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1 MDC are a large family of membrane-bound proteins contai
2 MDC blockade was deleterious in the survival of mice aft
3 MDC frequencies tended to be reduced in HIV infection (1
4 MDC mRNA and protein expression were markedly induced in
5 MDC-associated IL-12 production was markedly reduced in
6 MDC-enriched cells were either CD123(+) or CD123(-), but
7 MDC-expressing DCs specifically attracted T lymphocytes
8 MDC-L (ADAM 28), a member of the ADAM (a disintegrin and
9 MDC-staining and the GFP-AtATG8e fusion protein can now
10 MDCs activated with lactobacilli clearly skewed CD4(+) a
11 MDCs and PDCs exposed to either rAd5 or rAd35 encoding f
12 MDCs derived in vitro suppressed angiogenesis in vivo th
13 MDCs were more susceptible to both rAd35 and rAd5 than w
14 < .001), IL-10 (P = .01), IP-10 (P = .0001), MDC (P < .001), MIP-1alpha, (P < .001), MIP-1beta (P = .
15 of CC (I-309, Exodus-1, TARC, RANTES, MCP-1, MDC, and MIP-1 alpha and -1 beta), CXC (GRO-alpha, -beta
16 quence in the disintegrin domain of ADAM-15 (MDC-15; metargidin) highlighted ADAM-15 as a protein par
18 participants (21/112) did not meet accepted MDC for Moebius syndrome because they had abduction defi
19 st a potential role for the lymphocyte ADAM, MDC-L, in the interaction of lymphocytes with alpha(4)be
20 Consistent with this finding, virtually all MDCs isolated from the tissues or draining lymph nodes o
21 30 cross-sectional subjects revealed that an MDC:PDC ratio more than or equal to 1.78 was associated
30 ttern of CCR4 and its ligands TARC/CCL17 and MDC/CCL22 in the peripheral blood and skin of patients w
33 ndicate that lymph node Langerhans cells and MDC are APC needed for induction of the protective respo
35 determined in vitro, but only methylone and MDC (1, 3 mg/kg, iv) produced significant elevations in
36 or the PK study, we found that methylone and MDC peaked early (Tmax=15-45 min) and were short lived (
37 rea-under-the-curve values for methylone and MDC were greater than dose-proportional, suggesting non-
39 ess CCR4 and respond to its ligands TARC and MDC, whereas Th1 lymphocytes express CXC chemokine recep
41 covery of both recipient MDSCs (P < .01) and MDCs (P < .01) is significantly increased when the alkyl
44 Soon after HIV-1 exposure, both PDCs and MDCs were able to transfer the virus to T cells in the a
47 CLP, and neutralization of the MDC with anti-MDC Abs decreased CLP-induced recruitment of peritoneal
48 A positive association was observed between MDC-associated IL-12 production and HCV-specific T cell
49 T cells conditioned with the B7-H1-blocked MDCs had a more potent ability to inhibit autologous hum
50 g IAV infection, we stimulated primary blood MDCs and inflammatory monocyte-derived DCs (MDDCs) with
55 lone is extensively metabolized in rats, but MDC is the only centrally active metabolite that could c
56 4(+) T cells in a dose-dependent manner, but MDCs stimulated the highest frequencies of pp65-specific
57 resection is inhibited by gamma-H2AX and by MDC-1 (mediator of DNA damage checkpoint 1), which binds
58 action between RhoA and ROCK-2 is blocked by MDC and TGaseM, indicating a role for transglutaminase a
60 IL-12 production and CD80/CD86 expression by MDC/PDC mixtures was further enhanced by CD40L and ISS-O
61 Further, second-wave aggregation induced by MDC in platelet-rich plasma was inhibited by aspirin, AD
66 oprotein-pseudotyped lentivirus particles by MDCs were severely attenuated upon depletion of GSLs fro
69 s expressing mRNA encoding TSLP, TARC/CCL17, MDC/CCL22, and IP-10/CXCL10, but not I-TAC/CXCL11 and I-
70 mucosa expressing mRNA for TSLP, TARC/CCL17, MDC/CCL22, and IP-10/CXCL10, but not I-TAC/CXCL11 and I-
