ライフサイエンスコーパス: MCA

1 MCA fitness (1+s) was determined by comparing the change

2 MCA pseudofeeders and hydrocephalus were risk factors fo

3 MCA recanalization after endovascular treatment was achi

4 MCA V(mean), P(aCO2) and cerebral vascular conductance i

5 MCA-related symptoms may be mild, moderate, severe, or e

6 MCAs were mounted in a wire myograph.

7 sic electronic structure of the [B12X12](2-) MCAs provides the basis for a molecular level understand

8 , and t-butoxycarbonyl-Gln-Ala-Arg-7-amido-4-MCA fluorescent derivatives were used to assess chymotry

9 CA), benzyloxycarbonyl-Leu-Leu-Glu-7-amido-4-MCA, and t-butoxycarbonyl-Gln-Ala-Arg-7-amido-4-MCA fluo

10 with 5-methylphenazine-1-carboxylic acid (5-MCA) and pyocyanin (PYO) localized to the colony edge.

11 indicates an effect of O2 availability on 5-MCA synthesis.

12 A MCA prepared for chemical sensing was exposed to the sam

13 gas chromatograph and then introduced into a MCA flow cell.

14 ntional 7-methoxy-coumarin-4-yl acetic acid (MCA)/Lys(DNP) substrates; thus, substantially lower amou

15 he implementation of melamine-cyanuric acid (MCA) supramolecular aggregates and microwave-assisted th

16 of blood-brain barrier monocarboxylic acid (MCA) transport may contribute to the maintenance of brai

17 nonglucose substrates (monocarboxylic acids [MCAs]) by upregulation of the MCA transporters may contr

18 Mast cell activation (MCA) occurs in a number of different clinical conditions

19 rder to maximize its milk-clotting activity (MCA).

20 well as double mid1-complementing activity (MCA) channel mutants did not affect the response.

21 tive treatment option in patients with acute MCA occlusion with contraindication to IVT or after IVT

22 Neurologic deficit at admission, MCA recanalization, symptomatic intracerebral hemorrhage

23 ed threshold of 120 cm/sec for less advanced MCA spasm was significantly better than that of TCD at s

24 vanced, and 200 cm/sec for the more advanced MCA narrowing.

25 brain injury when administered 6 hours after MCA occlusion, suggesting that EP1 receptor inhibition m

26 nd show similar fibrosarcoma incidence after MCA inoculation compared to wild-type mice.

27 le, Medieval Climate Anomaly-Little Ice Age, MCA-LIA).

28 tivity and safety, microbial control agents (MCAs) appear to be ready-made components of integrated p

29 s were cultured as multicellular aggregates (MCA), cyclin D1 was induced through a serum-dependent EG

30 ure the growth of multi-cellular aggregates (MCAs), of increasing metastatic potential in different e

31 on the surface of multicellular aggregates (MCAs) are removed by treatment with LPA.

32 ng tumor cells and multicellular aggregates (MCAs) present in ascites fluid adhere to the peritoneum

33 dividual cells and multicellular aggregates (MCAs).

34 monstrated Km and kcat/Km values for Lys-Ala-MCA of 688 muM and 11.02 muM(-1) s(-1), respectively, an

35 rithm and a medoid classification algorithm (MCA), are examined.

36 well as tauro-alpha-muricholic acid (T-alpha-MCA) are potential markers for Geniposide-induced hepati

37 , making lactate the most likely alternative MCA as brain fuel.

38 ation enzyme mycothiol- S-conjugate amidase (MCA) from Mycobacterium tuberculosis are reported.

39 Michaelis complex analogue AK:Arg:Mg.AMPPNP (MCA), a product complex analogue AK:pAIE:Mg.ADP (PCA), a

40 We developed an MCA and an SCA that utilized either intact or env-delete

41 t the methods of metabolic control analysis (MCA) to determine which modifications are important for

42 d analysis using metabolic control analysis (MCA), metabolomics data, and statistical simulations.

43 is by multidimensional correlation analysis (MCA) is revisited.

