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1                                              ME is comprised of four genes in two adjacent operons, w
2                                              ME models present considerable computational challenges:
3                                              ME recruitment of leukocytes was delayed but persisted f
4                                              ME seems to be a common complication of longstanding OT
5                                              ME-Class is thus a powerful tool to identify genes that
6                                              ME-ECTs displayed the lowest dead cell ratio (p < 0.001)
7                                              ME-induced MOPr desensitization and resensitization did
8                                              ME-SIMS analysis followed by t-distributed stochastic ne
9 erity and fatigue duration, cytokines of 192 ME/CFS patients and 392 healthy controls were measured u
10 nd introducing AR into PC3-M cells confers 2-ME-induced mitotic arrest.
11 y small interfering RNA (siRNA) eliminates 2-ME-induced arrest and introducing AR into PC3-M cells co
12 ravitreal injection of 2-Methoxyestradiol (2-ME) nanoemulsion in regressing neovascularization of a R
13                  Intravitreal injection of 2-ME (in the two concentrations) caused marked regression
14 uced in 21 rats then two concentrations of 2-ME nanoparticles were injected in right eyes of 14 rats
15  84.1% for C. neoformans (4.5% VME and 11.4% ME).
16                                       The 6M-ME-ECTs implanted onto 1 week post-infarct immune tolera
17  subunit-containing GABAA receptors in adult ME mice specifically reduces the cross-modal aspect of r
18 ors or inflammatory cytokines may not affect ME in ERM patients.
19 xtensive recovery of cortical activity after ME fails as a result of suppression or functional immatu
20                     In both groups, VA after ME resolution correlated with baseline VA.
21 tward current produced by the opioid agonist ME was concentration dependent, reversed at the potassiu
22  a promising structural unit that carries an ME-active multipole moment.
23  ion beam-induced fragmentation of analytes, ME-SIMS has proven useful for detection of numerous bior
24 ty eyes of 60 patients with CRVO or BRVO and ME and 40 healthy subjects underwent measurements of sup
25 and 4 with a high probability of PT, CE, and ME resistance.
26 evalence rates for resistance to PT, CE, and ME were 28.6%, 21.8%, and 8.5%, respectively.
27  to the antibiotics of interest (PT, CE, and ME).
28 e, or high risk of resistance to PT, CE, and ME.
29 le the tunabilities of the magnetization and ME coupling due to the control of the charge transfer.
30  contribute to the adaptive pH response, and ME can influence the outcome of infection.
31 er increases Ser5P-RNAPII at LEF-1 sites and ME gene promoters, indicating that elongation remains li
32 nds under MAE as compared to 9 under UAE and ME.
33 ,290 isolates the CA was 97.1% (0.2% VME and ME, 2.5% minor errors) using anidulafungin as the surrog
34 e 25th, 50th, and 75th percentiles of annual ME were 1.8, 5.5, and 23.7 mg/kg, respectively.
35 ctric signals associated with an antiferroic ME-active magnetic quadrupole order in the real material
36 halamic arcuate-median eminence complex (Arc-ME) controls energy balance, fertility and growth throug
37 e identify 50 transcriptionally distinct Arc-ME cell populations, including a rare tanycyte populatio
38 dual cells in and around the adult mouse Arc-ME using Drop-seq.
39 luding a rare tanycyte population at the Arc-ME diffusion barrier, a new leptin-sensing neuron popula
40 experimental condition into constraint-based ME models.
41      Here, we show that dark exposure before ME in adulthood also prevents the late cross-modal react
42 bination was recorded following PeT for beta-ME ( approximately 100%), for Trolox, n-propyl gallate,
43 ng agents, namely beta-mercaptoethanol (beta-ME), Trolox (TX), n-propyl gallate (n-PG), and ascorbic
44 uantitative geminate recombination with beta-ME and efficient geminate recombination with TX.
45 ent a more symptomatic subset of the broader ME/CFS population.
46  a bulk linear ME vortex structure or a bulk ME coupling such that if P reverses so does M.
47 1S/T-A) abolishes desensitization induced by ME.
48    We concluded that killing of S. mutans by ME promotes effective remineralization of S. mutans-demi
49 ess of chemical or mechanical exfoliation (C/ME) followed by a rapid densification and restacking of
50 es of n-type Bi2Te2.7Se0.3 produced by the C/ME process, as a function of the flake thickness.