71 venance of TSLP, Th2-attracting (TARC/CCL17, MDC/CCL22, I-309/CCL1), and Th1-attracting (IP-10/CXCL10
72 erum concentration of CXCL10 (IP-10), CCL22 (MDC), CCL17 (TARC), CCL-2 (MCP-1) and CCL-13 (MCP-4) in
73 in the local chemokine production of CCL22 (MDC) and CCL17 (TARC), two chemokines previously shown t
74 uniformly suppressed the production of CCL22/MDC, a chemokine associated with infiltration of T regul
75 established that both CCR3/eotaxin and CCR4/MDC axes contribute to the recruitment of Th2 cells to t
76 the functional role of CCR3/eotaxin and CCR4/MDC during the in vivo recruitment of Th2 cells, we have
77 cells respond to both CCR3/eotaxin and CCR4/MDC pathways initially, but that a progressive increase
78 ells results in the predominance of the CCR4/MDC axis in the long-term recruitment of Th2 cells in vi
81 ude non-DCs, Lin(-)HLA-DR(+)CD11c(+)CD123(-) MDCs and Lin(-)HLA-DR(+)CD11c(-)CD123(+) PDCs were ident
83 Freshly purified myeloid dendritic cells (MDC) and plasmacytoid DC (PDC) obtained from subjects wi
85 nocytes and myeloid-derived dendritic cells (MDC) released the protein exclusively following stimulat
87 n of human CD11c(+) myeloid dendritic cells (MDCs) and CD123(+) plasmacytoid dendritic cells (PDCs),
88 od monocyte-derived myeloid dendritic cells (MDCs) express B7-H1, a member of the B7 family, on the c
89 capture of HIV-1 by mature dendritic cells (MDCs) is mediated by an interaction between the glycosph
90 ansion of recipient myeloid dendritic cells (MDCs) that induce contact-dependent expansion of donor T
95 eases in total leukocytes, Langerhans cells, MDC, LDC, and activated CD4+ T cells in draining lymph n
96 e receptor for macrophage-derived chemokine (MDC) and thymus and activation-regulated chemokine (TARC
98 tralization of macrophage-derived chemokine (MDC) with Ab caused a significant reduction of CCR4-posi
99 ine (TARC) and macrophage-derived chemokine (MDC)), CD (10 proteins), and PS (kynureninase (KYNU)).
100 tional role of macrophage-derived chemokine (MDC), a potent mononuclear cell chemoattractant, during
102 r (TNF)-alpha, macrophage-derived chemokine (MDC), and C10, known to enhance bacterial clearance.
103 ctor (GM-CSF), macrophage-derived chemokine (MDC), and macrophage inflammatory protein 1alpha (MIP-1a
104 kines, RANTES, macrophage-derived chemokine (MDC), macrophage inflammatory protein-1beta (MIP-1beta),
105 orted that the macrophage-derived chemokine (MDC), thymus activation-regulated chemokine (TARC), and
106 ; in addition, macrophage-derived chemokine (MDC), thymus and activation-regulated chemokine (TARC),
107 ta chemokines [macrophage-derived chemokine (MDC), thymus and activation-regulated chemokine (TARC),
108 t (MCP-1, MCP-5, monocyte-derived chemokine (MDC), thymus and activation-related chemokine (TARC), C1
112 10)/CXCL10 and macrophage-derived chemokine (MDC)/CCL22 production were evaluated at the mRNA or at t
113 (TARC)/CCL17, macrophage-derived chemokine (MDC)/CCL22, I-309/CCL1) and Th1-attracting (IFN-gamma-in
115 MCP-3/CCL7) or macrophage-derived chemokine (MDC/CCL22), elicited anti-gp120 antibodies with high tit
116 ARC/CCL17) and macrophage-derived chemokine (MDC/CCL22), which preferentially attract type 2 T helper
118 le protein-10, macrophage-derived chemokine [MDC], and platelet factor-4 [PF-4]) to be expressed by C
119 and CCL22 (or macrophage-derived chemokine; MDC), in Th2-type cytokine-dominated responses in the lu
121 OX40L, the production of the Th2-chemokines MDC (macrophage-derived chemokine/CCL22) and TARC (thymu
122 These findings suggest that the chemokines MDC, TARC, and SDF-1, which may be produced during infla