44 The DT and MCA classifiers are sufficiently flexible and can produc

45 /- 11 vs. 336 +/- 14 W), and lowered ICA and MCA Vmean by 12-23% without compromising CCA blood flow.

46 In all conditions, reductions in ICA and MCA Vmean were associated with declining cerebral vascul

47 38.5%), and 26 (54.2%) with ICA, MCA-M1, and MCA-M2 occlusions, respectively, achieved a modified Ran

48 of 22) among patients with ICA, MCA-M1, and MCA-M2 occlusions, respectively.

49 me 16q24.1q24.2 in patients with ACD/MPV and MCA.

50 unrelated patients with sporadic ACD/MPV and MCA.

51 Heart rate (HR), arterial blood pressure and MCA V(mean) were continuously recorded.

52 ciated behaviors of ovarian cancer cells and MCAs are influenced by cellular cadherin composition.

53 ymal N-cadherin-expressing (Ncad+) cells and MCAs invade much more efficiently than E-cadherin-expres

54 oulomb repulsion in multiply charged anions (MCAs) creates a potential barrier that provides dynamic

55 process of numerous multiply charged anions (MCAs) have been traditionally explained in terms of the

56 sic MA, i.e., magnetocrystalline anisotropy (MCA), typically depends upon crystal symmetry.

57 ers (ASD), or multiple congenital anomalies (MCA).

58 sociated with multiple congenital anomalies (MCA).

59 te that during the Medieval Climate Anomaly (MCA) (900-1300 AD) the Pacific Northwest experienced exc

60 nt) and during the Medieval Climate Anomaly (MCA) (~1 ka to 0.7 ka).

61 ynamics during the Medieval Climate Anomaly (MCA) and the Little Ice Age (LIA) are poorly characteriz

62 ability during the Medieval Climate Anomaly (MCA) between 1,200 and 850 y B.P., when temperatures wer

63 peaked during the Medieval Climate Anomaly (MCA) from A.D. 900 to 1100, providing evidence that the

64 routes during the Medieval Climate Anomaly (MCA), A.D. 800-1300, when the initial colonization of CE

65 states, during the Medieval Climate Anomaly (MCA).

66 During the Medieval Climate Anomaly (MCA; approximately 1,000-500 cal B.P.), the period most

67 B12X12](2-), X = F, Cl, Br, I, At, which are MCAs with not well-separated excess charges and cannot b

68 age site peptide (Ac-Asp-Arg-Glu-Ile-Nle-Arg-MCA) is used as the substrate of separase.

69 The AONP-modified MC array (MCA) were subsequently functionalized by being immersed

70 chromatograph with a microcantilever array (MCA) is demonstrated for enhanced selectivity and potent

71 nvestigated in rat middle cerebral arteries (MCA).

72 ed and pressurized middle cerebral arteries (MCAs) and PAs taken from the same animals.

73 halus, and so-called middle cerebral artery (MCA) "pseudofeeders" were correlated with cardiac failur

74 bserved in the right middle cerebral artery (MCA) (166 +/- 27 cm/s to 135 +/- 27 cm/s, P < .001) and

75 gations of the right middle cerebral artery (MCA) and both common carotid arteries (CCAs).

76 ary occlusion of the middle cerebral artery (MCA) causing damage to brain tissue occurs in the majori

77 mall branches of the middle cerebral artery (MCA) corresponding to the barrel cortex, local blood flo

78 he M1 segment of the middle cerebral artery (MCA) for 1 h.

79 ures after malignant middle cerebral artery (MCA) infarction with decompressive hemicraniectomy (DHC)

80 nal occlusion of the middle cerebral artery (MCA) is used extensively in cerebral ischemia research.

81 n, 10 min, or 2 h of middle cerebral artery (MCA) ischemia using the suture (thread) model.

82 ly attenuated in the middle cerebral artery (MCA) isolated from female compared to male rats.

83 ing 1-hour bilateral middle cerebral artery (MCA) microembolic signals (MES).

84 tients with isolated middle cerebral artery (MCA) occlusion and early recanalization at MR angiograph

85 jury associated with middle cerebral artery (MCA) occlusion and reperfusion.

