roscovitine, a Cdk i

169 iosides in the absence of inhibitors such as MAG is also shown to inhibit neurite outgrowth in cultur

170 Compared with LITA+SVG, MAG is associated with reduced mortality, repeated revas

171 We conclude that the inhibition site on MAG is carried by Ig domain 5 and that this site is dist

172 The mAG is composed of lipid mycolic acids, and arabinofuran

173 The ectodomain of MAG is comprised of five Ig-like domains and uses neuron

174 This increase in the size of MAG is contributed by an increase in cell size, but not

175 icated that the number of neurons in the MPN mag is greater in males than females but failed to find

176 ng lesion effect in overcoming inhibition by MAG is initially dependent on ongoing polyamine synthesi

177 muli in each AG subdivision, we propose that mAG is involved in semantic associations regardless of t

178 of the in vitro TAG synthesis initiated from MAG is mediated by DGAT1 in Caco-2 cell and rat intestin

179 ttraction to gradients of cAMP, netrin-1, or MAG is mediated by Epac.

180 The MPN mag is sexually differentiated in both neuron number and

181 Myelin-associated glycoprotein (MAG) is a sialic acid-binding Ig-family lectin that func

182 nous lectin, myelin-associated glycoprotein (MAG), is reported to bind to axonal gangliosides (GD1a a

183 oes not result in a substantial release of L-MAG (large MAG) inhibition.

184 which remained in an amorphous form, whereas MAG led to strong scattering, indicating the formation o

185 hat is possible given the disposition of the MAG linker.

186 Pretreatment with the monoacylglycerol (MAG) lipase inhibitor JZL-184 also reduced affective dis

187 olecule, the myelin-associated glycoprotein (MAG), located in the adaxonal plasmalemma of myelin-prod

188 At low concentrations of 2-MAG (<50 microm), the major acylation product by DGAT1 w

189 iGluR6 using a family of photoiosomerizable MAG (maleimide-azobenzene-glutamate) PTLs that covalentl

190 showed that the sialic acid binding site on MAG maps to arginine 118 in Ig domain 1.

191 r myelin genes, such as proteolipid protein, MAG, MBP, and myelin oligodendrocyte glycoprotein, were

192 An inhibitor of Rho kinase reversed MAG-mediated inhibition in all nerve cells, whereas a pe

193 Mechanistically, MT-I/II ability to overcome MAG-mediated inhibition is transcription-dependent, and

194 e relative roles of gangliosides and NgRs in MAG-mediated inhibition of neurite outgrowth from three

195 quired for dbcAMP and putrescine to overcome MAG-mediated inhibition.

196 CP-mag-micelles are biocompatible, can be delivered to vari

197 These 'theranostic' CP-mag-micelles are composed of monodisperse hydrophobic su

198 A single injection of CP-mag-micelles carrying reporter plasmids in vivo expresse

199 relaxivity of mag-micelles was similar to CP-mag-micelles confirming that coating with cationic polym

200 The T(2) relaxivity of mag-micelles was similar to CP-mag-micelles confirming t

201 e morphology and size distribution of the CP-mag-micelles were characterized and their potential for

202 leneimine (PEI) coated magnetic micelles (CP-mag-micelles) that can deliver nucleic acid-based therap

203 d modulate alternative exon inclusion from a MAG minigene reporter.

204 The Mag-MIP and Mag-NIP were characterized by FTIR, magnetic

205 The capacity of Mag-MIP for biotin adsorption, its kinetics and selectiv

206 lectivity experiments revealed that prepared Mag-MIP had higher selectivity toward biotin compared to

207 odified with molecularly imprinted polymers (Mag-MIP) through core-shell method for the determination

208 We show that when a truncated form of MAG missing Ig domains 1 and 2 is expressed by Chinese h

209 ficacy of two-photon (2P) excitation for two MAG molecules using nonlinear spectroscopy.

