ライフサイエンスコーパス: MAP kinase phosphatase-1

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1 ure to high shear stress by the induction of MAP kinase phosphatase-1.

2 chemokines as a result of rapid induction of MAP kinase phosphatase-1.

3 that protein-tyrosine-phosphatase, including MAP kinase phosphatase 1 activated by the AT2 receptor,

4 reatment arterializes venous ECs by inducing MAP kinase phosphatase-1 and may protect veins from infl

5 g with intensive, ET-1-induced expression of MAP kinase phosphatase-1, but does result in increased p

6 ligonucleotide of mitogen-activated protein (MAP) kinase phosphatase-1 enhanced the effects of NGF on

7 Activation of G-kinase induced MAP kinase phosphatase 1 expression, but this occurred l

8 hibiting its activation and (ii) by inducing MAP kinase phosphatase 1 expression.

9 ed expression of alpha- and beta-globins and MAP kinase phosphatase-1 in sodium butyrate-treated R6/2

10 (c) expression of mitogen-activated protein (MAP) kinase phosphatase-1 in SMC all led to increased mi

11 Antisense oligonucleotide to MAP kinase phosphatase 1 inhibited the AT2 receptor-medi

12 f the induced genes, such as CREM, CHOP, and MAP kinase phosphatase-1, may be components of a homeost

13 Another involves MAP kinase phosphatase 1 (MKP-1)-dependent inhibition of

14 se in the expression of the dual-specificity MAP kinase phosphatase 1 (MKP-1).

15 RA up-regulated the expression level of MAP kinase phosphatase-1 (MKP-1) and MKP-2, and the time

16 We found that MAP kinase phosphatase-1 (MKP-1) controls axon branching

17 Associated with p38 inhibition, Dex induced MAP kinase phosphatase-1 (MKP-1) in control, but not MGR

18 osphorylation and activity and expression of MAP kinase phosphatase-1 (MKP-1) in gastric mucosa of PH

19 present study, we have addressed the role of MAP kinase phosphatase-1 (MKP-1) in regulating proinflam

20 cineurin [protein phosphatase 2B (PP2B)] and MAP kinase phosphatase-1 (MKP-1) in the VTA.

21 The abundance of MAP kinase phosphatase-1 (MKP-1), a key upstream enzyme

22 gh dephosphorylation by the dual specificity MAP kinase phosphatase-1 (MKP-1), an immediate early gen

23 ression of the immediate early gene products MAP kinase phosphatase-1 (MKP-1), c-Fos and c-Jun.

24 of a dual-specificity tyrosine phosphatase, MAP kinase phosphatase-1 (MKP-1), in smooth muscle cell

25 a dual-specificity phosphatase also known as MAP kinase phosphatase-1 (MKP-1), showed greatest induct

26 e expression of ERK1/2-specific phosphatase, MAP kinase phosphatase-1 (MKP-1), which attenuates ERK1/

27 impairment is mediated by overexpression of MAP kinase phosphatase-1 (MKP-1), which results from the

28 poptosis, and the mitogen-activated protein (MAP) kinase phosphatase 1 (MKP-1) gene, that functions a

29 Mitogen-activated protein (MAP) kinase phosphatase 1 (MKP-1) has been shown to play

30 The mitogen-activated protein (MAP) kinase phosphatase 1 (MKP-1) plays a major role in

31 nitric oxide (NO)-donor led to induction of MAP-kinase phosphatase -1 (MKP-1) and dephosphorylation

32 we report the identification of Arabidopsis MAP Kinase Phosphatase 1 (MKP1) as a negative regulator

33 in adipocytes, which in turn phosphorylates MAP kinase phosphatase-1 (MKP1) at serine 334, initiatin

34 Overexpression of MAP kinase phosphatase-1 (MKP1) in mouse and rat muscle

35 ein expression of mitogen activated protein (MAP) kinase phosphatase 1 (MKP1) was also increased as d

36 induced genes was mitogen-activated protein (MAP) kinase phosphatase 1 (MKP1), a negative regulator o

37 PD98059, a MAP kinase inhibitor, as well as MAP kinase phosphatase-1 (MPK-1), in the transfection as

38 Unlike PP2A, MAP kinase phosphatase-1 or CYLD did not show an effect

39 treatment with dexamethasone, which induced MAP kinase phosphatase-1 to suppress proinflammatory act

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