ライフサイエンスコーパス: MBL

1 MBL and ficolin-2 were present in human carotid plaques,

2 MBL and L-ficolin were shown to interact with immobilize

3 MBL bound to CC in a calcium-dependent manner whereas fi

4 MBL box modeling calculations show that buildup of MClDM

5 MBL can be categorized as either low count or high count

6 MBL concentration, but not genotype, was a determinant o

7 MBL deficiency also significantly increased the number o

8 MBL deficiency is common (10-30% of the general populati

9 MBL did neither bind to LDL nor to acLDL.

10 MBL genotype frequencies and MBL serum concentrations di

11 MBL has also been identified in donated blood, but no sy

12 MBL ranged from 0 to 2.85 mm: 75.9% of mesial sites and

13 MBL rates at 6 and 18 months were mainly affected by the

14 MBL rates followed a non-linear trend, with a greater MB

15 MBL rates were higher for prosthetic abutment < 2 mm vs.

16 MBL serum concentrations in CDI and AAD subjects were de

17 MBL was assessed.

18 MBL was calculated from the difference between initial a

19 MBL was significantly (P <0.05) greater in open-flap com

20 MBL zinc fingers are the most highly conserved portion o

21 MBL-A was detected as early as 3.5 d of pregnancy, and M

22 MBL-A was observed in the implantation sites of CBA/J x

23 MBL-DGT accurately measured the concentration of trace m

24 MBL-DGT measured the Mn concentration accurately over th

25 MBL-GS muPADs offer direct quantitative analysis of vola

26 MBL-GS muPADs were designed to make fabrication of the d

27 nd deficient (26.3 versus 17.1%, p = 0.0361) MBL pathway functions were observed statistically more f

28 inhibition of the clinically relevant VIM-2 MBL by a rhodanine, which reveal that the rhodanine ring

29 by whole-exome sequencing of 456 CLL and 43 MBL patients, are either truncating or affect conserved

30 Of 51 MBL samples in which immunoglobulin heavy chain (IGH)V-D

31 was an outlier, and the IMP isolates and 6/7 MBL-negative isolates clustered separately from the main

32 A MBL-GS muPAD consists of three layers: "donor layer", "s

33 ficient concentration of biologically active MBL in body fluids is an indicator of proper function of

34 rs are micromolar competitive betaLIs of all MBL classes in vitro, with Kis of 6-15 microM or 36-84 m

35 hETHE1 contains an alphabetabetaalpha MBL-fold, which supports metal-binding by the side chain

36 s failed to demonstrate an association among MBL and confounding factors.

37 An MBL inhibitor could restore the effectiveness of beta-la

38 Accordingly, the mutation of an MBL conserved lysine residue essential for MASP binding

39 AL [P <0.0001], PD >/=4 mm [P <0.0001], and MBL [P <0.0001]) was significantly higher among WPs and

40 rameters (PI, BOP, PD, and AL) (P <0.01) and MBL (P <0.01) were worse among individuals in group A th

41 05], AL [P <0.05], PD >/=4 mm [P <0.05], and MBL [P <0.05]) were significantly higher in smokers than

42 we examined the deposition of ficolin-2 and MBL in human carotid plaques by immunohistochemistry and

43 activated on CC by binding of ficolin-2 and MBL in vitro, resulting in activation and deposition of

44 difference in number of MT, clinical AL, and MBL among all groups.

45 MBL genotype frequencies and MBL serum concentrations did not differ between patients

46 ng lectin (MBL) associated protein (MAP) and MBL associated serine protease (MASP) are scarcely inves

47 by the mandibular cortical index (MCI), and MBL and 2) to assess how various systemic diseases, peri

48 lth, severity of periodontal parameters, and MBL are worse in patients with prediabetes than controls

49 e thickness at time of implant placement and MBL with >/=12-month follow-up.

50 detected as early as 3.5 d of pregnancy, and MBL-A deficiency prevented pregnancy loss in the abortio

51 nce of the C/I ratio on the success rate and MBL of dental implants.

52 retrospective, that compared implant SR and MBL in patients with a history of GAgP versus those with

53 lopment of obesity, we studied wild-type and MBL(-/-) mice rendered obese using a high-fat diet (HFD)

54 We assessed MBL function with genotyping (low-expressing genotype [d

55 6-15 microM or 36-84 microM for subclass B1 MBLs (IMP-1 and BcII, respectively), and 10-12 microM fo

56 t closely resemble beta-lactam binding to B1 MBLs, but feature an unprecedented disruption of the D12

57 dominates interactions with the monozinc B2 MBL Sfh-I, with the thiol uninvolved.