210 Anti-MAG (myelin-associated glycoprotein) neuropathy is a dis

211 rts binding of the myelin inhibitors Nogo-A, MAG (myelin-associated glycoprotein), and OMgp (oligoden

212 These results suggest a novel role for MAG/myelin in poor SC-myelin interaction and identify p7

213 ry isolated coronary artery bypass grafting (MAG, n = 5580; LITA+SVG, n = 14496) in the province of B

214 evidence of a treatment effect for IgM anti-MAG neuropathy and diabetic amyotrophy (radiculoplexus n

215 immunological surrogate mouse model for anti-MAG neuropathy producing high levels of anti-MAG IgM was

216 Patients with anti-MAG neuropathy showed substantial clonal expansions of b

217 toward an antigen-specific therapy for anti-MAG neuropathy.

218 th anti-myelin-associated glycoprotein (anti-MAG) neuropathy, an autoimmune disease of the peripheral

219 ration beyond the injury site in either Nogo/MAG/NgR1 triple mutants or NgR1 single mutants.

220 The Mag-MIP and Mag-NIP were characterized by FTIR, magnetic hysteresis,

221 tors such as myelin-associated glycoprotein (MAG), Nogo-A, and oligodendrocyte-myelin glycoprotein.

222 -1beta) and myelin-associated glycoproteins (MAG, Nogo).

223 However, further investigation revealed that MAGEs not only drive tumorigenesis but also regulate pat

224 The Hb/MHAM@Mag-NPs biocomposite is captured at SPCE by a super magn

225 It shows that the MHAM@Mag-NPs composite could increase the adsorption ability

226 The entrapment of Hb at MHAM@Mag-NPs was confirmed by cyclic voltammetry (CV), electr

227 supported iron oxide magnetic nanoparticles (Mag-NPs), is reported.

228 ion of the mixed hemi/ad-micelles of CTAB at Mag-NPs, zeta-potential measurements were performed.

229 captured at SPCE by a super magnet (Hb/MHAM@Mag-NPs/SPCE).

230 We show here that incisures are present in MAG-null and absent from P(0)-null PNS internodes.

231 tially rescues neurite inhibition by Nogo66, MAG, OMgp, and myelin in cultured neurons.

232 tal dynamics after acute exposure to soluble MAG, OMgp, or Nogo-66, but is not required for these lig

233 We also examined the influence of DAG and MAG on the physical properties of SSO.

234 Finally, when adding SSL or MAG on top of BStA to starch suspensions, the effect of

235 vous system, myelin-associated glycoprotein (MAG) on residual myelin binds to receptors on axons, inh

236 f diacylglycerol (DAG) and monoacylglycerol (MAG) on the oxidative stability of stripped soybean oil

237 gamma-secretase activity before exposing to MAG or CNS myelin improves SC migration and survival in

238 Conversely, the repulsion conferred by MAG or netrin-1 on adult growth cones is mediated by pro

239 Conversely, specific inhibition of MAG or sialyl-T MUC1 partially blocked adhesion.

240 All of the results showed that Mag-PCMAs are reusable sorbents for fast, convenient, an

241 ned, and the regeneration and reusability of Mag-PCMAs for diuron removal was also investigated.

242 on of Mag-PCMAs for soil-washing, the use of Mag-PCMAs for removal of diuron from a contaminated soil

243 As a proof of principle for application of Mag-PCMAs for soil-washing, the use of Mag-PCMAs for rem

244 The synthesis of Mag-PCMAs involves coating a silica/surfactant mesostruc

245 ine, diuron, naphthalene, and biphenyl) onto Mag-PCMAs were determined, and the regeneration and reus

246 agnetic permanently confined micelle arrays (Mag-PCMAs) have been successfully synthesized as sorbent

247 MAG-PEI25 reached a maximum adsorption capacity of 6.11

248 MAG percentage was maximum (7mol%) at lower water activi

249 division of the medial Preoptic nucleus (MPN mag) plays a critical role in the regulation of male sex

250 erebral amyloid angiopathy, neither VEGF nor MAG:PLP1 correlated significantly with the severity of s

251 VEGF but not MAG:PLP1 increased with Alzheimer's disease severity, as

252 In frontal cortex the MAG:PLP1 ratio was significantly reduced in Alzheimer's

253 However, MAG:PLP1 showed a significant negative correlation with

254 The decline in MAG:PLP1 strongly suggests pathological hypoperfusion of

255 Although MAG:PLP1 tended to be lowest in cortex from patients wit

256 iated glycoprotein to proteolipid protein 1 (MAG:PLP1) ratio, which declines in chronically hypoperfu

257 ction in EDN1, and positive correlation with MAG:PLP1, in the hypoperfused white matter in Alzheimer'

258 Finally, we show that hnRNPA1 binds MAG pre-mRNA and modulates alternative inclusion of MAG

259 hly expressed on adhesion molecules, such as MAG, present in myelinated nerve fibers.

