1                                              MEB represented approximately 5% of all enterococcal bac

2      Mutations reported in genes of FCMD and MEB patients suggest that the genes may be involved in p

3 ion (heterogeneous) enterococcal bacteremia (MEB) is rarely reported.

4                        Here we show, in both MEB and FCMD patients, that alpha-dystroglycan is expres

5 h mutations in POMT2 had a Muscle-Eye-Brain (MEB)-like condition.

6  to detect this subpopulation and determined MEB frequency.

7                    Muscle eye brain disease (MEB) and Fukuyama congenital muscular dystrophy (FCMD) a

8 that patients with muscle-eye-brain disease (MEB) and Fukuyama congenital muscular dystrophy (FCMD),

9  dystrophy (FCMD), Muscle-Eye-Brain disease (MEB) and Walker-Warburg syndrome (WWS), which are associ

10 Syndrome (WWS) and muscle-eye-brain disease (MEB) are caused by mutations in two genes involved in O-

11  dystrophy (FCMD), muscle-eye-brain disease (MEB), and Walker-Warburg syndrome are congenital muscula

12                    Muscle-eye-brain disease (MEB), is caused by mutations in the POMGnT1 gene.

13 gs further define the phenotypic spectrum of MEB and its occurrence in the US population.

14 th fringe-like glycosyltransferases, and the MEB gene, POMGnT1, seems to be a new glycosyltransferase

15 tween these models, we compared rafts in the MEB-4 melanoma cell line and its GSL-deficient derivativ

16 hat is similar to that seen in patients with MEB and FCMD.

17                                         WWS, MEB, FCMD and the myd mouse are also associated with neu

18 rmalities with striking similarities to WWS, MEB, FCMD and the myd mouse.