123 ferent from that of secretion of chemokines (MDC, I-309, MIP-1alpha, MIP-1beta, and RANTES), cytokine
125 ecular Probes]), autophagic vacuole content (MDC), SA-beta-galactosidase (FDG), and cathepsin activit
128 that networks within multiple-demand cortex (MDC) become active when diverse skills and behaviors are
129 ing, networks within multiple-demand cortex (MDC), which control higher cognitive functions, such as
130 oebius syndrome minimum diagnostic criteria (MDC) (congenital, nonprogressive facial palsy, and abduc
131 ted that degrakines based on SDF-1 (CXCL12), MDC (CCL22) and RANTES (CCL5) specifically inactivate th
132 assays showed that SDF-1, which binds CXCR4; MDC, which binds CCR4; and ELC, which binds CCR7, mediat
135 ells (DCs), we evaluated myeloid-derived DC (MDC) and plasmacytoid-derived DC (PDC) frequencies and f
137 three subsets, Langerhans cells, myeloid DC (MDC), and lymphoid DC (LDC), based upon phenotypic and f
138 macytoid DCs (PDCs) 40-fold and myeloid DCs (MDCs) 20-fold less frequent in HIV(+) nodes than in cont
139 c cells (DCs); CD11c(+)CD123(-) myeloid DCs (MDCs) and CD11c(-)CD123(+) plasmacytoid DCs (PDCs).
140 ecies of Lactobacillus on human myeloid DCs (MDCs) and found that they modulated the phenotype and fu
141 how different subsets of human myeloid DCs (MDCs) involved in tissue inflammation are affected by in
143 acytoid DCs (PDCs) and CD11c(+) myeloid DCs (MDCs) were susceptible to both a CCR5- and a CXCR4-using
144 rformed to identify mature DCs, myeloid DCs (MDCs), and plasmacytoid DCs (PDCs) by using antibodies a
147 a biotin-independent malonate decarboxylase (MDC), which allows them to use malonate as the sole carb
153 PDCs to augment angiogenesis while excluding MDCs to prevent angiogenesis inhibition, demonstrating a
155 conversion of MDSCs to PD ligand-expressing MDCs, and increased donor naturally occurring Treg recov
156 n pediatric control muscle, there were fewer MDCs than PDCs, and the distributions of MDCs and PDCs w
157 e critical producers of cytokines (IL-12 for MDC and type I/II IFNs for PDC) and are functionally dif
159 this study that CCR4 is a major receptor for MDC and TARC on T lymphocytes, as anti-CCR4 mAbs signifi
161 his study, we demonstrate a pivotal role for MDC during experimental sepsis induced by cecal ligation
163 e infiltrating lymphocytes, and staining for MDC was present on scattered individual cells throughout
166 .004) was associated with numerically higher MDC counts and significantly lower PDC frequencies (P=0.
168 sures analysis showed a significantly higher MDC:PDC ratio (P=0.043, F-test) among rejectors, compare
182 hese results suggest that TNFR1 signaling in MDCs is a critical mediator of both the detrimental and
184 ignificantly, however, mice lacking TNFR1 in MDCs succumbed to listeria infection, although they disp
186 upernatant collected from long-term infected MDC, which had been exposed to an X4R5 virus 45 days ear
194 our unrelated participants, also not meeting MDC, had large-angle exotropia, vertical gaze deficiency
197 I metabolites: 3,4-methylenedioxycathinone (MDC), 3,4-dihydroxy-N-methylcathinone (HHMC), and 4-hydr
202 which is inhibited by monodansylcadaverine (MDC; an inhibitor of transglutaminase activity) and TGas
203 s, the fluorescent dye monodansylcadaverine (MDC) and a green fluorescent protein (GFP)-AtATG8e fusio
204 h the TGase inhibitor, monodansylcadaverine (MDC), converted RA from a differentiation factor to an a
205 Cs in 0.3 M sucrose or monodansylcadaverine (MDC), which both inhibit clathrin-coated pit formation,
206 d p62, GFP-LC3 puncta, monodansylcadaverine (MDC) staining, and transmission electron microscopy.