86 hemia was induced by middle cerebral artery (MCA) occlusion for 1 h followed by reperfusion in ovarie

87 stroke (AIS) due to middle cerebral artery (MCA) occlusion were enrolled; 75 underwent IVT.

88 induced by 60 min of middle cerebral artery (MCA) occlusion, elevated levels of JNK activity and phos

89 cose deprivation and middle cerebral artery (MCA) occlusion.

90 subjected to embolic middle cerebral artery (MCA) occlusion.

91 -rich thrombi in the middle cerebral artery (MCA) of mice.

92 al carotid artery or middle cerebral artery (MCA) stroke and to evaluate the relationship with treatm

93 ischemic changes in middle cerebral artery (MCA) stroke and to retrospectively compare admission non

94 acrificed for either middle cerebral artery (MCA) structure and function assessments via ex vivo pres

95 nist tirofiban, in a middle cerebral artery (MCA) thrombosis model in guinea pigs.

96 opic emboli into the middle cerebral artery (MCA) via an indwelling carotid artery catheter.

97 ent occlusion of the middle cerebral artery (MCA) which was produced either by inserting a filament v

98 Occlusion of the middle cerebral artery (MCA) with an endovascular filament is a widely used mode

99 clusion of the right middle cerebral artery (MCA), and anesthesia was either switched to alpha-chlora

100 ent occlusion of the middle cerebral artery (MCA).

101 cauterization of the middle cerebral artery (MCA).

102 CBF velocity at the middle cerebral artery (MCA).

103 emical injury to the middle cerebral artery (MCA).

104 oppler (aTCD) on the middle cerebral artery (MCA): MCA pulsatility index (PIa) and an estimator based

105 basilar artery (BA), middle cerebral artery (MCA)], the submandibular gland (SMG), and pineal gland w

106 Eln(+/+) mice in the middle cerebral artery (MCA, P < 0.001), but was similar between groups in the g

107 he M1 segment of the middle cerebral artery (MCA; 52 patients: median NIHSS score, 13 [interquartile

108 an those with tandem internal carotid artery-MCA occlusion and early recanalization (P = .05).

109 ither multiple-cycle or single-cycle assays (MCAs or SCAs); these assays have not been systematically

110 n the flanks of normal mice and mice bearing MCA 205 lung metastases.

111 g/kg) or vehicle was administered 2 h before MCA occlusion.

112 T-beta-MCA is an farnesoid X receptor (FXR) nuclear receptor an

113 ntestinal tauro-beta-muricholic acid (T-beta-MCA).

114 In addition, the phase and gain between MCA V(mean) and mean arterial pressure in the low freque

115 irculation modifies the relationship between MCA V(mean) and .

116 slope of the regression relationship between MCA V(mean) and at rest was greater (P = 0.035) than dur

117 alignant potential of lesions established by MCA inoculation, whereas inhibition of IL-12/23p40 enhan

118 by 25% following an acute stroke induced by MCA occlusion for 90 min.

119 velocity (MCA V(mean)), and calculated CBVC (MCA V(mean)/MAP) were assessed in seven healthy individu

120 he Medieval Climate Anomaly (950 to 1250 CE, MCA), a period which coincided with glacier retreat.

121 rowth and morphology of these cell clusters; MCAs grow larger and faster in the more rigid environmen

122 , OVA-expressing EG7 lymphoma cells and CMS5 MCA-induced fibrosarcoma cells naturally expressing muta

123 ial sheets and the formation of more compact MCAs relative to parental cells.

124 analysis of baseline variables, and complete MCA recanalization for the prediction of favorable clini

125 NKG2D ligand, in four independently derived MCA-205 transfectants.

126 nificantly better than that of end-diastolic MCA velocity alone (Az = 0.88 vs 0.84, P < .05).

127 e size and pattern of injury acutely, during MCA occlusion, and 7 days following reperfusion.

128 n-weighted (DW) MRI-detectable injury during MCA occlusion was similar following both surgical proced

129 t Thy1.2+ cells could specifically eliminate MCA-205 pulmonary metastases.

