ライフサイエンスコーパス: MDF

1 MDF during trophic transfer of MeHg leading to enrichmen

2 MDF encodes a putative RS domain protein with a predicte

3 MDF expression is not defective in the bodenlos, pin1 or

4 MDF identifies patients at risk of relapse and poor outc

5 all ions that fall outside of the GSH adduct MDF template windows, the processed full scan MS chromat

6 ither uniquely by one MDF or randomly by all MDFs.

7 We analyzed MDF of end-induction bone marrow samples from a historic

8 Moreover, the sensitivities with FIGS and MDF were equal for all As species, allowing for the poss

9 In general, any MDF protein that is expressed also is present on transcr

10 249 patients were prospectively evaluated by MDF for RD, and presence of RD was correlated with disea

11 of target genes correlates with occupancy by MDF, in particular, herculin.

12 y, the epsilon subunit gene becomes a common MDF target and begins to be expressed.

13 spindle assembly checkpoint (SAC) component MDF-1/MAD1 is required for the PGC arrest.

14 -1 or a second spindle checkpoint component, MDF-2, failed to arrest the cell cycle, exhibited chromo

15 ffects its binding to another SAC component, MDF-2/MAD2.

16 but the mass dependent isotope compositions (MDF; delta(202)Hg) were not (r(2) = 0.26, p = 0.16), ref

17 ase (RD) by multidimensional flow cytometry (MDF) in children treated on Children's Oncology Group AM

18 measured by multidimensional flow cytometry (MDF) is a key early prognostic indicator and is strongly

19 tions in the Arabidopsis MERISTEM-DEFECTIVE (MDF) gene lead to a loss of stem cell and meristematic a

20 Measurements of Hg isotopic mass-dependent (MDF) and mass-independent fractionation (MIF) in food we

21 indicating lower degrees of mass-dependent (MDF) and mass-independent Hg fractionation (MIF) (respec

22 appearance of myogenic determination factor (MDF) transcripts in developing chick limbs and other emb

23 The myogenic determination factors (MDFs) are transcriptional activators that target E boxes

24 of a single member of the MyoD gene family (MDF) is necessary and sufficient to establish the positi

25 raphene behave like massless Dirac fermions (MDFs) with linear energy-momentum dispersion (1, 2) , pr

26 as designed to apply the mass defect filter (MDF) approach to the screening and identification of rea

27 red to a standard miniature diffusion flame (MDF) atomizer.

28 y-derived metric (the Max-min Driving Force, MDF), which enables objective ranking of pathways by the

29 Mass-dependent fractionation (MDF) and mass-independent fractionation (MIF) may cause

30 Mass-dependent fractionation (MDF) and mass-independent fractionation (MIF) of Hg isot

31 ish Hg isotope mass-dependent fractionation (MDF) during biotic methylation (-1.20 to +0.58 per thous

32 leads to both mass-dependent fractionation (MDF) of Hg isotopes and mass-independent fractionation (

33 this is due to mass-dependent fractionation (MDF) of up to -0.9 per thousand between IHg and MMHg.

34 atively narrow mass-dependent fractionation (MDF, delta(202)Hg; +/- 0.08 per thousand, 2SD) ranges (-

35 ependent and mass-independent fractionation (MDF and MIF) of methylmercury (MeHg) during trophic tran

36 with calculation of mean dominant frequency (MDF) and relative power of delta, theta, alpha and beta

37 gic complete remission at EOI1, 46 (24%) had MDF-detectable disease.

38 The difference in delta(202)Hg (MDF) and Delta(199)Hg (MIF) between fish tissues and foo

39 Large variations in MDF and MIF are observed in fish and provide new insight

40 cipitation, have determined which individual MDFs reside at promoters of several receptor subunit gen

41 PGC arrest by two mechanisms, one involving MDF-2 and another that is independent of other SAC compo

42 West-Haven criteria) and various MMSE items, MDF showed no correlation with any of MMSE items as well

43 te checkpoint mechanism in which a core Mad1(MDF-1)/Mad2(MDF-2) signal generated at kinetochores is i

44 s orchestrate the integration of a core Mad1(MDF-1)/Mad2(MDF-2)-based signal, with a largely independ

45 tically and physically with SAC protein MAD1/MDF-1, whose nuclear envelope accumulation requires NPP-

46 t mechanism in which a core Mad1(MDF-1)/Mad2(MDF-2) signal generated at kinetochores is integrated wi

47 e the integration of a core Mad1(MDF-1)/Mad2(MDF-2)-based signal, with a largely independent Mad3(SAN

48 mes from the fact that subtly elevating Mad2(MDF-2) levels bypasses the requirement for BUB-3 and Mad

49 etochore localization is independent of Mad2(MDF-2).

50 uence on the Hg isotope composition of MMHg (MDF) in sediments and aquatic organisms.

51 Expression of a non-phosphorylatable mutant MDF-1 partially suppressed the defect in the starvation-

52 The poplar leaves exhibited negative MDF (-3.18 to -1.22 per thousand) and small positive MIF

53 We have analyzed expression of MDF and acetylcholine receptor subunits in cultured mous

54 lt2 double mutants have unaffected levels of MDF RNA, indicating that MDF acts upstream of PIN and PL

55 y defined as containing background levels of MDF transcripts which are thought to be nonfunctional.

56 The loss of MDF-2 or another SAC component also caused inappropriate

57 Overexpression of MDF leads to the activation of markers of embryonic iden

58 PDL-1 functions in a kinetochore receptor of MDF-1/MAD1 to induce SAC function.

59 The phosphorylation status of MDF-1 affects its binding to another SAC component, MDF-

60 ty of states consistent with the presence of MDFs.

61 tified 2 Akt kinase phosphorylation sites on MDF-1.

62 ive gene is occupied, either uniquely by one MDF or randomly by all MDFs.

63 n of stable oxidative metabolites with other MDF templates, and determination of metabolite molecular

64 This result is in contrast to net positive MDF (+0.4 to +0.8 per thousand) previously observed in l

65 lecular events and interactions that precede MDF expression in myogenic precursor cells.

66 mitotic delay and localizes the SAC protein MDF-1/MAD1 to the kinetochore facing away from the spind

67 s showed very similar Hg isotope signatures (MDF delta(202)Hg: -0.2 per thousand to -0.5 per thousand

68 ergence of a Dirac band structure supporting MDFs has been observed in AG using molecular (5) , atomi

69 naffected levels of MDF RNA, indicating that MDF acts upstream of PIN and PLT gene expression.

70 aused by mdf-1 hemizygosity, suggesting that MDF-1 causes the PGC arrest by two mechanisms, one invol

71 in L1 larvae lacking DAF-18, suggesting that MDF-1 regulates germ cell proliferation as a downstream

72 on-of-function allele of mdf-1 suggests that MDF-1 has a dual role during development.

73 types, that is not directly addressed by the MDF model.

74 uggests a unique pathway responsible for the MDF of Hg isotopes during methylation by this strain reg

75 se results demonstrate a requirement for the MDF-dependent pathway in regulating PIN/PLT- and WUS/CLV

76 scan by a triple quadrupole instrument, the MDF approach was more sensitive and selective in screeni

77 The GSH adduct screening capability of the MDF approach, together with the utility of accurate mass

78 tion energy, suggesting the emergence of the MDF linear dispersion in the AG.

79 while expressing the Pax3 gene and prior to MDF gene activation.

80 Therefore, while MDF family members act positively during myogenic differ

81 n addition, SPDL-1 coimmunoprecipitates with MDF-1/MAD1 in vivo.

82 Negative or zero MDF and MIF signatures are typical of geological Hg sour