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1 ats received pre-session injections of known MAO inhibitors.
2 onooxygenase (DbetaM) and monoamine oxidase (MAO) inhibitors.
3 ) revealed a surprisingly high percentage of MAO inhibitors (16%) with a low false hit rate (0.9%).
6 viors that can be reversed by treatment with MAO inhibitors, as well as the tricyclic antidepressant
8 rs were also treated with the antidepressant MAO inhibitor drug, tranylcypromine (10 mg/day for 3 day
9 line ligand agmatine, the monoamine oxidase (MAO) inhibitor harmane, the alpha(2)-adrenoceptor agonis
10 arge dietary intake of tyramine while taking MAO inhibitors has been reported; therefore, the measure
11 nal flexibility and structural properties of MAO inhibitors in tuning their isoform specificities.
12 show that (1) tranylcypromine (TCP), a known MAO inhibitor, increases sensitivity to the primary rein
13 unilaterally 2 h after pretreatment with an MAO inhibitor into left or right nucleus accumbens septi
15 ake; accordingly, it has been suggested that MAO inhibitors may represent a therapeutic alternative i
17 y the amine uptake inhibitor imipramine, the MAO inhibitor pargyline and the MEK inhibitor PD 98059.
21 It is believed that a monoamine oxidase (MAO) inhibitor specific for MAO A, which is reversibly b
23 vo were evaluated after application to three MAO inhibitors, tranylcypromine, clorgyline, and pargyli
24 effect" (hypertension during treatment with MAO inhibitors usually caused by ingestion of foods cont
25 ses (MAO) A and B following a single dose of MAO inhibitors was developed through the simultaneous de
26 cal features with a preferential response to MAO inhibitors, which is especially well-evidenced for r
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