51                                           CE-ME was a W/O ME and both LC-MEs were O/W type.
52  of (99m)Tc in a first-order manner, than CE-ME.
53                                       The CE-ME dispersed readily in water, forming a CE, whereas the
54 e rats received eight s.c. injections of Cit-ME on alternate days.
55 ed a multiepitope citrullinated peptide (Cit-ME), derived from major prevalent citrullinated autoanti
56 vant-induced arthritis rats treated with Cit-ME, disease severity was significantly reduced compared
57 lation-based Gene Expression Classification (ME-Class), to explain specific variation in methylation
58               ccl3(-/-) deletion compromised ME bacterial clearance and prolonged mucosal hyperplasia
59                       The sector-constrained ME model thus represents a generalist ME model reflectin
60 rocess, and that a single regulator controls ME expression in response to diverse signals.
61 t to be practical on large problems (current ME models have 70,000 constraints and variables and will
62 of clinical criteria are available to define ME/CFS, yet none of the current diagnostic methods have
63 ht case definitions have been used to define ME/CFS; a ninth, recently proposed by the Institute of M
64 ly lower percentage of patients demonstrated ME within 90 days after surgery with nepafenac 0.3% vers
65                                  In diabetic ME involving a multivariable model including baseline VA
66 after resolution of center-involved diabetic ME.
67 ctric and magnetic moments with differential ME coefficients on the order of 10(4) ps/m are achieved.
68                                        DRINK ME (DKM; bZIP30) is a member of the bZIP transcription f
69                              The middle ear (ME) contents were then harvested, amplified and the prep
70 role of CCL3 in OM, we evaluated middle ear (ME) responses of ccl3(-/-)mice to nontypeable Haemophilu
71                   Summer methane ebullition (ME) peaks were a factor of 4 to 10 times the winter mini
72 traction (57.1%), and chronic macular edema (ME) (71.4%).
73 es were: patients (%) in whom macular edema (ME) developed (>/=30% increase from preoperative baselin
74                               Macular edema (ME) is the leading cause of decreased visual acuity (VA)
75                               Macular edema (ME) prognosis and treatment response vary according to t
76 natives for the management of macular edema (ME) secondary to branch retinal vein occlusion (BRVO).
77 al vascular lesions including macular edema (ME), a leading cause of vision loss in DR.
78 clusion (BRVO) complicated by macular edema (ME).
79 cal steroids for postsurgical macular edema (ME).
80 na [i.e., the development of macular edema, (ME)].
81                           The matrix effect (ME) for each pesticide was evaluated through the study o
82                          The matrix effects (MEs) on the quantification of an analyte can be signific
83 re retention, microencapsulation efficiency (ME), particle size and moisture.
84  of nano-magnets using the magneto-electric (ME) effect.
85 amplification and microchip electrophoretic (ME) separation for rapid forensic short tandem repeat (S
86 uroendocrine signals at the median eminence (ME) to control long-lasting pituitary hormone rhythms es
87  produced by premix membrane emulsification (ME) enabled to produce microcapsules containing procyani
88       The FilmArray Meningitis/Encephalitis (ME) Panel is a multiplexed in vitro diagnostic test for
89                   Myalgic encephalomyelitis (ME)/chronic fatigue syndrome (CFS) is a debilitating mul
90  The diagnosis of myalgic encephalomyelitis (ME)/chronic fatigue syndrome (CFS) is based on clinical
91 ta on measures of myalgic encephalomyelitis (ME)/chronic fatigue syndrome (CFS).
92 eople living with myalgic encephalomyelitis (ME)/chronic fatigue syndrome (CFS).
93 iving contrast agent, magneto-endosymbionts (MEs) derived from magnetotactic bacteria for the labelin
94 mutans with high-frequency microwave energy (ME).
95 on as implemented in the Multilinear Engine (ME-2).
96 pioid agonists DAMGO or [Met(5) ]enkephalin (ME) into the KF reduced respiratory frequency and amplit
97 maximal concentrations of Met(5)-enkephalin (ME) and somatostatin (SST; coupling to native SST recept
98 n of the opioid agonist [Met(5)]-enkephalin (ME) caused significantly less desensitization in KF neur
99                       Monocular enucleation (ME) drastically affects the contralateral visual cortex,
100        In adult mice, monocular enucleation (ME) results in an immediate deactivation of the contrala
101              Mitochondrial NAD-malic enzyme (ME) and/or cytosolic/plastidic NADP-ME combined with the
102 ains possess the genes for the malic enzyme (ME) pathway, which allows malate to be used as a supplem
103 ion were normalized to morphine equivalents (ME) and expressed as mg/kg exposures.