58 Against the B2 MBL Sfh-I, the l-BTZ enantiomers exhibit 100-fold lower

59 kdown of conventional thermodynamics because MBL systems do not thermalize and show nonergodic time e

60 seed liquor-based beverage with maqui berry (MBL), characterising its bioactive and volatile composit

61 in regenerated sites (P <0.001) and between MBL and a previous history of periodontitis (P <0.05).

62 ificant associations were also found between MBL and placement of implants in regenerated sites (P <0

63 s related to the 'classical' di-zinc binding MBL hydrolases involved in antibiotic resistance, but ha

64 Both MBL and MCI were assessed from panoramic radiographs.

65 esented a low to moderate heterogeneity, but MBL presented a considerable heterogeneity among studies

66 alpha-Methylene-gamma-butyrolactone (MBL), a naturally occurring and biomass-sourced bifuncti

67 While regulation of specific target exons by MBL/MBNL has not been broadly conserved across these spe

68 measurement of trace metals and oxyanions by MBL-DGT was independent of pH (5.03-8.05) and ionic stre

69 observed in vitro as indicated by IgM, C1q, MBL, and factor Bb deposition on WT PAEC.

70 cases and AAD controls, but among CDI cases, MBL concentration, but not genotype, was associated with

71 ally, in humans, lower levels of circulating MBL were accompanied by enhanced macrophage influx in su

72 Cross-class MBL inhibition therefore arises from the unexpected vers

73 zinc 1 and zinc 2 binding sites in classical MBLs.

74 tion molecules, i.e., those of the collectin MBL-C, are involved in the Aspergillus-lectin complement

75 s, IMN can develop in patients with complete MBL deficiency, with complement activated mainly by the

76 otease-pattern recognition receptor complex, MBL-associated serine protease (MASP)-3/collectin-L1/K1

77 In conclusion, MBL prevalence is much higher in blood donors than previ

78 Although individuals with both high-count MBL and CLL Rai stage 0 are at increased risk of infecti

79 High-count MBL is distinguished from Rai 0 CLL based on whether the

80 unt MBL rarely progresses to CLL, high-count MBL progresses to CLL requiring therapy at a rate of 1%

81 lthough uncommon, the presence of high-count MBL warrants further investigations to define the biolog

82 istics distinguish low-count from high-count MBL.

83 Low-count MBL can be detected in approximately 5% of adults over t

84 Whereas low-count MBL rarely progresses to CLL, high-count MBL progresses

85 Most of the 149 donors had low-count MBL, including 99 chronic lymphocytic leukemia-like (66.

86 ere associated with diminished and deficient MBL pathway function, respectively.

87 allo-beta-lactamases (i.e., NDM-1 (New Delhi MBL), IMP-1 (Imipenemase), SPM-1 (Sao Paulo MBL), and VI

88 and specific laboratory methods, we detected MBL in 149 (7.1%; 95% confidence interval, 6.0% to 8.3%)

89 ohort study, the radiographically determined MBL was related to the height of the abutments of intern

90 icrofluidic paper-based analytical devices" (MBL-GS muPADs).

91 ion cut-off level associated with diminished MBL pathway function in a group of 201 individuals.

92 Four donors (2.7%) had 2 distinct MBL clones.

93 Active-site architecture divides MBLs into three classes (B1, B2, and B3), complicating d

94 In conclusion, when donor MBL was approximated by subsequent donor CLL diagnosis,

95 influence of soft tissue thickness on early MBL around dental implants.

96 e the influence of tissue thickness on early MBL.

97 There was no difference in either MBL concentrations or genotypes between cases and contro

98 ulo MBL), and VIM-2 (Verona integron-encoded MBL)) and the identification of suitable fluorogenic sub

99 mediated by metallo-beta-lactamase enzymes (MBLs).

100 y using logistic regression, controlling for MBL cut-off, age, gender, and age and gender interaction

101 ved across these species, genes enriched for MBL/MBNL binding sites in their introns may play roles i

102 We propose a novel role for MBL in the recognition and clearance of apoptotic adipoc

103 M) was identified to be highly selective for MBLs when compared to other Zn(II) metalloenzymes.

104 followed a non-linear trend, with a greater MBL rate during the first 6 months post-loading than dur

105 t pose a risk for the development of greater MBL.