130 72 h later in neurons throughout the entire MCA territory in many but not all animals.

131 effectively suppressed growth of established MCA-induced tumors or TRAMP-C1 prostate tumors and inhib

132 Despite restoration of P(ETCO2), MCA V(mean) (P = 0.005) and CBVC (P = 0.03) remained red

133 C, which resulted in decreases in P(ETCO2), MCA V(mean) and calculated CBVC (P < 0.001 for all varia

134 MP levels were comparable in male and female MCAs (1.7 pmol/mg protein).

135 After 1 h of focal MCA ischemia, the coil was retrieved to cause reperfusio

136 Tumors from MCA/BHT-treated Rosa26-Foxm1 mice displayed a significan

137 he CARS signals from cellular sheddings from MCAs with LPA treatment are consistent with cleavage of

138 Furthermore, MCAs formed from MT1-MMP-T567E-expressing cells adhere a

139 Gly-MCA is a selective high-affinity FXR inhibitor that can

140 mice with glycine-beta-muricholic acid (Gly-MCA) inhibits FXR signalling exclusively in intestine, a

141 high-affinity FXR agonist GW4064 blocks Gly-MCA action in the gut, and intestine-specific Fxr-null m

142 eramide treatment reverses the action of Gly-MCA in high-fat diet-induced obese mice.

143 nresponsive to the beneficial effects of Gly-MCA.

144 These data suggest that Gly-MCA may be a candidate for the treatment of metabolic di

145 tically, the metabolic improvements with Gly-MCA depend on reduced biosynthesis of intestinal-derived

146 Thirty-one fetuses (53%) had MCA pseudofeeders.

147 tom onset in 51 patients suspected of having MCA stroke were reviewed.

148 To our knowledge, togetherness of hyperdense MCA sign and pulmonary thromboembolism is extremely rare

149 We conclude that during hypoglycemia MCA transport in the brain was increased by approximatel

150 ate day, however, exercise capacity and ICA, MCA Vmean and CCA dynamics were preserved.

151 38.5%), 20 (38.5%), and 26 (54.2%) with ICA, MCA-M1, and MCA-M2 occlusions, respectively, achieved a

152 and 40.9% (9 of 22) among patients with ICA, MCA-M1, and MCA-M2 occlusions, respectively.

153 Research toward improving MCA efficacy and economic competitiveness is required to

154 d by P(ETCO2), contribute to the decrease in MCA V(mean) and CBVC; however, other factors (e.g. perha

155 are known to play important roles either in MCA tumorigenesis or in the elimination phase of cancer

156 We show that the endothelium in MCA/129 fibrosarcomas and B16 melanomas exhibits a wild-

157 The cantilevers in MCAs are differentially coated on one side with responsi

158 ve stress marker nitrotyrosine was higher in MCAs from Eln(+/-) compared with Eln(+/+) mice (P < 0.05

159 6 mum in vehicle; P<0.05) in PAs, but not in MCAs.

160 An understanding of these processes in MCAs with not well-separated excess charges is still lac

161 normothermic level (P = 0.99) and increased MCA V(mean) (P = 0.002) and CBVC (P = 0.008) relative to

162 e data suggest that in addition to increased MCA transport at the blood-brain barrier, there may be a

163 O2) and cerebral vascular conductance index (MCA V(mean)/MAP) were lower in the older group at each e

164 ts previously shown to competitively inhibit MCA.

165 INTERPRETATION: MCA pseudofeeders are the result of impaired cerebral bl

166 Fischer rats were divided into: MCA tumor bearing fed chow (TB-Chow) or omega-3FA diet (

167 cm/s to 135 +/- 27 cm/s, P < .001) and left (MCA) (168 +/- 26 cm/s to 142 +/- 27 cm/s, P < .001) velo

168 hly tumorigenic mouse fibrosarcoma cell line MCA-205 with Ad5-E1A or a mutant form of E1A that does n

169 risk of developing seizures after malignant MCA stroke with DHC, especially after cranioplasty.