104                        Fifteen major errors (MEs) (0.4%) were noted using the Vitek 2 breakpoints and
105 ry major errors [VME] and 2.6% major errors [ME]) and 84.1% for C. neoformans (4.5% VME and 11.4% ME)
106 Mi gilts in diestrus (MD, n = 2) and estrus (ME, n = 3) were investigated using RNA sequencing.
107 ty for Rzhetsky and Nei's minimum evolution (ME) approach, one of the earliest methods for constructi
108 of Metabolism and macromolecular Expression (ME) at genome-scale.
109 of Metabolism and macromolecular Expression (ME) compute proteome allocation linked to metabolism and
110 of metabolism and macromolecular expression (ME) significantly expand the scope and predictive capabi
111          Orange juice (OJ) and malt extract (ME) samples were treated using an electrokinetic (EK) ap
112  power (FRAP) assays found methanol extract (ME) to be the most active.
113  ultrasonic (UAE) and maceration extraction (ME), MAE showed significantly (p<0.05) higher recovery o
114                                The FilmArray ME Panel demonstrated a sensitivity or positive percenta
115                                The FilmArray ME Panel is a sensitive and specific test to aid in diag
116 HAID tree models, with both being better for ME than for PT and CE.
117                Patients meeting criteria for ME represent a more symptomatic subset of the broader ME
118 en and 23 women) met diagnostic criteria for ME/CFS by Institute of Medicine, Canadian, and Fukuda cr
119 that achieves reliability and efficiency for ME models and other challenging problems tested here.
120 l growth factor (VEGF) pharmacotherapies for ME associated with CRVO, including intravitreal ranibizu
121 l growth factor (VEGF) pharmacotherapies for ME, including intravitreal bevacizumab (5), aflibercept
122  These results demonstrate the potential for ME-SIMS tandem MS development in bottom-up proteomics im
123 of the nasopharyngeal niche, a reservoir for ME and upper respiratory infections.
124 h anti-VEGF agents is effective and safe for ME secondary to BRVO.
125 erapy is safe and effective over 2 years for ME associated with CRVO and that delay in treatment is a
126  water, forming a CE, whereas the LC-forming MEs formed 'depots' in water.
127  samples were then exposed to high-frequency ME, and the other half were used as controls.
128 rained ME model thus represents a generalist ME model reflecting both growth rate maximization and "h
129  T, Yang GX, Leung PS, Matsumura F, Gershwin ME.
130                         Here we report giant ME effects in a polar magnet Fe2Mo3O8 at temperatures as
131         The terms instrumental ME and global ME were introduced, and the term recovery was subdivided
132 cho-Planar J-resolved Spectroscopic Imaging (ME-EP-JRESI), was evaluated in 10 healthy controls and a
133 ficiency under EK by 7.8% in OJ and 11.8% in ME.
134 medium, pH/LCP<1) and towards the cathode in ME samples (alkaline medium, pH/LCP>1).
135 e solution method for growth maximization in ME models using a quad-precision NLP solver (Quad MINOS)
136                   The bulk acoustic waves in ME antennas stimulate magnetization oscillations of the
137 eeting different case definitions, including ME, and providing subgroup analysis are needed to fill r
138  similar to those seen in human DR including ME and microaneurysms.
139                       The terms instrumental ME and global ME were introduced, and the term recovery
140   We attribute the current-induced interface ME effect to modulations of the strong double-exchange i
141 d 55 patients (70 eyes) with center-involved ME that had resolved during an 8-month period.
142 piezoelectric and magnetostrictive laminate (ME composite) structure shows an equivalent magnetic noi
143       Unfortunately, in these systems, large ME response is revealed only upon elaborate poling proce
144                                  While large ME coupling achieved through interfacial strain-induced
145                                      Largest ME coefficients are often associated with phase transiti
146               CE-ME was a W/O ME and both LC-MEs were O/W type.
147 g SC injection into the animal thigh, the LC-MEs had more prolonged release of (99m)Tc in a first-ord
148 is results in the existence of a bulk linear ME vortex structure or a bulk ME coupling such that if P
149              Temkin AM, Bowers RR, Magaletta ME, Holshouser S, Maggi A, Ciana P, Guillette LJ, Bowden
150                             Magnetoelectric (ME) effect is recognized for its utility for low-power e
151                             Magnetoelectric (ME) nanoparticles (MENs) intrinsically couple magnetic a
152 perature ferromagnetism and magnetoelectric (ME) coupling.