207 effects, we found that monodansylcadaverine (MDC), which binds to the enzyme (transamidase) active si
208 to block tTG function (monodansylcadaverine; MDC) or c-Src kinase activity (PP2) disrupted the format
211 DC from HCV-infected persons, the ability of MDC to activate naive CD4 T cells in the presence or abs
212 ults indicate novel regulatory activities of MDC in innate immunity during sepsis and suggest that MD
214 e report here that the disintegrin domain of MDC-L is recognized by the leukocyte integrin alpha(4)be
215 roteins possessing the disintegrin domain of MDC-L supported adhesion of the T-lymphoma cell line, Ju
216 of Jurkat cells to the disintegrin domain of MDC-L was inhibited by an anti-MDC-L monoclonal antibody
219 the absolute number and median frequency of MDC in HIV-infected individuals were similar to those ob
222 tion, mice treated with an i.p. injection of MDC cleared bacteria more effectively than those in the
224 tion is available concerning the presence of MDC in the kidney or their role in renal pathophysiology
227 secreted cytokines and chemokines typical of MDC upon stimulation with a TLR-4 agonist and both subse
228 atical model to simulate dynamic behavior of MDCs for the first time through evaluating multiple fact
229 data highlight the functional capacities of MDCs and PDCs in naive as well as healthy, infected maca
230 wer MDCs than PDCs, and the distributions of MDCs and PDCs were similar in pediatric control skin sam
232 ty and thus has highlighted the potential of MDCs as an energy-efficient technology to address water-
233 re was increased CD83 and CD86 expression on MDC from HCV-infected persons, the ability of MDC to act
236 rontal midline node of the cingulo-opercular MDC affected learning rates specifically during the init
238 into a mitochondrial-derived compartment, or MDC, followed by release through mitochondrial fission a
239 n with recombinant RABV expressing GM-CSF or MDC protected significantly more mice against intracereb
242 Optimally TLR ligand-stimulated PDCs or MDCs exposed to CMV or HIV-1 Ags enhanced autologous CMV
243 nsduced dendritic cells (DCs) overexpressing MDC would enhance the T cell-mediated humoral immune res
246 HLA DR expression; however, it does promote MDC maturation, as NS-398 significantly reduces CD83 exp
248 esent the crystal structure of a Pseudomonas MDC and give insights into its catalytic mechanism and f
252 on the nature of hepatitis; the data specify MDCs as the essential cell type of TNFR1 function in T c
256 latory T cells (Tregs) that express the TARC/MDC-specific chemokine receptor CCR4, thus generating an
260 and male human participants to confirm that MDC was most active in the initial stages of learning a
264 rating characteristic analysis revealed that MDC:PDC ratio more than or equal to 1.52 was associated
267 al of malignant T cells and demonstrate that MDCs are abundant within the tumor microenvironment of T
274 ongitudinal and cross-sectional cohorts, the MDC:PDC ratio was higher and was associated with decreas
275 ermore, optimal operating conditions for the MDC used in this study were estimated to have an acetate
277 e inverse association with viral load of the MDC number, but not of IFN-alpha secretion or the number
278 itoneum after CLP, and neutralization of the MDC with anti-MDC Abs decreased CLP-induced recruitment
279 It was able to predict the response of the MDC with time under various conditions, and also provide
281 pression of the glomerular expression of the MDC/CCL22 chemokine and down-regulation of MIP-1alpha/CC
283 tochondrial dysfunction, suggesting that the MDC pathway provides protection to mitochondria in times
289 rthermore, adhesion of various cell lines to MDC-L correlated with expression of the integrin alpha(4
290 ide evidence that noninvasive stimulation to MDC nodes can enhance learning rates, thereby demonstrat
292 pression of TLR4, TRIF, and MyD88 in the two MDC subsets regulated the ability of the cells to enter
294 nd COX-2 when activated, we examined whether MDC express these enzymes and produce prostanoids that a
299 resent study demonstrated that a node within MDC, located in midline frontal cortex, becomes active d
300 these cells in the presence of NS-398 yields MDC that stimulate significantly more IFN-gamma in an al
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