106 LC- and HC-MBL had similar somatic hypermutation status, yet differ

107 this study, we investigated whether impaired MBL pathway function, represented by reduced serum MBL c

108 C/I ratio was performed on the peri-implant MBL while considering follow-up period, type of implants

109 her the C/I ratio, the less the peri-implant MBL.

110 bony defects of >/=5 mm regarding change in MBL.

111 with data on comparison of SR and changes in MBL between the flapless and conventional flap procedure

112 meta-analyses were performed for changes in MBL.

113 study was powered to detect a difference in MBL of >0.9 mm.

114 The abutment height is a key factor in MBL.

115 ohn's disease patients have an impairment in MBL-MASP functional activity and that this defect is ass

116 gnificantly correlated with an impairment in MBL-MASP functional activity.

117 However, the clonal size in MBL is extremely variable and allows discrimination of t

118 rsely affecting the development of increased MBL are a previous history of periodontitis and especial

119 INTERPRETATION: MBL is common in both Uganda and the UK, but the substan

120 with the titer of the commercial ELISA kit (MBL, Japan) (rho = 0.5680, P = 0.0011).

121 zinc centers of B1 (IMP-1; BcII) and B3 (L1) MBLs via the free thiol, but orient differently dependin

122 inding member of the metallo-beta-lactamase (MBL) fold superfamily.

123 d to the prokaryotic metallo-beta-lactamase (MBL) fold.

124 18, and 7 carried no metallo-beta-lactamase (MBL) gene.

125 Metallo-beta-lactamase (MBL) inhibitors can restore the function of carbapenem a

126 The metallo-beta-lactamase (MBL) is a candidate protein family that includes ribo- a

127 and global spread of metallo-beta-lactamase (MBL) mediated resistance, specifically New Delhi metallo

128 carbapenemases and metallo-beta-lactamases (MBL).

129 Metallo-beta-lactamases (MBLs) are a growing threat to the use of almost all clin

130 Metallo-beta-lactamases (MBLs) are particularly problematic because of their abil

131 Metallo-beta-lactamases (MBLs) hydrolyze almost all beta-lactam antibiotics and a

132 Development of metallo-beta-lactamases (MBLs) inhibitors has proven challenging, due to their co

133 biotics mediated by metallo-beta-lactamases (MBLs) is a growing problem.

134 The acquisition of metallo-beta-lactamases (MBLs) such as NDM-1 is a principle contributor to the em

135 The mixed binding layer (MBL) DGT technique was evaluated against the Chelex-DGT

136 nd has been used in a marine boundary layer (MBL) box model to determine the enhancement of SO2 produ

137 t O3 reduction in the marine boundary layer (MBL).

138 ular, stereotyped BcRs were infrequent in LC-MBL and were often not CLL specific.

139 entities (high-count [HC] and low-count [LC]-MBL) based on a cutoff value of 0.5 x 10(9)/L clonal B c

140 ses functional human mannose-binding lectin (MBL) 2 under the control of Mbl1 promoter.

141 tion molecules (PRMs) mannan-binding lectin (MBL) and ficolins complexed with the MBL-associated seri

142 Measurement of mannan binding lectin (MBL) antigenic level and activity revealed MBL deficienc

143 e immune functions of mannan-binding lectin (MBL) associated protein (MAP) and MBL associated serine

144 interact with C1q and mannan-binding lectin (MBL) likely through its C-terminal region (CCP22-30).

145 e similar to C1q and mannose-binding lectin (MBL) participate in microbe infection and apoptotic cell

146 The mannose-binding lectin (MBL) pathway of the complement cascade has an essential

147 Mannose-binding lectin (MBL) plays a key role in the activation of the lectin-co

148 Mannose-binding lectin (MBL), a soluble mediator of innate immunity, promotes ph

149 ctors C3b, IgM, C1q, mannose-binding lectin (MBL), and properdin to LDL and acLDL were investigated b

150 binding of the PRMs mannose-binding lectin (MBL), ficolin-1, ficolin-2, and ficolin-3, the associate

151 eered human opsonin--mannose-binding lectin (MBL)--that captures a broad range of pathogens and toxin

152 complement inhibitor, mannan-binding lectin (MBL)-associated protein (MAp)44, in regulating the compo

153 wn that mice lacking mannose-binding lectin (MBL)-associated serine protease-1 (MASP-1) and MASP-3 co

154 Mannan-binding lectin (MBL)-associated serine proteases, MASP-1 and MASP-2, hav

155 Mannose-binding lectin (MBL)/ficolin/collectin-11-associated protein-1 (MAP-1) i

156 s, and that flapless surgery results in less MBL compared with traditional open-flap surgery.