153  and electric properties in magnetoelectric (ME) composite heterostructures is crucial for multiple t
154 injection induced interface magnetoelectric (ME) effect.
155  near-theoretical intrinsic magnetoelectric (ME) coupling of 7 V cm(-1) Oe(-1) is achieved in a heter
156 an therefore exhibit linear magnetoelectric (ME) activity.
157 lly actuated nanomechanical magnetoelectric (ME) antennas with a suspended ferromagnetic/piezoelectri
158 oits recent advances in the magnetoelectric (ME) composite sensors that exhibit an ultrahigh AC magne
159                         The magnetoelectric (ME) effect, the phenomenon of inducing magnetization by
160 rs in realizing such a giant strain-mediated ME coupling.
161 zobactam (PT), cefepime (CE), and meropenem (ME).
162 hESCs) can be directed toward a mesendoderm (ME) or neuroectoderm (NE) fate, the first decision durin
163 promoter looping and activate mesendodermal (ME) lineage genes.
164 h Wnt/beta-catenin and induce mesendodermal (ME) differentiation genes.
165  rectangular internal staggered posts (mesh, ME), without posts (plain sheet, PS), or long parallel p
166 BSTARCT: Microbial enhanced coalbed methane (ME-CBM) recovery is critically examined as a viable tech
167 (SECM) like approach of a Pt microelectrode (ME), which was leveled with the WE toward the IRE surfac
168 he selection of the association with minimum MEs.
169           We generated a larger (30 x 30 mm) ME-ECT to confirm scalability.
170 VA CS and 2 of 15 (13%) receiving ChAd63-MVA ME-TRAP achieved sterile protection after CHMI.
171 VA CS and 5 of 15 (33%) receiving ChAd63-MVA ME-TRAP demonstrated a delay in time to treatment, compa
172 ce the liver parasite burden, but ChAd63-MVA ME-TRAP remains the most promising antigenic insert for
173 9%-79%, compared with 79%-84% for ChAd63-MVA ME-TRAP.
174 al efficacy of ChAd63-MVA CS with ChAd63-MVA ME-TRAP.
175  as a consequence of knocking out either NAD-ME or PPDK activity, particularly phosphoenolpyruvate ca
176 d greater than the kcatc of Rubisco from NAD-ME species across all temperatures.
177 ppear to compensate for the reduction in NAD-ME, suggesting that NAD-ME was the key decarboxylase for
178  fedtschenkoi with reduced activities of NAD-ME or PPDK.
179 the reduction in NAD-ME, suggesting that NAD-ME was the key decarboxylase for CAM.
180 geous in warmer climates relative to the NAD-ME grasses.
181 erature was less pronounced for PCK and NADP-ME Rubisco, which would be advantageous in warmer climat
182   The alternative malate decarboxylase, NADP-ME, did not appear to compensate for the reduction in NA
183 ucleotide (NAD) phosphate malic enzyme (NADP-ME) and phosphoenolpyruvate carboxykinase (PCK) photosyn
184  enzyme (ME) and/or cytosolic/plastidic NADP-ME combined with the cytosolic/plastidic pyruvate orthop
185 useful guide for exploring and designing new ME-active materials based on vortex-like spin arrangemen
186    Morphine-induced desensitization (but not ME) was reduced by protein kinase C inhibition in wild-t
187                              CE-ME was a W/O ME and both LC-MEs were O/W type.
188 nd autumn simulated ME exceeded the observed ME.
189  will accelerate the wide-spread adoption of ME models for researchers in these fields.
190                     Adjusted associations of ME level with posttransplant graft and patient survival
191 ds (CS) (n = 7) were administered in case of ME and/or posterior segment inflammation.
192 etinal vascular permeability, development of ME, and morphological characteristics including acellula
193 ive and specific test to aid in diagnosis of ME.
194 questionnaires to measure illness domains of ME/CFS and to evaluate patient heterogeneity overall and
195 gation factor and block SMAD2,3 induction of ME genes.