170 s increases growth rates, and change maximum MCA sizes.

171 raction conditions, corresponding to maximum MCA (9.550CAU/mL), were selected as follows: Grinding ti

172 (aTCD) on the middle cerebral artery (MCA): MCA pulsatility index (PIa) and an estimator based on di

173 recently observed as a long-lived metastable MCA with a large negative electron binding energy of -0.

174 expression of H60 on 3'-methylcholanthrene (MCA) sarcomas from 129/Sv mice.

175 rbamate (urethane), or 3-methylcholanthrene (MCA) and butylated hydroxytoluene (BHT) was used to indu

176 Using the carcinogen 3-methylcholanthrene (MCA), we demonstrate with Fourier transform-infrared spe

177 sed the development of 3-methylcholanthrene (MCA)-induced fibrosarcomas through a mechanism relying u

178 6-Foxm1 mice using the 3-methylcholanthrene (MCA)/butylated hydroxytoluene (BHT) lung tumor initiatio

179 tal tumor metastases and methylcholanthrene (MCA)-induced sarcomas in mice deficient for NLRP3 was NK

180 nged with the carcinogen methylcholanthrene (MCA).

181 cal skin carcinogenesis [methylcholanthrene (MCA) or 7,12-dimethylbenz(a)anthracene (DMBA)/12-O-tetra

182 We inoculated methylcholanthrene (MCA) 205 in the flanks of normal mice and mice bearing M

183 during immune control of methylcholanthrene (MCA)-induced or p53 mutant cancers and showed the critic

184 uccinyl-Leu-Leu-Val-Tyr-methylcoumarinamide (MCA), benzyloxycarbonyl-Leu-Leu-Glu-7-amido-4-MCA, and t

185 end-diastolic velocity for diagnosis of mild MCA narrowing was significantly better than that of end-

186 Following 5- and 10-minute MCA occlusions, 9 of 32 animals (28%) had microinfarcts

187 g a separate tumor initiator/promoter model (MCA+BHT) indicated that NF-kappaB functions as an indepe

188 The prion state, which we name [MCA], was transmitted to the chromosomally encoded Mca1p

189 Ncad+ MCAs exhibit rapid lateral dispersal prior to penetratio

190 Using intact tissue explants, Ncad+ MCAs were also shown to efficiently rupture peritoneal m

191 reased above rest (P < 0.001), while neither MCA V(mean) nor P(a,CO2) was altered (P > 0.05).

192 cerebral/common carotid arteries occlusion (MCA/CCAo) induced up-regulation of PPARgamma messenger R

193 ry 2002 and 31 June 2011 for space-occupying MCA ischaemic stroke and who underwent DHC.

194 more sensitive than TCD in the detection of MCA spasm.

195 detected TP in the endothelial cell layer of MCA.

196 ial, s.c., or 10-day pulmonary metastases of MCA 205 received total body irradiation conditioning or

197 rarchical framework and the methodologies of MCA may prove useful both for thinking about aging and f

198 Neuroimaging results showed that 0 min of MCA occlusion did not produce a lesion, whereas occlusio

199 we previously developed an embolic model of MCA occlusion in the rat, which recapitulates the key co

200 ism from acetate (P = 0.015) and the rate of MCA transport (P = 0.01) were also approximately twofold

201 ant thrombi resulting in fast restoration of MCA patency and consequently reduced cerebral infarct si

202 The severity of MCA depends on a number of different factors, including

203 ent with the hypothesis that upregulation of MCA transport may contribute to the maintenance of brain

204 Use of MCA velocity measurements and V(MCA)/V(ICA) ratio can in

205 itiveness is required to enhance the role of MCAs in IPM.

206 In this series of MCAs, comprising a "boron core" surrounded by a "halogen

207 respectively; P = .004) and was dependent on MCA recanalization (odds ratio, 5.55; P = .006).

208 individuals with unexplained DD/ID, ASD, or MCA than a G-banded karyotype ( approximately 3%, exclud

209 nostic test for patients with DD/ID, ASD, or MCA.