157 ating the success rate, marginal bone level (MBL), soft tissue stability, and subjective patient eval

158 assess, with regard to marginal bone level (MBL), whether the outcome of immediate implant placement

159 rvival rates (SRs) and marginal bone levels (MBLs) compared with the conventional flap approach.

160 ted the use of the molecular bacterial load (MBL) assay, for measuring viable Mycobacterium tuberculo

161 Many-body localization (MBL), the disorder-induced localization of interacting p

162 loss (AL) were measured; marginal bone loss (MBL) around all teeth was measured on digital radiograph

163 hetic abutment height on marginal bone loss (MBL) around implants in the posterior maxilla.

164 luate survival rates and marginal bone loss (MBL) around implants placed in sites treated with maxill

165 of the C/I ratio on the marginal bone loss (MBL) has not yet been examined extensively.

166 iodontal parameters, and marginal bone loss (MBL) in patients with prediabetes.

167 survival rates (SRs) and marginal bone loss (MBL) when compared with patients with chronic periodonti

168 al attachment loss (AL), marginal bone loss (MBL), and number of missing teeth, were recorded.

169 en shown to affect early marginal bone loss (MBL).

170 relating osteoporosis to marginal bone loss (MBL).

171 cations; 3) peri-implant marginal bone loss (MBL); 4) esthetic and periodontal parameters; and 5) pat

172 OP], probing depth [PD], marginal bone loss [MBL]) and fasting blood glucose levels (FBGLs) were reco

173 ttachment loss [AL], and marginal bone loss [MBL]) and numbers of missing teeth (MT) were recorded.

174 ttachment loss [AL], and marginal bone loss [MBL]) were measured.

175 Monoclonal B lymphocytosis (MBL) is defined as the presence of a clonal B-cell popul

176 LL (5.3%) and in monoclonal B lymphocytosis (MBL, 7%), a B-cell expansion that can evolve to CLL.

177 Monoclonal B-cell lymphocytosis (MBL) is a precursor lesion that can be screened for in a

178 Monoclonal B-cell lymphocytosis (MBL) is a precursor of chronic lymphocytic leukemia (CLL

179 (CLL) -like monoclonal B-cell lymphocytosis (MBL) shares common immunophenotype and cytogenetic abnor

180 sition from monoclonal B-cell lymphocytosis (MBL) to CLL and tested miR-15a/16-1 cluster, miR-21, and

181 tion called monoclonal B-cell lymphocytosis (MBL).

182 A statistically higher mean MBL was found in the control group compared with the tes

183 demonstrated statistically significant mean MBL in the control group, but not in the test group.

184 y beta-lactamases belonging to both metallo (MBL)- and serine (SBL)-beta-lactamase subfamilies.

185 Premolar and molar MBLs were measured on panoramic radiographs.

186 The Muscleblind (MBL) protein family is a deeply conserved family of RNA

187 hat the catalytic activities of these mutant MBLs were highly increased.

188 The prevalence of CD5-negative MBL was higher in the Ugandan cohort (41 [14%], of whom

189 ukaemia (CLL)-phenotype MBL and CD5-negative MBL, as well as differences in absolute monoclonal B-cel

190 However, neither MBL concentration nor MBL2 genotype was linked with the

191 phage abundance and polarity, the absence of MBL did not affect systemic insulin resistance.

192 CBA/J x DBA/2 combination in the absence of MBL-C and was undetectable in BALB/c-mated CBA/J mice.

193 ent in human carotid plaques, and binding of MBL to CC was confirmed in vivo by immunohistochemistry,

194 The majority of cases of MBL have the immunophenotype of chronic lymphocytic leuk

195 tential to address the clinical challenge of MBL-positive carbapenem-resistant Gram-negative pathogen

196 prevalence and phenotypic characteristics of MBL in age-and-sex-matched populations from rural Uganda

197 Colocalization of MBL/MASP2 with IgM, C3b/c, C4b/c, and C6 was investigate

198 Meta-analysis for the comparison of MBL among selected studies showed a WMD of -0.80 mm (95%

199 e led to observation of a ternary complex of MBL, a thioenolate fragment and rhodanine.

