ライフサイエンスコーパス: MAP kinase kinase 1

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1 by transfection with a constitutively active MAP kinase kinase 1.

2 158780 potently inhibited the LPA-stimulated MAP kinase kinase 1/2 (MKK1/2) activation and EGF recept

3 The in vivo administration of the MAP kinase kinase 1/2 inhibitor U0126 [1,4-diamino-2,3-d

4 ted protein kinases 1 and 2 (ERK1, ERK2) and MAP kinase kinases 1 and 2 (MKK1, MKK2), is well-known t

5 , an inhibitor of mitogen-activated protein (MAP) kinase kinase 1 and 2 (MEK1/2) that blocks phosphor

6 of C-Raf and the mitogen-activated protein (MAP) kinase kinases 1 and 2 (MEK1/2).

7 osphorylation and p90 ribosomal S6 kinase 2, MAP kinase kinase 1, and Raf-1 activities were increased

8 Since the only known targets of MAP kinase kinase-1 are Erk1 and Erk2, these findings ar

9 ion of mKsr-1 and mitogen-activated protein (MAP) kinase kinase 1 (MAPKK-1 or MEK-1).

10 it, respectively, mitogen-activated protein (MAP) kinase kinase 1 (MEK 1) and calcineurin, to determi

11 The transfection of a MAP kinase kinase 1 (MEK1) dominant negative mutant cDNA

12 induced phenotype, we utilized the selective MAP kinase kinase 1 (MEK1) inhibitor, PD 098059.

13 tion, PD098059, a synthetic inhibitor of the MAP kinase kinase 1 (MEK1) was employed.

14 UO126, an inhibitor of MAP kinase kinase 1 (MEK1), suppressed focal adhesion ta

15 We report that cdk5 phosphorylates the MAP kinase kinase-1 (MEK1) in vivo as well as the Ras-ac

16 Pretreatment of cells with the MAP kinase kinase-1 (MEK1) inhibitor PD 98059 blocked IL

17 We show that both mitogen-activated protein (MAP) kinase kinase 1 (MEK1) and MAP kinase kinase 2 (MEK

18 MNK1 activation is blocked by inhibitors of MAP kinase kinase 1 (Mkk1) and p38, demonstrating that M

19 transfection of constitutively active mutant MAP kinase kinase 1 (MKK1) enhanced endogenous topoisome

20 e and constitutively active mutants of human MAP kinase kinase-1 (MKK1) were analyzed by deuterium ex

21 Conversely, a dominant-negative MAP kinase kinase 1 mutant decreased adhesiveness and st

22 In addition, a MAP kinase kinase 1 specific inhibitor blocked the expan

23 Upon dual phosphorylation by MAP kinase kinase 1, the dynamics of assigned methyls in

24 Coinjection of constitutively active MAP kinase kinase-1, which opposes XCL100's effects on M

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