210 r twofold increase in the transport of other MCAs, including lactate, during insulin-induced hypoglyc

211 Recruitment of tissue outside MCA territory produced a steep increase in the volume of

212 Vascular diameter was measured in perfused MCA segments using videomicroscopy in the presence and a

213 After induction of permanent MCA occlusion (pMCAO), the rats received an initial intr

214 PFR-MCA hydrolysis was greater for patients' IgG than for a

215 nylalanine-arginine-methylcoumarinamide (PFR-MCA) substrate.

216 tor VIII, did not correlate with that of PFR-MCA prior transplantation, whereas it did 12 mo posttran

217 Prenatal MCA pseudofeeders were a risk factor for encephalomalaci

218 ALX-0081 prevented MCA thrombosis and induced reperfusion when given immedi

219 pH 3 had the best milk-clotting properties (MCA/PA ratio).

220 g protein) and female (11.2 pmol/mg protein) MCAs.

221 There were 160 patients with proximal MCA occlusion at six centers in this institutional revie

222 te that reduced EDHF dilations in female rat MCA cannot be solely attributed to impaired cAMP signali

223 nhance EDHF-mediated dilations in female rat MCA.

224 duced in the female compared to the male rat MCA.

225 In severe reactions, MCA can be documented by a substantial increase in the s

226 The right MCA was occluded in spontaneously hypertensive rats for

227 A second MCA functionalized with two different immunological rece

228 Furthermore, we show that in serum-starved MCA, expression of constitutively active AKT re-establis

229 In serum-starved MCAs, EGFR activation was associated with active ERK1/2,

230 s has been achieved, in most orchard systems MCAs account for a relatively small proportion of the pe

231 elial collagen matrix on which MT1-MMP-T567E MCAs rapidly disperse.

232 The up-regulation of RAE-1 by E1A targeted MCA-205 tumor cells to lysis by NK cells, resulting in N

233 Following temporary MCA occlusion EEG was monitored from a screw electrode a

234 The MCA V(mean) and were linearly related at rest (P < 0.001

235 The MCA was graded as normal, narrowed <50%, and >50% using

236 femoral artery catheterization to access the MCA and therefore eliminated the occurrence of head trau

237 tatistically different fitness values by the MCA versus the SCA: the D30N, G48V, I50V, I54L, and I54M

238 est dataset (300 Cases and 300 Controls) the MCA procedure finds several unique classifiers that have

239 Diminished SASM precipitation during the MCA and CWP tracks reconstructed Northern Hemisphere and

240 Lake-based fire rotation during the MCA decreased to an estimated 120 y, representing a 260%

241 imilar to feedbacks that occurred during the MCA may stabilize the fire regime, despite additional wa

242 patterns at bidecadal resolution during the MCA.

243 We propose that discordant results in the MCA and SCA are due to alterations in late events in the

244 tion did not affect tumor development in the MCA model; however, CXCR3 was important in the DMBA/TPA

245 atio after flow velocity measurements in the MCA resulted in a decreased number of false-negative fin

246 evels of JNK activity and phospho-JNK in the MCA territory.

247 In the MCA, after incubation with the superoxide scavenger TEMP

248 protects the input of K(Ca)2.3 to EDH in the MCA, and represents a beneficial pleiotropic effect of s

249 In the MCA, EDD did not differ between groups after incubation

250 hanges in central blood volume influence the MCA V(mean) at rest and during exercise and its regulati

251 By occluding the MCA with a fibrin-rich allogeneic clot, we previously de

252 e range, 6-16]), or of the M2 segment of the MCA (48 patients: median NIHSS score, 7 [interquartile r

253 The peak of the MCA (900-1150 CE) was a warm period in the eastern Pacif

254 or flushed into the superior division of the MCA (M2).

255 ti-decadal cold conditions at the end of the MCA are neither resolved in temperature reconstructions

256 The average lifetime of the MCA is 36 ms and the free energy difference to the TSA-l

257 on of infarcts smaller than one-third of the MCA territory (32%, 48%, and 69% for ASITN/SIR grades of

258 boxylic acids [MCAs]) by upregulation of the MCA transporters may contribute metabolic substrates dur

259 lium-dependent dilatation (carbachol) of the MCA were not different between groups.