200 Serum concentrations of MBL were determined using a commercial enzyme-linked imm

201 Mean differences of MBL were retrieved from five RCTs and two retrospective

202 ons, yet the predominant oligomeric forms of MBL carry only a single MASP homodimer.

203 To assess the function of MBL in the development of obesity, we studied wild-type

204 mmunohistochemistry, showing localization of MBL around CC clefts.

205 es and 83.4% of distal sites showed <1 mm of MBL, whereas 35.2% of mesial sites and 37% of distal sit

206 changes in the adipose tissue morphology of MBL(-/-) mice.

207 Observations of MBL in blood donors raise concern that transmitted MBL m

208 flow cytometry to determine the presence of MBL, defined according to standard diagnostic criteria,

209 e first ring-opening polymerization (ROP) of MBL, thereby producing exclusively unsaturated polyester

210 paves the way to further detailed studies of MBL, such as in noncorrelated disorder or higher dimensi

211 vestigated their ability, as well as that of MBL homologs from human/mouse, fly and worm, to regulate

212 nalyze the weighted mean difference (WMD) of MBL between groups of thick and thin peri-implant soft t

213 cations for the mechanisms and inhibition of MBLs by beta-lactams and non-beta-lactams and illustrate

214 To date, a clinical inhibitor of MBLs that could reverse resistance and re-sensitize resi

215 Clinically approved inhibitors of MBLs are currently unavailable as design has been limite

216 BTZs therefore inhibit the full range of MBLs and potentiate beta-lactam activity against produce

217 erence in UWSFR, number of missing teeth, or MBL among habitual gutka chewers and controls.

218 discovered a function for LACTB2, an orphan MBL protein found in mammalian mitochondria.

219 e screening approach can be adapted to other MBLs and in this way improve the drug discovery process

220 method described will be applicable to other MBLs and more generally to monitoring ligand-induced con

221 Overall MBL prevalence was higher in the Ugandan participants (4

222 pes of polymers, P(MBL)VAP, P(MBL)CLP, and P(MBL)ROP, can be readily controlled by adjusting the cata

223 roducing exclusively unsaturated polyester P(MBL)ROP with Mn up to 21.0 kg/mol.

224 esses, thus affording cross-linked polymer P(MBL)CLP.

225 formation of the three types of polymers, P(MBL)VAP, P(MBL)CLP, and P(MBL)ROP, can be readily contro

226 inyl-addition polymerization (VAP) product P(MBL)VAP.

227 The resulting P(MBL)ROP is degradable and can be readily postfunctionali

228 of the three types of polymers, P(MBL)VAP, P(MBL)CLP, and P(MBL)ROP, can be readily controlled by adj

229 was no difference in periodontal parameters, MBL, and self-perceived oral symptoms among patients wit

230 NMR) to study the dynamics of the Sao Paulo MBL (SPM-1) from beta-lactam-resistant Pseudomonas aerug

231 MBL), IMP-1 (Imipenemase), SPM-1 (Sao Paulo MBL), and VIM-2 (Verona integron-encoded MBL)) and the i

232 hronic lymphocytic leukaemia (CLL)-phenotype MBL and CD5-negative MBL, as well as differences in abso

233 CLL-phenotype MBL was detected in three (1%) Ugandan participants and

234 4%], of whom two [5%] also had CLL-phenotype MBL) than in the UK cohort (six [2%], of whom two [33%]

235 %], of whom two [33%] also had CLL-phenotype MBL; p<0.0001), but the median absolute B-cell count was

236 One year after definitive crown placement, MBL loss was 0.56 +/- 0.39 mm mesially and 0.74 +/- 0.51

237 -rich conditions are typical of the polluted MBL, near coastlines and ship plumes.

238 and RIS on O3 mixing ratios in the polluted MBL.

239 ood starting points for the design of potent MBL inhibitors.

240 susceptibility of Escherichia coli-producing MBLs (IMP-1, Sfh-1, BcII, and GOB-18) and, significantly

241 g lectin-mannose-associated serine protease (MBL-MASP) functional activity in Crohn's disease patient

242 We identified putative MBL homologs in Ciona intestinalis and Trichoplax adhaer

243 i-implant soft tissue have less radiographic MBL in the short term.