196 are effective and safe for the management of ME associated with BRVO; however, corticosteroids are as
197 eatment was associated with a higher odds of ME resolution (OR, 3.59; 95% CI, 2.22-5.80; P < .001) an
198 ted out 2 clusters with a low probability of ME resistance and 4 with a high probability of PT, CE, a
199 ll previously implicated in the promotion of ME in ischemic retinal disease, were not elevated by qua
200 ive donor recipients with upper quartiles of ME use, compared with 92.0% among those who did not rece
201  seemed to be associated with higher rate of ME.
202                                 The rates of ME- and morphine-induced desensitization did not correla
203  CST less than 300 microm, and resolution of ME.
204 Patients were grouped based on the source of ME (diabetic vs nondiabetic).
205 ctivin-induced SMADs to activate a subset of ME genes that is required to form cardiac mesoderm.
206 e management decisions in different types of ME.
207                       Water fraction (WF) of ME was a stronger alpha-glucosidase inhibitor (EC50 2.9
208                                     Based on ME property, for the first time we show that MENs can di
209 tokine's preprocessed data were regressed on ME/CFS severity plus covariates for age, sex, race, and
210 re found in LD vs MD, LE vs ME, LE vs LD, or ME vs MD comparisons.
211                                      The PCR-ME genetic profiles were analyzed using binning palettes
212 phiphile required to form microemulsion (Phi(ME)), which was the analytical signal of the method.
213           Most of the compounds had positive ME.
214               Concomitantly, at 7 weeks post ME, somatosensory and auditory cortex shifted from a hyp
215 ehicle in reducing the risk of postoperative ME, with the integrated analysis showing improved BCVA a
216 data suggest that patients with postsurgical ME should initially be treated with ketorolac and PA qid
217                   Subjects with postsurgical ME stratified into post-cataract surgery ME (PCSME) and
218 , thus representing a step towards practical ME transduction devices.
219                              Although premix ME produced W1/O/W2 emulsions with a narrow droplet size
220 issues from the Roadmap Epigenomics Project, ME-Class significantly outperforms standard methods usin
221 ope-thrombospondin-related adhesion protein (ME-TRAP).
222  whether GABAergic inhibition also regulates ME-induced cross-modal plasticity.
223 ate that these fast and numerically reliable ME solution methods will accelerate the wide-spread adop
224      Eight subjects in Group 2 with residual ME at week 12 were switched to PA q1hWA and at week 24,
225 nalysis of the trajectories, "localized RRKM-ME" and "competitive localized noncanonical" rate models
226 in retinal vein occlusion macular edema (RVO-ME).
227                          Treatment-naive RVO-ME receiving monthly intravitreal bevacizumab.
228 y serve as a biomarker for patients with RVO-ME.
229              We show that Rzhetsky and Nei's ME problem is NP-complete, and so probably computational
230               The key driver of the seasonal ME dynamics was temperature.
231                        Virulence of selected ME mutants was assessed in a murine model of soft tissue
232 d in a subset of ERM patients who had severe ME (central subfield thickness >/=450 mum on spectral do
233 r, although late summer and autumn simulated ME exceeded the observed ME.
234 ix-enhanced secondary ion mass spectrometry (ME-SIMS) has overcome one of the biggest disadvantages o
235 ix-enhanced-secondary ion mass spectrometry (ME-SIMS) to investigate the lipid profiles of neuronal c
236 cal ME stratified into post-cataract surgery ME (PCSME) and post-other surgery ME (POSME) were random
237 ct surgery ME (PCSME) and post-other surgery ME (POSME) were randomized to ketorolac 4 times a day (q
238 cephalomyelitis or chronic fatigue syndrome (ME/CFS), the data are limited and contradictory.
239  encephalomyelitis/chronic fatigue syndrome (ME/CFS).
240 eport here the first demonstration of tandem ME-SIMS for de novo sequencing of endogenous neuropeptid
241 tment of cross-modal recovery upon long-term ME coincides with the transition from adolescence to adu
242  thereby converting the outcome of long-term ME into a more P45-like response.
243 onocular and binocular cortices to long-term ME or dark exposure or a combinatorial deprivation revea
244 he Tobacco, Exercise and Diet Messages (TEXT ME) trial was a parallel-group, single-blind, randomized
245 nantly by their phase transition rather than ME type or oil content.
