ライフサイエンスコーパス: MCP-1 receptor

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1 ce of the amino-terminal domain of CCR2, the MCP-1 receptor.

2 SMC, indicating that they possess a distinct MCP-1 receptor.

3 t a two-step mechanism for activation of the MCP-1 receptor.

4 The C-C chemokine receptor-1 (CKR-1), the MCP-1 receptor-A (MCP-1Ra), and MCP-1Rb can reconstitute

5 eceptors revealed that the third loop of the MCP-1 receptor accounted for virtually all of the coupli

6 In HEK-293 cells, however, the MCP-1 receptors and C-CR1 coupled to Galphaq but failed

7 either an MCP-1-neutralizing antibody nor an MCP-1 receptor antagonist inhibited ET-1-induced collage

8 proteins that couple to the two forms of the MCP-1 receptor, as well as to related chemokine receptor

9 ell specificities and because CCR2, a cloned MCP-1 receptor, binds other ligands, it has been uncerta

10 and VSMCs invasion that are inhibited by the MCP-1 receptor blocker vCCI.

11 retained the amino-terminal extension of the MCP-1 receptor bound MCP-1 with high affinity.

12 ude that the amino-terminal extension of the MCP-1 receptor, but not the RANTES/MIP-1alpha receptor,

13 -1 bound with high affinity to the wild-type MCP-1 receptor, but not to the RANTES/MIP-1alpha recepto

14 Mice deficient for the major MCP-1 receptor, CC chemokine receptor (CCR)2, did not de

15 e type A monocyte chemoattractant protein 1 (MCP-1) receptor CCR-2A and the type B MCP-1 receptor (CC

16 ells transfected with the monocyte selective MCP-1 receptor (CCR-2B) and the eosinophil selective eot

17 ein 1 (MCP-1) receptor CCR-2A and the type B MCP-1 receptor (CCR-2B) have been recently cloned and fo

18 Finally, the MCP-1 receptor CCR2 was expressed in neurons and some no

19 ecific blockade achieved via blockade of the MCP-1 receptor CCR2, was effective at reducing neointima

20 nfusion into mice lacking either IL-6 or the MCP-1 receptor CCR2.

21 ability to down-modulate and internalize the MCP-1 receptor (CCR2) in response to exposure to this ch

22 m of the monocyte chemoattractant protein-1 (MCP-1) receptor (CCR2) gene on human chromosome 3p21.

23 , atherosclerosis, and asthma, by use of the MCP-1 receptor, CCR2, a member of the G-protein-coupled

24 the alternatively spliced form of the human MCP-1 receptor (CCR2A) binds MCP-1 with high affinity an

25 However, mRNA encoding the macrophage MCP-1 receptors, CCR2A and B, is not present in SMC, ind

26 nd J-beta 2.7 transfectants coexpressing the MCP-1 receptor CCR2B and wild-type alpha L.

27 nts to dimerize and to bind and activate the MCP-1 receptor, CCR2b.

28 THP-1 cells expressed on average about 5000 MCP-1 receptors/cell.

29 In COS-7 cells, the type A and type B MCP-1 receptors coupled to Galphai, Galphaq, and Galpha1

30 e alternatively spliced variants of a single MCP-1 receptor gene.

31 To better understand the role of the human MCP-1 receptor (hCCR2) in monocyte recruitment, we have

32 In contrast, chimeric MCP-1 receptors, in which the wild-type amino terminus w

33 by pertussis toxin, suggesting that the SMC MCP-1 receptor is coupled to a Gi-protein.

34 data indicate that the amino terminus of the MCP-1 receptor is necessary for high affinity binding of

35 The monocyte chemotactic protein-1 (MCP-1) receptor (MCP-1R) is expressed on monocytes, a su

36 lts show that in the 3B4 M1.9 T cell hybrid, MCP-1 receptors mediate intracellular calcium mobilizati

37 This cell line was used to examine MCP-1 receptor-mediated signal transduction events in a

38 hat the detection of a high concentration of MCP-1 receptors on inflammatory cells should noninvasive

39 In contrast to the MCP-1 receptor, substitution of the wild-type amino-term

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