244 sistent with these observations, recombinant MBL enhanced phagocytic capacity of the stromal vascular

245 lement activation via its ability to recruit MBL to MASP, and other hand as a modulator of immune cel

246 Significantly reduced MBL deposition and complement activation was observed on

247 fragments inhibiting the clinically relevant MBL Verona integron-encoded metallo-beta-lactamase (VIM-

248 NDM-1 enzyme and another clinically relevant MBL, VIM-2.

249 substrates and a set of clinically relevant MBLs.

250 eins (PBPs), SBLs and, as recently reported, MBLs.

251 (MBL) antigenic level and activity revealed MBL deficiency.

252 In synthetic seawater, MBL-DGT accurately measured the concentration of metals

253 ll be useful in the development of selective MBL inhibitors.

254 inant CsMAP34 (rCsMAP34) bound C. semilaevis MBL (rCsBML) when the latter was activated by bacteria,

255 Serum MBL concentration did not differentiate between CDI case

256 Serum MBL concentrations < 787 and < 445 ng/ml were associated

257 First, we determined a serum MBL concentration cut-off level associated with diminish

258 rchives of our laboratory and measured serum MBL concentrations in both sample groups.

259 Also, the age-adjusted median serum MBL concentrations were significantly lower in the LB pa

260 thway function, represented by reduced serum MBL concentration, predisposes individuals to LB.

261 In parallel with increased adipocyte size, MBL(-/-) mice displayed an increased influx of macrophag

262 ed thioenolate to be a potent broad-spectrum MBL inhibitor and a lead structure for the development o

263 The identification of broad-spectrum MBL inhibitors is hampered by the lack of a suitable scr

264 tocols should be based on factors other than MBL.

265 rience over more than 30 years indicate that MBL/CLL transmission does not contribute importantly to

266 ociated with CDI recurrence, indicating that MBL acts as a modulator of disease, rather than a predis

267 icolins are new CR1 ligands and propose that MBL/L-ficolin binding involves major ionic interactions

268 The results showed that MBL had attractive colour, exhibited higher anthocyanin

269 The MBL assay provides early information on the rate of decl

270 The MBL research training courses Neurobiology and Neural Sy

271 The MBL weighted mean difference for each subgroup was 0.15

272 The MBL-DGT method described in this study offers significan

273 zone, and cefotaxime) were isolated, and the MBL variants were characterized.

274 ve association between the C/I ratio and the MBL was found (P = 0.012).

275 asirandom optical lattice and identified the MBL transition through the relaxation dynamics of an ini

276 r findings indicate that a deficiency in the MBL pathway of the complement cascade is a risk factor f

277 onstrain chemical processes occurring in the MBL.

278 ure revealed almost perfect alignment of the MBL domain with CPSF73, as well as to other ribonuclease

279 ds is an indicator of proper function of the MBL pathway.

280 73, as well as to other ribonucleases of the MBL superfamily.

281 Optimization of the MBL-GS muPADs was carried out for direct determination o

282 Proof of concept of the MBL-GS muPADs was demonstrated by analyzing standard sol

283 ant differences appear to relate only to the MBL with the placement of implants in regenerated bone s

284 lectin (MBL) and ficolins complexed with the MBL-associated serine proteases (MASP)-1 and MASP-2 clea

285 y been shown that MAP-1 can compete with the MBL/ficolin/collectin-11-associated serine proteases (MA

286 Therefore, MBL can be an acceptable new liquor, with better quality

287 iated serine proteases (MASPs) in binding to MBL and the ficolins.

288 mated CBA/J mice with Polyman2 that binds to MBL-A with high affinity proved to be highly effective i

289 nt harbored the LXA/LYC haplotypes linked to MBL deficiency.

290 Binding of NS1 to MBL protects DENV against mannose-binding lectin-mediate

291 blood donors raise concern that transmitted MBL may cause recipient CLL.

292 of reactive bromine species in the tropical MBL showed unexpectedly high levels that could potential

293 While both proteins contain a typical MBL-beta-CASP domain, a region of positive charge surrou

294 evelopment of new types of clinically useful MBL inhibitors.

295 Diffusion coefficients (D) measured using MBL-DGT generally agreed well with those measured by Che

296 SP-2, have been thought to autoactivate when MBL/ficolin.MASP complexes bind to pathogens triggering

297 and coding region variations associated with MBL deficiency and the same complement pattern in immune

298 ants in regenerated bone are correlated with MBL and MCI.

299 ells of normal individuals, individuals with MBL, and patients with CLL.

300 gement, and supportive care of patients with MBL and CLL.