260 millennium, the first one at the end of the MCA, in ~1200 CE, was of similar amplitude to two other

261 60 minutes after the total occlusion of the MCA.

262 e abrupt and sustained than the onset of the MCA.

263 re restricted to cortical territories of the MCA.

264 unts of mus-ms time scale dynamics, only the MCA samples a dominant excited state that resembles the

265 After T3-SCI, the MCA had more collagen I (42%), collagen III (24%), trans

266 ing, however, did not persist throughout the MCA.

267 ging wind field patterns associated with the MCA provided conditions in which voyaging to and from th

268 Infusion of t-PA did not dissolve these MCA occlusions.

269 e trend in the electronic stability of these MCAs is determined by the interplay between the Coulomb

270 gm for understanding the properties of these MCAs with not well-separated charges that departs from t

271 Interaction of these MCAs with peritoneal mesothelium disrupts mesothelial in

272 cetate is transported into the brain through MCA transporters that are also used for lactate and keto

273 re difficult to elucidate when introduced to MCA systems as mixtures, although pattern recognition te

274 The apparent limit to MCA burning has been surpassed by the regional fire regi

275 l barrier that provides dynamic stability to MCAs and allows electronically metastable species to be

276 exercise were attenuated following transient MCA occlusion (MCAO) and reperfusion, mediated via alter

277 IFN-gamma-treated MCA sarcomas with high levels of H60 were resistant to k

278 B. thuringiensis is the most used MCA for control of lepidopteran orchard pests.

279 ontrol of several orchard pest insects using MCAs, including viruses, Bacillus thuringiensis, fungi,

280 V(MCA), V(tICA), and V(tICA)/V(ICA) ratio were not age dep

281 Use of MCA velocity measurements and V(MCA)/V(ICA) ratio can increase the accuracy of Doppler U

282 not age dependent, contrary to V(ICA) and V(MCA)/V(ICA) ratio, after controlling for hematocrit and

283 parameters were peak systolic velocity and V(MCA)/V(ICA) ratio.

284 ow velocity in the middle cerebral artery (V(MCA)) to flow velocity in the ipsilateral extracranial i

285 meters per second for mean angle-corrected V(MCA) on the left and right sides were 62-198 and 69-153;

286 Reference ranges for mean angle-corrected V(MCA)/V(ICA) ratio on the left and right sides were 1.2-4

287 nography (US), and carotid US to determine V(MCA)/V(ICA) and V(tICA)/V(ICA) ratios from angle-correct

288 The study provides reference limits for V(MCA), V(tICA), V(ICA), and velocity ratios obtained from

289 y of 94 and 108 cm/sec and a peak systolic V(MCA)/V(ICA) ratio of 3.6 and 3.9 for diagnosis of mild a

290 The stepwise approach with use of the V(MCA)/V(ICA) ratio after flow velocity measurements in th

291 Accuracy of the V(MCA)/V(ICA) ratio calculated on the basis of end-diastol

292 lood flow), middle cerebral artery velocity (MCA Vmean), arterial-venous differences and blood temper

293 middle cerebral artery mean blood velocity (MCA V(mean)) in seven healthy volunteer men at rest and

294 CO2), middle cerebral artery blood velocity (MCA V(mean)), and calculated CBVC (MCA V(mean)/MAP) were

295 Mean middle cerebral artery blood velocity (MCA V(mean)), mean arterial pressure (MAP), cardiac outp

296 imal intracranial arterial occlusion (ICA vs MCA-M1 vs MCA-M2) was not.

297 cranial arterial occlusion (ICA vs MCA-M1 vs MCA-M2) was not.

298 y involved intracranial stenosis segment was MCA, present in 10 (41.6%) out of 24 intracranial segmen

299 formed in 26 (35%) IVT-treated patients with MCA recanalization (group 1).

300 l to use pattern recognition techniques with MCAs as a new type of detector for chromatography with a