260 ) of an acylglycerol mixture (containing 67% MAGs) produced by enzymatic glycerolysis of sardine oil

261 pathways to regulate myelination by means of MAG protein stability in myelin-forming cells of the aud

262 "MAG" PTLs for ionotropic and metabotropic glutamate rece

263 The MILLIPLEX-MAG Rat cytokine-chemokine magnetic bead array was used

264 ain that results in sn-2 monoacylglycerol (2-MAG) rather than LPA as the major product.

265 -enhancing mechanism in which C1q binding to MAG reduces MAG signaling to neurons, complement C1q blo

266 lin, such as myelin-associated glycoprotein (MAG), represent major obstacles to axonal regeneration f

267 Comparison of nine Sulfurovum MAGs reveals two high-coverage, low-diversity MAGs from

268 Production of monoacylglycerols (MAGs) rich in omega-3 polyunsaturated fatty acids (n-3 P

269 MAG's preferred ligands are derivatives of the monosialy

270 tween a tetrazine-functionalized pro-sensor, Mag-S-Tz, and a strained bicyclononyne conjugated to a g

271 Antibodies against the MPZ, MAG, S100, and SCMP proteins immunostained along pericor

272 that hydrolyzes sialic acids and eliminates MAG-sialoglycan binding.

273 echanism in which C1q binding to MAG reduces MAG signaling to neurons, complement C1q blocked both th

274 MAG significantly decreased the interfacial tension of S

275 an upper limit in the g filter of about -4.1 mag, so it is unlikely to be a giant eruption from a lum

276 iency was thought to underlie the defects of MAG splicing in the qk(v) mutant.

277 OLs completely rescues the dysregulation of MAG splicing without increasing expression or nuclear ab

278 ing I (QKI) leads to severe dysregulation of MAG splicing.

279 ics and magnitude of F-actin accumulation in mAg-stimulated B cells are greater than those in sAg-sti

280 tional resistance, as well as slowly growing MAG strains and also strains displaying an inducible res

281 ardless of the NgR1 genotype, membrane-bound MAG strongly inhibits neurite outgrowth of primary cereb

282 ive to perceptual matching: (1) a midregion (mAG) that overlapped with the default network because it

283 aired trafficking of plasma membrane-derived MAG through the endolysosomal system in primary cells an

284 MASS1 inhibits the ubiquitylation of MAG, thus enhancing the stability of this protein, and t

285 improved patients were those with high anti-MAG titers and most severe sensory deficits at baseline.

286 At month 8, IgM was reduced by 34% and anti-MAG titers by 50%.

287 IgM levels, anti-MAG titers, B cells, antigen-presenting cells, and immun

288 ing was neither necessary nor sufficient for MAG to bring about inhibition of neurite outgrowth.

289 is, we now map a distinct inhibition site on MAG to Ig domain 5 (Ig-5).

290 on emulsion presented a higher conversion of MAGs to FFAs during digestion, which led to a higher con

291 (OMgp), and myelin-associated glycoprotein (MAG) to mediate neurite outgrowth inhibition by these li

292 A synthesized short version of MAG turns the channel on in either the cis or trans stat

293 MASS1 RNAi or a specific inhibitor abrogates MAG up-regulation.

294 compare the safety and long-term outcomes of MAG vs LITA+SVG among overall and selected subgroups of

295 val advantage of multiple arterial grafting (MAG) vs the standard use of left internal thoracic arter

296 Compared with LITA+SVG, MAG was associated with reduced mortality rates (hazard

297 The growth of MAG was impaired after double-stranded RNA (dsRNA)-media

298 nts encoding myelin-associated glycoprotein (MAG), which generates two protein isoforms that associat

299 ral HNK-1 epitope blocked the interaction of MAG with pathogenic IgM antibodies from patient sera but

300 diacylglycerols (DAG) and monoacylglycerols (MAG) with a high content of polyunsaturated fatty acids