246  the present study, after demonstrating that ME kills >99% of S. mutans in planktonic cultures, 8 ena
247         Taken together, these data show that ME contributes to S. pyogenes' carbon source repertory,
248                         We further show that ME-Class can detect functionally relevant cancer-specifi
249                                          The ME antennas (with sizes as small as one-thousandth of a
250                                          The ME-labeled iCMs were injected into the infarcted area of
251 tegration time window is detected across the ME and consists of high-frequency DA discharges that are
252 I), as a PtsI(-) mutant fails to express the ME genes and is unable to utilize malate.
253 al/spectral profiles of metabolites from the ME-EP-JRESI sequence were in good agreement with that of
254                         So far, however, the ME activity of these multipole moments has only been est
255  constraints for key proteome sectors in the ME model.
256 offers a new drug delivery platform into the ME.
257                    We do this by linking the ME problem to a graph clustering problem called the quas
258 f SECM and revealed the contact point of the ME and WE on the IRE, which was used as reference point
259                              The sign of the ME coefficients can be switched by changing the applied
260            Here, we report a new form of the ME effect based on the valley DOF in two-dimensional Dir
261                               Studies of the ME effect have so far focused on the control of the elec
262                       Due to presence of the ME effect, these nanoparticles can significantly enhance
263 ed ion beam (FIB) cutting of the apex of the ME was used to expose an electroactive BDD disk.
264                  The precise lowering of the ME/WE plane toward the IRE was enabled by a micrometer s
265 e a voltage driven logic-device based on the ME induced switching of nano-magnets.
266 e have implemented, validated and tested the ME-EP-JRESI sequence, demonstrating that multi-echo acqu
267 d transmit electromagnetic waves through the ME effect at their acoustic resonance frequencies.
268 f peptides that transit across the TM to the ME in a time and temperature dependent manner.
269            Our findings demonstrate that the MEs are robust and effective biological contrast agents
270                             We show that the MEs clear within one week of cell death whereas the SPIO
271                                        These ME antennas have potential implications for portable wir
272                                        These ME antennas receive and transmit electromagnetic waves t
273                                         This ME (microelectrode) assembly consists of an inner boron
274 on and in improving reduced VA attributed to ME in a majority of patients.
275 mong the subset with VA <20/40 attributed to ME, 33.1% (95% CI, 25.2-42.7) improved to >/= 20/40.
276 alteration of the nano-pore structure due to ME-CBM treatment.
277 amine desensitization induced by exposure to ME and morphine for 5 minutes at 37 degrees C.
278                                Resistance to ME was specifically linked with infection attributed to
279 tion computes steady-state flux solutions to ME models, but flux values are spread over many orders o
280                        Cellular tolerance to ME and morphine was also lacking in KF neurons from morp
281                                     Training ME-Class on normal-tumor pairs from The Cancer Genome At
282 f pharmacologic therapeutic options to treat ME in patients with ERMs, we examine here the expression
283 y injection of anti-VEGF agents for treating ME due to CRVO and HRVO.
284  to counteract YAP repression and upregulate ME genes during early hESC differentiation.
285          To demonstrate its utility, we used ME-Class to analyze 32 datasets from different hematopoi
286 d genes (DEGs) were found in LD vs MD, LE vs ME, LE vs LD, or ME vs MD comparisons.
287  a single tertiary referral center for which ME was the principal cause of reduced visual acuity.
288  of serum cytokines could be associated with ME/CFS and correlated with disease severity and fatigue
289 ant upward linear trend that correlated with ME/CFS severity: CCL11 (Eotaxin-1), CXCL1 (GROalpha), CX
290 ther illnesses that share some features with ME/CFS were enrolled in comparison groups.
291 on in the vitreous of most ERM patients with ME compared to control patients.
292 th factor) were similar in ERM patients with ME compared to controls.
293     The medical records of all patients with ME from December 1, 2010, to December 31, 2012, were rev
294 eported symptoms differentiate patients with ME/CFS from healthy controls under study conditions but
295  adequately tested to identify patients with ME/CFS when diagnostic uncertainty exists.
296 initial description of the 471 patients with ME/CFS who were enrolled in stage 1.
297  measures that best distinguish persons with ME/CFS from those in the comparison groups and detect su
298  groups and detect subgroups of persons with ME/CFS who may have different underlying causes.
299 y expert clinicians to care for persons with ME/CFS; 4) collect biospecimens for future hypothesis te
300  Optically guided single cell profiling with ME-SIMS is suitable for a range of cell sizes, from Aply

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