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1 are not required for linking GPCRs with the MAP kinase signaling cascade.
2 tor that is negatively regulated by the Rasl-MAP kinase signaling cascade.
3 n as essential elements in the GPCR-mediated MAP kinase signaling cascade.
4 1 and 2, consistent with the activation of a MAP kinase signaling cascade.
5 sponses, including the Jak-Stat and Rac1/p38 Map kinase signaling cascades.
6 ors (GPCR) to the mitogen-activated protein (MAP) kinase signaling cascade.
7 nd transducers of mitogen-activated protein (MAP) kinase signaling cascades.
8 ding a miRNA that suppresses a target in the MAP kinase signaling cascade, a central signal transduct
9 protein signaling accounts for D2R canonical MAP kinase signaling cascade activation, whereas beta-ar
10 of ERK in the Ras/mitogen-activated protein (MAP) kinase signaling cascade, also interacts directly w
11 RKIP or PEBP) is an inhibitor of the Raf/MEK/MAP kinase signaling cascade and a suppressor of cancer
12 n by PKC relieves a key inhibitor of the Raf/MAP kinase signaling cascade and may represent a general
13 smotic stress via CaMKII and a conserved p38 MAP kinase signaling cascade and regulates osmotic avoid
14 described an important link between the ERK MAP kinase signaling cascade and the translational machi
15 ine phosphatase (HePTP) regulates orthogonal MAP kinase signaling cascades by dephosphorylating both
16 inhibition of the mitogen-activated protein (MAP) kinase signaling cascade by wild-type but not mutan
17 regulation of the mitogen-activated protein (MAP) kinase signaling cascades by hormonal stimulation i
18 se 2A (PP2A) positively regulates an RTK-Ras-MAP kinase signaling cascade during Caenorhabditis elega
21 e against neoplastic transformation when the MAP kinase signaling cascade is inappropriately active.
22 ein kinase kinase/mitogen-activated protein (MAP) kinase signaling cascade is initiated by activation
23 activation of the canonical Ras/Raf/MEK/ERK Map kinase signaling cascade, likely because of PAK co-a
24 se data provide the first demonstration that MAP kinase signaling cascades (MEK-->ERK) are intracellu
26 her these data demonstrate that the Rac1/p38 MAP kinase signaling cascade plays a critical role in ty
27 ur data establish that activation of the p38 MAP kinase signaling cascade plays an important role in
32 iated p38 and JNK mitogen-activated protein (MAP) kinase signaling cascades trigger specific cellular
33 rane TGF-alpha through activation of the Erk MAP kinase signaling cascade without the need for new pr
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