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1 ion of undifferentiated ES cells via the ERK/MAP kinase signaling pathway.
2 on factor within a regulatory circuit in the MAP kinase signaling pathway.
3 apse by regulating gene expression through a MAP kinase signaling pathway.
4 adherens junction protein complexes and the MAP kinase signaling pathway.
5 ore appears to be a new component of the p38 MAP kinase signaling pathway.
6 cludens-1 (ZO-1) at the BTB site via the p38 MAP kinase signaling pathway.
7 e caused by Ang II is mediated by an ERK 1/2 MAP kinase signaling pathway.
8 ivation is associated with disruption of the MAP kinase signaling pathway.
9 ion are differentially controlled by the p38 MAP kinase signaling pathway.
10 kinase Rsk1, a downstream target of the MEK-MAP kinase signaling pathway.
11 Drosophila indeed possesses a conserved p38 MAP kinase signaling pathway.
12 scaffold protein can mediate activation of a MAP kinase signaling pathway.
13 ide a novel mechanism for modulating the Ras-MAP kinase signaling pathway.
14 nuclei contain EGF receptors, which use the MAP kinase signaling pathway.
15 IA in C2C12 cells requires activation of the MAP kinase signaling pathway.
16 mitted efficient signaling through the yeast MAP kinase-signaling pathway.
17 affecting most notably genes of the NFkB and MAP kinase signaling pathways.
18 ication requires input from both the Wnt and MAP kinase signaling pathways.
19 d by up-regulation of MKPs and inhibition of MAP kinase signaling pathways.
20 at least in part, through the activation of MAP kinase signaling pathways.
21 -independent pathways, and may also involve MAP kinase signaling pathways.
22 ibitors of phosphatidylinositol 3-kinase and MAP kinase signaling pathways.
23 K alpha/beta-I kappa B alpha and MKK3/6--p38 MAP kinase signaling pathways.
24 negative components of the ERK, JNK, and p38 MAP kinase signaling pathways.
25 at is activated through both the ERK and p38 MAP kinase signaling pathways.
26 ed the upstream and downstream parameters in MAP kinase signaling pathways.
27 e BTB via the p38 mitogen activated protein (MAP) kinase signaling pathway.
28 vation of the p38 mitogen-activated protein (MAP) kinase signaling pathway.
29 activation of the mitogen-activated protein (MAP) kinase signaling pathway.
30 regulated by the mitogen-activated protein (MAP) kinase signaling pathway.
31 tors with the Ras/mitogen-activated protein (MAP) kinase signaling pathway.
32 ase (ERK) and p38 mitogen-activated protein (MAP) kinase signaling pathways.
33 pled receptors to mitogen-activated protein (MAP) kinase signaling pathways.
34 ve opposing effects on the regulation of the MAP kinase signaling pathway and a downstream target of
35 CB1 receptor, with an involvement of p42/44 MAP kinase signaling pathway and changes in actin cytosk
37 lish Ets-2 as a direct target of the Raf-MEK-MAP kinase signaling pathway and strongly implicate Ets-
38 E, the 15-LOX-1 metabolite, up-regulates the MAP kinase signaling pathway and subsequently down-regul
39 nfirm that bromelain is a novel inhibitor of MAP kinase signaling pathways and suggest a novel role f
40 ion of the mitogen-activated protein kinase (MAP kinase) signaling pathway and the subsequent increas
41 o induce the EGFR-mediated activation of the MAP-kinase signaling pathway and consequently the expres
42 e differentially affects the PI 3-kinase and MAP kinase signaling pathways, and insulin-stimulated IR
43 brane dynamics, the antagonistic activity of MAP kinase signaling pathways, and the role of stress in
44 aphragm muscle we tested the hypothesis that MAP kinase signaling pathways are activated by mechanica
46 with activation of the p38, Erk1/2, and JNK MAP kinase signaling pathways as well as up-regulated ex
47 ity is required in the G beta gamma-mediated MAP kinase signaling pathway at a point upstream of Sos
48 We have examined the function of the p38 MAP kinase signaling pathway by investigating the effect
49 ry and inhibitory factors to act through the MAP kinase signaling pathway by studying the effects of
52 protection is mediated by VEGFR2, and by the MAP kinase signaling pathway downstream of the receptor.
54 ated kinase (ERK)/mitogen-activated protein (MAP) kinase signaling pathway during infection and requi
55 /2-extracellular-signal regulated kinase 1/2 MAP kinase signaling pathway following Toll-like recepto
56 dy reveals a critical role for the MEK5-ERK5 MAP kinase signaling pathway in BAFF-induced mature B ce
58 T-2, functions through the conserved RAS/ERK MAP kinase signaling pathway in the C. elegans germline
59 owth factor receptor (EGFR)/LET-60 Ras/MPK-1 MAP kinase signaling pathway in the vulval precursor cel
60 82 and 70 kDa proteins that function in the MAP kinase signaling pathway in transient expression ass
62 in and epinephrine have different effects on MAP kinase signaling pathways in skeletal muscle, which
63 These findings establish a role for multiple MAP kinase signaling pathways in the cellular response t
64 C1 to potentiate EGF-dependent activation of MAP kinase signaling pathways in the lactating mammary g
66 on of Bad at serine 112 was mediated through MAP kinase signaling pathways in which JNK1, RSK2, and M
67 an unexpected role and connection of the p38 MAP kinase-signaling pathway in cell cycle control, sene
68 rent study the role of a stress-specific p38 MAP kinase-signaling pathway in mediating these effects
69 nvolvement of the mitogen-activated protein (MAP) kinase signaling pathway in Ang II stimulation of t
70 onents of the Ras-mitogen-activated protein (MAP) kinase signaling pathway in epidermal growth factor
71 nts of the mitogen-activated protein kinase (MAP kinase) signaling pathway, including Ras, Raf, and M
73 , which encode components of a conserved p38 MAP kinase signaling pathway involved in nematode defens
75 show that the p38 mitogen-activated protein (MAP) kinase signaling pathway is activated in immature t
76 show that the p38 mitogen-activated protein (MAP) kinase signaling pathway is strictly regulated duri
77 um may be due to the specific stimulation of MAP kinase signaling pathways leading to nuclear respons
79 3a and C5a both strongly activate downstream MAP kinase signaling pathways (p44 and p42 Erk kinases).
81 oals of this study were to determine whether MAP kinase signaling pathways play a role in the formati
82 n inductive interaction mediated through Wnt/MAP kinase signaling pathways, POP-1, a Lef/Tcf-type tra
83 imultaneous disruption of cell cycle and MEK/MAP kinase signaling pathways provides a potent stimulus
84 uggest that in some cell types the MEK5/BMK1 MAP kinase signaling pathway regulates serum-induced ear
85 we show that the mitogen-activated protein (MAP) kinase signaling pathway regulates cytokine-mediate
86 differential utilization of TCF proteins and MAP kinase signaling pathways represents a potential mec
87 nts probably converge into the LPS-modulated MAP kinase signaling pathway resulting in greater activa
90 exhibit mutations in other components of the MAP kinase signaling pathway such as the Her-2/neu and r
91 ase (ERK) and the c-jun kinase (JNK) are two MAP kinase signaling pathways that could play a role in
92 kinase (MAP3K) family, Cot can also activate MAP kinase signaling pathways that target AP-1 and NFAT
93 tein that integrates G protein and H-Ras/ERK/MAP kinase signaling pathways, thereby making it well po
95 s, suggesting that the activation of the Ras/MAP kinase signaling pathway through Grb2 may be essenti
96 cells, where it acts through a conserved Ras/MAP kinase signaling pathway to induce vulval differenti
97 or in conjunction with other factors through MAP kinase signaling pathways to maximally induce UGT1A1
98 Significantly modulated pathways included: MAP kinase signaling pathway, Toll receptor pathway, TGF
99 des generated during phagocytosis act on the MAP kinase signaling pathway, ultimately "turning off" t
100 es belonging to a mitogen-activated protein (MAP) kinase signaling pathway, was regulated by vision a
101 llular signal-regulated kinase 1/2 (ERK-1/2) MAP kinase signaling pathways were used to assess their
102 e branches of the mitogen-activated protein (MAP) kinase signaling pathway were abnormally hyperactiv
103 nstrate that three different branches of the MAP kinase signaling pathway with overlapping consequenc
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